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ACE for all - a molecular perspective.

Journal of cell communication and signaling

Harrison C, Acharya KR.
PMID: 25027949
J Cell Commun Signal. 2014 Sep;8(3):195-210. doi: 10.1007/s12079-014-0236-8. Epub 2014 Jul 16.

Angiotensin-I converting enzyme (ACE, EC 3.4.15.1) is a zinc dependent dipeptidyl carboxypeptidase with an essential role in mammalian blood pressure regulation as part of the renin-angiotensin aldosterone system (RAAS). As such, it has long been targeted in the treatment...

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Harrison C, Acharya KR. ACE for all - a molecular perspective. J Cell Commun Signal. 2014;8(3):195-210doi: 10.1007/s12079-014-0236-8.
Harrison, C., & Acharya, K. R. (2014). ACE for all - a molecular perspective. Journal of cell communication and signaling, 8(3), 195-210. https://doi.org/10.1007/s12079-014-0236-8
Harrison, Charlotte, and Acharya, K Ravi. "ACE for all - a molecular perspective." Journal of cell communication and signaling vol. 8,3 (2014): 195-210. doi: https://doi.org/10.1007/s12079-014-0236-8
Harrison C, Acharya KR. ACE for all - a molecular perspective. J Cell Commun Signal. 2014 Sep;8(3):195-210. doi: 10.1007/s12079-014-0236-8. Epub 2014 Jul 16. PMID: 25027949; PMCID: PMC4165820.
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Recent advances in managing and understanding diabetic nephropathy.

F1000Research

Tang SC, Chan GC, Lai KN.
PMID: 27303648
F1000Res. 2016 May 31;5. doi: 10.12688/f1000research.7693.1. eCollection 2016.

Diabetic nephropathy is the commonest cause of end-stage renal disease in most developed economies. Current standard of care for diabetic nephropathy embraces stringent blood pressure control via blockade of the renin-angiotensin-aldosterone system and glycemia control. Recent understanding of the...

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Tang SC, Chan GC, Lai KN. Recent advances in managing and understanding diabetic nephropathy. F1000Res. 2016;5doi: 10.12688/f1000research.7693.1.
Tang, S. C., Chan, G. C., & Lai, K. N. (2016). Recent advances in managing and understanding diabetic nephropathy. F1000Research, 5. https://doi.org/10.12688/f1000research.7693.1
Tang, Sydney C W, et al. "Recent advances in managing and understanding diabetic nephropathy." F1000Research vol. 5 (2016). doi: https://doi.org/10.12688/f1000research.7693.1
Tang SC, Chan GC, Lai KN. Recent advances in managing and understanding diabetic nephropathy. F1000Res. 2016 May 31;5. doi: 10.12688/f1000research.7693.1. eCollection 2016. PMID: 27303648; PMCID: PMC4892357.
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Recurrence of Acute Page Kidney in a Renal Transplant Allograft.

Case reports in medicine

Kapoor R, Zayas C, Mulloy L, Jagadeesan M.
PMID: 27725836
Case Rep Med. 2016;2016:3898307. doi: 10.1155/2016/3898307. Epub 2016 Sep 20.

Acute Page Kidney (APK) phenomenon is a rare cause of secondary hypertension, mediated by activation of renin-angiotensin-aldosterone system (RAAS). Timely intervention is of great importance to prevent any end organ damage from hypertension. We present a unique case of...

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Kapoor R, Zayas C, Mulloy L, et al. Recurrence of Acute Page Kidney in a Renal Transplant Allograft. Case Rep Med. 2016;2016:3898307doi: 10.1155/2016/3898307.
Kapoor, R., Zayas, C., Mulloy, L., & Jagadeesan, M. (2016). Recurrence of Acute Page Kidney in a Renal Transplant Allograft. Case reports in medicine, 20163898307. https://doi.org/10.1155/2016/3898307
Kapoor, Rajan, et al. "Recurrence of Acute Page Kidney in a Renal Transplant Allograft." Case reports in medicine vol. 2016 (2016): 3898307. doi: https://doi.org/10.1155/2016/3898307
Kapoor R, Zayas C, Mulloy L, Jagadeesan M. Recurrence of Acute Page Kidney in a Renal Transplant Allograft. Case Rep Med. 2016;2016:3898307. doi: 10.1155/2016/3898307. Epub 2016 Sep 20. PMID: 27725836; PMCID: PMC5048039.
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Addition of a renin-angiotensin-aldosterone system inhibitor to a calcium channel blocker ameliorates arterial stiffness.

