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NOX4 stimulates ANF secretion via activation of the Sirt1/Nrf2/ATF3/4 axis in hypoxic beating rat atria.

Molecular medicine reports

Li ZY, Liu Y, Wang YY, Li X, Han ZN, Hong L, Li YS, Cui X.
PMID: 35029280
Mol Med Rep. 2022 Mar;25(3). doi: 10.3892/mmr.2022.12600. Epub 2022 Jan 14.

Silent information regulator factor 2‑related enzyme 1 (Sirt1) is involved in the regulation of cell senescence, gene transcription, energy balance and oxidative stress. However, the effect of Sirt1 on atrial natriuretic factor (ANF) secretion, especially under hypoxic conditions is...

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Li ZY, Liu Y, Wang YY, et al. NOX4 stimulates ANF secretion via activation of the Sirt1/Nrf2/ATF3/4 axis in hypoxic beating rat atria. Mol Med Rep. 2022;25(3)doi: 10.3892/mmr.2022.12600.
Li, Z. Y., Liu, Y., Wang, Y. Y., Li, X., Han, Z. N., Hong, L., Li, Y. S., & Cui, X. (2022). NOX4 stimulates ANF secretion via activation of the Sirt1/Nrf2/ATF3/4 axis in hypoxic beating rat atria. Molecular medicine reports, 25(3), . https://doi.org/10.3892/mmr.2022.12600
Li, Zhi-Yu, et al. "NOX4 stimulates ANF secretion via activation of the Sirt1/Nrf2/ATF3/4 axis in hypoxic beating rat atria." Molecular medicine reports vol. 25,3 (2022). doi: https://doi.org/10.3892/mmr.2022.12600
Li ZY, Liu Y, Wang YY, Li X, Han ZN, Hong L, Li YS, Cui X. NOX4 stimulates ANF secretion via activation of the Sirt1/Nrf2/ATF3/4 axis in hypoxic beating rat atria. Mol Med Rep. 2022 Mar;25(3). doi: 10.3892/mmr.2022.12600. Epub 2022 Jan 14. PMID: 35029280.
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MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling.

Cell biology international

Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P.
PMID: 34854520
Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02.

MicroRNAs (miRNAs) regulate multiple biological processes and participate in various cardiovascular diseases. This study aims to investigate the role of miR-339-5p in cardiomyocyte hypertrophy and the involved mechanism. Neonatal rat cardiomyocytes (NRCMs) were cultured and stimulated with isoproterenol (ISO)....

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Bi X, Zhang Y, Yu Y, et al. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021;doi: 10.1002/cbin.11731.
Bi, X., Zhang, Y., Yu, Y., Yuan, J., Xu, S., Liu, F., Ye, J., & Liu, P. (2021). MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell biology international, . https://doi.org/10.1002/cbin.11731
Bi, Xueying, et al. "MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling." Cell biology international vol. (2021). doi: https://doi.org/10.1002/cbin.11731
Bi X, Zhang Y, Yu Y, Yuan J, Xu S, Liu F, Ye J, Liu P. MiRNA-339-5p promotes isoproterenol-induced cardiomyocyte hypertrophy by targeting VCP to activate the mTOR signaling. Cell Biol Int. 2021 Dec 02; doi: 10.1002/cbin.11731. Epub 2021 Dec 02. PMID: 34854520.
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Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments.

Frontiers in physiology

Cerrudo CS, Cavallero S, Rodríguez Fermepín M, González GE, Donato M, Kouyoumdzian NM, Gelpi RJ, Hertig CM, Choi MR, Fernández BE.
PMID: 34113261
Front Physiol. 2021 May 25;12:651246. doi: 10.3389/fphys.2021.651246. eCollection 2021.

The involvement of natriuretic peptides was studied during the hypertrophic remodeling transition mediated by sequential exposure to chronic hemodynamic overload. We induced hypertension in rats by pressure (renovascular) or volume overload (DOCA-salt) during 6 and 12 weeks of treatment....

