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Enhancement by retinoic acid and dibutyryl cyclic adenosine 3':5'-monophosphate of the differentiation and gene expression of human neuroblastoma cells induced by interferon.

Cancer research

Higuchi T, Hannigan GE, Malkin D, Yeger H, Williams BR.
PMID: 1677311
Cancer Res. 1991 Aug 01;51(15):3958-64.
Free Article

Human neuroblastoma cell lines are induced to differentiate and display neuronal phenotypes when treated with interferon (IFN)-alpha 2, retinoic acid (RA), or dibutyryl cyclic AMP (dbcAMP). We investigated the effects of combinations of these agents in induction-differentiation in the...

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Higuchi T, Hannigan GE, Malkin D, et al. Enhancement by retinoic acid and dibutyryl cyclic adenosine 3':5'-monophosphate of the differentiation and gene expression of human neuroblastoma cells induced by interferon. Cancer Res. 1991;51(15):3958-64
Higuchi, T., Hannigan, G. E., Malkin, D., Yeger, H., & Williams, B. R. (1991). Enhancement by retinoic acid and dibutyryl cyclic adenosine 3':5'-monophosphate of the differentiation and gene expression of human neuroblastoma cells induced by interferon. Cancer research, 51(15), 3958-64.
Higuchi, T, et al. "Enhancement by retinoic acid and dibutyryl cyclic adenosine 3':5'-monophosphate of the differentiation and gene expression of human neuroblastoma cells induced by interferon." Cancer research vol. 51,15 (1991): 3958-64.
Higuchi T, Hannigan GE, Malkin D, Yeger H, Williams BR. Enhancement by retinoic acid and dibutyryl cyclic adenosine 3':5'-monophosphate of the differentiation and gene expression of human neuroblastoma cells induced by interferon. Cancer Res. 1991 Aug 01;51(15):3958-64. PMID: 1677311.
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Novel metaphase chromosome anomaly in cells with specialized products.

Clinical bulletin

[No authors listed]
PMID: 57837
Clin Bull. 1976;6(1):30-1.

No abstract available.

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Novel metaphase chromosome anomaly in cells with specialized products. Clin Bull. 1976;6(1):30-1
(1976). Novel metaphase chromosome anomaly in cells with specialized products. Clinical bulletin, 6(1), 30-1.
"Novel metaphase chromosome anomaly in cells with specialized products." Clinical bulletin vol. 6,1 (1976): 30-1.
Novel metaphase chromosome anomaly in cells with specialized products. Clin Bull. 1976;6(1):30-1. PMID: 57837.
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A translocation (1;10)(p32;q24) in a neuroblastoma cell line derived from marrow metastases.

Cancer genetics and cytogenetics

Skinnider L, Wang SY, Wang HC, Krespin HI.
PMID: 2302681
Cancer Genet Cytogenet. 1990 Mar;45(1):131-5. doi: 10.1016/0165-4608(90)90076-m.

Chromosomes of a cell line derived from neuroblastoma metastases were analysed. Ninety-nine percent of the cells had 46 chromosomes, and of 10 cells analysed by G-banding, all had a t(1;10)(p32;q24).

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Skinnider L, Wang SY, Wang HC, et al. A translocation (1;10)(p32;q24) in a neuroblastoma cell line derived from marrow metastases. Cancer Genet Cytogenet. 1990;45(1):131-5doi: 10.1016/0165-4608(90)90076-m.
Skinnider, L., Wang, S. Y., Wang, H. C., & Krespin, H. I. (1990). A translocation (1;10)(p32;q24) in a neuroblastoma cell line derived from marrow metastases. Cancer genetics and cytogenetics, 45(1), 131-5. https://doi.org/10.1016/0165-4608(90)90076-m
Skinnider, L, et al. "A translocation (1;10)(p32;q24) in a neuroblastoma cell line derived from marrow metastases." Cancer genetics and cytogenetics vol. 45,1 (1990): 131-5. doi: https://doi.org/10.1016/0165-4608(90)90076-m
Skinnider L, Wang SY, Wang HC, Krespin HI. A translocation (1;10)(p32;q24) in a neuroblastoma cell line derived from marrow metastases. Cancer Genet Cytogenet. 1990 Mar;45(1):131-5. doi: 10.1016/0165-4608(90)90076-m. PMID: 2302681.
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Effect of persistent measles virus infection on protein kinase C activity and c-fos protooncogene expression in neuroblastoma cells.