Clinical pharmacology : advances and applications

Kiuchi S, Hisatake S, Kawasaki M, Hirashima O, Kabuki T, Yamazaki J, Ikeda T.
PMID: 26487804
Clin Pharmacol. 2015 Oct 08;7:97-102. doi: 10.2147/CPAA.S81880. eCollection 2015.

BACKGROUND: The aim of controlling hypertension is to protect against arteriosclerosis. Calcium channel blockers (CCBs) and renin-angiotensin-aldosterone system (RAAS) inhibitors have been reported to have antihypertensive effects, but their effect on the progression of arteriosclerosis is not fully understood....

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Kiuchi S, Hisatake S, Kawasaki M, et al. Addition of a renin-angiotensin-aldosterone system inhibitor to a calcium channel blocker ameliorates arterial stiffness. Clin Pharmacol. 2015;7:97-102doi: 10.2147/CPAA.S81880.
Kiuchi, S., Hisatake, S., Kawasaki, M., Hirashima, O., Kabuki, T., Yamazaki, J., & Ikeda, T. (2015). Addition of a renin-angiotensin-aldosterone system inhibitor to a calcium channel blocker ameliorates arterial stiffness. Clinical pharmacology : advances and applications, 797-102. https://doi.org/10.2147/CPAA.S81880
Kiuchi, Shunsuke, et al. "Addition of a renin-angiotensin-aldosterone system inhibitor to a calcium channel blocker ameliorates arterial stiffness." Clinical pharmacology : advances and applications vol. 7 (2015): 97-102. doi: https://doi.org/10.2147/CPAA.S81880
Kiuchi S, Hisatake S, Kawasaki M, Hirashima O, Kabuki T, Yamazaki J, Ikeda T. Addition of a renin-angiotensin-aldosterone system inhibitor to a calcium channel blocker ameliorates arterial stiffness. Clin Pharmacol. 2015 Oct 08;7:97-102. doi: 10.2147/CPAA.S81880. eCollection 2015. PMID: 26487804; PMCID: PMC4607055.
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Editorial: ACTH Action in the Adrenal Cortex: From Molecular Biology to Pathophysiology.

Frontiers in endocrinology

Gallo-Payet N, Martinez A, Lacroix A.
PMID: 28659864
Front Endocrinol (Lausanne). 2017 Jun 12;8:101. doi: 10.3389/fendo.2017.00101. eCollection 2017.

No abstract available.

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Gallo-Payet N, Martinez A, Lacroix A. Editorial: ACTH Action in the Adrenal Cortex: From Molecular Biology to Pathophysiology. Front Endocrinol (Lausanne). 2017;8:101doi: 10.3389/fendo.2017.00101.
Gallo-Payet, N., Martinez, A., & Lacroix, A. (2017). Editorial: ACTH Action in the Adrenal Cortex: From Molecular Biology to Pathophysiology. Frontiers in endocrinology, 8101. https://doi.org/10.3389/fendo.2017.00101
Gallo-Payet, Nicole, et al. "Editorial: ACTH Action in the Adrenal Cortex: From Molecular Biology to Pathophysiology." Frontiers in endocrinology vol. 8 (2017): 101. doi: https://doi.org/10.3389/fendo.2017.00101
Gallo-Payet N, Martinez A, Lacroix A. Editorial: ACTH Action in the Adrenal Cortex: From Molecular Biology to Pathophysiology. Front Endocrinol (Lausanne). 2017 Jun 12;8:101. doi: 10.3389/fendo.2017.00101. eCollection 2017. PMID: 28659864; PMCID: PMC5467431.
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Baseline Serum Aldosterone-to-Renin Ratio is Associated with the Add-on Effect of Thiazide Diuretics in Non-Diabetic Essential Hypertensives.

Acta Cardiologica Sinica

Huang CC, Leu HB, Huang PH, Wu TC, Lin SJ, Chen JW.
PMID: 27122683
Acta Cardiol Sin. 2013 Jan;29(1):37-48.

BACKGROUND: The baseline status of renin-angiotensin-aldosterone system (RAAS) might modify the blood pressure (BP) lowering effects of thiazide diuretics. This study aimed to determine if baseline RAAS indicated by serum aldosterone-to-renin ratio (ARR) could be associated with the add-on...

