Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Journal
Age
Showing 1 to 12 of 10129 entries
Sorted by: Best Match Show Resources per page
Effects of heart failure on baroreflex control of sympathetic neural activity.

The American journal of cardiology

Ferguson DW, Berg WJ, Roach PJ, Oren RM, Mark AL.
PMID: 1736618
Am J Cardiol. 1992 Feb 15;69(5):523-31. doi: 10.1016/0002-9149(92)90998-e.

Baroreflex control of heart rate, vascular resistance and norepinephrine is impaired in patients with heart failure, but recent animal studies demonstrate preserved baroreflex control of sympathetic nerve activity in this disorder. Studies were therefore performed to compare baroreflex control...

Cite
Ferguson DW, Berg WJ, Roach PJ, et al. Effects of heart failure on baroreflex control of sympathetic neural activity. Am J Cardiol. 1992;69(5):523-31doi: 10.1016/0002-9149(92)90998-e.
Ferguson, D. W., Berg, W. J., Roach, P. J., Oren, R. M., & Mark, A. L. (1992). Effects of heart failure on baroreflex control of sympathetic neural activity. The American journal of cardiology, 69(5), 523-31. https://doi.org/10.1016/0002-9149(92)90998-e
Ferguson, D W, et al. "Effects of heart failure on baroreflex control of sympathetic neural activity." The American journal of cardiology vol. 69,5 (1992): 523-31. doi: https://doi.org/10.1016/0002-9149(92)90998-e
Ferguson DW, Berg WJ, Roach PJ, Oren RM, Mark AL. Effects of heart failure on baroreflex control of sympathetic neural activity. Am J Cardiol. 1992 Feb 15;69(5):523-31. doi: 10.1016/0002-9149(92)90998-e. PMID: 1736618.
Copy Download .nbib Format: NLM
The effects of adrenalectomy, corticotropin releasing factor and vasopressin on the sympathetic firing rate of nerves to interscapular brown adipose tissue in the Zucker rat.

Physiology & behavior

Holt SJ, York DA.
PMID: 2554347
Physiol Behav. 1989 Jun;45(6):1123-9. doi: 10.1016/0031-9384(89)90098-x.

The firing rate of the sympathetic efferent nerves to interscapular brown adipose tissue (IBAT) is lower in the obese rat compared with the lean rat. The present experiments show that adrenalectomy has no effect on nerve firing rate in...

Cite
Holt SJ, York DA. The effects of adrenalectomy, corticotropin releasing factor and vasopressin on the sympathetic firing rate of nerves to interscapular brown adipose tissue in the Zucker rat. Physiol Behav. 1989;45(6):1123-9doi: 10.1016/0031-9384(89)90098-x.
Holt, S. J., & York, D. A. (1989). The effects of adrenalectomy, corticotropin releasing factor and vasopressin on the sympathetic firing rate of nerves to interscapular brown adipose tissue in the Zucker rat. Physiology & behavior, 45(6), 1123-9. https://doi.org/10.1016/0031-9384(89)90098-x
Holt, S J, and York, D A. "The effects of adrenalectomy, corticotropin releasing factor and vasopressin on the sympathetic firing rate of nerves to interscapular brown adipose tissue in the Zucker rat." Physiology & behavior vol. 45,6 (1989): 1123-9. doi: https://doi.org/10.1016/0031-9384(89)90098-x
Holt SJ, York DA. The effects of adrenalectomy, corticotropin releasing factor and vasopressin on the sympathetic firing rate of nerves to interscapular brown adipose tissue in the Zucker rat. Physiol Behav. 1989 Jun;45(6):1123-9. doi: 10.1016/0031-9384(89)90098-x. PMID: 2554347.
Copy Download .nbib Format: NLM
[Systolic time intervals in mitral valve prolapse syndrome].

Journal of cardiology. Supplement

Yazaki N, Niwayama H, Onishi M, Sunami Y, Nishimoto Y, Masuda Y, Inagaki Y.
PMID: 2778645
J Cardiol Suppl. 1989;21:75-80, discussion 81-3.

Systolic time intervals (STI) were measured in 135 patients with mitral valve prolapse syndrome (MVP). These patients were categorized as Group II (no or trivial mitral regurgitation) and Group III (moderate or severe regurgitation). The controls consisted of 120...

