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Repeated immobilization stress alters tyrosine hydroxylase, corticotropin-releasing hormone and corticosteroid receptor messenger ribonucleic Acid levels in rat brain.

Journal of neuroendocrinology

Mamalaki E, Kvetnansky R, Brady LS, Gold PW, Herkenham M.
PMID: 21554656
J Neuroendocrinol. 1992 Dec;4(6):689-99. doi: 10.1111/j.1365-2826.1992.tb00220.x.

In situ hybridization histochemistry was used to localize and quantify the effects of acute and repeated immobilization stress on mRNA levels of tyrosine hydroxylase (TH) in catecholaminergic neurons in the locus ceruleus and substantia nigra and on mRNA levels...

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Mamalaki E, Kvetnansky R, Brady LS, et al. Repeated immobilization stress alters tyrosine hydroxylase, corticotropin-releasing hormone and corticosteroid receptor messenger ribonucleic Acid levels in rat brain. J Neuroendocrinol. 1992;4(6):689-99doi: 10.1111/j.1365-2826.1992.tb00220.x.
Mamalaki, E., Kvetnansky, R., Brady, L. S., Gold, P. W., & Herkenham, M. (1992). Repeated immobilization stress alters tyrosine hydroxylase, corticotropin-releasing hormone and corticosteroid receptor messenger ribonucleic Acid levels in rat brain. Journal of neuroendocrinology, 4(6), 689-99. https://doi.org/10.1111/j.1365-2826.1992.tb00220.x
Mamalaki, E, et al. "Repeated immobilization stress alters tyrosine hydroxylase, corticotropin-releasing hormone and corticosteroid receptor messenger ribonucleic Acid levels in rat brain." Journal of neuroendocrinology vol. 4,6 (1992): 689-99. doi: https://doi.org/10.1111/j.1365-2826.1992.tb00220.x
Mamalaki E, Kvetnansky R, Brady LS, Gold PW, Herkenham M. Repeated immobilization stress alters tyrosine hydroxylase, corticotropin-releasing hormone and corticosteroid receptor messenger ribonucleic Acid levels in rat brain. J Neuroendocrinol. 1992 Dec;4(6):689-99. doi: 10.1111/j.1365-2826.1992.tb00220.x. PMID: 21554656.
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An integrated map of corticotropin-releasing hormone signaling pathway.

Journal of cell communication and signaling

Subbannayya T, Balakrishnan L, Sudarshan G, Advani J, Kumar S, Mahmood R, Nair B, Sirdeshmukh R, Mukherjee KK, Umathe SN, Raju R, Prasad TS.
PMID: 23504413
J Cell Commun Signal. 2013 Dec;7(4):295-300. doi: 10.1007/s12079-013-0197-3. Epub 2013 Mar 16.

No abstract available.

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Subbannayya T, Balakrishnan L, Sudarshan G, et al. An integrated map of corticotropin-releasing hormone signaling pathway. J Cell Commun Signal. 2013;7(4):295-300doi: 10.1007/s12079-013-0197-3.
Subbannayya, T., Balakrishnan, L., Sudarshan, G., Advani, J., Kumar, S., Mahmood, R., Nair, B., Sirdeshmukh, R., Mukherjee, K. K., Umathe, S. N., Raju, R., & Prasad, T. S. (2013). An integrated map of corticotropin-releasing hormone signaling pathway. Journal of cell communication and signaling, 7(4), 295-300. https://doi.org/10.1007/s12079-013-0197-3
Subbannayya, Tejaswini, et al. "An integrated map of corticotropin-releasing hormone signaling pathway." Journal of cell communication and signaling vol. 7,4 (2013): 295-300. doi: https://doi.org/10.1007/s12079-013-0197-3
Subbannayya T, Balakrishnan L, Sudarshan G, Advani J, Kumar S, Mahmood R, Nair B, Sirdeshmukh R, Mukherjee KK, Umathe SN, Raju R, Prasad TS. An integrated map of corticotropin-releasing hormone signaling pathway. J Cell Commun Signal. 2013 Dec;7(4):295-300. doi: 10.1007/s12079-013-0197-3. Epub 2013 Mar 16. PMID: 23504413; PMCID: PMC3889255.
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The neuroendocrine functions of the parathyroid hormone 2 receptor.