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Huang CC, Leu HB, Huang PH, et al. Baseline Serum Aldosterone-to-Renin Ratio is Associated with the Add-on Effect of Thiazide Diuretics in Non-Diabetic Essential Hypertensives. Acta Cardiol Sin. 2013;29(1):37-48
Huang, C. C., Leu, H. B., Huang, P. H., Wu, T. C., Lin, S. J., & Chen, J. W. (2013). Baseline Serum Aldosterone-to-Renin Ratio is Associated with the Add-on Effect of Thiazide Diuretics in Non-Diabetic Essential Hypertensives. Acta Cardiologica Sinica, 29(1), 37-48.
Huang, Chin-Chou, et al. "Baseline Serum Aldosterone-to-Renin Ratio is Associated with the Add-on Effect of Thiazide Diuretics in Non-Diabetic Essential Hypertensives." Acta Cardiologica Sinica vol. 29,1 (2013): 37-48.
Huang CC, Leu HB, Huang PH, Wu TC, Lin SJ, Chen JW. Baseline Serum Aldosterone-to-Renin Ratio is Associated with the Add-on Effect of Thiazide Diuretics in Non-Diabetic Essential Hypertensives. Acta Cardiol Sin. 2013 Jan;29(1):37-48. PMID: 27122683; PMCID: PMC4804959.
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Effect of aldosterone on epithelial-to-mesenchymal transition of human peritoneal mesothelial cells.

Kidney research and clinical practice

Yu M, Shin HS, Lee HK, Ryu DR, Kim SJ, Choi KB, Kang DH.
PMID: 26484027
Kidney Res Clin Pract. 2015 Jun;34(2):83-92. doi: 10.1016/j.krcp.2015.03.005. Epub 2015 Apr 25.

BACKGROUND: Peritoneal fibrosis is one of the major causes of technical failure in patients on peritoneal dialysis. Epithelial-to-mesenchymal transition (EMT) of the peritoneum is an early and reversible mechanism of peritoneal fibrosis. Human peritoneal mesothelial cells (HPMCs) have their...

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Yu M, Shin HS, Lee HK, et al. Effect of aldosterone on epithelial-to-mesenchymal transition of human peritoneal mesothelial cells. Kidney Res Clin Pract. 2015;34(2):83-92doi: 10.1016/j.krcp.2015.03.005.
Yu, M., Shin, H. S., Lee, H. K., Ryu, D. R., Kim, S. J., Choi, K. B., & Kang, D. H. (2015). Effect of aldosterone on epithelial-to-mesenchymal transition of human peritoneal mesothelial cells. Kidney research and clinical practice, 34(2), 83-92. https://doi.org/10.1016/j.krcp.2015.03.005
Yu, Mina, et al. "Effect of aldosterone on epithelial-to-mesenchymal transition of human peritoneal mesothelial cells." Kidney research and clinical practice vol. 34,2 (2015): 83-92. doi: https://doi.org/10.1016/j.krcp.2015.03.005
Yu M, Shin HS, Lee HK, Ryu DR, Kim SJ, Choi KB, Kang DH. Effect of aldosterone on epithelial-to-mesenchymal transition of human peritoneal mesothelial cells. Kidney Res Clin Pract. 2015 Jun;34(2):83-92. doi: 10.1016/j.krcp.2015.03.005. Epub 2015 Apr 25. PMID: 26484027; PMCID: PMC4570652.
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Renin-Angiotensin-Aldosterone System Blockade in Diabetic Nephropathy. Present Evidences.

Journal of clinical medicine

Lozano-Maneiro L, Puente-García A.
PMID: 26569322
J Clin Med. 2015 Nov 09;4(11):1908-37. doi: 10.3390/jcm4111908.

Diabetic Kidney Disease (DKD) is the leading cause of chronic kidney disease in developed countries and its prevalence has increased dramatically in the past few decades. These patients are at an increased risk for premature death, cardiovascular disease, and...

