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Showing 37 to 44 of 44 entries
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[Idiopathic edema].

Nihon rinsho. Japanese journal of clinical medicine

Kuwahara M.
PMID: 15675327
Nihon Rinsho. 2005 Jan;63(1):109-12.

Idiopathic edema is an orthostatic edema accompanied by excessive weight gain from morning to evening. It occurs almost exclusively in post pubertal women and is independent of menstral cycle. Although the cause is not clear, a number of hormonal...

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Kuwahara M. [Idiopathic edema]. Nihon Rinsho. 2005;63(1):109-12
Kuwahara, M. (2005). [Idiopathic edema]. Nihon rinsho. Japanese journal of clinical medicine, 63(1), 109-12.
Kuwahara, Michio. "[Idiopathic edema]." Nihon rinsho. Japanese journal of clinical medicine vol. 63,1 (2005): 109-12.
Kuwahara M. [Idiopathic edema]. Nihon Rinsho. 2005 Jan;63(1):109-12. Japanese. PMID: 15675327.
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The heart as an endocrine organ.

Endocrine connections

Ogawa T, de Bold AJ.
PMID: 24562677
Endocr Connect. 2014 Apr 15;3(2):R31-44. doi: 10.1530/EC-14-0012. Print 2014.

The concept of the heart as an endocrine organ arises from the observation that the atrial cardiomyocytes in the mammalian heart display a phenotype that is partly that of endocrine cells. Investigations carried out between 1971 and 1983 characterised,...

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Ogawa T, de Bold AJ. The heart as an endocrine organ. Endocr Connect. 2014;3(2):R31-44doi: 10.1530/EC-14-0012.
Ogawa, T., & de Bold, A. J. (2014). The heart as an endocrine organ. Endocrine connections, 3(2), R31-44. https://doi.org/10.1530/EC-14-0012
Ogawa, Tsuneo, and de Bold, Adolfo J. "The heart as an endocrine organ." Endocrine connections vol. 3,2 (2014): R31-44. doi: https://doi.org/10.1530/EC-14-0012
Ogawa T, de Bold AJ. The heart as an endocrine organ. Endocr Connect. 2014 Apr 15;3(2):R31-44. doi: 10.1530/EC-14-0012. Print 2014. PMID: 24562677; PMCID: PMC3987289.
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Brain natriuretic Peptide production and secretion in inflammation.

Journal of transplantation

Ogawa T, de Bold AJ.
PMID: 23251786
J Transplant. 2012;2012:962347. doi: 10.1155/2012/962347. Epub 2012 Nov 28.

Gene expression and secretion of the cardiac polypeptide hormones atrial natriuretic factor (ANF) and brain natriuretic peptide (BNP) are simultaneously upregulated in various cardiac disorders such as congestive heart failure, ischemic heart disease, and hypertensive heart disease, in which...

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Ogawa T, de Bold AJ. Brain natriuretic Peptide production and secretion in inflammation. J Transplant. 2012;2012:962347doi: 10.1155/2012/962347.
Ogawa, T., & de Bold, A. J. (2012). Brain natriuretic Peptide production and secretion in inflammation. Journal of transplantation, 2012962347. https://doi.org/10.1155/2012/962347
Ogawa, Tsuneo, and de Bold, Adolfo J. "Brain natriuretic Peptide production and secretion in inflammation." Journal of transplantation vol. 2012 (2012): 962347. doi: https://doi.org/10.1155/2012/962347
Ogawa T, de Bold AJ. Brain natriuretic Peptide production and secretion in inflammation. J Transplant. 2012;2012:962347. doi: 10.1155/2012/962347. Epub 2012 Nov 28. PMID: 23251786; PMCID: PMC3515950.
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Effects of buyang huanwu decoction on ventricular remodeling and differential protein profile in a rat model of myocardial infarction.

Evidence-based complementary and alternative medicine : eCAM

Zhou YC, Liu B, Li YJ, Jing LL, Wen G, Tang J, Xu X, Lv ZP, Sun XG.
PMID: 23049607
Evid Based Complement Alternat Med. 2012;2012:385247. doi: 10.1155/2012/385247. Epub 2012 Sep 19.

Buyang Huanwu decoction (BYHWD) is a well-known and canonical Chinese medicine formula from "Correction on Errors in Medical Classics" in Qing dynasty. Here, we show that BYHWD could alleviate the ventricular remodeling induced by left anterior descending (LAD) artery...

