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Increased serum dehydroepiandrosterone sulfate in the first episode but not in subsequent episodes in male patients with schizophrenia.

Neuropsychiatric disease and treatment

Beyazyüz M, Albayrak Y, Beyazyüz E, Unsal C, Göka E.
PMID: 24812513
Neuropsychiatr Dis Treat. 2014 Apr 29;10:687-93. doi: 10.2147/NDT.S61406. eCollection 2014.

BACKGROUND: Many studies have investigated the relationship between blood levels of dehydroepiandrosterone (DHEA) and its sulfate ester (DHEA-S), cortisol, progesterone, and testosterone and the onset, prognosis, symptom severity, and treatment response of schizophrenia. In the present study, we assessed...

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Beyazyüz M, Albayrak Y, Beyazyüz E, et al. Increased serum dehydroepiandrosterone sulfate in the first episode but not in subsequent episodes in male patients with schizophrenia. Neuropsychiatr Dis Treat. 2014;10:687-93doi: 10.2147/NDT.S61406.
Beyazyüz, M., Albayrak, Y., Beyazyüz, E., Unsal, C., & Göka, E. (2014). Increased serum dehydroepiandrosterone sulfate in the first episode but not in subsequent episodes in male patients with schizophrenia. Neuropsychiatric disease and treatment, 10687-93. https://doi.org/10.2147/NDT.S61406
Beyazyüz, Murat, et al. "Increased serum dehydroepiandrosterone sulfate in the first episode but not in subsequent episodes in male patients with schizophrenia." Neuropsychiatric disease and treatment vol. 10 (2014): 687-93. doi: https://doi.org/10.2147/NDT.S61406
Beyazyüz M, Albayrak Y, Beyazyüz E, Unsal C, Göka E. Increased serum dehydroepiandrosterone sulfate in the first episode but not in subsequent episodes in male patients with schizophrenia. Neuropsychiatr Dis Treat. 2014 Apr 29;10:687-93. doi: 10.2147/NDT.S61406. eCollection 2014. PMID: 24812513; PMCID: PMC4011925.
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Adrenocorticotropic Hormone-Dependent Cushing's Syndrome: Use of an octreotide trial to distinguish between pituitary or ectopic sources.

Sultan Qaboos University medical journal

El-Shafie OT, Al-Saffi N, Al-Sajwani A, Woodhouse N.
PMID: 25685371
Sultan Qaboos Univ Med J. 2015 Feb;15(1):e120-3. Epub 2015 Jan 21.

OBJECTIVES: Adrenocorticotropic hormone (ACTH) overproduction is usually due to a pituitary tumour which is often not visible on magnetic resonance imaging (MRI). However, ACTH overproduction may be due to an ectopic source. This study aimed to develop a simple...

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El-Shafie OT, Al-Saffi N, Al-Sajwani A, et al. Adrenocorticotropic Hormone-Dependent Cushing's Syndrome: Use of an octreotide trial to distinguish between pituitary or ectopic sources. Sultan Qaboos Univ Med J. 2015;15(1):e120-3
El-Shafie, O. T., Al-Saffi, N., Al-Sajwani, A., & Woodhouse, N. (2015). Adrenocorticotropic Hormone-Dependent Cushing's Syndrome: Use of an octreotide trial to distinguish between pituitary or ectopic sources. Sultan Qaboos University medical journal, 15(1), e120-3.
El-Shafie, Omayma T, et al. "Adrenocorticotropic Hormone-Dependent Cushing's Syndrome: Use of an octreotide trial to distinguish between pituitary or ectopic sources." Sultan Qaboos University medical journal vol. 15,1 (2015): e120-3.
El-Shafie OT, Al-Saffi N, Al-Sajwani A, Woodhouse N. Adrenocorticotropic Hormone-Dependent Cushing's Syndrome: Use of an octreotide trial to distinguish between pituitary or ectopic sources. Sultan Qaboos Univ Med J. 2015 Feb;15(1):e120-3. Epub 2015 Jan 21. PMID: 25685371; PMCID: PMC4318592.
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Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus.

Obesity research & clinical practice

Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H.
PMID: 24351449
Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002.