FEBS letters

Wolfson M, Katorza A, Segal S, Rager-Zisman B.
PMID: 2502430
FEBS Lett. 1989 Jul 03;250(2):367-70. doi: 10.1016/0014-5793(89)80757-4.
Free Article

The effect of persistent measles virus infection on c-fos protooncogene and protein kinase C (PKC-I) gene expression in a murine neuroblastoma cell line was studied. Overexpression of c-fos protooncogene by infected NS20Y/MS cells was detected when compared with uninfected...

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Wolfson M, Katorza A, Segal S, et al. Effect of persistent measles virus infection on protein kinase C activity and c-fos protooncogene expression in neuroblastoma cells. FEBS Lett. 1989;250(2):367-70doi: 10.1016/0014-5793(89)80757-4.
Wolfson, M., Katorza, A., Segal, S., & Rager-Zisman, B. (1989). Effect of persistent measles virus infection on protein kinase C activity and c-fos protooncogene expression in neuroblastoma cells. FEBS letters, 250(2), 367-70. https://doi.org/10.1016/0014-5793(89)80757-4
Wolfson, M, et al. "Effect of persistent measles virus infection on protein kinase C activity and c-fos protooncogene expression in neuroblastoma cells." FEBS letters vol. 250,2 (1989): 367-70. doi: https://doi.org/10.1016/0014-5793(89)80757-4
Wolfson M, Katorza A, Segal S, Rager-Zisman B. Effect of persistent measles virus infection on protein kinase C activity and c-fos protooncogene expression in neuroblastoma cells. FEBS Lett. 1989 Jul 03;250(2):367-70. doi: 10.1016/0014-5793(89)80757-4. PMID: 2502430.
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src expression in small-cell lung carcinoma and other neuroendocrine malignancies.

The American review of respiratory disease

Påhlman S, Hammerling U.
PMID: 2174663
Am Rev Respir Dis. 1990 Dec;142(6):S54-6. doi: 10.1164/ajrccm/142.6_Pt_2.S54.

The proto-oncogene c-src codes for two tyrosine kinases, pp60c-src and pp60c-srcN. The latter protein appears to be exclusively expressed in neurons and neuronally differentiated tumors. In cell lines derived from neuroblastoma and small-cell lung carcinoma, src expression correlates positively...

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Påhlman S, Hammerling U. src expression in small-cell lung carcinoma and other neuroendocrine malignancies. Am Rev Respir Dis. 1990;142(6):S54-6doi: 10.1164/ajrccm/142.6_Pt_2.S54.
Påhlman, S., & Hammerling, U. (1990). src expression in small-cell lung carcinoma and other neuroendocrine malignancies. The American review of respiratory disease, 142(6), S54-6. https://doi.org/10.1164/ajrccm/142.6_Pt_2.S54
Påhlman, S, and Hammerling, U. "src expression in small-cell lung carcinoma and other neuroendocrine malignancies." The American review of respiratory disease vol. 142,6 (1990): S54-6. doi: https://doi.org/10.1164/ajrccm/142.6_Pt_2.S54
Påhlman S, Hammerling U. src expression in small-cell lung carcinoma and other neuroendocrine malignancies. Am Rev Respir Dis. 1990 Dec;142(6):S54-6. doi: 10.1164/ajrccm/142.6_Pt_2.S54. PMID: 2174663.
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[The temperature dependence of protein kinase A and C activity in variant mouse neuroblastoma cells differing genetically in their heat resistance].

Tsitologiia

Konstantinova MF, Nekrasova TP, Nisman BKh.
PMID: 1841458
Tsitologiia. 1991;33(12):54-8.

The ability of the mouse neuroblastoma cell line NTR to proliferate at 40 degrees C correlates with the position of the temperature optimum of protein kinases A and C activities in the region of higher temperatures compared to those...

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Konstantinova MF, Nekrasova TP, Nisman BKh. Temperaturnaia zavisimost' aktivnosti proteinkinaz A i C u variantov kletok neĭroblastomy myshi, geneticheski razlichaiushchikhsia po teploustoĭchivosti. [The temperature dependence of protein kinase A and C activity in variant mouse neuroblastoma cells differing genetically in their heat resistance]. Tsitologiia. 1991;33(12):54-8
Konstantinova, M. F., Nekrasova, T. P., & Nisman, B. K. h. (1991). Temperaturnaia zavisimost' aktivnosti proteinkinaz A i C u variantov kletok neĭroblastomy myshi, geneticheski razlichaiushchikhsia po teploustoĭchivosti. [The temperature dependence of protein kinase A and C activity in variant mouse neuroblastoma cells differing genetically in their heat resistance]. Tsitologiia, 33(12), 54-8.
Konstantinova, M F, et al. "Temperaturnaia zavisimost' aktivnosti proteinkinaz A i C u variantov kletok neĭroblastomy myshi, geneticheski razlichaiushchikhsia po teploustoĭchivosti." [The temperature dependence of protein kinase A and C activity in variant mouse neuroblastoma cells differing genetically in their heat resistance]. Tsitologiia vol. 33,12 (1991): 54-8.
Konstantinova MF, Nekrasova TP, Nisman BKh. Temperaturnaia zavisimost' aktivnosti proteinkinaz A i C u variantov kletok neĭroblastomy myshi, geneticheski razlichaiushchikhsia po teploustoĭchivosti. [The temperature dependence of protein kinase A and C activity in variant mouse neuroblastoma cells differing genetically in their heat resistance]. Tsitologiia. 1991;33(12):54-8. Russian. PMID: 1841458.
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Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line.