Cite
Yazaki N, Niwayama H, Onishi M, et al. [Systolic time intervals in mitral valve prolapse syndrome]. J Cardiol Suppl. 1989;21:75-80, discussion 81-3
Yazaki, N., Niwayama, H., Onishi, M., Sunami, Y., Nishimoto, Y., Masuda, Y., & Inagaki, Y. (1989). [Systolic time intervals in mitral valve prolapse syndrome]. Journal of cardiology. Supplement, 2175-80, discussion 81-3.
Yazaki, N, et al. "[Systolic time intervals in mitral valve prolapse syndrome]." Journal of cardiology. Supplement vol. 21 (1989): 75-80, discussion 81-3.
Yazaki N, Niwayama H, Onishi M, Sunami Y, Nishimoto Y, Masuda Y, Inagaki Y. [Systolic time intervals in mitral valve prolapse syndrome]. J Cardiol Suppl. 1989;21:75-80, discussion 81-3. Japanese. PMID: 2778645.
Copy Download .nbib Format: NLM
Neural influences and sudden cardiac death.

Advances in cardiology

Verrier RL, Lown B.
PMID: 30259
Adv Cardiol. 1978;25:155-68. doi: 10.1159/000402015.

No abstract available.

Cite
Verrier RL, Lown B. Neural influences and sudden cardiac death. Adv Cardiol. 1978;25:155-68doi: 10.1159/000402015.
Verrier, R. L., & Lown, B. (1978). Neural influences and sudden cardiac death. Advances in cardiology, 25155-68. https://doi.org/10.1159/000402015
Verrier, R L, and Lown, B. "Neural influences and sudden cardiac death." Advances in cardiology vol. 25 (1978): 155-68. doi: https://doi.org/10.1159/000402015
Verrier RL, Lown B. Neural influences and sudden cardiac death. Adv Cardiol. 1978;25:155-68. doi: 10.1159/000402015. PMID: 30259.
Copy Download .nbib Format: NLM
Oculosympathetic paralysis (Horner's syndrome) in the dog.

Veterinary medicine, small animal clinician : VM, SAC

Wowk BJ, Olson GA.
PMID: 256375
Vet Med Small Anim Clin. 1979 Apr;74(4):521-7.

No abstract available.

Cite
Wowk BJ, Olson GA. Oculosympathetic paralysis (Horner's syndrome) in the dog. Vet Med Small Anim Clin. 1979;74(4):521-7
Wowk, B. J., & Olson, G. A. (1979). Oculosympathetic paralysis (Horner's syndrome) in the dog. Veterinary medicine, small animal clinician : VM, SAC, 74(4), 521-7.
Wowk, B J, and Olson, G A. "Oculosympathetic paralysis (Horner's syndrome) in the dog." Veterinary medicine, small animal clinician : VM, SAC vol. 74,4 (1979): 521-7.
Wowk BJ, Olson GA. Oculosympathetic paralysis (Horner's syndrome) in the dog. Vet Med Small Anim Clin. 1979 Apr;74(4):521-7. PMID: 256375.
Copy Download .nbib Format: NLM
Development of individual differences of cardiovascular regulation in the Bailey recombinant inbred mice.

Behavior genetics

Harshfield GA, Simmel EC.
PMID: 262626
Behav Genet. 1979 Nov;9(6):495-504. doi: 10.1007/BF01067346.

Previous research has demonstrated that the cardiovascular system (CVS) is not completely developed in the rat until 20 days of age. Prior to this age the sympathetic nervous system (SNS) is dominant during resting conditions and the stress response...

Cite
Harshfield GA, Simmel EC. Development of individual differences of cardiovascular regulation in the Bailey recombinant inbred mice. Behav Genet. 1979;9(6):495-504doi: 10.1007/BF01067346.
Harshfield, G. A., & Simmel, E. C. (1979). Development of individual differences of cardiovascular regulation in the Bailey recombinant inbred mice. Behavior genetics, 9(6), 495-504. https://doi.org/10.1007/BF01067346
Harshfield, G A, and Simmel, E C. "Development of individual differences of cardiovascular regulation in the Bailey recombinant inbred mice." Behavior genetics vol. 9,6 (1979): 495-504. doi: https://doi.org/10.1007/BF01067346
Harshfield GA, Simmel EC. Development of individual differences of cardiovascular regulation in the Bailey recombinant inbred mice. Behav Genet. 1979 Nov;9(6):495-504. doi: 10.1007/BF01067346. PMID: 262626.
Copy Download .nbib Format: NLM
Hypothesis: the nervous system may contribute to the pathophysiology of rheumatoid arthritis.

