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Showing 13 to 18 of 18 entries
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Genetic markers in the diagnosis of Alzheimer's disease.

Journal of Alzheimer's disease : JAD

Rogaeva E, Tandon A, St George-Hyslop PH.
PMID: 12214050
J Alzheimers Dis. 2001 Jun;3(3):293-304. doi: 10.3233/jad-2001-3304.

Abbreviations: AD, Alzheimer's disease; AbetaPP, amyloid beta protein precursor; BACE, beta-site AbetaPP cleaving enzyme; PS1, presenilin-1; PS2, presenilin-2; APOE, apolipoprotein E; LRP, low density lipoprotein receptor-related protein; SNPs, single-nucleotide polymorphisms; A2M, alpha-2-macroglobulin. Alzheimer's disease (AD) is the most common...

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Rogaeva E, Tandon A, St George-Hyslop PH. Genetic markers in the diagnosis of Alzheimer's disease. J Alzheimers Dis. 2001;3(3):293-304doi: 10.3233/jad-2001-3304.
Rogaeva, E., Tandon, A., & St George-Hyslop, P. H. (2001). Genetic markers in the diagnosis of Alzheimer's disease. Journal of Alzheimer's disease : JAD, 3(3), 293-304. https://doi.org/10.3233/jad-2001-3304
Rogaeva, Ekaterina, et al. "Genetic markers in the diagnosis of Alzheimer's disease." Journal of Alzheimer's disease : JAD vol. 3,3 (2001): 293-304. doi: https://doi.org/10.3233/jad-2001-3304
Rogaeva E, Tandon A, St George-Hyslop PH. Genetic markers in the diagnosis of Alzheimer's disease. J Alzheimers Dis. 2001 Jun;3(3):293-304. doi: 10.3233/jad-2001-3304. PMID: 12214050.
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Commentary: Abeta N- Terminal Isoforms: Critical contributors in the course of AD pathophysiology.

Journal of Alzheimer's disease : JAD

Tekirian TL.
PMID: 12214065
J Alzheimers Dis. 2001 Apr;3(2):241-248. doi: 10.3233/jad-2001-3209.

The assessment of protein or amino acid variations across evolution allows one to glean divergent features of disease-specific pathology. Within the Alzheimer's disease (AD) literature, extensive differences in Abeta processing across cell lines and evolution have clearly been observed....

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Tekirian TL. Commentary: Abeta N- Terminal Isoforms: Critical contributors in the course of AD pathophysiology. J Alzheimers Dis. 2001;3(2):241-248doi: 10.3233/jad-2001-3209.
Tekirian, T. L. (2001). Commentary: Abeta N- Terminal Isoforms: Critical contributors in the course of AD pathophysiology. Journal of Alzheimer's disease : JAD, 3(2), 241-248. https://doi.org/10.3233/jad-2001-3209
Tekirian, Tina L.. "Commentary: Abeta N- Terminal Isoforms: Critical contributors in the course of AD pathophysiology." Journal of Alzheimer's disease : JAD vol. 3,2 (2001): 241-248. doi: https://doi.org/10.3233/jad-2001-3209
Tekirian TL. Commentary: Abeta N- Terminal Isoforms: Critical contributors in the course of AD pathophysiology. J Alzheimers Dis. 2001 Apr;3(2):241-248. doi: 10.3233/jad-2001-3209. PMID: 12214065.
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Recent advances in the understanding of the role of synaptic proteins in Alzheimer's Disease and other neurodegenerative disorders.

Journal of Alzheimer's disease : JAD

Masliah E.
PMID: 12214081
J Alzheimers Dis. 2001 Feb;3(1):121-129. doi: 10.3233/jad-2001-3117.

Synaptic damage is an early pathological event common to many neurodegenerative disorders such as Alzheimer's disease (AD) and is the best correlate to the cognitive impairment. Several molecules involved in AD and in other neurodegenerative disorders play an important...

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Masliah E. Recent advances in the understanding of the role of synaptic proteins in Alzheimer's Disease and other neurodegenerative disorders. J Alzheimers Dis. 2001;3(1):121-129doi: 10.3233/jad-2001-3117.
Masliah, E. (2001). Recent advances in the understanding of the role of synaptic proteins in Alzheimer's Disease and other neurodegenerative disorders. Journal of Alzheimer's disease : JAD, 3(1), 121-129. https://doi.org/10.3233/jad-2001-3117
Masliah, Eliezer. "Recent advances in the understanding of the role of synaptic proteins in Alzheimer's Disease and other neurodegenerative disorders." Journal of Alzheimer's disease : JAD vol. 3,1 (2001): 121-129. doi: https://doi.org/10.3233/jad-2001-3117
Masliah E. Recent advances in the understanding of the role of synaptic proteins in Alzheimer's Disease and other neurodegenerative disorders. J Alzheimers Dis. 2001 Feb;3(1):121-129. doi: 10.3233/jad-2001-3117. PMID: 12214081.
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Dexamethasone diminishes the pro-inflammatory and cytotoxic effects of amyloid beta-protein in cerebrovascular smooth muscle cells.

Journal of neuroinflammation

Previti ML, Zhang W, Van Nostrand WE.
PMID: 16887032
J Neuroinflammation. 2006 Aug 03;3:18. doi: 10.1186/1742-2094-3-18.

