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Harbin TS, Pollack IP. Glaucoma in episcleritis. Arch Ophthalmol. 1975;93(10):948-50doi: 10.1001/archopht.1975.01010020746002.
Harbin, T. S., & Pollack, I. P. (1975). Glaucoma in episcleritis. Archives of ophthalmology (Chicago, Ill. : 1960), 93(10), 948-50. https://doi.org/10.1001/archopht.1975.01010020746002
Harbin, T S, and Pollack, I P. "Glaucoma in episcleritis." Archives of ophthalmology (Chicago, Ill. : 1960) vol. 93,10 (1975): 948-50. doi: https://doi.org/10.1001/archopht.1975.01010020746002
Harbin TS, Pollack IP. Glaucoma in episcleritis. Arch Ophthalmol. 1975 Oct;93(10):948-50. doi: 10.1001/archopht.1975.01010020746002. PMID: 1180753.
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Arch Ophthalmol. 1975 Oct;93(10):948-50. doi: 10.1001/archopht.1975.01010020746002.

Glaucoma in episcleritis.

Archives of ophthalmology (Chicago, Ill. : 1960)

T S Harbin, I P Pollack

PMID: 1180753 DOI: 10.1001/archopht.1975.01010020746002

Abstract

Two patients had episcleritis and a secondary open angle glaucoma. Anterior uveitis was absent in three of the four eyes. The low facility of outflow suggests that increased episcleral venous pressure was not the mechanism of the glaucoma. The response of the intraocular pressure and, in one case, the outflow facility to steroid therapy suggests that inflammation of angle structures was the pathogenesis of the raised intraocular pressure.

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