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Is there more to a healthy-heart diet than cholesterol?.

Nature

Healey N.
PMID: 34108707
Nature. 2021 Jun;594(7862):S12-S13. doi: 10.1038/d41586-021-01455-4.

No abstract available.

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Healey N. Is there more to a healthy-heart diet than cholesterol?. Nature. 2021;594(7862):S12-S13doi: 10.1038/d41586-021-01455-4.
Healey, N. (2021). Is there more to a healthy-heart diet than cholesterol?. Nature, 594(7862), S12-S13. https://doi.org/10.1038/d41586-021-01455-4
Healey, Natalie. "Is there more to a healthy-heart diet than cholesterol?." Nature vol. 594,7862 (2021): S12-S13. doi: https://doi.org/10.1038/d41586-021-01455-4
Healey N. Is there more to a healthy-heart diet than cholesterol?. Nature. 2021 Jun;594(7862):S12-S13. doi: 10.1038/d41586-021-01455-4. PMID: 34108707.
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The Prevalence of Heterozygous Familial Hypercholesterolemia in Selected Regions of the Russian Federation: The FH-ESSE-RF Study.

Journal of personalized medicine

Meshkov AN, Ershova AI, Kiseleva AV, Shalnova SA, Drapkina OM, Boytsov SA, On Behalf Of The Fh-Esse-Rf Investigators.
PMID: 34074024
J Pers Med. 2021 May 24;11(6). doi: 10.3390/jpm11060464.

Heterozygous familial hypercholesterolemia (HeFH) is one of the most common genetic conditions but remains substantially underdiagnosed. The aim of our study was to investigate the prevalence of HeFH in the population of 11 different regions of Russia. Individuals were...

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Meshkov AN, Ershova AI, Kiseleva AV, et al. The Prevalence of Heterozygous Familial Hypercholesterolemia in Selected Regions of the Russian Federation: The FH-ESSE-RF Study. J Pers Med. 2021;11(6)doi: 10.3390/jpm11060464.
Meshkov, A. N., Ershova, A. I., Kiseleva, A. V., Shalnova, S. A., Drapkina, O. M., Boytsov, S. A., & On Behalf Of The Fh-Esse-Rf Investigators. (2021). The Prevalence of Heterozygous Familial Hypercholesterolemia in Selected Regions of the Russian Federation: The FH-ESSE-RF Study. Journal of personalized medicine, 11(6), . https://doi.org/10.3390/jpm11060464
Meshkov, Alexey N, et al. "The Prevalence of Heterozygous Familial Hypercholesterolemia in Selected Regions of the Russian Federation: The FH-ESSE-RF Study." Journal of personalized medicine vol. 11,6 (2021). doi: https://doi.org/10.3390/jpm11060464
Meshkov AN, Ershova AI, Kiseleva AV, Shalnova SA, Drapkina OM, Boytsov SA, On Behalf Of The Fh-Esse-Rf Investigators. The Prevalence of Heterozygous Familial Hypercholesterolemia in Selected Regions of the Russian Federation: The FH-ESSE-RF Study. J Pers Med. 2021 May 24;11(6). doi: 10.3390/jpm11060464. PMID: 34074024; PMCID: PMC8225162.
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Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome.

Nutrients

Galié S, Papandreou C, Arcelin P, Garcia D, Palau-Galindo A, Gutiérrez-Tordera L, Folch À, Bulló M.
PMID: 34959869
Nutrients. 2021 Nov 29;13(12). doi: 10.3390/nu13124318.

(1) Background: The microbiota-host cross-talk has been previously investigated, while its role in health is not yet clear. This study aimed to unravel the network of microbial-host interactions and correlate it with cardiometabolic risk factors. (2) Methods: A total...