BACKGROUND: Cerebrovascular deposition of fibrillar amyloid beta-protein (Abeta), a condition known as cerebral amyloid angiopathy (CAA), is a prominent pathological feature of Alzheimer's disease (AD) and related disorders. Accumulation of cerebral vascular fibrillar Abeta is implicated in promoting local...

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Previti ML, Zhang W, Van Nostrand WE. Dexamethasone diminishes the pro-inflammatory and cytotoxic effects of amyloid beta-protein in cerebrovascular smooth muscle cells. J Neuroinflammation. 2006;3:18doi: 10.1186/1742-2094-3-18.
Previti, M. L., Zhang, W., & Van Nostrand, W. E. (2006). Dexamethasone diminishes the pro-inflammatory and cytotoxic effects of amyloid beta-protein in cerebrovascular smooth muscle cells. Journal of neuroinflammation, 318. https://doi.org/10.1186/1742-2094-3-18
Previti, Mary Lou, et al. "Dexamethasone diminishes the pro-inflammatory and cytotoxic effects of amyloid beta-protein in cerebrovascular smooth muscle cells." Journal of neuroinflammation vol. 3 (2006): 18. doi: https://doi.org/10.1186/1742-2094-3-18
Previti ML, Zhang W, Van Nostrand WE. Dexamethasone diminishes the pro-inflammatory and cytotoxic effects of amyloid beta-protein in cerebrovascular smooth muscle cells. J Neuroinflammation. 2006 Aug 03;3:18. doi: 10.1186/1742-2094-3-18. PMID: 16887032; PMCID: PMC1557842.
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Developing BACE-1 inhibitors for FXS.

Frontiers in cellular neuroscience

Westmark CJ, Berry-Kravis EM, Ikonomidou C, Yin JC, Puglielli L.
PMID: 23754978
Front Cell Neurosci. 2013 May 28;7:77. doi: 10.3389/fncel.2013.00077. eCollection 2013.

Fragile X syndrome (FXS) is a debilitating genetic disorder with no cure and few therapeutic options. Excessive signaling through metabotropic glutamate receptor 5 in FXS leads to increased translation of numerous synaptic proteins and exaggerated long-term depression. Two of...

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Westmark CJ, Berry-Kravis EM, Ikonomidou C, et al. Developing BACE-1 inhibitors for FXS. Front Cell Neurosci. 2013;7:77doi: 10.3389/fncel.2013.00077.
Westmark, C. J., Berry-Kravis, E. M., Ikonomidou, C., Yin, J. C., & Puglielli, L. (2013). Developing BACE-1 inhibitors for FXS. Frontiers in cellular neuroscience, 777. https://doi.org/10.3389/fncel.2013.00077
Westmark, Cara J, et al. "Developing BACE-1 inhibitors for FXS." Frontiers in cellular neuroscience vol. 7 (2013): 77. doi: https://doi.org/10.3389/fncel.2013.00077
Westmark CJ, Berry-Kravis EM, Ikonomidou C, Yin JC, Puglielli L. Developing BACE-1 inhibitors for FXS. Front Cell Neurosci. 2013 May 28;7:77. doi: 10.3389/fncel.2013.00077. eCollection 2013. PMID: 23754978; PMCID: PMC3664772.
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L-3-n-butylphthalide improves cognitive impairment of APP/PS1 mice by BDNF/TrkB/PI3K/AKT pathway.

International journal of clinical and experimental medicine

Xiang J, Pan J, Chen F, Zheng L, Chen Y, Zhang S, Feng W.
PMID: 25126168
Int J Clin Exp Med. 2014 Jul 15;7(7):1706-13. eCollection 2014.

L-3-n-butylphthalide (L-NBP), an extract from seeds of Apium graveolens Linn (Chinese celery), has been shown to have neuroprotective effects on cerebral ischemic, vascular dementia and amyloid-beta (Abeta)-induced animal models by inhibiting oxidative injury, neuronal apoptosis and glial activation, regulating...

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Xiang J, Pan J, Chen F, et al. L-3-n-butylphthalide improves cognitive impairment of APP/PS1 mice by BDNF/TrkB/PI3K/AKT pathway. Int J Clin Exp Med. 2014;7(7):1706-13
Xiang, J., Pan, J., Chen, F., Zheng, L., Chen, Y., Zhang, S., & Feng, W. (2014). L-3-n-butylphthalide improves cognitive impairment of APP/PS1 mice by BDNF/TrkB/PI3K/AKT pathway. International journal of clinical and experimental medicine, 7(7), 1706-13.
Xiang, Jing, et al. "L-3-n-butylphthalide improves cognitive impairment of APP/PS1 mice by BDNF/TrkB/PI3K/AKT pathway." International journal of clinical and experimental medicine vol. 7,7 (2014): 1706-13.
Xiang J, Pan J, Chen F, Zheng L, Chen Y, Zhang S, Feng W. L-3-n-butylphthalide improves cognitive impairment of APP/PS1 mice by BDNF/TrkB/PI3K/AKT pathway. Int J Clin Exp Med. 2014 Jul 15;7(7):1706-13. eCollection 2014. PMID: 25126168; PMCID: PMC4132132.
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Showing 13 to 18 of 18 entries