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Cerrudo CS, Cavallero S, Rodríguez Fermepín M, et al. Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments. Front Physiol. 2021;12:651246doi: 10.3389/fphys.2021.651246.
Cerrudo, C. S., Cavallero, S., Rodríguez Fermepín, M., González, G. E., Donato, M., Kouyoumdzian, N. M., Gelpi, R. J., Hertig, C. M., Choi, M. R., & Fernández, B. E. (2021). Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments. Frontiers in physiology, 12651246. https://doi.org/10.3389/fphys.2021.651246
Cerrudo, Carolina S, et al. "Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments." Frontiers in physiology vol. 12 (2021): 651246. doi: https://doi.org/10.3389/fphys.2021.651246
Cerrudo CS, Cavallero S, Rodríguez Fermepín M, González GE, Donato M, Kouyoumdzian NM, Gelpi RJ, Hertig CM, Choi MR, Fernández BE. Cardiac Natriuretic Peptide Profiles in Chronic Hypertension by Single or Sequentially Combined Renovascular and DOCA-Salt Treatments. Front Physiol. 2021 May 25;12:651246. doi: 10.3389/fphys.2021.651246. eCollection 2021. PMID: 34113261; PMCID: PMC8185994.
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Clinicopathological features of clinically undiagnosed sporadic transthyretin cardiac amyloidosis: a forensic autopsy-based series.

Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis

Ichimata S, Hata Y, Hirono K, Yamaguchi Y, Nishida N.
PMID: 33554665
Amyloid. 2021 Jun;28(2):125-133. doi: 10.1080/13506129.2021.1882979. Epub 2021 Feb 08.

OBJECTIVE: To investigate the clinicopathological features of sporadic amyloid transthyretin (ATTR) amyloidosis.METHODS: We evaluated 1698 serial Japanese forensic autopsy patients. The extent and amount of ATTR deposition in the 16 cardiac regions, including the conduction system, were semiquantitatively evaluated....

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Ichimata S, Hata Y, Hirono K, et al. Clinicopathological features of clinically undiagnosed sporadic transthyretin cardiac amyloidosis: a forensic autopsy-based series. Amyloid. 2021;28(2):125-133doi: 10.1080/13506129.2021.1882979.
Ichimata, S., Hata, Y., Hirono, K., Yamaguchi, Y., & Nishida, N. (2021). Clinicopathological features of clinically undiagnosed sporadic transthyretin cardiac amyloidosis: a forensic autopsy-based series. Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis, 28(2), 125-133. https://doi.org/10.1080/13506129.2021.1882979
Ichimata, Shojiro, et al. "Clinicopathological features of clinically undiagnosed sporadic transthyretin cardiac amyloidosis: a forensic autopsy-based series." Amyloid : the international journal of experimental and clinical investigation : the official journal of the International Society of Amyloidosis vol. 28,2 (2021): 125-133. doi: https://doi.org/10.1080/13506129.2021.1882979
Ichimata S, Hata Y, Hirono K, Yamaguchi Y, Nishida N. Clinicopathological features of clinically undiagnosed sporadic transthyretin cardiac amyloidosis: a forensic autopsy-based series. Amyloid. 2021 Jun;28(2):125-133. doi: 10.1080/13506129.2021.1882979. Epub 2021 Feb 08. PMID: 33554665.
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Alpha Mangostin Derived from Garcinia magostana Linn Ameliorates Cardiomyocyte Hypertrophy and Fibroblast Phenotypes in Vitro.

Biological & pharmaceutical bulletin

Sari N, Katanasaka Y, Sugiyama Y, Miyazaki Y, Sunagawa Y, Funamoto M, Shimizu K, Shimizu S, Hasegawa K, Morimoto T.
PMID: 34602555
Biol Pharm Bull. 2021;44(10):1465-1472. doi: 10.1248/bpb.b21-00294.

Cardiac hypertrophy and fibrosis are significant risk factors for chronic heart failure (HF). Since pharmacotherapy agents targeting these processes have not been established, we investigated the effect of alpha-magostin (α-man) on cardiomyocyte hypertrophy and fibrosis in vitro. Primary cultured...