Under restraint stress conditions, prepro-thyrotropin releasing hormone (TRH) 178-199 suppresses adrenocorticotropic hormone (ACTH) secretion from the rat pituitary, which indicates that prepro-TRH 178-199 is a candidate endogenous corticotropin releasing inhibitory factor (CRIF). Restraint stress also activates the release of...

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Fukagawa T, Fukagawa K, Gotoh K, et al. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007;1(2):I-IIdoi: 10.1016/j.orcp.2007.01.002.
Fukagawa, T., Fukagawa, K., Gotoh, K., Noguchi, H., & Yoshimatsu, H. (2007). Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obesity research & clinical practice, 1(2), I-II. https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa, Tomiyo, et al. "Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus." Obesity research & clinical practice vol. 1,2 (2007): I-II. doi: https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002. PMID: 24351449.
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CD4 count as a predictor of adrenocortical insufficiency in persons with human immunodeficiency virus infection: How useful?.

Indian journal of endocrinology and metabolism

Odeniyi IA, Fasanmade OA, Ajala MO, Ohwovoriole AE.
PMID: 24381877
Indian J Endocrinol Metab. 2013 Nov;17(6):1012-7. doi: 10.4103/2230-8210.122615.

OBJECTIVE: To determine the usefulness of CD4 count in predicting adrenocortical insufficiency (AI) in persons with HIV infection.DESIGN: Experimental study involving people with HIV infection and healthy people.PARTICIPANTS: The participants were recruited from the Lagos University Teaching Hospital. Forty-three...

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Odeniyi IA, Fasanmade OA, Ajala MO, et al. CD4 count as a predictor of adrenocortical insufficiency in persons with human immunodeficiency virus infection: How useful?. Indian J Endocrinol Metab. 2013;17(6):1012-7doi: 10.4103/2230-8210.122615.
Odeniyi, I. A., Fasanmade, O. A., Ajala, M. O., & Ohwovoriole, A. E. (2013). CD4 count as a predictor of adrenocortical insufficiency in persons with human immunodeficiency virus infection: How useful?. Indian journal of endocrinology and metabolism, 17(6), 1012-7. https://doi.org/10.4103/2230-8210.122615
Odeniyi, Ifedayo A, et al. "CD4 count as a predictor of adrenocortical insufficiency in persons with human immunodeficiency virus infection: How useful?." Indian journal of endocrinology and metabolism vol. 17,6 (2013): 1012-7. doi: https://doi.org/10.4103/2230-8210.122615
Odeniyi IA, Fasanmade OA, Ajala MO, Ohwovoriole AE. CD4 count as a predictor of adrenocortical insufficiency in persons with human immunodeficiency virus infection: How useful?. Indian J Endocrinol Metab. 2013 Nov;17(6):1012-7. doi: 10.4103/2230-8210.122615. PMID: 24381877; PMCID: PMC3872678.
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Case report: a rare cause of metabolic alkalosis.

NDT plus

Titan SM, Gebara OC, Callas SH, Hoff AO, Hoff PM, Galvão PC.
PMID: 25984146
NDT Plus. 2011 Jun;4(3):164-6. doi: 10.1093/ndtplus/sfr036. Epub 2011 Mar 29.

A case of a 66-year-old white man with recent onset of oedema, hypertension, metabolic alkalosis and profound hypokalaemia is described. The initial laboratorial workup showed that urinary chloride concentration and potassium excretion were increased, suggesting a state of hyperaldosteronism....

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Titan SM, Gebara OC, Callas SH, et al. Case report: a rare cause of metabolic alkalosis. NDT Plus. 2011;4(3):164-6doi: 10.1093/ndtplus/sfr036.
Titan, S. M., Gebara, O. C., Callas, S. H., Hoff, A. O., Hoff, P. M., & Galvão, P. C. (2011). Case report: a rare cause of metabolic alkalosis. NDT plus, 4(3), 164-6. https://doi.org/10.1093/ndtplus/sfr036
Titan, Silvia M, et al. "Case report: a rare cause of metabolic alkalosis." NDT plus vol. 4,3 (2011): 164-6. doi: https://doi.org/10.1093/ndtplus/sfr036
Titan SM, Gebara OC, Callas SH, Hoff AO, Hoff PM, Galvão PC. Case report: a rare cause of metabolic alkalosis. NDT Plus. 2011 Jun;4(3):164-6. doi: 10.1093/ndtplus/sfr036. Epub 2011 Mar 29. PMID: 25984146; PMCID: PMC4421598.
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Nonfunctioning giant pituitary adenomas: Invasiveness and recurrence.