The Journal of rheumatology

Levine JD, Collier DH, Basbaum AI, Moskowitz MA, Helms CA.
PMID: 2413204
J Rheumatol. 1985 Jun;12(3):406-11.

No current theory of the mechanisms involved in the pathophysiology of rheumatoid arthritis (RA) explains its important clinical features. We hypothesize that neural mechanisms are involved in this pathophysiology and they explain at least 3 clinical features: specific high...

Cite
Levine JD, Collier DH, Basbaum AI, et al. Hypothesis: the nervous system may contribute to the pathophysiology of rheumatoid arthritis. J Rheumatol. 1985;12(3):406-11
Levine, J. D., Collier, D. H., Basbaum, A. I., Moskowitz, M. A., & Helms, C. A. (1985). Hypothesis: the nervous system may contribute to the pathophysiology of rheumatoid arthritis. The Journal of rheumatology, 12(3), 406-11.
Levine, J D, et al. "Hypothesis: the nervous system may contribute to the pathophysiology of rheumatoid arthritis." The Journal of rheumatology vol. 12,3 (1985): 406-11.
Levine JD, Collier DH, Basbaum AI, Moskowitz MA, Helms CA. Hypothesis: the nervous system may contribute to the pathophysiology of rheumatoid arthritis. J Rheumatol. 1985 Jun;12(3):406-11. PMID: 2413204.
Copy Download .nbib Format: NLM
Sympathetic skin response in diabetic neuropathy.

Electromyography and clinical neurophysiology

Watahiki Y, Baba M, Matsunaga M, Takebe K, Onuma T.
PMID: 2721428
Electromyogr Clin Neurophysiol. 1989 Apr;29(3):155-9.

Sympathetic function test was undertaken using the sympathetic skin response (SSR), a technique for assessment of sudomotor activity, in 67 diabetics, and the results were compared with those from 45 age-matched normal subjects. The SSR was readily elicitable in...

Cite
Watahiki Y, Baba M, Matsunaga M, et al. Sympathetic skin response in diabetic neuropathy. Electromyogr Clin Neurophysiol. 1989;29(3):155-9
Watahiki, Y., Baba, M., Matsunaga, M., Takebe, K., & Onuma, T. (1989). Sympathetic skin response in diabetic neuropathy. Electromyography and clinical neurophysiology, 29(3), 155-9.
Watahiki, Y, et al. "Sympathetic skin response in diabetic neuropathy." Electromyography and clinical neurophysiology vol. 29,3 (1989): 155-9.
Watahiki Y, Baba M, Matsunaga M, Takebe K, Onuma T. Sympathetic skin response in diabetic neuropathy. Electromyogr Clin Neurophysiol. 1989 Apr;29(3):155-9. PMID: 2721428.
Copy Download .nbib Format: NLM
Neuroendocrine activation in congestive heart failure.

The American journal of cardiology

Forfar JC.
PMID: 2021117
Am J Cardiol. 1991 May 06;67(12):3C-5C. doi: 10.1016/0002-9149(91)90065-s.

Several changes in neuroendocrine activity follow failure of cardiac function to satisfy peripheral requirements and contribute to the clinical syndromes of heart failure. Afferent pathways are poorly understood and triggers are both central and peripheral, involving attenuation of atrial...

Cite
Forfar JC. Neuroendocrine activation in congestive heart failure. Am J Cardiol. 1991;67(12):3C-5Cdoi: 10.1016/0002-9149(91)90065-s.
Forfar, J. C. (1991). Neuroendocrine activation in congestive heart failure. The American journal of cardiology, 67(12), 3C-5C. https://doi.org/10.1016/0002-9149(91)90065-s
Forfar, J C. "Neuroendocrine activation in congestive heart failure." The American journal of cardiology vol. 67,12 (1991): 3C-5C. doi: https://doi.org/10.1016/0002-9149(91)90065-s
Forfar JC. Neuroendocrine activation in congestive heart failure. Am J Cardiol. 1991 May 06;67(12):3C-5C. doi: 10.1016/0002-9149(91)90065-s. PMID: 2021117.
Copy Download .nbib Format: NLM
No impairment of sympathetic neurotransmission in stunned myocardium.

Basic research in cardiology

Schulz R, Frehen D, Heusch G.
PMID: 1965400
Basic Res Cardiol. 1990;85:267-80. doi: 10.1007/978-3-662-11038-6_22.

Reversibly injured myocardium after short periods of ischemia is characterized by a prolonged depression of contractile function which can, however, be enhanced by inotropic interventions. Thus, a lack of inotropic stimulation due to ischemic damage of cardiac sympathetic nerves...