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Sari N, Katanasaka Y, Sugiyama Y, et al. Alpha Mangostin Derived from Garcinia magostana Linn Ameliorates Cardiomyocyte Hypertrophy and Fibroblast Phenotypes in Vitro. Biol Pharm Bull. 2021;44(10):1465-1472doi: 10.1248/bpb.b21-00294.
Sari, N., Katanasaka, Y., Sugiyama, Y., Miyazaki, Y., Sunagawa, Y., Funamoto, M., Shimizu, K., Shimizu, S., Hasegawa, K., & Morimoto, T. (2021). Alpha Mangostin Derived from Garcinia magostana Linn Ameliorates Cardiomyocyte Hypertrophy and Fibroblast Phenotypes in Vitro. Biological & pharmaceutical bulletin, 44(10), 1465-1472. https://doi.org/10.1248/bpb.b21-00294
Sari, Nurmila, et al. "Alpha Mangostin Derived from Garcinia magostana Linn Ameliorates Cardiomyocyte Hypertrophy and Fibroblast Phenotypes in Vitro." Biological & pharmaceutical bulletin vol. 44,10 (2021): 1465-1472. doi: https://doi.org/10.1248/bpb.b21-00294
Sari N, Katanasaka Y, Sugiyama Y, Miyazaki Y, Sunagawa Y, Funamoto M, Shimizu K, Shimizu S, Hasegawa K, Morimoto T. Alpha Mangostin Derived from Garcinia magostana Linn Ameliorates Cardiomyocyte Hypertrophy and Fibroblast Phenotypes in Vitro. Biol Pharm Bull. 2021;44(10):1465-1472. doi: 10.1248/bpb.b21-00294. PMID: 34602555.
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cGMP-Producing, Atrial Natriuretic Factor-Responding Cells in the Rat Brain.

The European journal of neuroscience

De Vente J, Bol JG, Steinbusch HW.
PMID: 12106130
Eur J Neurosci. 1989 Sep;1(5):436-460. doi: 10.1111/j.1460-9568.1989.tb00351.x.

Using an in vitro incubation method, we stimulated cGMP production in rat brain slices by rat ANF-(103 - 126). The localization of the cells responding to this ANF stimulation with an increase in cGMP production was studied by cGMP...

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De Vente J, Bol JG, Steinbusch HW. cGMP-Producing, Atrial Natriuretic Factor-Responding Cells in the Rat Brain. Eur J Neurosci. 1989;1(5):436-460doi: 10.1111/j.1460-9568.1989.tb00351.x.
De Vente, J., Bol, J. G., & Steinbusch, H. W. (1989). cGMP-Producing, Atrial Natriuretic Factor-Responding Cells in the Rat Brain. The European journal of neuroscience, 1(5), 436-460. https://doi.org/10.1111/j.1460-9568.1989.tb00351.x
De Vente, J., et al. "cGMP-Producing, Atrial Natriuretic Factor-Responding Cells in the Rat Brain." The European journal of neuroscience vol. 1,5 (1989): 436-460. doi: https://doi.org/10.1111/j.1460-9568.1989.tb00351.x
De Vente J, Bol JG, Steinbusch HW. cGMP-Producing, Atrial Natriuretic Factor-Responding Cells in the Rat Brain. Eur J Neurosci. 1989 Sep;1(5):436-460. doi: 10.1111/j.1460-9568.1989.tb00351.x. PMID: 12106130.
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Forebrain binding sites for atrial natriuretic factor: Alterations in spontaneously hypertensive (SHR) rats.

Neurochemistry international

McCarty R, Plunkett LM.
PMID: 20493115
Neurochem Int. 1986;9(1):177-83. doi: 10.1016/0197-0186(86)90046-x.

Binding sites for atrial natriuretic factor (ANF-28) were studied in forebrain areas of spontaneously hypertensive (SHR) and Wistar Kyoto (WKY) normotensive male rats by quantitative autoradiography. The maximum binding capacity of [(125)I]ANF-28 was significantly reduced in the subfornical organ...

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McCarty R, Plunkett LM. Forebrain binding sites for atrial natriuretic factor: Alterations in spontaneously hypertensive (SHR) rats. Neurochem Int. 1986;9(1):177-83doi: 10.1016/0197-0186(86)90046-x.
McCarty, R., & Plunkett, L. M. (1986). Forebrain binding sites for atrial natriuretic factor: Alterations in spontaneously hypertensive (SHR) rats. Neurochemistry international, 9(1), 177-83. https://doi.org/10.1016/0197-0186(86)90046-x
McCarty, R, and Plunkett, L M. "Forebrain binding sites for atrial natriuretic factor: Alterations in spontaneously hypertensive (SHR) rats." Neurochemistry international vol. 9,1 (1986): 177-83. doi: https://doi.org/10.1016/0197-0186(86)90046-x
McCarty R, Plunkett LM. Forebrain binding sites for atrial natriuretic factor: Alterations in spontaneously hypertensive (SHR) rats. Neurochem Int. 1986;9(1):177-83. doi: 10.1016/0197-0186(86)90046-x. PMID: 20493115.
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Adult rat cardiomyocytes in culture A model system to study the plasticity of the differentiated cardiac phenotype at the molecular and cellular levels.