Surgical neurology international

Landeiro JA, Fonseca EO, Monnerat AL, Taboada GF, Cabral GA, Antunes F.
PMID: 26674325
Surg Neurol Int. 2015 Nov 26;6:179. doi: 10.4103/2152-7806.170536. eCollection 2015.

BACKGROUND: We report our surgical series of 35 patients with giant nonfunctioning pituitary adenomas (GNFPA). We analyzed the rule of Ki-67 antigen expression in predicting recurrence.METHODS: Thirty-five patients were operated between 2000 and 2010. Suprassellar extension of the tumors...

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Landeiro JA, Fonseca EO, Monnerat AL, et al. Nonfunctioning giant pituitary adenomas: Invasiveness and recurrence. Surg Neurol Int. 2015;6:179doi: 10.4103/2152-7806.170536.
Landeiro, J. A., Fonseca, E. O., Monnerat, A. L., Taboada, G. F., Cabral, G. A., & Antunes, F. (2015). Nonfunctioning giant pituitary adenomas: Invasiveness and recurrence. Surgical neurology international, 6179. https://doi.org/10.4103/2152-7806.170536
Landeiro, José Alberto, et al. "Nonfunctioning giant pituitary adenomas: Invasiveness and recurrence." Surgical neurology international vol. 6 (2015): 179. doi: https://doi.org/10.4103/2152-7806.170536
Landeiro JA, Fonseca EO, Monnerat AL, Taboada GF, Cabral GA, Antunes F. Nonfunctioning giant pituitary adenomas: Invasiveness and recurrence. Surg Neurol Int. 2015 Nov 26;6:179. doi: 10.4103/2152-7806.170536. eCollection 2015. PMID: 26674325; PMCID: PMC4665135.
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Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders.

Acta neuropsychiatrica

Juruena MF, Cleare AJ, Bauer ME, Pariante CM.
PMID: 26983771
Acta Neuropsychiatr. 2003 Dec;15(6):354-67. doi: 10.1046/j.1601-5215.2003.00051.x.

Changes in the hypothalamic-pituitary-adrenocortical (HPA) system are characteristic of depression, and in the majority of these patients these result in HPA axis hyperactivity. This is further supported by the reduced sensitivity to the inhibitory effects of the glucocorticoid, dexamethasone...

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Juruena MF, Cleare AJ, Bauer ME, et al. Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta Neuropsychiatr. 2003;15(6):354-67doi: 10.1046/j.1601-5215.2003.00051.x.
Juruena, M. F., Cleare, A. J., Bauer, M. E., & Pariante, C. M. (2003). Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta neuropsychiatrica, 15(6), 354-67. https://doi.org/10.1046/j.1601-5215.2003.00051.x
Juruena, Mario F, et al. "Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders." Acta neuropsychiatrica vol. 15,6 (2003): 354-67. doi: https://doi.org/10.1046/j.1601-5215.2003.00051.x
Juruena MF, Cleare AJ, Bauer ME, Pariante CM. Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta Neuropsychiatr. 2003 Dec;15(6):354-67. doi: 10.1046/j.1601-5215.2003.00051.x. PMID: 26983771.
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Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals.

Journal of neurogastroenterology and motility

Yamasaki T, Tomita T, Takimoto M, Kondo T, Tozawa K, Ohda Y, Oshima T, Fukui H, Watari J, Miwa H.
PMID: 28992676
J Neurogastroenterol Motil. 2017 Oct 30;23(4):526-532. doi: 10.5056/jnm17067.

BACKGROUND/AIMS: When a person is experiencing stress, corticotropin-releasing hormone (CRH) can modulate gut physiologies, such as visceral sensation or gastrointestinal motility, and its intravenous administration mimics stress-induced physiological changes. However, the influence of CRH on the esophagus is yet...