Cite
Schulz R, Frehen D, Heusch G. No impairment of sympathetic neurotransmission in stunned myocardium. Basic Res Cardiol. 1990;85:267-80doi: 10.1007/978-3-662-11038-6_22.
Schulz, R., Frehen, D., & Heusch, G. (1990). No impairment of sympathetic neurotransmission in stunned myocardium. Basic research in cardiology, 85267-80. https://doi.org/10.1007/978-3-662-11038-6_22
Schulz, R, et al. "No impairment of sympathetic neurotransmission in stunned myocardium." Basic research in cardiology vol. 85 (1990): 267-80. doi: https://doi.org/10.1007/978-3-662-11038-6_22
Schulz R, Frehen D, Heusch G. No impairment of sympathetic neurotransmission in stunned myocardium. Basic Res Cardiol. 1990;85:267-80. doi: 10.1007/978-3-662-11038-6_22. PMID: 1965400.
Copy Download .nbib Format: NLM
Skeletal muscle metaboreceptor exercise responses are attenuated in heart failure.

Circulation

Sterns DA, Ettinger SM, Gray KS, Whisler SK, Mosher TJ, Smith MB, Sinoway LI.
PMID: 1934378
Circulation. 1991 Nov;84(5):2034-9. doi: 10.1161/01.cir.84.5.2034.

BACKGROUND: Resting sympathetic nervous system activity is increased in heart failure. Whether sympathetic nervous system responses during exercise are increased is controversial. Furthermore, the role of muscle metaboreceptors and central command in regulating sympathetic outflow has been largely unexplored.METHODS...

Cite
Sterns DA, Ettinger SM, Gray KS, et al. Skeletal muscle metaboreceptor exercise responses are attenuated in heart failure. Circulation. 1991;84(5):2034-9doi: 10.1161/01.cir.84.5.2034.
Sterns, D. A., Ettinger, S. M., Gray, K. S., Whisler, S. K., Mosher, T. J., Smith, M. B., & Sinoway, L. I. (1991). Skeletal muscle metaboreceptor exercise responses are attenuated in heart failure. Circulation, 84(5), 2034-9. https://doi.org/10.1161/01.cir.84.5.2034
Sterns, D A, et al. "Skeletal muscle metaboreceptor exercise responses are attenuated in heart failure." Circulation vol. 84,5 (1991): 2034-9. doi: https://doi.org/10.1161/01.cir.84.5.2034
Sterns DA, Ettinger SM, Gray KS, Whisler SK, Mosher TJ, Smith MB, Sinoway LI. Skeletal muscle metaboreceptor exercise responses are attenuated in heart failure. Circulation. 1991 Nov;84(5):2034-9. doi: 10.1161/01.cir.84.5.2034. PMID: 1934378.
Copy Download .nbib Format: NLM
Primary erythromelalgia: the role of skin sympathetic nerve activity.

Japanese journal of medicine

Sugiyama Y, Hakusui S, Takahashi A, Iwase S, Mano T.
PMID: 1798217
Jpn J Med. 1991 Nov-Dec;30(6):564-7. doi: 10.2169/internalmedicine1962.30.564.

A 54-year-old man complained of burning pain, warm skin and erythema in his extremities. A diagnosis of primary erythromelalgia was made. Microneurography was used to clarify the role of skin sympathetic nerve activity in the pathophysiology of primary erythromelalgia....

Cite
Sugiyama Y, Hakusui S, Takahashi A, et al. Primary erythromelalgia: the role of skin sympathetic nerve activity. Jpn J Med. 1991;30(6):564-7doi: 10.2169/internalmedicine1962.30.564.
Sugiyama, Y., Hakusui, S., Takahashi, A., Iwase, S., & Mano, T. (1991). Primary erythromelalgia: the role of skin sympathetic nerve activity. Japanese journal of medicine, 30(6), 564-7. https://doi.org/10.2169/internalmedicine1962.30.564
Sugiyama, Y, et al. "Primary erythromelalgia: the role of skin sympathetic nerve activity." Japanese journal of medicine vol. 30,6 (1991): 564-7. doi: https://doi.org/10.2169/internalmedicine1962.30.564
Sugiyama Y, Hakusui S, Takahashi A, Iwase S, Mano T. Primary erythromelalgia: the role of skin sympathetic nerve activity. Jpn J Med. 1991 Nov-Dec;30(6):564-7. doi: 10.2169/internalmedicine1962.30.564. PMID: 1798217.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 10129 entries