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The stripe rust fungal effector PEC6 suppresses pattern-triggered immunity in a host species-independent manner and interacts with adenosine kinases.

The New phytologist

Liu C, Pedersen C, Schultz-Larsen T, Aguilar GB, Madriz-Ordeñana K, Hovmøller MS, Thordal-Christensen H.
PMID: 27252028
New Phytol. 2016 Jun 02; doi: 10.1111/nph.14034. Epub 2016 Jun 02.

We identified a wheat stripe rust (Puccinia striiformis) effector candidate (PEC6) with pattern-triggered immunity (PTI) suppression function and its corresponding host target. PEC6 compromised PTI host species-independently. In Nicotiana benthamiana, it hampers reactive oxygen species (ROS) accumulation and callose...

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Liu C, Pedersen C, Schultz-Larsen T, et al. The stripe rust fungal effector PEC6 suppresses pattern-triggered immunity in a host species-independent manner and interacts with adenosine kinases. New Phytol. 2016;doi: 10.1111/nph.14034.
Liu, C., Pedersen, C., Schultz-Larsen, T., Aguilar, G. B., Madriz-Ordeñana, K., Hovmøller, M. S., & Thordal-Christensen, H. (2016). The stripe rust fungal effector PEC6 suppresses pattern-triggered immunity in a host species-independent manner and interacts with adenosine kinases. The New phytologist, . https://doi.org/10.1111/nph.14034
Liu, Changhai, et al. "The stripe rust fungal effector PEC6 suppresses pattern-triggered immunity in a host species-independent manner and interacts with adenosine kinases." The New phytologist vol. (2016). doi: https://doi.org/10.1111/nph.14034
Liu C, Pedersen C, Schultz-Larsen T, Aguilar GB, Madriz-Ordeñana K, Hovmøller MS, Thordal-Christensen H. The stripe rust fungal effector PEC6 suppresses pattern-triggered immunity in a host species-independent manner and interacts with adenosine kinases. New Phytol. 2016 Jun 02; doi: 10.1111/nph.14034. Epub 2016 Jun 02. PMID: 27252028.
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The effects of Aminophylline on clinical recovery and bispectral index in patients anesthetized with total intravenous anaesthesia.

Pakistan journal of medical sciences

Ghaffaripour S, Khosravi MB, Rahimi A, Sahmedini MA, Chohedri A, Mahmoudi H, Kazemi MR.
PMID: 25674137
Pak J Med Sci. 2014 Nov-Dec;30(6):1351-5. doi: 10.12669/pjms.306.5853.

OBJECTIVE: Aminophylline, which is clinically used as a bronchodilator, antagonizes the action of adenosine, so it can be used to shorten the recovery time after general anesthesia. Therefore, we wanted to test the hypothesis that the administration of aminophylline...

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Ghaffaripour S, Khosravi MB, Rahimi A, et al. The effects of Aminophylline on clinical recovery and bispectral index in patients anesthetized with total intravenous anaesthesia. Pak J Med Sci. 2014;30(6):1351-5doi: 10.12669/pjms.306.5853.
Ghaffaripour, S., Khosravi, M. B., Rahimi, A., Sahmedini, M. A., Chohedri, A., Mahmoudi, H., & Kazemi, M. R. (2014). The effects of Aminophylline on clinical recovery and bispectral index in patients anesthetized with total intravenous anaesthesia. Pakistan journal of medical sciences, 30(6), 1351-5. https://doi.org/10.12669/pjms.306.5853
Ghaffaripour, Sina, et al. "The effects of Aminophylline on clinical recovery and bispectral index in patients anesthetized with total intravenous anaesthesia." Pakistan journal of medical sciences vol. 30,6 (2014): 1351-5. doi: https://doi.org/10.12669/pjms.306.5853
Ghaffaripour S, Khosravi MB, Rahimi A, Sahmedini MA, Chohedri A, Mahmoudi H, Kazemi MR. The effects of Aminophylline on clinical recovery and bispectral index in patients anesthetized with total intravenous anaesthesia. Pak J Med Sci. 2014 Nov-Dec;30(6):1351-5. doi: 10.12669/pjms.306.5853. PMID: 25674137; PMCID: PMC4320729.
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Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory.