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Yamasaki T, Tomita T, Takimoto M, et al. Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. J Neurogastroenterol Motil. 2017;23(4):526-532doi: 10.5056/jnm17067.
Yamasaki, T., Tomita, T., Takimoto, M., Kondo, T., Tozawa, K., Ohda, Y., Oshima, T., Fukui, H., Watari, J., & Miwa, H. (2017). Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. Journal of neurogastroenterology and motility, 23(4), 526-532. https://doi.org/10.5056/jnm17067
Yamasaki, Takahisa, et al. "Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals." Journal of neurogastroenterology and motility vol. 23,4 (2017): 526-532. doi: https://doi.org/10.5056/jnm17067
Yamasaki T, Tomita T, Takimoto M, Kondo T, Tozawa K, Ohda Y, Oshima T, Fukui H, Watari J, Miwa H. Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. J Neurogastroenterol Motil. 2017 Oct 30;23(4):526-532. doi: 10.5056/jnm17067. PMID: 28992676; PMCID: PMC5628984.
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The impact of butylphthalide on the hypothalamus-pituitary-adrenal axis of patients suffering from cerebral infarction in the basal ganglia.

Electronic physician

Niu H, Zhang Z, Wang H, Wang H, Zhang J, Li C, Zhao L.
PMID: 26955446
Electron Physician. 2016 Jan 15;8(1):1759-63. doi: 10.19082/1759. eCollection 2016 Jan.

BACKGROUND: Butylphthalide sodium chloride injection for patients with acute cerebral infarction has a certain effect. Although there are several proposed mechanisms of drug action, no related research on improving the inflammatory cytokines that regulate the body's immune system through...

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Niu H, Zhang Z, Wang H, et al. The impact of butylphthalide on the hypothalamus-pituitary-adrenal axis of patients suffering from cerebral infarction in the basal ganglia. Electron Physician. 2016;8(1):1759-63doi: 10.19082/1759.
Niu, H., Zhang, Z., Wang, H., Wang, H., Zhang, J., Li, C., & Zhao, L. (2016). The impact of butylphthalide on the hypothalamus-pituitary-adrenal axis of patients suffering from cerebral infarction in the basal ganglia. Electronic physician, 8(1), 1759-63. https://doi.org/10.19082/1759
Niu, Huiyan, et al. "The impact of butylphthalide on the hypothalamus-pituitary-adrenal axis of patients suffering from cerebral infarction in the basal ganglia." Electronic physician vol. 8,1 (2016): 1759-63. doi: https://doi.org/10.19082/1759
Niu H, Zhang Z, Wang H, Wang H, Zhang J, Li C, Zhao L. The impact of butylphthalide on the hypothalamus-pituitary-adrenal axis of patients suffering from cerebral infarction in the basal ganglia. Electron Physician. 2016 Jan 15;8(1):1759-63. doi: 10.19082/1759. eCollection 2016 Jan. PMID: 26955446; PMCID: PMC4768925.
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Metachronous Bilateral Testicular Leydig-Like Tumors Leading to the Diagnosis of Congenital Adrenal Hyperplasia (Adrenogenital Syndrome).

Case reports in pathology

Vukina J, Chism DD, Sharpless JL, Raynor MC, Milowsky MI, Funkhouser WK.
PMID: 26351608
Case Rep Pathol. 2015;2015:459318. doi: 10.1155/2015/459318. Epub 2015 Aug 16.

A 33-year-old male with a history of left testis Leydig cell tumor (LCT), 3-month status after left radical orchiectomy, presented with a rapidly enlarging (0.6 cm to 3.7 cm) right testicular mass. He underwent a right radical orchiectomy, sections...

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Vukina J, Chism DD, Sharpless JL, et al. Metachronous Bilateral Testicular Leydig-Like Tumors Leading to the Diagnosis of Congenital Adrenal Hyperplasia (Adrenogenital Syndrome). Case Rep Pathol. 2015;2015:459318doi: 10.1155/2015/459318.
Vukina, J., Chism, D. D., Sharpless, J. L., Raynor, M. C., Milowsky, M. I., & Funkhouser, W. K. (2015). Metachronous Bilateral Testicular Leydig-Like Tumors Leading to the Diagnosis of Congenital Adrenal Hyperplasia (Adrenogenital Syndrome). Case reports in pathology, 2015459318. https://doi.org/10.1155/2015/459318
Vukina, Josip, et al. "Metachronous Bilateral Testicular Leydig-Like Tumors Leading to the Diagnosis of Congenital Adrenal Hyperplasia (Adrenogenital Syndrome)." Case reports in pathology vol. 2015 (2015): 459318. doi: https://doi.org/10.1155/2015/459318
Vukina J, Chism DD, Sharpless JL, Raynor MC, Milowsky MI, Funkhouser WK. Metachronous Bilateral Testicular Leydig-Like Tumors Leading to the Diagnosis of Congenital Adrenal Hyperplasia (Adrenogenital Syndrome). Case Rep Pathol. 2015;2015:459318. doi: 10.1155/2015/459318. Epub 2015 Aug 16. PMID: 26351608; PMCID: PMC4553183.
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Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case.