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Galié S, Papandreou C, Arcelin P, et al. Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome. Nutrients. 2021;13(12)doi: 10.3390/nu13124318.
Galié, S., Papandreou, C., Arcelin, P., Garcia, D., Palau-Galindo, A., Gutiérrez-Tordera, L., Folch, �. �., & Bulló, M. (2021). Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome. Nutrients, 13(12), . https://doi.org/10.3390/nu13124318
Galié, Serena, et al. "Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome." Nutrients vol. 13,12 (2021). doi: https://doi.org/10.3390/nu13124318
Galié S, Papandreou C, Arcelin P, Garcia D, Palau-Galindo A, Gutiérrez-Tordera L, Folch À, Bulló M. Examining the Interaction of the Gut Microbiome with Host Metabolism and Cardiometabolic Health in Metabolic Syndrome. Nutrients. 2021 Nov 29;13(12). doi: 10.3390/nu13124318. PMID: 34959869; PMCID: PMC8706982.
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Impact of the Main Cardiovascular Risk Factors on Plasma Extracellular Vesicles and Their Influence on the Heart's Vulnerability to Ischemia-Reperfusion Injury.

Cells

Majka M, Kleibert M, Wojciechowska M.
PMID: 34943838
Cells. 2021 Nov 27;10(12). doi: 10.3390/cells10123331.

The majority of cardiovascular deaths are associated with acute coronary syndrome, especially ST-elevation myocardial infarction. Therapeutic reperfusion alone can contribute up to 40 percent of total infarct size following coronary artery occlusion, which is called ischemia-reperfusion injury (IRI). Its...

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Majka M, Kleibert M, Wojciechowska M. Impact of the Main Cardiovascular Risk Factors on Plasma Extracellular Vesicles and Their Influence on the Heart's Vulnerability to Ischemia-Reperfusion Injury. Cells. 2021;10(12)doi: 10.3390/cells10123331.
Majka, M., Kleibert, M., & Wojciechowska, M. (2021). Impact of the Main Cardiovascular Risk Factors on Plasma Extracellular Vesicles and Their Influence on the Heart's Vulnerability to Ischemia-Reperfusion Injury. Cells, 10(12), . https://doi.org/10.3390/cells10123331
Majka, Miłosz, et al. "Impact of the Main Cardiovascular Risk Factors on Plasma Extracellular Vesicles and Their Influence on the Heart's Vulnerability to Ischemia-Reperfusion Injury." Cells vol. 10,12 (2021). doi: https://doi.org/10.3390/cells10123331
Majka M, Kleibert M, Wojciechowska M. Impact of the Main Cardiovascular Risk Factors on Plasma Extracellular Vesicles and Their Influence on the Heart's Vulnerability to Ischemia-Reperfusion Injury. Cells. 2021 Nov 27;10(12). doi: 10.3390/cells10123331. PMID: 34943838.
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Lauric Acid, a Dietary Saturated Medium-Chain Fatty Acid, Elicits Calcium-Dependent Eryptosis.

Cells

Alfhili MA, Aljuraiban GS.
PMID: 34943896
Cells. 2021 Dec 01;10(12). doi: 10.3390/cells10123388.

Cardiovascular diseases (CVD) are a leading cause of mortality worldwide, and dietary habits represent a major risk factor for dyslipidemia; a hallmark of CVD. Saturated fatty acids contribute to CVD by aggravating dyslipidemia, and, in particular, lauric acid (LA)...

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Alfhili MA, Aljuraiban GS. Lauric Acid, a Dietary Saturated Medium-Chain Fatty Acid, Elicits Calcium-Dependent Eryptosis. Cells. 2021;10(12)doi: 10.3390/cells10123388.
Alfhili, M. A., & Aljuraiban, G. S. (2021). Lauric Acid, a Dietary Saturated Medium-Chain Fatty Acid, Elicits Calcium-Dependent Eryptosis. Cells, 10(12), . https://doi.org/10.3390/cells10123388
Alfhili, Mohammad A, and Aljuraiban, Ghadeer S. "Lauric Acid, a Dietary Saturated Medium-Chain Fatty Acid, Elicits Calcium-Dependent Eryptosis." Cells vol. 10,12 (2021). doi: https://doi.org/10.3390/cells10123388
Alfhili MA, Aljuraiban GS. Lauric Acid, a Dietary Saturated Medium-Chain Fatty Acid, Elicits Calcium-Dependent Eryptosis. Cells. 2021 Dec 01;10(12). doi: 10.3390/cells10123388. PMID: 34943896.
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Lipid Levels and Short-Term Risk of Recurrent Brain Infarcts in Symptomatic Intracranial Stenosis.

Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association

Prabhakaran S, Liebeskind DS, Cotsonis G, Nizam A, Feldmann E, Sangha RS, Campo-Bustillo I, Romano JG.
PMID: 34710776
J Stroke Cerebrovasc Dis. 2021 Oct 25;31(1):106141. doi: 10.1016/j.jstrokecerebrovasdis.2021.106141. Epub 2021 Oct 25.

OBJECTIVES: Hyperlipidemia is a strong risk factor for intracranial atherosclerotic disease (ICAD) and clinical stroke recurrence. We explored the effect of serum lipid levels on subclinical infarct recurrence in the Mechanisms of earlY Recurrence in Intracranial Atherosclerotic Disease (MYRIAD)...

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Prabhakaran S, Liebeskind DS, Cotsonis G, et al. Lipid Levels and Short-Term Risk of Recurrent Brain Infarcts in Symptomatic Intracranial Stenosis. J Stroke Cerebrovasc Dis. 2021;31(1):106141doi: 10.1016/j.jstrokecerebrovasdis.2021.106141.
Prabhakaran, S., Liebeskind, D. S., Cotsonis, G., Nizam, A., Feldmann, E., Sangha, R. S., Campo-Bustillo, I., Romano, J. G. (2021). Lipid Levels and Short-Term Risk of Recurrent Brain Infarcts in Symptomatic Intracranial Stenosis. Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association, 31(1), 106141. https://doi.org/10.1016/j.jstrokecerebrovasdis.2021.106141
Prabhakaran, Shyam, et al. "Lipid Levels and Short-Term Risk of Recurrent Brain Infarcts in Symptomatic Intracranial Stenosis." Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association vol. 31,1 (2021): 106141. doi: https://doi.org/10.1016/j.jstrokecerebrovasdis.2021.106141
Prabhakaran S, Liebeskind DS, Cotsonis G, Nizam A, Feldmann E, Sangha RS, Campo-Bustillo I, Romano JG. Lipid Levels and Short-Term Risk of Recurrent Brain Infarcts in Symptomatic Intracranial Stenosis. J Stroke Cerebrovasc Dis. 2021 Oct 25;31(1):106141. doi: 10.1016/j.jstrokecerebrovasdis.2021.106141. Epub 2021 Oct 25. PMID: 34710776.
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Associations between SNPs in Intestinal Cholesterol Absorption and Endogenous Cholesterol Synthesis Genes with Cholesterol Metabolism.

Biomedicines

Schroor MM, Mokhtar FBA, Plat J, Mensink RP.
PMID: 34680591
Biomedicines. 2021 Oct 14;9(10). doi: 10.3390/biomedicines9101475.

Single nucleotide polymorphisms (SNPs) have been associated with cholesterol metabolism and may partly explain large inter-individual variability in intestinal cholesterol absorption and endogenous cholesterol synthesis rates. This cross-sectional study therefore examined whether SNPs in genes encoding for proteins involved...