Frontiers in endocrinology

Dobolyi A, Dimitrov E, Palkovits M, Usdin TB.
PMID: 23060860
Front Endocrinol (Lausanne). 2012 Oct 08;3:121. doi: 10.3389/fendo.2012.00121. eCollection 2012.

The G-protein coupled parathyroid hormone 2 receptor (PTH2R) is concentrated in endocrine and limbic regions in the forebrain. Its endogenous ligand, tuberoinfundibular peptide of 39 residues (TIP39), is synthesized in only two brain regions, within the posterior thalamus and...

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Dobolyi A, Dimitrov E, Palkovits M, et al. The neuroendocrine functions of the parathyroid hormone 2 receptor. Front Endocrinol (Lausanne). 2012;3:121doi: 10.3389/fendo.2012.00121.
Dobolyi, A., Dimitrov, E., Palkovits, M., & Usdin, T. B. (2012). The neuroendocrine functions of the parathyroid hormone 2 receptor. Frontiers in endocrinology, 3121. https://doi.org/10.3389/fendo.2012.00121
Dobolyi, Arpád, et al. "The neuroendocrine functions of the parathyroid hormone 2 receptor." Frontiers in endocrinology vol. 3 (2012): 121. doi: https://doi.org/10.3389/fendo.2012.00121
Dobolyi A, Dimitrov E, Palkovits M, Usdin TB. The neuroendocrine functions of the parathyroid hormone 2 receptor. Front Endocrinol (Lausanne). 2012 Oct 08;3:121. doi: 10.3389/fendo.2012.00121. eCollection 2012. PMID: 23060860; PMCID: PMC3465808.
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Contributions of the paraventricular thalamic nucleus in the regulation of stress, motivation, and mood.

Frontiers in behavioral neuroscience

Hsu DT, Kirouac GJ, Zubieta JK, Bhatnagar S.
PMID: 24653686
Front Behav Neurosci. 2014 Mar 11;8:73. doi: 10.3389/fnbeh.2014.00073. eCollection 2014.

The purpose of this review is to describe how the function and connections of the paraventricular thalamic nucleus (Pa) may play a role in the regulation of stress and negative emotional behavior. Located in the dorsal midline thalamus, the...

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Hsu DT, Kirouac GJ, Zubieta JK, et al. Contributions of the paraventricular thalamic nucleus in the regulation of stress, motivation, and mood. Front Behav Neurosci. 2014;8:73doi: 10.3389/fnbeh.2014.00073.
Hsu, D. T., Kirouac, G. J., Zubieta, J. K., & Bhatnagar, S. (2014). Contributions of the paraventricular thalamic nucleus in the regulation of stress, motivation, and mood. Frontiers in behavioral neuroscience, 873. https://doi.org/10.3389/fnbeh.2014.00073
Hsu, David T, et al. "Contributions of the paraventricular thalamic nucleus in the regulation of stress, motivation, and mood." Frontiers in behavioral neuroscience vol. 8 (2014): 73. doi: https://doi.org/10.3389/fnbeh.2014.00073
Hsu DT, Kirouac GJ, Zubieta JK, Bhatnagar S. Contributions of the paraventricular thalamic nucleus in the regulation of stress, motivation, and mood. Front Behav Neurosci. 2014 Mar 11;8:73. doi: 10.3389/fnbeh.2014.00073. eCollection 2014. PMID: 24653686; PMCID: PMC3949320.
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Deciphering the Contributions of CRH Receptors in the Brain and Pituitary to Stress-Induced Inhibition of the Reproductive Axis.