Molecular psychiatry

Li P, Rial D, Canas PM, Yoo JH, Li W, Zhou X, Wang Y, van Westen GJ, Payen MP, Augusto E, Gonçalves N, Tomé AR, Li Z, Wu Z, Hou X, Zhou Y, PIJzerman A, Boyden ES, Cunha RA, Qu J, Chen JF.
PMID: 25802981
Mol Psychiatry. 2015 Nov;20(11):1481. doi: 10.1038/mp.2015.43. Epub 2015 Mar 24.

No abstract available.

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Li P, Rial D, Canas PM, et al. Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory. Mol Psychiatry. 2015;20(11):1481doi: 10.1038/mp.2015.43.
Li, P., Rial, D., Canas, P. M., Yoo, J. H., Li, W., Zhou, X., Wang, Y., van Westen, G. J., Payen, M. P., Augusto, E., Gonçalves, N., Tomé, A. R., Li, Z., Wu, Z., Hou, X., Zhou, Y., PIJzerman, A., Boyden, E. S., Cunha, R. A., Qu, J., & Chen, J. F. (2015). Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory. Molecular psychiatry, 20(11), 1481. https://doi.org/10.1038/mp.2015.43
Li, P, et al. "Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory." Molecular psychiatry vol. 20,11 (2015): 1481. doi: https://doi.org/10.1038/mp.2015.43
Li P, Rial D, Canas PM, Yoo JH, Li W, Zhou X, Wang Y, van Westen GJ, Payen MP, Augusto E, Gonçalves N, Tomé AR, Li Z, Wu Z, Hou X, Zhou Y, PIJzerman A, Boyden ES, Cunha RA, Qu J, Chen JF. Optogenetic activation of intracellular adenosine A2A receptor signaling in the hippocampus is sufficient to trigger CREB phosphorylation and impair memory. Mol Psychiatry. 2015 Nov;20(11):1481. doi: 10.1038/mp.2015.43. Epub 2015 Mar 24. PMID: 25802981.
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Induction of apoptosis by FFJ-5, a novel naphthoquinone compound, occurs via downregulation of PKM2 in A549 and HepG2 cells.

Oncology letters

Wei X, Li M, Ma M, Jia H, Zhang Y, Kang W, Wang T, Shi X.
PMID: 28356960
Oncol Lett. 2017 Feb;13(2):791-799. doi: 10.3892/ol.2016.5522. Epub 2016 Dec 20.

Pyruvate kinase isoenzyme M2 (PKM2) has previously been identified as a tumor biomarker and as a potential target for cancer therapy. In this study, F§FJ-5, a characterized naphthoquinone modifier of mollugin, was synthesized in order to investigate its anticancer...

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Wei X, Li M, Ma M, et al. Induction of apoptosis by FFJ-5, a novel naphthoquinone compound, occurs via downregulation of PKM2 in A549 and HepG2 cells. Oncol Lett. 2016;13(2):791-799doi: 10.3892/ol.2016.5522.
Wei, X., Li, M., Ma, M., Jia, H., Zhang, Y., Kang, W., Wang, T., & Shi, X. (2017). Induction of apoptosis by FFJ-5, a novel naphthoquinone compound, occurs via downregulation of PKM2 in A549 and HepG2 cells. Oncology letters, 13(2), 791-799. https://doi.org/10.3892/ol.2016.5522
Wei, Xiaoli, et al. "Induction of apoptosis by FFJ-5, a novel naphthoquinone compound, occurs via downregulation of PKM2 in A549 and HepG2 cells." Oncology letters vol. 13,2 (2017): 791-799. doi: https://doi.org/10.3892/ol.2016.5522
Wei X, Li M, Ma M, Jia H, Zhang Y, Kang W, Wang T, Shi X. Induction of apoptosis by FFJ-5, a novel naphthoquinone compound, occurs via downregulation of PKM2 in A549 and HepG2 cells. Oncol Lett. 2017 Feb;13(2):791-799. doi: 10.3892/ol.2016.5522. Epub 2016 Dec 20. PMID: 28356960; PMCID: PMC5351257.
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AFP-producing acinar cell carcinoma treated by pancreaticoduodenectomy in a patient with a previous radical subtotal gastrectomy by gastric cancer.