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Lozano-Maneiro L, Puente-García A. Renin-Angiotensin-Aldosterone System Blockade in Diabetic Nephropathy. Present Evidences. J Clin Med. 2015;4(11):1908-37doi: 10.3390/jcm4111908.
Lozano-Maneiro, L., & Puente-García, A. (2015). Renin-Angiotensin-Aldosterone System Blockade in Diabetic Nephropathy. Present Evidences. Journal of clinical medicine, 4(11), 1908-37. https://doi.org/10.3390/jcm4111908
Lozano-Maneiro, Luz, and Puente-García, Adriana. "Renin-Angiotensin-Aldosterone System Blockade in Diabetic Nephropathy. Present Evidences." Journal of clinical medicine vol. 4,11 (2015): 1908-37. doi: https://doi.org/10.3390/jcm4111908
Lozano-Maneiro L, Puente-García A. Renin-Angiotensin-Aldosterone System Blockade in Diabetic Nephropathy. Present Evidences. J Clin Med. 2015 Nov 09;4(11):1908-37. doi: 10.3390/jcm4111908. PMID: 26569322; PMCID: PMC4663476.
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Aldosterone Signals the Onset of Depressive Behaviour in a Female Rat Model of Depression along with SSRI Treatment Resistance.

Neuroendocrinology

Franklin M, Hlavacova N, Babic S, Pokusa M, Bermudez I, Jezova D.
PMID: 25968351
Neuroendocrinology. 2015;102(4):274-287. doi: 10.1159/000431152. Epub 2015 May 09.

Depression is a serious condition that occurs more frequently in women and is often associated with treatment resistance. The main hypotheses of this study are that (a) aldosterone is an early marker of depression onset and (b) a tryptophan...

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Franklin M, Hlavacova N, Babic S, et al. Aldosterone Signals the Onset of Depressive Behaviour in a Female Rat Model of Depression along with SSRI Treatment Resistance. Neuroendocrinology. 2015;102(4):274-287doi: 10.1159/000431152.
Franklin, M., Hlavacova, N., Babic, S., Pokusa, M., Bermudez, I., & Jezova, D. (2015). Aldosterone Signals the Onset of Depressive Behaviour in a Female Rat Model of Depression along with SSRI Treatment Resistance. Neuroendocrinology, 102(4), 274-287. https://doi.org/10.1159/000431152
Franklin, Michael, et al. "Aldosterone Signals the Onset of Depressive Behaviour in a Female Rat Model of Depression along with SSRI Treatment Resistance." Neuroendocrinology vol. 102,4 (2015): 274-287. doi: https://doi.org/10.1159/000431152
Franklin M, Hlavacova N, Babic S, Pokusa M, Bermudez I, Jezova D. Aldosterone Signals the Onset of Depressive Behaviour in a Female Rat Model of Depression along with SSRI Treatment Resistance. Neuroendocrinology. 2015;102(4):274-287. doi: 10.1159/000431152. Epub 2015 May 09. PMID: 25968351.
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Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis.

Annals of rehabilitation medicine

Lee JH, Kim E, Chon S.
PMID: 26605182
Ann Rehabil Med. 2015 Oct;39(5):826-32. doi: 10.5535/arm.2015.39.5.826. Epub 2015 Oct 26.

We describes a patient with hypokalemia-induced rhabdomyolysis due to primary aldosteronism (PA), who suffered from slowly progressive muscle weakness after laparoscopic adrenalectomy, and was later diagnosed with coexisting sporadic inclusion body myositis (sIBM). A 54-year-old Asian male presented with...

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Lee JH, Kim E, Chon S. Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis. Ann Rehabil Med. 2015;39(5):826-32doi: 10.5535/arm.2015.39.5.826.
Lee, J. H., Kim, E., & Chon, S. (2015). Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis. Annals of rehabilitation medicine, 39(5), 826-32. https://doi.org/10.5535/arm.2015.39.5.826
Lee, Jong Ha, et al. "Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis." Annals of rehabilitation medicine vol. 39,5 (2015): 826-32. doi: https://doi.org/10.5535/arm.2015.39.5.826
Lee JH, Kim E, Chon S. Hypokalemia-Induced Rhabdomyolysis by Primary Aldosteronism Coexistent With Sporadic Inclusion Body Myositis. Ann Rehabil Med. 2015 Oct;39(5):826-32. doi: 10.5535/arm.2015.39.5.826. Epub 2015 Oct 26. PMID: 26605182; PMCID: PMC4654090.
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Telmisartan reduced abdominal circumference and body weight with decreasing triglyceride level in patients with type 2 diabetes and metabolic syndrome.