Frontiers in molecular neuroscience

Raftogianni A, Roth LC, García-González D, Bus T, Kühne C, Monyer H, Spergel DJ, Deussing JM, Grinevich V.
PMID: 30214395
Front Mol Neurosci. 2018 Aug 30;11:305. doi: 10.3389/fnmol.2018.00305. eCollection 2018.

Based on pharmacological studies, corticotropin-releasing hormone (CRH) and its receptors play a leading role in the inhibition of the hypothalamic-pituitary-gonadal (HPG) axis during acute stress. To further study the effects of CRH receptor signaling on the HPG axis, we...

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Raftogianni A, Roth LC, García-González D, et al. Deciphering the Contributions of CRH Receptors in the Brain and Pituitary to Stress-Induced Inhibition of the Reproductive Axis. Front Mol Neurosci. 2018;11:305doi: 10.3389/fnmol.2018.00305.
Raftogianni, A., Roth, L. C., García-González, D., Bus, T., Kühne, C., Monyer, H., Spergel, D. J., Deussing, J. M., & Grinevich, V. (2018). Deciphering the Contributions of CRH Receptors in the Brain and Pituitary to Stress-Induced Inhibition of the Reproductive Axis. Frontiers in molecular neuroscience, 11305. https://doi.org/10.3389/fnmol.2018.00305
Raftogianni, Androniki, et al. "Deciphering the Contributions of CRH Receptors in the Brain and Pituitary to Stress-Induced Inhibition of the Reproductive Axis." Frontiers in molecular neuroscience vol. 11 (2018): 305. doi: https://doi.org/10.3389/fnmol.2018.00305
Raftogianni A, Roth LC, García-González D, Bus T, Kühne C, Monyer H, Spergel DJ, Deussing JM, Grinevich V. Deciphering the Contributions of CRH Receptors in the Brain and Pituitary to Stress-Induced Inhibition of the Reproductive Axis. Front Mol Neurosci. 2018 Aug 30;11:305. doi: 10.3389/fnmol.2018.00305. eCollection 2018. PMID: 30214395; PMCID: PMC6125327.
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Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons.

Frontiers in cellular neuroscience

Dos-Santos RC, Grover HM, Reis LC, Ferguson AV, Mecawi AS.
PMID: 30210300
Front Cell Neurosci. 2018 Aug 24;12:275. doi: 10.3389/fncel.2018.00275. eCollection 2018.

The paraventricular nucleus (PVN) is involved in the control of sympathetic tone and the secretion of hormones, both functions known to be influenced by ghrelin, suggesting direct effect of ghrelin in this nucleus. However, the effects of ghrelin on...

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Dos-Santos RC, Grover HM, Reis LC, et al. Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons. Front Cell Neurosci. 2018;12:275doi: 10.3389/fncel.2018.00275.
Dos-Santos, R. C., Grover, H. M., Reis, L. C., Ferguson, A. V., & Mecawi, A. S. (2018). Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons. Frontiers in cellular neuroscience, 12275. https://doi.org/10.3389/fncel.2018.00275
Dos-Santos, Raoni C, et al. "Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons." Frontiers in cellular neuroscience vol. 12 (2018): 275. doi: https://doi.org/10.3389/fncel.2018.00275
Dos-Santos RC, Grover HM, Reis LC, Ferguson AV, Mecawi AS. Electrophysiological Effects of Ghrelin in the Hypothalamic Paraventricular Nucleus Neurons. Front Cell Neurosci. 2018 Aug 24;12:275. doi: 10.3389/fncel.2018.00275. eCollection 2018. PMID: 30210300; PMCID: PMC6121211.
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Chronic Stress Reduces Nectin-1 mRNA Levels and Disrupts Dendritic Spine Plasticity in the Adult Mouse Perirhinal Cortex.

Frontiers in cellular neuroscience

Gong Q, Su YA, Wu C, Si TM, Deussing JM, Schmidt MV, Wang XD.
PMID: 29593501
Front Cell Neurosci. 2018 Mar 13;12:67. doi: 10.3389/fncel.2018.00067. eCollection 2018.