DNA and cell biology

Bernards A, Haase VH, Murthy AE, Menon A, Hannigan GE, Gusella JF.
PMID: 1457041
DNA Cell Biol. 1992 Dec;11(10):727-34. doi: 10.1089/dna.1992.11.727.

Neurofibromatosis type 1 (NF1) is caused by mutations in a large gene on chromosome 17q11.2. Previously described partial cDNAs for this gene predicted a protein related to yeast IRA1/IRA2 and the mammalian RAS GTPase activator protein GAP. To initiate...

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Bernards A, Haase VH, Murthy AE, et al. Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line. DNA Cell Biol. 1992;11(10):727-34doi: 10.1089/dna.1992.11.727.
Bernards, A., Haase, V. H., Murthy, A. E., Menon, A., Hannigan, G. E., & Gusella, J. F. (1992). Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line. DNA and cell biology, 11(10), 727-34. https://doi.org/10.1089/dna.1992.11.727
Bernards, A, et al. "Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line." DNA and cell biology vol. 11,10 (1992): 727-34. doi: https://doi.org/10.1089/dna.1992.11.727
Bernards A, Haase VH, Murthy AE, Menon A, Hannigan GE, Gusella JF. Complete human NF1 cDNA sequence: two alternatively spliced mRNAs and absence of expression in a neuroblastoma line. DNA Cell Biol. 1992 Dec;11(10):727-34. doi: 10.1089/dna.1992.11.727. PMID: 1457041.
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Chromosome studies of solid tumours.

Journal of clinical pathology

Bown NP.
PMID: 1517454
J Clin Pathol. 1992 Jul;45(7):556-60. doi: 10.1136/jcp.45.7.556.
Free PMC Article

No abstract available.

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Bown NP. Chromosome studies of solid tumours. J Clin Pathol. 1992;45(7):556-60doi: 10.1136/jcp.45.7.556.
Bown, N. P. (1992). Chromosome studies of solid tumours. Journal of clinical pathology, 45(7), 556-60. https://doi.org/10.1136/jcp.45.7.556
Bown, N P. "Chromosome studies of solid tumours." Journal of clinical pathology vol. 45,7 (1992): 556-60. doi: https://doi.org/10.1136/jcp.45.7.556
Bown NP. Chromosome studies of solid tumours. J Clin Pathol. 1992 Jul;45(7):556-60. doi: 10.1136/jcp.45.7.556. PMID: 1517454; PMCID: PMC495176.
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Neuroblastoma and ganglioneuroma in a child with multiple neurofibromatosis. Implications for the mutational origin of neuroblastoma.

Cancer

Knudson AG, Amromin GD.
PMID: 4957245
Cancer. 1966 Jul;19(7):1032-7. doi: 10.1002/1097-0142(196607)19:7<1032::aid-cncr2820190719>3.0.co;2-7.

No abstract available.

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Knudson AG, Amromin GD. Neuroblastoma and ganglioneuroma in a child with multiple neurofibromatosis. Implications for the mutational origin of neuroblastoma. Cancer. 1966;19(7):1032-7doi: 10.1002/1097-0142(196607)19:7<1032::aid-cncr2820190719>3.0.co;2-7.
Knudson, A. G., & Amromin, G. D. (1966). Neuroblastoma and ganglioneuroma in a child with multiple neurofibromatosis. Implications for the mutational origin of neuroblastoma. Cancer, 19(7), 1032-7. https://doi.org/10.1002/1097-0142(196607)19:7<1032::aid-cncr2820190719>3.0.co;2-7
Knudson, A G, and Amromin, G D. "Neuroblastoma and ganglioneuroma in a child with multiple neurofibromatosis. Implications for the mutational origin of neuroblastoma." Cancer vol. 19,7 (1966): 1032-7. doi: https://doi.org/10.1002/1097-0142(196607)19:7<1032::aid-cncr2820190719>3.0.co;2-7
Knudson AG, Amromin GD. Neuroblastoma and ganglioneuroma in a child with multiple neurofibromatosis. Implications for the mutational origin of neuroblastoma. Cancer. 1966 Jul;19(7):1032-7. doi: 10.1002/1097-0142(196607)19:7<1032::aid-cncr2820190719>3.0.co;2-7. PMID: 4957245.
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Dup 3(q) syndrome and neuroblastoma.

