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Showing 1 to 12 of 16485 entries
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Growth regulation of human neuroblastoma.

Cancer treatment and research

el-Badry OM, Israel MA.
PMID: 1363353
Cancer Treat Res. 1992;63:105-28. doi: 10.1007/978-1-4615-3088-6_5.

No abstract available.

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el-Badry OM, Israel MA. Growth regulation of human neuroblastoma. Cancer Treat Res. 1992;63:105-28doi: 10.1007/978-1-4615-3088-6_5.
el-Badry, O. M., & Israel, M. A. (1992). Growth regulation of human neuroblastoma. Cancer treatment and research, 63105-28. https://doi.org/10.1007/978-1-4615-3088-6_5
el-Badry, O M, and Israel, M A. "Growth regulation of human neuroblastoma." Cancer treatment and research vol. 63 (1992): 105-28. doi: https://doi.org/10.1007/978-1-4615-3088-6_5
el-Badry OM, Israel MA. Growth regulation of human neuroblastoma. Cancer Treat Res. 1992;63:105-28. doi: 10.1007/978-1-4615-3088-6_5. PMID: 1363353.
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Functional antagonism between members of the myb family: B-myb inhibits v-myb-induced gene activation.

The EMBO journal

Foos G, Grimm S, Klempnauer KH.
PMID: 1425593
EMBO J. 1992 Dec;11(12):4619-29.
Free PMC Article

The oncogene v-myb and its cellular progenitor c-myb encode nuclear, DNA binding phosphoproteins that control the expression of certain target genes in immature hematopoietic cells. Here, we report the isolation of a myb-related chicken gene, chicken B-myb. We show...

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Foos G, Grimm S, Klempnauer KH. Functional antagonism between members of the myb family: B-myb inhibits v-myb-induced gene activation. EMBO J. 1992;11(12):4619-29
Foos, G., Grimm, S., & Klempnauer, K. H. (1992). Functional antagonism between members of the myb family: B-myb inhibits v-myb-induced gene activation. The EMBO journal, 11(12), 4619-29.
Foos, G, et al. "Functional antagonism between members of the myb family: B-myb inhibits v-myb-induced gene activation." The EMBO journal vol. 11,12 (1992): 4619-29.
Foos G, Grimm S, Klempnauer KH. Functional antagonism between members of the myb family: B-myb inhibits v-myb-induced gene activation. EMBO J. 1992 Dec;11(12):4619-29. PMID: 1425593; PMCID: PMC557038.
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Genes and signal transduction in tumor promotion: conclusions from studies with promoter resistant variants of JB-6 mouse epidermal cells.

Carcinogenesis; a comprehensive survey

Gindhart TD, Nakamura Y, Stevens LA, Hegameyer GA, West MW, Smith BM, Colburn NH.
PMID: 3157452
Carcinog Compr Surv. 1985;8:341-67.

No abstract available.

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Gindhart TD, Nakamura Y, Stevens LA, et al. Genes and signal transduction in tumor promotion: conclusions from studies with promoter resistant variants of JB-6 mouse epidermal cells. Carcinog Compr Surv. 1985;8:341-67
Gindhart, T. D., Nakamura, Y., Stevens, L. A., Hegameyer, G. A., West, M. W., Smith, B. M., & Colburn, N. H. (1985). Genes and signal transduction in tumor promotion: conclusions from studies with promoter resistant variants of JB-6 mouse epidermal cells. Carcinogenesis; a comprehensive survey, 8341-67.
Gindhart, T D, et al. "Genes and signal transduction in tumor promotion: conclusions from studies with promoter resistant variants of JB-6 mouse epidermal cells." Carcinogenesis; a comprehensive survey vol. 8 (1985): 341-67.
Gindhart TD, Nakamura Y, Stevens LA, Hegameyer GA, West MW, Smith BM, Colburn NH. Genes and signal transduction in tumor promotion: conclusions from studies with promoter resistant variants of JB-6 mouse epidermal cells. Carcinog Compr Surv. 1985;8:341-67. PMID: 3157452.
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[Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc].

Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie

Pompidou A, Corral M, Michel P, Kruh J, Curran T.
PMID: 3096504
C R Acad Sci III. 1986;303(11):445-7.