In adulthood, chronic exposure to stressful experiences disrupts synaptic plasticity and cognitive function. Previous studies have shown that perirhinal cortex-dependent object recognition memory is impaired by chronic stress. However, the stress effects on molecular expression and structural plasticity in...

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Gong Q, Su YA, Wu C, et al. Chronic Stress Reduces Nectin-1 mRNA Levels and Disrupts Dendritic Spine Plasticity in the Adult Mouse Perirhinal Cortex. Front Cell Neurosci. 2018;12:67doi: 10.3389/fncel.2018.00067.
Gong, Q., Su, Y. A., Wu, C., Si, T. M., Deussing, J. M., Schmidt, M. V., & Wang, X. D. (2018). Chronic Stress Reduces Nectin-1 mRNA Levels and Disrupts Dendritic Spine Plasticity in the Adult Mouse Perirhinal Cortex. Frontiers in cellular neuroscience, 1267. https://doi.org/10.3389/fncel.2018.00067
Gong, Qian, et al. "Chronic Stress Reduces Nectin-1 mRNA Levels and Disrupts Dendritic Spine Plasticity in the Adult Mouse Perirhinal Cortex." Frontiers in cellular neuroscience vol. 12 (2018): 67. doi: https://doi.org/10.3389/fncel.2018.00067
Gong Q, Su YA, Wu C, Si TM, Deussing JM, Schmidt MV, Wang XD. Chronic Stress Reduces Nectin-1 mRNA Levels and Disrupts Dendritic Spine Plasticity in the Adult Mouse Perirhinal Cortex. Front Cell Neurosci. 2018 Mar 13;12:67. doi: 10.3389/fncel.2018.00067. eCollection 2018. PMID: 29593501; PMCID: PMC5859075.
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Child sexual abuse and the development of psychiatric disorders: a neurobiological trajectory of pathogenesis.

Industrial psychiatry journal

Shrivastava AK, Karia SB, Sonavane SS, De Sousa AA.
PMID: 29456314
Ind Psychiatry J. 2017 Jan-Jun;26(1):4-12. doi: 10.4103/ipj.ipj_38_15.

Child sexual abuse (CSA) is an important public health problem with long-standing neurobiological, developmental, and psychiatric abnormalities. The present review analyzes the long-term effects of CSA from a developmental, psychiatric morbidity, neurochemical and neurobiological perspective and then tries to...

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Shrivastava AK, Karia SB, Sonavane SS, et al. Child sexual abuse and the development of psychiatric disorders: a neurobiological trajectory of pathogenesis. Ind Psychiatry J. 2017;26(1):4-12doi: 10.4103/ipj.ipj_38_15.
Shrivastava, A. K., Karia, S. B., Sonavane, S. S., & De Sousa, A. A. (2017). Child sexual abuse and the development of psychiatric disorders: a neurobiological trajectory of pathogenesis. Industrial psychiatry journal, 26(1), 4-12. https://doi.org/10.4103/ipj.ipj_38_15
Shrivastava, Amresh K, et al. "Child sexual abuse and the development of psychiatric disorders: a neurobiological trajectory of pathogenesis." Industrial psychiatry journal vol. 26,1 (2017): 4-12. doi: https://doi.org/10.4103/ipj.ipj_38_15
Shrivastava AK, Karia SB, Sonavane SS, De Sousa AA. Child sexual abuse and the development of psychiatric disorders: a neurobiological trajectory of pathogenesis. Ind Psychiatry J. 2017 Jan-Jun;26(1):4-12. doi: 10.4103/ipj.ipj_38_15. PMID: 29456314; PMCID: PMC5810166.
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Dominance relationships in Syrian hamsters modulate neuroendocrine and behavioral responses to social stress.