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Schroor MM, Mokhtar FBA, Plat J, et al. Associations between SNPs in Intestinal Cholesterol Absorption and Endogenous Cholesterol Synthesis Genes with Cholesterol Metabolism. Biomedicines. 2021;9(10)doi: 10.3390/biomedicines9101475.
Schroor, M. M., Mokhtar, F. B. A., Plat, J., & Mensink, R. P. (2021). Associations between SNPs in Intestinal Cholesterol Absorption and Endogenous Cholesterol Synthesis Genes with Cholesterol Metabolism. Biomedicines, 9(10), . https://doi.org/10.3390/biomedicines9101475
Schroor, Maite M, et al. "Associations between SNPs in Intestinal Cholesterol Absorption and Endogenous Cholesterol Synthesis Genes with Cholesterol Metabolism." Biomedicines vol. 9,10 (2021). doi: https://doi.org/10.3390/biomedicines9101475
Schroor MM, Mokhtar FBA, Plat J, Mensink RP. Associations between SNPs in Intestinal Cholesterol Absorption and Endogenous Cholesterol Synthesis Genes with Cholesterol Metabolism. Biomedicines. 2021 Oct 14;9(10). doi: 10.3390/biomedicines9101475. PMID: 34680591; PMCID: PMC8533139.
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Rhubarb hydroxyanthraquinones act as antiobesity agents to inhibit adipogenesis and enhance lipolysis.

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Fang JY, Huang TH, Chen WJ, Aljuffali IA, Hsu CY.
PMID: 34891117
Biomed Pharmacother. 2021 Dec 07;146:112497. doi: 10.1016/j.biopha.2021.112497. Epub 2021 Dec 07.

Rhubarb as an herbal medicine has been shown to exhibit antiadipogenic activity. This study evaluated and compared the lipid-lowering activity of five rhubarb hydroxyanthraquinones (HAQs), including chrysophanol, aloe emodin, emodin, physcion, and rhein, aiming to identify candidate compounds for...

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Fang JY, Huang TH, Chen WJ, et al. Rhubarb hydroxyanthraquinones act as antiobesity agents to inhibit adipogenesis and enhance lipolysis. Biomed Pharmacother. 2021;146:112497doi: 10.1016/j.biopha.2021.112497.
Fang, J. Y., Huang, T. H., Chen, W. J., Aljuffali, I. A., & Hsu, C. Y. (2021). Rhubarb hydroxyanthraquinones act as antiobesity agents to inhibit adipogenesis and enhance lipolysis. Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 146112497. https://doi.org/10.1016/j.biopha.2021.112497
Fang, Jia-You, et al. "Rhubarb hydroxyanthraquinones act as antiobesity agents to inhibit adipogenesis and enhance lipolysis." Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie vol. 146 (2021): 112497. doi: https://doi.org/10.1016/j.biopha.2021.112497
Fang JY, Huang TH, Chen WJ, Aljuffali IA, Hsu CY. Rhubarb hydroxyanthraquinones act as antiobesity agents to inhibit adipogenesis and enhance lipolysis. Biomed Pharmacother. 2021 Dec 07;146:112497. doi: 10.1016/j.biopha.2021.112497. Epub 2021 Dec 07. PMID: 34891117.
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Amyloid beta increases ABCA1 and HMGCR protein expression, and cholesterol synthesis and accumulation in mice neurons and astrocytes.

Biochimica et biophysica acta. Molecular and cell biology of lipids

Azizidoost S, Babaahmadi-Rezaei H, Nazeri Z, Cheraghzadeh M, Kheirollah A.
PMID: 34744007
Biochim Biophys Acta Mol Cell Biol Lipids. 2022 Jan;1867(1):159069. doi: 10.1016/j.bbalip.2021.159069. Epub 2021 Oct 28.

INTRODUCTION: Imbalanced cholesterol metabolism in the brain is one of the main pathophysiological mechanisms involved in Alzheimer's disease. We investigated the effect of amyloid-beta (Aβ) on the main proteins involved in regulation of cholesterol metabolism along with cholesterol content...