Korean journal of hepato-biliary-pancreatic surgery

Kim CY, Lee SH, Jeon HM, Kim HK, Kang CM, Lee WJ.
PMID: 26155245
Korean J Hepatobiliary Pancreat Surg. 2014 Feb;18(1):33-7. doi: 10.14701/kjhbps.2014.18.1.33. Epub 2014 Feb 24.

We report a case of alpha-fetoprotein (AFP)-producing acinar cell carcinoma (ACC) of the pancreas. The tumor was diagnosed in a 72 yearold female after radical subtotal gastrectomy (Billroth I) due to early gastric cancer six months before. The initial...

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Kim CY, Lee SH, Jeon HM, et al. AFP-producing acinar cell carcinoma treated by pancreaticoduodenectomy in a patient with a previous radical subtotal gastrectomy by gastric cancer. Korean J Hepatobiliary Pancreat Surg. 2014;18(1):33-7doi: 10.14701/kjhbps.2014.18.1.33.
Kim, C. Y., Lee, S. H., Jeon, H. M., Kim, H. K., Kang, C. M., & Lee, W. J. (2014). AFP-producing acinar cell carcinoma treated by pancreaticoduodenectomy in a patient with a previous radical subtotal gastrectomy by gastric cancer. Korean journal of hepato-biliary-pancreatic surgery, 18(1), 33-7. https://doi.org/10.14701/kjhbps.2014.18.1.33
Kim, Chang Young, et al. "AFP-producing acinar cell carcinoma treated by pancreaticoduodenectomy in a patient with a previous radical subtotal gastrectomy by gastric cancer." Korean journal of hepato-biliary-pancreatic surgery vol. 18,1 (2014): 33-7. doi: https://doi.org/10.14701/kjhbps.2014.18.1.33
Kim CY, Lee SH, Jeon HM, Kim HK, Kang CM, Lee WJ. AFP-producing acinar cell carcinoma treated by pancreaticoduodenectomy in a patient with a previous radical subtotal gastrectomy by gastric cancer. Korean J Hepatobiliary Pancreat Surg. 2014 Feb;18(1):33-7. doi: 10.14701/kjhbps.2014.18.1.33. Epub 2014 Feb 24. PMID: 26155245; PMCID: PMC4492330.
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Acupuncture for visceral pain: neural substrates and potential mechanisms.

Evidence-based complementary and alternative medicine : eCAM

Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J.
PMID: 25614752
Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29.

Visceral pain is the most common form of pain caused by varied diseases and a major reason for patients to seek medical consultation. Despite much advances, the pathophysiological mechanism is still poorly understood comparing with its somatic counterpart and,...

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Chen S, Wang S, Rong P, et al. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594doi: 10.1155/2014/609594.
Chen, S., Wang, S., Rong, P., Wang, J., Qiao, L., Feng, X., Liu, J., & Zhang, J. (2014). Acupuncture for visceral pain: neural substrates and potential mechanisms. Evidence-based complementary and alternative medicine : eCAM, 2014609594. https://doi.org/10.1155/2014/609594
Chen, Shuping, et al. "Acupuncture for visceral pain: neural substrates and potential mechanisms." Evidence-based complementary and alternative medicine : eCAM vol. 2014 (2014): 609594. doi: https://doi.org/10.1155/2014/609594
Chen S, Wang S, Rong P, Wang J, Qiao L, Feng X, Liu J, Zhang J. Acupuncture for visceral pain: neural substrates and potential mechanisms. Evid Based Complement Alternat Med. 2014;2014:609594. doi: 10.1155/2014/609594. Epub 2014 Dec 29. PMID: 25614752; PMCID: PMC4295157.
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Novel Aptasensor Platform Based on Ratiometric Surface-Enhanced Raman Spectroscopy.