Stress (Amsterdam, Netherlands)

Dulka BN, Koul-Tiwari R, Grizzell JA, Harvey ML, Datta S, Cooper MA.
PMID: 29932809
Stress. 2018 Jun 22;1-6. doi: 10.1080/10253890.2018.1485646. Epub 2018 Jun 22.

Stress is a well-known risk factor for psychopathology and rodent models of social defeat have strong face, etiological, construct and predictive validity for these conditions. Syrian hamsters are highly aggressive and territorial, but after an acute social defeat experience...

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Dulka BN, Koul-Tiwari R, Grizzell JA, et al. Dominance relationships in Syrian hamsters modulate neuroendocrine and behavioral responses to social stress. Stress. 2018;1-6doi: 10.1080/10253890.2018.1485646.
Dulka, B. N., Koul-Tiwari, R., Grizzell, J. A., Harvey, M. L., Datta, S., & Cooper, M. A. (2018). Dominance relationships in Syrian hamsters modulate neuroendocrine and behavioral responses to social stress. Stress (Amsterdam, Netherlands), 1-6. https://doi.org/10.1080/10253890.2018.1485646
Dulka, Brooke N, et al. "Dominance relationships in Syrian hamsters modulate neuroendocrine and behavioral responses to social stress." Stress (Amsterdam, Netherlands) vol. (2018): 1-6. doi: https://doi.org/10.1080/10253890.2018.1485646
Dulka BN, Koul-Tiwari R, Grizzell JA, Harvey ML, Datta S, Cooper MA. Dominance relationships in Syrian hamsters modulate neuroendocrine and behavioral responses to social stress. Stress. 2018 Jun 22;1-6. doi: 10.1080/10253890.2018.1485646. Epub 2018 Jun 22. PMID: 29932809; PMCID: PMC6309596.
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Effects of corticotropin-releasing hormone on the expression of adenosine triphosphate-sensitive potassium channels (Kir6.1/SUR2B) in human term pregnant myometrium.

Obstetrics & gynecology science

Kim JY, Wu WH, Jun JH, Sohn J, Seo YS.
PMID: 29372145
Obstet Gynecol Sci. 2018 Jan;61(1):14-22. doi: 10.5468/ogs.2018.61.1.14. Epub 2017 Dec 11.

OBJECTIVE: Corticotropin-releasing hormone (CRH) is a crucial regulator of human pregnancy and parturition. Adenosine triphosphate (ATP)-sensitive potassium (KATP) channels are important for regulating myometrial quiescence during pregnancy. We investigated regulatory effects of different concentrations of CRH on KATP channel...

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Kim JY, Wu WH, Jun JH, et al. Effects of corticotropin-releasing hormone on the expression of adenosine triphosphate-sensitive potassium channels (Kir6.1/SUR2B) in human term pregnant myometrium. Obstet Gynecol Sci. 2017;61(1):14-22doi: 10.5468/ogs.2018.61.1.14.
Kim, J. Y., Wu, W. H., Jun, J. H., Sohn, J., & Seo, Y. S. (2018). Effects of corticotropin-releasing hormone on the expression of adenosine triphosphate-sensitive potassium channels (Kir6.1/SUR2B) in human term pregnant myometrium. Obstetrics & gynecology science, 61(1), 14-22. https://doi.org/10.5468/ogs.2018.61.1.14
Kim, Joo Young, et al. "Effects of corticotropin-releasing hormone on the expression of adenosine triphosphate-sensitive potassium channels (Kir6.1/SUR2B) in human term pregnant myometrium." Obstetrics & gynecology science vol. 61,1 (2018): 14-22. doi: https://doi.org/10.5468/ogs.2018.61.1.14
Kim JY, Wu WH, Jun JH, Sohn J, Seo YS. Effects of corticotropin-releasing hormone on the expression of adenosine triphosphate-sensitive potassium channels (Kir6.1/SUR2B) in human term pregnant myometrium. Obstet Gynecol Sci. 2018 Jan;61(1):14-22. doi: 10.5468/ogs.2018.61.1.14. Epub 2017 Dec 11. PMID: 29372145; PMCID: PMC5780309.
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Mistrustful and Misunderstood: A Review of Paranoid Personality Disorder.