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Zhou YC, Liu B, Li YJ, et al. Effects of buyang huanwu decoction on ventricular remodeling and differential protein profile in a rat model of myocardial infarction. Evid Based Complement Alternat Med. 2012;2012:385247doi: 10.1155/2012/385247.
Zhou, Y. C., Liu, B., Li, Y. J., Jing, L. L., Wen, G., Tang, J., Xu, X., Lv, Z. P., & Sun, X. G. (2012). Effects of buyang huanwu decoction on ventricular remodeling and differential protein profile in a rat model of myocardial infarction. Evidence-based complementary and alternative medicine : eCAM, 2012385247. https://doi.org/10.1155/2012/385247
Zhou, Ying Chun, et al. "Effects of buyang huanwu decoction on ventricular remodeling and differential protein profile in a rat model of myocardial infarction." Evidence-based complementary and alternative medicine : eCAM vol. 2012 (2012): 385247. doi: https://doi.org/10.1155/2012/385247
Zhou YC, Liu B, Li YJ, Jing LL, Wen G, Tang J, Xu X, Lv ZP, Sun XG. Effects of buyang huanwu decoction on ventricular remodeling and differential protein profile in a rat model of myocardial infarction. Evid Based Complement Alternat Med. 2012;2012:385247. doi: 10.1155/2012/385247. Epub 2012 Sep 19. PMID: 23049607; PMCID: PMC3459299.
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Sphingosine 1 phosphate receptor-1 (S1PR1) signaling protects cardiac function by inhibiting cardiomyocyte autophagy.

Journal of geriatric cardiology : JGC

Chen YZ, Wang F, Wang HJ, Liu HB.
PMID: 30083186
J Geriatr Cardiol. 2018 May;15(5):334-345. doi: 10.11909/j.issn.1671-5411.2018.05.003.

OBJECTIVE: To investigate the role of sphingosine-1-phosphate (S1P) and its receptors in cardiomyocyte autophagy, cardiomyocyte hypertrophy and cardiac function.METHODS: Cardiomyocytes were isolated from neonatal Vista rats. Autophagy and hypertrophy of cardiomyocytes were induced via starvation culture and phenylephrine (PE),...

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Chen YZ, Wang F, Wang HJ, et al. Sphingosine 1 phosphate receptor-1 (S1PR1) signaling protects cardiac function by inhibiting cardiomyocyte autophagy. J Geriatr Cardiol. 2018;15(5):334-345doi: 10.11909/j.issn.1671-5411.2018.05.003.
Chen, Y. Z., Wang, F., Wang, H. J., & Liu, H. B. (2018). Sphingosine 1 phosphate receptor-1 (S1PR1) signaling protects cardiac function by inhibiting cardiomyocyte autophagy. Journal of geriatric cardiology : JGC, 15(5), 334-345. https://doi.org/10.11909/j.issn.1671-5411.2018.05.003
Chen, Yong-Zeng, et al. "Sphingosine 1 phosphate receptor-1 (S1PR1) signaling protects cardiac function by inhibiting cardiomyocyte autophagy." Journal of geriatric cardiology : JGC vol. 15,5 (2018): 334-345. doi: https://doi.org/10.11909/j.issn.1671-5411.2018.05.003
Chen YZ, Wang F, Wang HJ, Liu HB. Sphingosine 1 phosphate receptor-1 (S1PR1) signaling protects cardiac function by inhibiting cardiomyocyte autophagy. J Geriatr Cardiol. 2018 May;15(5):334-345. doi: 10.11909/j.issn.1671-5411.2018.05.003. PMID: 30083186; PMCID: PMC6064777.
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Effect of early versus delayed activation of thoracic epidural anesthesia on plasma pro-atrial natriuretic peptide to indicate deviations in central blood volume during esophagectomy.

Regional anesthesia and pain medicine

Strandby RB, Ambrus R, Achiam MP, Goetze JP, Secher NH, Svendsen LB.
PMID: 31129617
Reg Anesth Pain Med. 2019 May 24; doi: 10.1136/rapm-2019-100508. Epub 2019 May 24.

BACKGROUND AND OBJECTIVES: A side effect to thoracic epidural anesthesia (TEA) is hypotension induced by central hypovolemia. This study addressed whether early activation (EA) versus late activation (LA) of TEA affects plasma pro-atrial natriuretic peptide (proANP) reflecting deviations in...