Analytical chemistry

Wu Y, Xiao F, Wu Z, Yu R.
PMID: 28225598
Anal Chem. 2017 Mar 07;89(5):2852-2858. doi: 10.1021/acs.analchem.6b04010. Epub 2017 Feb 22.

A novel aptasensor platform has been developed for quantitative detection of adenosine triphosphate (ATP) based on a ratiometric surface-enhanced Raman scattering (SERS) strategy. The thiolated 3'-Rox-labeled complementary DNA (cDNA) is first immobilized on the gold nanoparticle (AuNP) surface and...

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Wu Y, Xiao F, Wu Z, et al. Novel Aptasensor Platform Based on Ratiometric Surface-Enhanced Raman Spectroscopy. Anal Chem. 2017;89(5):2852-2858doi: 10.1021/acs.analchem.6b04010.
Wu, Y., Xiao, F., Wu, Z., & Yu, R. (2017). Novel Aptasensor Platform Based on Ratiometric Surface-Enhanced Raman Spectroscopy. Analytical chemistry, 89(5), 2852-2858. https://doi.org/10.1021/acs.analchem.6b04010
Wu, Yan, et al. "Novel Aptasensor Platform Based on Ratiometric Surface-Enhanced Raman Spectroscopy." Analytical chemistry vol. 89,5 (2017): 2852-2858. doi: https://doi.org/10.1021/acs.analchem.6b04010
Wu Y, Xiao F, Wu Z, Yu R. Novel Aptasensor Platform Based on Ratiometric Surface-Enhanced Raman Spectroscopy. Anal Chem. 2017 Mar 07;89(5):2852-2858. doi: 10.1021/acs.analchem.6b04010. Epub 2017 Feb 22. PMID: 28225598.
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Cachexia: a problem of energetic inefficiency.

Journal of cachexia, sarcopenia and muscle

Argilés JM, Fontes-Oliveira CC, Toledo M, López-Soriano FJ, Busquets S.
PMID: 25118829
J Cachexia Sarcopenia Muscle. 2014 Dec;5(4):279-86. doi: 10.1007/s13539-014-0154-x. Epub 2014 Aug 15.

An alteration of energy balance is the immediate cause of the so-called cachexia. Although alterations of energy intake are often associated with cachexia, it has lately became clear that an increased energy expenditure is the main cause of wasting...

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Argilés JM, Fontes-Oliveira CC, Toledo M, et al. Cachexia: a problem of energetic inefficiency. J Cachexia Sarcopenia Muscle. 2014;5(4):279-86doi: 10.1007/s13539-014-0154-x.
Argilés, J. M., Fontes-Oliveira, C. C., Toledo, M., López-Soriano, F. J., & Busquets, S. (2014). Cachexia: a problem of energetic inefficiency. Journal of cachexia, sarcopenia and muscle, 5(4), 279-86. https://doi.org/10.1007/s13539-014-0154-x
Argilés, Josep M, et al. "Cachexia: a problem of energetic inefficiency." Journal of cachexia, sarcopenia and muscle vol. 5,4 (2014): 279-86. doi: https://doi.org/10.1007/s13539-014-0154-x
Argilés JM, Fontes-Oliveira CC, Toledo M, López-Soriano FJ, Busquets S. Cachexia: a problem of energetic inefficiency. J Cachexia Sarcopenia Muscle. 2014 Dec;5(4):279-86. doi: 10.1007/s13539-014-0154-x. Epub 2014 Aug 15. PMID: 25118829; PMCID: PMC4248416.
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Increased adenosine levels contribute to ischemic kidney fibrosis in the unilateral ureteral obstruction model.