Current behavioral neuroscience reports

Lee R.
PMID: 29399432
Curr Behav Neurosci Rep. 2017 Jun;4(2):151-165. doi: 10.1007/s40473-017-0116-7. Epub 2017 May 18.

PURPOSE OF REVIEW: Paranoid Personality Disorder (PPD) has historically been neglected by science out of proportion to its prevalence or its association with negative clinical outcomes. This review provides an update on what is known about PPD regarding its...

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Lee R. Mistrustful and Misunderstood: A Review of Paranoid Personality Disorder. Curr Behav Neurosci Rep. 2017;4(2):151-165doi: 10.1007/s40473-017-0116-7.
Lee, R. (2017). Mistrustful and Misunderstood: A Review of Paranoid Personality Disorder. Current behavioral neuroscience reports, 4(2), 151-165. https://doi.org/10.1007/s40473-017-0116-7
Lee, Royce. "Mistrustful and Misunderstood: A Review of Paranoid Personality Disorder." Current behavioral neuroscience reports vol. 4,2 (2017): 151-165. doi: https://doi.org/10.1007/s40473-017-0116-7
Lee R. Mistrustful and Misunderstood: A Review of Paranoid Personality Disorder. Curr Behav Neurosci Rep. 2017 Jun;4(2):151-165. doi: 10.1007/s40473-017-0116-7. Epub 2017 May 18. PMID: 29399432; PMCID: PMC5793931.
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Utility of a corticotropin-releasing hormone test to differentiate pituitary-dependent hyperadrenocorticism from cortisol-producing adrenal tumors in dogs.

Journal of veterinary internal medicine

Tanaka S, Suzuki S, Sato A, Teshima T, Mori A, Sako T, Tanaka A, Hara Y.
PMID: 34859496
J Vet Intern Med. 2021 Dec 03; doi: 10.1111/jvim.16336. Epub 2021 Dec 03.

BACKGROUND: Hyperadrenocorticism (HAC) is a common endocrine disorder in dogs; however, there are no reports on the use of the corticotropin-releasing hormone test (CRHT) to differentiate between pituitary-dependent hyperadrenocorticism (PDH) and cortisol-producing adrenal tumors (CPATs), both causative of HAC.OBJECTIVES:...

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Tanaka S, Suzuki S, Sato A, et al. Utility of a corticotropin-releasing hormone test to differentiate pituitary-dependent hyperadrenocorticism from cortisol-producing adrenal tumors in dogs. J Vet Intern Med. 2021;doi: 10.1111/jvim.16336.
Tanaka, S., Suzuki, S., Sato, A., Teshima, T., Mori, A., Sako, T., Tanaka, A., & Hara, Y. (2021). Utility of a corticotropin-releasing hormone test to differentiate pituitary-dependent hyperadrenocorticism from cortisol-producing adrenal tumors in dogs. Journal of veterinary internal medicine, . https://doi.org/10.1111/jvim.16336
Tanaka, Sachiyo, et al. "Utility of a corticotropin-releasing hormone test to differentiate pituitary-dependent hyperadrenocorticism from cortisol-producing adrenal tumors in dogs." Journal of veterinary internal medicine vol. (2021). doi: https://doi.org/10.1111/jvim.16336
Tanaka S, Suzuki S, Sato A, Teshima T, Mori A, Sako T, Tanaka A, Hara Y. Utility of a corticotropin-releasing hormone test to differentiate pituitary-dependent hyperadrenocorticism from cortisol-producing adrenal tumors in dogs. J Vet Intern Med. 2021 Dec 03; doi: 10.1111/jvim.16336. Epub 2021 Dec 03. PMID: 34859496.
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Showing 25 to 36 of 386 entries