After stimulation of human lymphocytes by 12-0 tetradecanoyl phorbol acetate (TPA) and by the calcium ionophore A23187 an early transient expression of proto-oncogene fos followed by an expression of c-myc is observed, as has been described in rodent fibroblast...

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Pompidou A, Corral M, Michel P, et al. La stimulation de lymphocytes humains par l'acétate de phorbol et un ionophore calcique induit une expression transitoire précoce de l'oncogéne c-fos suivie de l'oncogéne c-myc. [Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc]. C R Acad Sci III. 1986;303(11):445-7
Pompidou, A., Corral, M., Michel, P., Kruh, J., & Curran, T. (1986). La stimulation de lymphocytes humains par l'acétate de phorbol et un ionophore calcique induit une expression transitoire précoce de l'oncogéne c-fos suivie de l'oncogéne c-myc. [Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc]. Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie, 303(11), 445-7.
Pompidou, A, et al. "La stimulation de lymphocytes humains par l'acétate de phorbol et un ionophore calcique induit une expression transitoire précoce de l'oncogéne c-fos suivie de l'oncogéne c-myc." [Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc]. Comptes rendus de l'Academie des sciences. Serie III, Sciences de la vie vol. 303,11 (1986): 445-7.
Pompidou A, Corral M, Michel P, Kruh J, Curran T. La stimulation de lymphocytes humains par l'acétate de phorbol et un ionophore calcique induit une expression transitoire précoce de l'oncogéne c-fos suivie de l'oncogéne c-myc. [Stimulation of human lymphocytes by phorbol acetate and a calcium ionophore induces an early transient expression of the c-fos oncogene followed by c-myc]. C R Acad Sci III. 1986;303(11):445-7. French. PMID: 3096504.
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Growth factor, viruses, and oncogenes in human lymphoid neoplasia.

Lymphokine research

Sharma S, Mehta SR, Ford RJ.
PMID: 3306177
Lymphokine Res. 1987;6(3):245-65.

No abstract available.

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Sharma S, Mehta SR, Ford RJ. Growth factor, viruses, and oncogenes in human lymphoid neoplasia. Lymphokine Res. 1987;6(3):245-65
Sharma, S., Mehta, S. R., & Ford, R. J. (1987). Growth factor, viruses, and oncogenes in human lymphoid neoplasia. Lymphokine research, 6(3), 245-65.
Sharma, S, et al. "Growth factor, viruses, and oncogenes in human lymphoid neoplasia." Lymphokine research vol. 6,3 (1987): 245-65.
Sharma S, Mehta SR, Ford RJ. Growth factor, viruses, and oncogenes in human lymphoid neoplasia. Lymphokine Res. 1987;6(3):245-65. PMID: 3306177.
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Selected concepts of cancer as a disease: from 1900 to oncogenes.

Oncology nursing forum

Gallucci BB.
PMID: 3898032
Oncol Nurs Forum. 1985 Sep-Oct;12(5):69-78.

No abstract available.

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Gallucci BB. Selected concepts of cancer as a disease: from 1900 to oncogenes. Oncol Nurs Forum. 1985;12(5):69-78
Gallucci, B. B. (1985). Selected concepts of cancer as a disease: from 1900 to oncogenes. Oncology nursing forum, 12(5), 69-78.
Gallucci, B B. "Selected concepts of cancer as a disease: from 1900 to oncogenes." Oncology nursing forum vol. 12,5 (1985): 69-78.
Gallucci BB. Selected concepts of cancer as a disease: from 1900 to oncogenes. Oncol Nurs Forum. 1985 Sep-Oct;12(5):69-78. PMID: 3898032.
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Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters.

Laboratory investigation; a journal of technical methods and pathology

Noguera R, Triche TJ, Navarro S, Tsokos M, Llombart-Bosch A.
PMID: 1310513
Lab Invest. 1992 Feb;66(2):143-51.

We report the establishment of a model of neural differentiation in four well-characterized Ewing's sarcoma cell lines. This process was induced by serum-depleted medium (1% fetal bovine serum) and agents such as dibutyryl cyclic AMP and retinoic acid. The...