Obstetric medicine

Trinh A, Chan I, Alexiadis M, Pell M, Kumar B, Fuller PJ.
PMID: 27512490
Obstet Med. 2016 Mar;9(1):43-5. doi: 10.1177/1753495X15618542. Epub 2015 Dec 09.

BACKGROUND: Cushing's syndrome is rare during pregnancy and more commonly due to adrenal pathology, in contrast to the non-pregnant population. Increased levels of cortisol-binding globulin and placental production of corticotropin-releasing hormone and adrenocorticotropic hormone complicate the diagnostic strategies usually...

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Trinh A, Chan I, Alexiadis M, et al. Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstet Med. 2015;9(1):43-5doi: 10.1177/1753495X15618542.
Trinh, A., Chan, I., Alexiadis, M., Pell, M., Kumar, B., & Fuller, P. J. (2016). Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstetric medicine, 9(1), 43-5. https://doi.org/10.1177/1753495X15618542
Trinh, Anne, et al. "Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case." Obstetric medicine vol. 9,1 (2016): 43-5. doi: https://doi.org/10.1177/1753495X15618542
Trinh A, Chan I, Alexiadis M, Pell M, Kumar B, Fuller PJ. Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstet Med. 2016 Mar;9(1):43-5. doi: 10.1177/1753495X15618542. Epub 2015 Dec 09. PMID: 27512490; PMCID: PMC4950436.
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Effect of steroid replacement on thyroid function and thyroid autoimmunity in Addison's disease with primary hypothyroidism.

Indian journal of endocrinology and metabolism

Sahoo JP, Selviambigapathy J, Kamalanathan S, Nagarajan K, Vivekanandan M.
PMID: 27042409
Indian J Endocrinol Metab. 2016 Mar-Apr;20(2):162-6. doi: 10.4103/2230-8210.176356.

BACKGROUND: Steroid replacement without thyroxine supplementation normalizes thyroid function test (TFT) in some but not all Addison's disease patients with primary hypothyroidism. Both autoimmune and nonautoimmune mechanisms contribute to this improvement in TFT. However, the documentation of the change...

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Sahoo JP, Selviambigapathy J, Kamalanathan S, et al. Effect of steroid replacement on thyroid function and thyroid autoimmunity in Addison's disease with primary hypothyroidism. Indian J Endocrinol Metab. 2016;20(2):162-6doi: 10.4103/2230-8210.176356.
Sahoo, J. P., Selviambigapathy, J., Kamalanathan, S., Nagarajan, K., & Vivekanandan, M. (2016). Effect of steroid replacement on thyroid function and thyroid autoimmunity in Addison's disease with primary hypothyroidism. Indian journal of endocrinology and metabolism, 20(2), 162-6. https://doi.org/10.4103/2230-8210.176356
Sahoo, Jaya Prakash, et al. "Effect of steroid replacement on thyroid function and thyroid autoimmunity in Addison's disease with primary hypothyroidism." Indian journal of endocrinology and metabolism vol. 20,2 (2016): 162-6. doi: https://doi.org/10.4103/2230-8210.176356
Sahoo JP, Selviambigapathy J, Kamalanathan S, Nagarajan K, Vivekanandan M. Effect of steroid replacement on thyroid function and thyroid autoimmunity in Addison's disease with primary hypothyroidism. Indian J Endocrinol Metab. 2016 Mar-Apr;20(2):162-6. doi: 10.4103/2230-8210.176356. PMID: 27042409; PMCID: PMC4792014.
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Showing 13 to 24 of 1041 entries