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Azizidoost S, Babaahmadi-Rezaei H, Nazeri Z, et al. Amyloid beta increases ABCA1 and HMGCR protein expression, and cholesterol synthesis and accumulation in mice neurons and astrocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2021;1867(1):159069doi: 10.1016/j.bbalip.2021.159069.
Azizidoost, S., Babaahmadi-Rezaei, H., Nazeri, Z., Cheraghzadeh, M., & Kheirollah, A. (2022). Amyloid beta increases ABCA1 and HMGCR protein expression, and cholesterol synthesis and accumulation in mice neurons and astrocytes. Biochimica et biophysica acta. Molecular and cell biology of lipids, 1867(1), 159069. https://doi.org/10.1016/j.bbalip.2021.159069
Azizidoost, Shirin, et al. "Amyloid beta increases ABCA1 and HMGCR protein expression, and cholesterol synthesis and accumulation in mice neurons and astrocytes." Biochimica et biophysica acta. Molecular and cell biology of lipids vol. 1867,1 (2022): 159069. doi: https://doi.org/10.1016/j.bbalip.2021.159069
Azizidoost S, Babaahmadi-Rezaei H, Nazeri Z, Cheraghzadeh M, Kheirollah A. Amyloid beta increases ABCA1 and HMGCR protein expression, and cholesterol synthesis and accumulation in mice neurons and astrocytes. Biochim Biophys Acta Mol Cell Biol Lipids. 2022 Jan;1867(1):159069. doi: 10.1016/j.bbalip.2021.159069. Epub 2021 Oct 28. PMID: 34744007.
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Impaired Retromer Function in Niemann-Pick Type C Disease Is Dependent on Intracellular Cholesterol Accumulation.

International journal of molecular sciences

Dominko K, Rastija A, Sobocanec S, Vidatic L, Meglaj S, Lovincic Babic A, Hutter-Paier B, Colombo AV, Lichtenthaler SF, Tahirovic S, Hecimovic S.
PMID: 34948052
Int J Mol Sci. 2021 Dec 09;22(24). doi: 10.3390/ijms222413256.

Niemann-Pick type C disease (NPC) is a rare inherited neurodegenerative disorder characterized by an accumulation of intracellular cholesterol within late endosomes and lysosomes due to NPC1 or NPC2 dysfunction. In this work, we tested the hypothesis that retromer impairment...

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Dominko K, Rastija A, Sobocanec S, et al. Impaired Retromer Function in Niemann-Pick Type C Disease Is Dependent on Intracellular Cholesterol Accumulation. Int J Mol Sci. 2021;22(24)doi: 10.3390/ijms222413256.
Dominko, K., Rastija, A., Sobocanec, S., Vidatic, L., Meglaj, S., Lovincic Babic, A., Hutter-Paier, B., Colombo, A. V., Lichtenthaler, S. F., Tahirovic, S., & Hecimovic, S. (2021). Impaired Retromer Function in Niemann-Pick Type C Disease Is Dependent on Intracellular Cholesterol Accumulation. International journal of molecular sciences, 22(24), . https://doi.org/10.3390/ijms222413256
Dominko, Kristina, et al. "Impaired Retromer Function in Niemann-Pick Type C Disease Is Dependent on Intracellular Cholesterol Accumulation." International journal of molecular sciences vol. 22,24 (2021). doi: https://doi.org/10.3390/ijms222413256
Dominko K, Rastija A, Sobocanec S, Vidatic L, Meglaj S, Lovincic Babic A, Hutter-Paier B, Colombo AV, Lichtenthaler SF, Tahirovic S, Hecimovic S. Impaired Retromer Function in Niemann-Pick Type C Disease Is Dependent on Intracellular Cholesterol Accumulation. Int J Mol Sci. 2021 Dec 09;22(24). doi: 10.3390/ijms222413256. PMID: 34948052; PMCID: PMC8705785.
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Effectiveness of a new rutin Cu(II) complex in the prevention of lipid peroxidation and hepatotoxicity in hypercholesterolemic rats.

Journal of food biochemistry

de Oliveira DAF, Diniz SN, Pereira RMS, Gonçalves ID, Rennó AL, Gorjão R, Vieira EG, da C Ferreira AM, Okuyama CE.
PMID: 34747031
J Food Biochem. 2021 Nov 07;e13999. doi: 10.1111/jfbc.13999. Epub 2021 Nov 07.