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Strandby RB, Ambrus R, Achiam MP, et al. Effect of early versus delayed activation of thoracic epidural anesthesia on plasma pro-atrial natriuretic peptide to indicate deviations in central blood volume during esophagectomy. Reg Anesth Pain Med. 2019;doi: 10.1136/rapm-2019-100508.
Strandby, R. B., Ambrus, R., Achiam, M. P., Goetze, J. P., Secher, N. H., & Svendsen, L. B. (2019). Effect of early versus delayed activation of thoracic epidural anesthesia on plasma pro-atrial natriuretic peptide to indicate deviations in central blood volume during esophagectomy. Regional anesthesia and pain medicine, . https://doi.org/10.1136/rapm-2019-100508
Strandby, Rune B, et al. "Effect of early versus delayed activation of thoracic epidural anesthesia on plasma pro-atrial natriuretic peptide to indicate deviations in central blood volume during esophagectomy." Regional anesthesia and pain medicine vol. (2019). doi: https://doi.org/10.1136/rapm-2019-100508
Strandby RB, Ambrus R, Achiam MP, Goetze JP, Secher NH, Svendsen LB. Effect of early versus delayed activation of thoracic epidural anesthesia on plasma pro-atrial natriuretic peptide to indicate deviations in central blood volume during esophagectomy. Reg Anesth Pain Med. 2019 May 24; doi: 10.1136/rapm-2019-100508. Epub 2019 May 24. PMID: 31129617.
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Fibroblast growth factor 21 inhibition aggravates cardiac dysfunction in diabetic cardiomyopathy by improving lipid accumulation.

Experimental and therapeutic medicine

Chen C, Meng Z, Zheng Y, Hu B, Shen E.
PMID: 29375676
Exp Ther Med. 2018 Jan;15(1):75-84. doi: 10.3892/etm.2017.5375. Epub 2017 Oct 25.

Diabetic cardiomyopathy (DCM) is one of the major causes of morbidity and mortality in diabetic patients. Recent studies have demonstrated an increased level of fibroblast growth factor 21 (FGF21) in the plasma of DCM patients, and FGF21 has been...

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Chen C, Meng Z, Zheng Y, et al. Fibroblast growth factor 21 inhibition aggravates cardiac dysfunction in diabetic cardiomyopathy by improving lipid accumulation. Exp Ther Med. 2017;15(1):75-84doi: 10.3892/etm.2017.5375.
Chen, C., Meng, Z., Zheng, Y., Hu, B., & Shen, E. (2018). Fibroblast growth factor 21 inhibition aggravates cardiac dysfunction in diabetic cardiomyopathy by improving lipid accumulation. Experimental and therapeutic medicine, 15(1), 75-84. https://doi.org/10.3892/etm.2017.5375
Chen, Cui, et al. "Fibroblast growth factor 21 inhibition aggravates cardiac dysfunction in diabetic cardiomyopathy by improving lipid accumulation." Experimental and therapeutic medicine vol. 15,1 (2018): 75-84. doi: https://doi.org/10.3892/etm.2017.5375
Chen C, Meng Z, Zheng Y, Hu B, Shen E. Fibroblast growth factor 21 inhibition aggravates cardiac dysfunction in diabetic cardiomyopathy by improving lipid accumulation. Exp Ther Med. 2018 Jan;15(1):75-84. doi: 10.3892/etm.2017.5375. Epub 2017 Oct 25. PMID: 29375676; PMCID: PMC5763648.
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MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling.

Cell biology international

Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P.
PMID: 34854520
Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02.

MicroRNAs (miRNAs) regulate multiple biological processes and participate in various cardiovascular diseases. This study aims to investigate the role of miR-339-5p in cardiomyocyte hypertrophy and the involved mechanism. Neonatal rat cardiomyocytes (NRCMs) were cultured and stimulated with isoproterenol (ISO)....

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Bi X, Zhang Y, Yu Y, et al. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021;doi: 10.1002/cbin.11731.
Bi, X., Zhang, Y., Yu, Y., Yuan, J., Xu, S., Liu, F., Ye, J., & Liu, P. (2021). MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell biology international, . https://doi.org/10.1002/cbin.11731
Bi, Xueying, et al. "MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling." Cell biology international vol. (2021). doi: https://doi.org/10.1002/cbin.11731
Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02. PMID: 34854520.
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Showing 37 to 44 of 44 entries