Trends in cardiovascular medicine

Eppenberger HM, Hertig C, Eppenberger-Eberhardt M.
PMID: 21244885
Trends Cardiovasc Med. 1994 Jul-Aug;4(4):187-93. doi: 10.1016/1050-1738(94)90056-6.

Adult rat cardiomyocytes (ARCs) in long-term culture, which show a distinct adaptive flexibility, are presented as a system to study cardiac cell hypertrophy in vitro. In the first 1-2 weeks after isolation, ARCs undergo a process of de- and...

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Eppenberger HM, Hertig C, Eppenberger-Eberhardt M. Adult rat cardiomyocytes in culture A model system to study the plasticity of the differentiated cardiac phenotype at the molecular and cellular levels. Trends Cardiovasc Med. 1994;4(4):187-93doi: 10.1016/1050-1738(94)90056-6.
Eppenberger, H. M., Hertig, C., & Eppenberger-Eberhardt, M. (1994). Adult rat cardiomyocytes in culture A model system to study the plasticity of the differentiated cardiac phenotype at the molecular and cellular levels. Trends in cardiovascular medicine, 4(4), 187-93. https://doi.org/10.1016/1050-1738(94)90056-6
Eppenberger, H M, et al. "Adult rat cardiomyocytes in culture A model system to study the plasticity of the differentiated cardiac phenotype at the molecular and cellular levels." Trends in cardiovascular medicine vol. 4,4 (1994): 187-93. doi: https://doi.org/10.1016/1050-1738(94)90056-6
Eppenberger HM, Hertig C, Eppenberger-Eberhardt M. Adult rat cardiomyocytes in culture A model system to study the plasticity of the differentiated cardiac phenotype at the molecular and cellular levels. Trends Cardiovasc Med. 1994 Jul-Aug;4(4):187-93. doi: 10.1016/1050-1738(94)90056-6. PMID: 21244885.
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Flavonoids Extraction from Propolis Attenuates Pathological Cardiac Hypertrophy through PI3K/AKT Signaling Pathway.

Evidence-based complementary and alternative medicine : eCAM

Sun GW, Qiu ZD, Wang WN, Sui X, Sui DJ.
PMID: 27213000
Evid Based Complement Alternat Med. 2016;2016:6281376. doi: 10.1155/2016/6281376. Epub 2016 Apr 24.

Propolis, a traditional medicine, has been widely used for a thousand years as an anti-inflammatory and antioxidant drug. The flavonoid fraction is the main active component of propolis, which possesses a wide range of biological activities, including activities related...

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Sun GW, Qiu ZD, Wang WN, et al. Flavonoids Extraction from Propolis Attenuates Pathological Cardiac Hypertrophy through PI3K/AKT Signaling Pathway. Evid Based Complement Alternat Med. 2016;2016:6281376doi: 10.1155/2016/6281376.
Sun, G. W., Qiu, Z. D., Wang, W. N., Sui, X., & Sui, D. J. (2016). Flavonoids Extraction from Propolis Attenuates Pathological Cardiac Hypertrophy through PI3K/AKT Signaling Pathway. Evidence-based complementary and alternative medicine : eCAM, 20166281376. https://doi.org/10.1155/2016/6281376
Sun, Guang-Wei, et al. "Flavonoids Extraction from Propolis Attenuates Pathological Cardiac Hypertrophy through PI3K/AKT Signaling Pathway." Evidence-based complementary and alternative medicine : eCAM vol. 2016 (2016): 6281376. doi: https://doi.org/10.1155/2016/6281376
Sun GW, Qiu ZD, Wang WN, Sui X, Sui DJ. Flavonoids Extraction from Propolis Attenuates Pathological Cardiac Hypertrophy through PI3K/AKT Signaling Pathway. Evid Based Complement Alternat Med. 2016;2016:6281376. doi: 10.1155/2016/6281376. Epub 2016 Apr 24. PMID: 27213000; PMCID: PMC4860246.
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Genetics and epigenetics of arrhythmia and heart failure.

Frontiers in genetics

Duygu B, Poels EM, da Costa Martins PA.
PMID: 24198825
Front Genet. 2013 Oct 30;4:219. doi: 10.3389/fgene.2013.00219.