Obesity research & clinical practice

Kakuma T, Gotoh K, Masaki T, Itateyama E, Abe N, Yoshimatsu H.
PMID: 24345653
Obes Res Clin Pract. 2010 Apr-Jun;4(2):e83-e162. doi: 10.1016/j.orcp.2009.12.003.

SUMMARY: Telmisartan has the dual ability to inhibit angiotensin II type 1 receptors and activate peroxisome proliferator-activated receptor gamma (PPAR), especially as a selective PPAR modulator that does not strongly promote adipogenesis for weight gain. Accordingly, Telmisartan is expected...

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Kakuma T, Gotoh K, Masaki T, et al. Telmisartan reduced abdominal circumference and body weight with decreasing triglyceride level in patients with type 2 diabetes and metabolic syndrome. Obes Res Clin Pract. 2010;4(2):e83-e162doi: 10.1016/j.orcp.2009.12.003.
Kakuma, T., Gotoh, K., Masaki, T., Itateyama, E., Abe, N., & Yoshimatsu, H. (2010). Telmisartan reduced abdominal circumference and body weight with decreasing triglyceride level in patients with type 2 diabetes and metabolic syndrome. Obesity research & clinical practice, 4(2), e83-e162. https://doi.org/10.1016/j.orcp.2009.12.003
Kakuma, Tetsuya, et al. "Telmisartan reduced abdominal circumference and body weight with decreasing triglyceride level in patients with type 2 diabetes and metabolic syndrome." Obesity research & clinical practice vol. 4,2 (2010): e83-e162. doi: https://doi.org/10.1016/j.orcp.2009.12.003
Kakuma T, Gotoh K, Masaki T, Itateyama E, Abe N, Yoshimatsu H. Telmisartan reduced abdominal circumference and body weight with decreasing triglyceride level in patients with type 2 diabetes and metabolic syndrome. Obes Res Clin Pract. 2010 Apr-Jun;4(2):e83-e162. doi: 10.1016/j.orcp.2009.12.003. PMID: 24345653.
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Impact of aldosterone-producing adenoma on cardiac structures in echocardiography.

Journal of echocardiography

Hidaka T, Shiwa T, Fujii Y, Nishioka K, Utsunomiya H, Ishibashi K, Mitsuba N, Dohi Y, Oda N, Noma K, Kurisu S, Nakano Y, Yamamoto H, Iishida T, Higashi Y, Kihara Y.
PMID: 24319340
J Echocardiogr. 2013;11:123-9. doi: 10.1007/s12574-013-0168-y. Epub 2013 Feb 05.

BACKGROUND: Primary aldosteronism (PA) is a most common cause of secondary hypertension. In PA, left ventricular hypertrophy (LVH) is more progressive than in any other cause of hypertension. Aldosterone-producing adenoma (APA) and idiopathic hyperaldosteronism (IHA) are major subtypes of...

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Hidaka T, Shiwa T, Fujii Y, et al. Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. J Echocardiogr. 2013;11:123-9doi: 10.1007/s12574-013-0168-y.
Hidaka, T., Shiwa, T., Fujii, Y., Nishioka, K., Utsunomiya, H., Ishibashi, K., Mitsuba, N., Dohi, Y., Oda, N., Noma, K., Kurisu, S., Nakano, Y., Yamamoto, H., Iishida, T., Higashi, Y., & Kihara, Y. (2013). Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. Journal of echocardiography, 11123-9. https://doi.org/10.1007/s12574-013-0168-y
Hidaka, Takayuki, et al. "Impact of aldosterone-producing adenoma on cardiac structures in echocardiography." Journal of echocardiography vol. 11 (2013): 123-9. doi: https://doi.org/10.1007/s12574-013-0168-y
Hidaka T, Shiwa T, Fujii Y, Nishioka K, Utsunomiya H, Ishibashi K, Mitsuba N, Dohi Y, Oda N, Noma K, Kurisu S, Nakano Y, Yamamoto H, Iishida T, Higashi Y, Kihara Y. Impact of aldosterone-producing adenoma on cardiac structures in echocardiography. J Echocardiogr. 2013;11:123-9. doi: 10.1007/s12574-013-0168-y. Epub 2013 Feb 05. PMID: 24319340; PMCID: PMC3851697.
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Showing 13 to 24 of 2536 entries