Experimental and therapeutic medicine

Tang J, Jiang X, Zhou Y, Xia B, Dai Y.
PMID: 25667621
Exp Ther Med. 2015 Mar;9(3):737-743. doi: 10.3892/etm.2015.2177. Epub 2015 Jan 13.

Renal interstitial fibrosis (RIF) occurs as a result of chronic kidney disease (CKD) and is a common pathway leading to end-stage renal failure. Renal tissue hypoxia and ischemia are present during CKD. Adenosine (ADO) is an important signaling molecule...

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Tang J, Jiang X, Zhou Y, et al. Increased adenosine levels contribute to ischemic kidney fibrosis in the unilateral ureteral obstruction model. Exp Ther Med. 2015;9(3):737-743doi: 10.3892/etm.2015.2177.
Tang, J., Jiang, X., Zhou, Y., Xia, B., & Dai, Y. (2015). Increased adenosine levels contribute to ischemic kidney fibrosis in the unilateral ureteral obstruction model. Experimental and therapeutic medicine, 9(3), 737-743. https://doi.org/10.3892/etm.2015.2177
Tang, Jin, et al. "Increased adenosine levels contribute to ischemic kidney fibrosis in the unilateral ureteral obstruction model." Experimental and therapeutic medicine vol. 9,3 (2015): 737-743. doi: https://doi.org/10.3892/etm.2015.2177
Tang J, Jiang X, Zhou Y, Xia B, Dai Y. Increased adenosine levels contribute to ischemic kidney fibrosis in the unilateral ureteral obstruction model. Exp Ther Med. 2015 Mar;9(3):737-743. doi: 10.3892/etm.2015.2177. Epub 2015 Jan 13. PMID: 25667621; PMCID: PMC4316984.
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Tuberculous pleural effusions: advances and controversies.

Journal of thoracic disease

Vorster MJ, Allwood BW, Diacon AH, Koegelenberg CF.
PMID: 26150911
J Thorac Dis. 2015 Jun;7(6):981-91. doi: 10.3978/j.issn.2072-1439.2015.02.18.

On a global scale, tuberculosis (TB) remains one of the most frequent causes of pleural effusions. Our understanding of the pathogenesis of the disease has evolved and what was once thought to be an effusion as a result of...

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Vorster MJ, Allwood BW, Diacon AH, et al. Tuberculous pleural effusions: advances and controversies. J Thorac Dis. 2015;7(6):981-91doi: 10.3978/j.issn.2072-1439.2015.02.18.
Vorster, M. J., Allwood, B. W., Diacon, A. H., & Koegelenberg, C. F. (2015). Tuberculous pleural effusions: advances and controversies. Journal of thoracic disease, 7(6), 981-91. https://doi.org/10.3978/j.issn.2072-1439.2015.02.18
Vorster, Morné J, et al. "Tuberculous pleural effusions: advances and controversies." Journal of thoracic disease vol. 7,6 (2015): 981-91. doi: https://doi.org/10.3978/j.issn.2072-1439.2015.02.18
Vorster MJ, Allwood BW, Diacon AH, Koegelenberg CF. Tuberculous pleural effusions: advances and controversies. J Thorac Dis. 2015 Jun;7(6):981-91. doi: 10.3978/j.issn.2072-1439.2015.02.18. PMID: 26150911; PMCID: PMC4466424.
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Caloric restriction protects against electrical kindling of the amygdala by inhibiting the mTOR signaling pathway.

Frontiers in cellular neuroscience

Phillips-Farfán BV, Rubio Osornio Mdel C, Custodio Ramírez V, Paz Tres C, Carvajal Aguilera KG.
PMID: 25814935
Front Cell Neurosci. 2015 Mar 11;9:90. doi: 10.3389/fncel.2015.00090. eCollection 2015.

Caloric restriction (CR) has been shown to possess antiepileptic properties; however its mechanism of action is poorly understood. CR might inhibit the activity of the mammalian or mechanistic target of rapamycin (mTOR) signaling cascade, which seems to participate crucially...