A new rutin copper(II) complex (R-Cu2) was prepared and characterized by spectroscopic methods and elemental analysis. The effects of rutin and R-Cu2 were evaluated on the prevention of hypercholesterolemia in animals feed with high-cholesterol diet (HCD) for 8 weeks....

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de Oliveira DAF, Diniz SN, Pereira RMS, et al. Effectiveness of a new rutin Cu(II) complex in the prevention of lipid peroxidation and hepatotoxicity in hypercholesterolemic rats. J Food Biochem. 2021;e13999doi: 10.1111/jfbc.13999.
de Oliveira, D. A. F., Diniz, S. N., Pereira, R. M. S., Gonçalves, I. D., Rennó, A. L., Gorjão, R., Vieira, E. G., da C Ferreira, A. M., & Okuyama, C. E. (2021). Effectiveness of a new rutin Cu(II) complex in the prevention of lipid peroxidation and hepatotoxicity in hypercholesterolemic rats. Journal of food biochemistry, e13999. https://doi.org/10.1111/jfbc.13999
de Oliveira, Daniel A F, et al. "Effectiveness of a new rutin Cu(II) complex in the prevention of lipid peroxidation and hepatotoxicity in hypercholesterolemic rats." Journal of food biochemistry vol. (2021): e13999. doi: https://doi.org/10.1111/jfbc.13999
de Oliveira DAF, Diniz SN, Pereira RMS, Gonçalves ID, Rennó AL, Gorjão R, Vieira EG, da C Ferreira AM, Okuyama CE. Effectiveness of a new rutin Cu(II) complex in the prevention of lipid peroxidation and hepatotoxicity in hypercholesterolemic rats. J Food Biochem. 2021 Nov 07;e13999. doi: 10.1111/jfbc.13999. Epub 2021 Nov 07. PMID: 34747031.
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The emerging role of the sigma-1 receptor in autophagy: hand-in-hand targets for the treatment of Alzheimer's.

Expert opinion on therapeutic targets

Prasanth MI, Malar DS, Tencomnao T, Brimson JM.
PMID: 34110944
Expert Opin Ther Targets. 2021 May;25(5):401-414. doi: 10.1080/14728222.2021.1939681. Epub 2021 Jun 17.

INTRODUCTION: Autophagy is a cellular catabolic mechanism that helps clear damaged cellular components and is essential for normal cellular and tissue function. The sigma-1 receptor (σ-1R) is a chaperone protein involved in signal transduction, neurite outgrowth, and plasticity, improving...

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Prasanth MI, Malar DS, Tencomnao T, et al. The emerging role of the sigma-1 receptor in autophagy: hand-in-hand targets for the treatment of Alzheimer's. Expert Opin Ther Targets. 2021;25(5):401-414doi: 10.1080/14728222.2021.1939681.
Prasanth, M. I., Malar, D. S., Tencomnao, T., & Brimson, J. M. (2021). The emerging role of the sigma-1 receptor in autophagy: hand-in-hand targets for the treatment of Alzheimer's. Expert opinion on therapeutic targets, 25(5), 401-414. https://doi.org/10.1080/14728222.2021.1939681
Prasanth, Mani Iyer, et al. "The emerging role of the sigma-1 receptor in autophagy: hand-in-hand targets for the treatment of Alzheimer's." Expert opinion on therapeutic targets vol. 25,5 (2021): 401-414. doi: https://doi.org/10.1080/14728222.2021.1939681
Prasanth MI, Malar DS, Tencomnao T, Brimson JM. The emerging role of the sigma-1 receptor in autophagy: hand-in-hand targets for the treatment of Alzheimer's. Expert Opin Ther Targets. 2021 May;25(5):401-414. doi: 10.1080/14728222.2021.1939681. Epub 2021 Jun 17. PMID: 34110944.
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