European journal of pediatrics

Maier B, Beck JD.
PMID: 1396939
Eur J Pediatr. 1992 Sep;151(9):715-6. doi: 10.1007/BF01957583.

No abstract available.

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Maier B, Beck JD. Dup 3(q) syndrome and neuroblastoma. Eur J Pediatr. 1992;151(9):715-6doi: 10.1007/BF01957583.
Maier, B., & Beck, J. D. (1992). Dup 3(q) syndrome and neuroblastoma. European journal of pediatrics, 151(9), 715-6. https://doi.org/10.1007/BF01957583
Maier, B, and Beck, J D. "Dup 3(q) syndrome and neuroblastoma." European journal of pediatrics vol. 151,9 (1992): 715-6. doi: https://doi.org/10.1007/BF01957583
Maier B, Beck JD. Dup 3(q) syndrome and neuroblastoma. Eur J Pediatr. 1992 Sep;151(9):715-6. doi: 10.1007/BF01957583. PMID: 1396939.
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[Gene expression of NGFR, EGFR, CGA, NPY in 4 neuroblastoma cell lines].

Zhonghua bing li xue za zhi = Chinese journal of pathology

Chen J, Liu TH.
PMID: 1323422
Zhonghua Bing Li Xue Za Zhi. 1992 Apr;21(2):69-72.

Gene expression of nerve growth factor receptor (NGFR), epidermal growth factor receptor(EGFR), chromogranin A (CGA) and neuropeptide Y (NPY) in 4 neuroblastoma cell Lines without N-myc amplification was studied by using Northern blot technique. N type cells expressed more...

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Chen J, Liu TH. [Gene expression of NGFR, EGFR, CGA, NPY in 4 neuroblastoma cell lines]. Zhonghua Bing Li Xue Za Zhi. 1992;21(2):69-72
Chen, J., & Liu, T. H. (1992). [Gene expression of NGFR, EGFR, CGA, NPY in 4 neuroblastoma cell lines]. Zhonghua bing li xue za zhi = Chinese journal of pathology, 21(2), 69-72.
Chen, J, and Liu, T H. "[Gene expression of NGFR, EGFR, CGA, NPY in 4 neuroblastoma cell lines]." Zhonghua bing li xue za zhi = Chinese journal of pathology vol. 21,2 (1992): 69-72.
Chen J, Liu TH. [Gene expression of NGFR, EGFR, CGA, NPY in 4 neuroblastoma cell lines]. Zhonghua Bing Li Xue Za Zhi. 1992 Apr;21(2):69-72. Chinese. PMID: 1323422.
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Growth factors and oncogenes in human solid tumors: clinical aspects.

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Bonneterre J, Peyrat JP, Demaille A.
PMID: 1369689
Biomed Pharmacother. 1990;44(1):25-34. doi: 10.1016/0753-3322(90)90066-i.

Growth factors, growth factor receptors and oncogenes have been extensively studied in human tumors for some years. The purpose of this paper is to review the clinical results obtained in human cancers and their predisposing conditions or high risk...

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Bonneterre J, Peyrat JP, Demaille A. Growth factors and oncogenes in human solid tumors: clinical aspects. Biomed Pharmacother. 1990;44(1):25-34doi: 10.1016/0753-3322(90)90066-i.
Bonneterre, J., Peyrat, J. P., & Demaille, A. (1990). Growth factors and oncogenes in human solid tumors: clinical aspects. Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 44(1), 25-34. https://doi.org/10.1016/0753-3322(90)90066-i
Bonneterre, J, et al. "Growth factors and oncogenes in human solid tumors: clinical aspects." Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie vol. 44,1 (1990): 25-34. doi: https://doi.org/10.1016/0753-3322(90)90066-i
Bonneterre J, Peyrat JP, Demaille A. Growth factors and oncogenes in human solid tumors: clinical aspects. Biomed Pharmacother. 1990;44(1):25-34. doi: 10.1016/0753-3322(90)90066-i. PMID: 1369689.
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Showing 1 to 12 of 4780 entries