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Noguera R, Triche TJ, Navarro S, et al. Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters. Lab Invest. 1992;66(2):143-51
Noguera, R., Triche, T. J., Navarro, S., Tsokos, M., & Llombart-Bosch, A. (1992). Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters. Laboratory investigation; a journal of technical methods and pathology, 66(2), 143-51.
Noguera, R, et al. "Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters." Laboratory investigation; a journal of technical methods and pathology vol. 66,2 (1992): 143-51.
Noguera R, Triche TJ, Navarro S, Tsokos M, Llombart-Bosch A. Dynamic model of differentiation in Ewing's sarcoma cells. Comparative analysis of morphologic, immunocytochemical, and oncogene expression parameters. Lab Invest. 1992 Feb;66(2):143-51. PMID: 1310513.
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Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction.

Blood

Biondi A, Rambaldi A, Pandolfi PP, Rossi V, Giudici G, Alcalay M, Lo Coco F, Diverio D, Pogliani EM, Lanzi EM.
PMID: 1320955
Blood. 1992 Jul 15;80(2):492-7.

The acute promyelocytic leukemia (APL) t(15;17) translocation generates a myl/retinoic acid receptor-alpha (RAR-alpha) chimeric gene that is transcribed as a fusion myl/RAR-alpha messenger RNA. Using primer sets derived from RAR-alpha and myl cDNAs, we were able to amplify the...

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Biondi A, Rambaldi A, Pandolfi PP, et al. Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction. Blood. 1992;80(2):492-7
Biondi, A., Rambaldi, A., Pandolfi, P. P., Rossi, V., Giudici, G., Alcalay, M., Lo Coco, F., Diverio, D., Pogliani, E. M., & Lanzi, E. M. (1992). Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction. Blood, 80(2), 492-7.
Biondi, A, et al. "Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction." Blood vol. 80,2 (1992): 492-7.
Biondi A, Rambaldi A, Pandolfi PP, Rossi V, Giudici G, Alcalay M, Lo Coco F, Diverio D, Pogliani EM, Lanzi EM. Molecular monitoring of the myl/retinoic acid receptor-alpha fusion gene in acute promyelocytic leukemia by polymerase chain reaction. Blood. 1992 Jul 15;80(2):492-7. PMID: 1320955.
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myb oncogene in human hematopoietic neoplasia with 6q- anomaly.

Cancer genetics and cytogenetics

Ohyashiki K, Ohyashiki JH, Kinniburgh AJ, Toyama K, Ito H, Minowada J, Sandberg AA.
PMID: 2838159
Cancer Genet Cytogenet. 1988 Jul 01;33(1):83-92. doi: 10.1016/0165-4608(88)90053-2.

Molecular and cytogenetic analyses were performed on human T-cell leukemia cell lines (PEER and MOLT-4) with the 6q- anomaly. The PEER cells contained an interstitial deletion of the long arm of chromosome 6, that is, del(6)(q13q21), as well as...

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Ohyashiki K, Ohyashiki JH, Kinniburgh AJ, et al. myb oncogene in human hematopoietic neoplasia with 6q- anomaly. Cancer Genet Cytogenet. 1988;33(1):83-92doi: 10.1016/0165-4608(88)90053-2.
Ohyashiki, K., Ohyashiki, J. H., Kinniburgh, A. J., Toyama, K., Ito, H., Minowada, J., & Sandberg, A. A. (1988). myb oncogene in human hematopoietic neoplasia with 6q- anomaly. Cancer genetics and cytogenetics, 33(1), 83-92. https://doi.org/10.1016/0165-4608(88)90053-2
Ohyashiki, K, et al. "myb oncogene in human hematopoietic neoplasia with 6q- anomaly." Cancer genetics and cytogenetics vol. 33,1 (1988): 83-92. doi: https://doi.org/10.1016/0165-4608(88)90053-2
Ohyashiki K, Ohyashiki JH, Kinniburgh AJ, Toyama K, Ito H, Minowada J, Sandberg AA. myb oncogene in human hematopoietic neoplasia with 6q- anomaly. Cancer Genet Cytogenet. 1988 Jul 01;33(1):83-92. doi: 10.1016/0165-4608(88)90053-2. PMID: 2838159.
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Expression of c-erbB-2 in in situ and in adjacent invasive ductal adenocarcinomas of the female breast.

Pathobiology : journal of immunopathology, molecular and cellular biology

Maguire HC, Hellman ME, Greene MI, Yeh I.
PMID: 1352687
Pathobiology. 1992;60(3):117-21. doi: 10.1159/000163709.