Heart failure (HF) is the end stage of several pathological cardiac conditions including myocardial infarction, cardiac hypertrophy and hypertension. Various molecular and cellular mechanisms are involved in the development of HF. At the molecular level, the onset of HF...

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Duygu B, Poels EM, da Costa Martins PA. Genetics and epigenetics of arrhythmia and heart failure. Front Genet. 2013;4:219doi: 10.3389/fgene.2013.00219.
Duygu, B., Poels, E. M., & da Costa Martins, P. A. (2013). Genetics and epigenetics of arrhythmia and heart failure. Frontiers in genetics, 4219. https://doi.org/10.3389/fgene.2013.00219
Duygu, Burcu, et al. "Genetics and epigenetics of arrhythmia and heart failure." Frontiers in genetics vol. 4 (2013): 219. doi: https://doi.org/10.3389/fgene.2013.00219
Duygu B, Poels EM, da Costa Martins PA. Genetics and epigenetics of arrhythmia and heart failure. Front Genet. 2013 Oct 30;4:219. doi: 10.3389/fgene.2013.00219. PMID: 24198825; PMCID: PMC3812794.
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Gestational diabetes influences the expression of hypertrophic genes in left ventricle of rat's offspring.

Iranian journal of basic medical sciences

Kermani ES, Nazari Z, Mehdizadeh M, Shahbazi M, Golalipour MJ.
PMID: 29922434
Iran J Basic Med Sci. 2018 May;21(5):525-528. doi: 10.22038/IJBMS.2018.25116.6233.

OBJECTIVES: Gestational diabetes increases the risk of congenital heart disease in the offspring, but the molecular mechanism underlying this process remains unclear. Therefore, the current study was conducted to assess the effects of induced gestational diabetes on expression of...

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Kermani ES, Nazari Z, Mehdizadeh M, et al. Gestational diabetes influences the expression of hypertrophic genes in left ventricle of rat's offspring. Iran J Basic Med Sci. 2018;21(5):525-528doi: 10.22038/IJBMS.2018.25116.6233.
Kermani, E. S., Nazari, Z., Mehdizadeh, M., Shahbazi, M., & Golalipour, M. J. (2018). Gestational diabetes influences the expression of hypertrophic genes in left ventricle of rat's offspring. Iranian journal of basic medical sciences, 21(5), 525-528. https://doi.org/10.22038/IJBMS.2018.25116.6233
Kermani, Elia Saragard, et al. "Gestational diabetes influences the expression of hypertrophic genes in left ventricle of rat's offspring." Iranian journal of basic medical sciences vol. 21,5 (2018): 525-528. doi: https://doi.org/10.22038/IJBMS.2018.25116.6233
Kermani ES, Nazari Z, Mehdizadeh M, Shahbazi M, Golalipour MJ. Gestational diabetes influences the expression of hypertrophic genes in left ventricle of rat's offspring. Iran J Basic Med Sci. 2018 May;21(5):525-528. doi: 10.22038/IJBMS.2018.25116.6233. PMID: 29922434; PMCID: PMC6000218.
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Atrial natriuretic peptides in the diagnosis and treatment of congestive heart failure.

Congestive heart failure (Greenwich, Conn.)

Vesely DL.
PMID: 12189309
Congest Heart Fail. 1999 Jul-Aug;5(4):171-179.

Atrial natriuretic peptides (ANPs) consist of a family of peptides (atrial natriuretic factor [ANF], long acting natriuretic peptide, vessel dilator, kaliuretic peptide, urodilatin, brain natriuretic peptide [BNP], and C type natriuretic peptide [CNP]) which are synthesized within the heart,...

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Vesely DL. Atrial natriuretic peptides in the diagnosis and treatment of congestive heart failure. Congest Heart Fail. 1999;5(4):171-179
Vesely, D. L. (1999). Atrial natriuretic peptides in the diagnosis and treatment of congestive heart failure. Congestive heart failure (Greenwich, Conn.), 5(4), 171-179.
Vesely, D L. "Atrial natriuretic peptides in the diagnosis and treatment of congestive heart failure." Congestive heart failure (Greenwich, Conn.) vol. 5,4 (1999): 171-179.
Vesely DL. Atrial natriuretic peptides in the diagnosis and treatment of congestive heart failure. Congest Heart Fail. 1999 Jul-Aug;5(4):171-179. PMID: 12189309.
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Showing 13 to 24 of 44 entries