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Phillips-Farfán BV, Rubio Osornio Mdel C, Custodio Ramírez V, et al. Caloric restriction protects against electrical kindling of the amygdala by inhibiting the mTOR signaling pathway. Front Cell Neurosci. 2015;9:90doi: 10.3389/fncel.2015.00090.
Phillips-Farfán, B. V., Rubio Osornio, M. d. e. l. . C., Custodio Ramírez, V., Paz Tres, C., & Carvajal Aguilera, K. G. (2015). Caloric restriction protects against electrical kindling of the amygdala by inhibiting the mTOR signaling pathway. Frontiers in cellular neuroscience, 990. https://doi.org/10.3389/fncel.2015.00090
Phillips-Farfán, Bryan V, et al. "Caloric restriction protects against electrical kindling of the amygdala by inhibiting the mTOR signaling pathway." Frontiers in cellular neuroscience vol. 9 (2015): 90. doi: https://doi.org/10.3389/fncel.2015.00090
Phillips-Farfán BV, Rubio Osornio Mdel C, Custodio Ramírez V, Paz Tres C, Carvajal Aguilera KG. Caloric restriction protects against electrical kindling of the amygdala by inhibiting the mTOR signaling pathway. Front Cell Neurosci. 2015 Mar 11;9:90. doi: 10.3389/fncel.2015.00090. eCollection 2015. PMID: 25814935; PMCID: PMC4356078.
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Total saponin from Korean Red Ginseng inhibits binding of adhesive proteins to glycoprotein IIb/IIIa via phosphorylation of VASP (Ser(157)) and dephosphorylation of PI3K and Akt.

Journal of ginseng research

Kwon HW, Shin JH, Cho HJ, Rhee MH, Park HJ.
PMID: 26843825
J Ginseng Res. 2016 Jan;40(1):76-85. doi: 10.1016/j.jgr.2015.05.004. Epub 2015 May 21.

BACKGROUND: Binding of adhesive proteins (i.e., fibrinogen, fibronectin, vitronectin) to platelet integrin glycoprotein IIb/IIIa (αIIb/β3) by various agonists (thrombin, collagen, adenosine diphosphate) involve in strength of thrombus. This study was carried out to evaluate the antiplatelet effect of total...

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Kwon HW, Shin JH, Cho HJ, et al. Total saponin from Korean Red Ginseng inhibits binding of adhesive proteins to glycoprotein IIb/IIIa via phosphorylation of VASP (Ser(157)) and dephosphorylation of PI3K and Akt. J Ginseng Res. 2015;40(1):76-85doi: 10.1016/j.jgr.2015.05.004.
Kwon, H. W., Shin, J. H., Cho, H. J., Rhee, M. H., & Park, H. J. (2016). Total saponin from Korean Red Ginseng inhibits binding of adhesive proteins to glycoprotein IIb/IIIa via phosphorylation of VASP (Ser(157)) and dephosphorylation of PI3K and Akt. Journal of ginseng research, 40(1), 76-85. https://doi.org/10.1016/j.jgr.2015.05.004
Kwon, Hyuk-Woo, et al. "Total saponin from Korean Red Ginseng inhibits binding of adhesive proteins to glycoprotein IIb/IIIa via phosphorylation of VASP (Ser(157)) and dephosphorylation of PI3K and Akt." Journal of ginseng research vol. 40,1 (2016): 76-85. doi: https://doi.org/10.1016/j.jgr.2015.05.004
Kwon HW, Shin JH, Cho HJ, Rhee MH, Park HJ. Total saponin from Korean Red Ginseng inhibits binding of adhesive proteins to glycoprotein IIb/IIIa via phosphorylation of VASP (Ser(157)) and dephosphorylation of PI3K and Akt. J Ginseng Res. 2016 Jan;40(1):76-85. doi: 10.1016/j.jgr.2015.05.004. Epub 2015 May 21. PMID: 26843825; PMCID: PMC4703804.
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