Several lines of evidence have demonstrated that expression of the c-erbB-2 gene product contributes to the malignant phenotype. We and others have determined that c-erbB-2 is substantially expressed in most ductal in situ carcinomas of the comedo type, but...

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Maguire HC, Hellman ME, Greene MI, et al. Expression of c-erbB-2 in in situ and in adjacent invasive ductal adenocarcinomas of the female breast. Pathobiology. 1992;60(3):117-21doi: 10.1159/000163709.
Maguire, H. C., Hellman, M. E., Greene, M. I., & Yeh, I. (1992). Expression of c-erbB-2 in in situ and in adjacent invasive ductal adenocarcinomas of the female breast. Pathobiology : journal of immunopathology, molecular and cellular biology, 60(3), 117-21. https://doi.org/10.1159/000163709
Maguire, H C, et al. "Expression of c-erbB-2 in in situ and in adjacent invasive ductal adenocarcinomas of the female breast." Pathobiology : journal of immunopathology, molecular and cellular biology vol. 60,3 (1992): 117-21. doi: https://doi.org/10.1159/000163709
Maguire HC, Hellman ME, Greene MI, Yeh I. Expression of c-erbB-2 in in situ and in adjacent invasive ductal adenocarcinomas of the female breast. Pathobiology. 1992;60(3):117-21. doi: 10.1159/000163709. PMID: 1352687.
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Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts.

Proceedings of the National Academy of Sciences of the United States of America

Dike LE, Farmer SR.
PMID: 3045824
Proc Natl Acad Sci U S A. 1988 Sep;85(18):6792-6. doi: 10.1073/pnas.85.18.6792.
Free PMC Article

A methylcellulose suspension system that prevents cell-surface contact with the substrate was used to study the role of cell adhesion in the regulation of proliferation. The nonadhesive conditions established by suspension culture cause BALB/c 3T3 (A31) cells to enter...

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Dike LE, Farmer SR. Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts. Proc Natl Acad Sci U S A. 1988;85(18):6792-6doi: 10.1073/pnas.85.18.6792.
Dike, L. E., & Farmer, S. R. (1988). Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts. Proceedings of the National Academy of Sciences of the United States of America, 85(18), 6792-6. https://doi.org/10.1073/pnas.85.18.6792
Dike, L E, and Farmer, S R. "Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts." Proceedings of the National Academy of Sciences of the United States of America vol. 85,18 (1988): 6792-6. doi: https://doi.org/10.1073/pnas.85.18.6792
Dike LE, Farmer SR. Cell adhesion induces expression of growth-associated genes in suspension-arrested fibroblasts. Proc Natl Acad Sci U S A. 1988 Sep;85(18):6792-6. doi: 10.1073/pnas.85.18.6792. PMID: 3045824; PMCID: PMC282064.
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Sequence rearrangements and genome instability. A possible step in carcinogenesis.

Journal of cancer research and clinical oncology

Chorazy M.
PMID: 2989295
J Cancer Res Clin Oncol. 1985;109(3):159-72. doi: 10.1007/BF00390351.

A substantial part of the mammalian genome is composed of sequences that do not contain structural genes. These sequences may constitute the major target for physical, chemical and biological DNA-damaging agents and can be involved in carcinogenesis. DNA-damaging agents...

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Chorazy M. Sequence rearrangements and genome instability. A possible step in carcinogenesis. J Cancer Res Clin Oncol. 1985;109(3):159-72doi: 10.1007/BF00390351.
Chorazy, M. (1985). Sequence rearrangements and genome instability. A possible step in carcinogenesis. Journal of cancer research and clinical oncology, 109(3), 159-72. https://doi.org/10.1007/BF00390351
Chorazy, M. "Sequence rearrangements and genome instability. A possible step in carcinogenesis." Journal of cancer research and clinical oncology vol. 109,3 (1985): 159-72. doi: https://doi.org/10.1007/BF00390351
Chorazy M. Sequence rearrangements and genome instability. A possible step in carcinogenesis. J Cancer Res Clin Oncol. 1985;109(3):159-72. doi: 10.1007/BF00390351. PMID: 2989295.
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Showing 1 to 12 of 16485 entries