Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 5 of 5 entries
Sorted by: Best Match Show Resources per page
A Net Mold-based Method of Scaffold-free Three-Dimensional Cardiac Tissue Creation.

Journal of visualized experiments : JoVE

Bai Y, Yeung E, Lui C, Ong CS, Pitaktong I, Huang C, Inoue T, Matsushita H, Ma C, Hibino N.
PMID: 30124650
J Vis Exp. 2018 Aug 05;(138). doi: 10.3791/58252.

This protocol describes a novel and easy net mold-based method to create three-dimensional (3-D) cardiac tissues without additional scaffold material. Human-induced pluripotent stem-cell-derived cardiomyocytes (iPSC-CMs), human cardiac fibroblasts (HCFs), and human umbilical vein endothelial cells (HUVECs) are isolated and...

Cite
Bai Y, Yeung E, Lui C, et al. A Net Mold-based Method of Scaffold-free Three-Dimensional Cardiac Tissue Creation. J Vis Exp. 2018;(138)doi: 10.3791/58252.
Bai, Y., Yeung, E., Lui, C., Ong, C. S., Pitaktong, I., Huang, C., Inoue, T., Matsushita, H., Ma, C., & Hibino, N. (2018). A Net Mold-based Method of Scaffold-free Three-Dimensional Cardiac Tissue Creation. Journal of visualized experiments : JoVE, (138), . https://doi.org/10.3791/58252
Bai, Yang, et al. "A Net Mold-based Method of Scaffold-free Three-Dimensional Cardiac Tissue Creation." Journal of visualized experiments : JoVE vol. ,138 (2018). doi: https://doi.org/10.3791/58252
Bai Y, Yeung E, Lui C, Ong CS, Pitaktong I, Huang C, Inoue T, Matsushita H, Ma C, Hibino N. A Net Mold-based Method of Scaffold-free Three-Dimensional Cardiac Tissue Creation. J Vis Exp. 2018 Aug 05;(138). doi: 10.3791/58252. PMID: 30124650; PMCID: PMC6126624.
Copy Download .nbib Format: NLM
Protein trafficking in cardiovascular disease: How the science has evolved and where it must go.

Trends in cardiovascular medicine

Saffitz JE.
PMID: 25666491
Trends Cardiovasc Med. 2015 Jul;25(5):390-1. doi: 10.1016/j.tcm.2015.01.002. Epub 2015 Jan 12.

No abstract available.

Cite
Saffitz JE. Protein trafficking in cardiovascular disease: How the science has evolved and where it must go. Trends Cardiovasc Med. 2015;25(5):390-1doi: 10.1016/j.tcm.2015.01.002.
Saffitz, J. E. (2015). Protein trafficking in cardiovascular disease: How the science has evolved and where it must go. Trends in cardiovascular medicine, 25(5), 390-1. https://doi.org/10.1016/j.tcm.2015.01.002
Saffitz, Jeffrey E. "Protein trafficking in cardiovascular disease: How the science has evolved and where it must go." Trends in cardiovascular medicine vol. 25,5 (2015): 390-1. doi: https://doi.org/10.1016/j.tcm.2015.01.002
Saffitz JE. Protein trafficking in cardiovascular disease: How the science has evolved and where it must go. Trends Cardiovasc Med. 2015 Jul;25(5):390-1. doi: 10.1016/j.tcm.2015.01.002. Epub 2015 Jan 12. PMID: 25666491.
Copy Download .nbib Format: NLM
Metabolic profiling using HPLC allows classification of drugs according to their mechanisms of action in HL-1 cardiomyocytes.

Toxicology and applied pharmacology

Strigun A, Wahrheit J, Beckers S, Heinzle E, Noor F.
PMID: 21320520
Toxicol Appl Pharmacol. 2011 Apr 15;252(2):183-91. doi: 10.1016/j.taap.2011.02.008. Epub 2011 Feb 12.

Along with hepatotoxicity, cardiotoxic side effects remain one of the major reasons for drug withdrawals and boxed warnings. Prediction methods for cardiotoxicity are insufficient. High content screening comprising of not only electrophysiological characterization but also cellular molecular alterations are...

Cite
Strigun A, Wahrheit J, Beckers S, et al. Metabolic profiling using HPLC allows classification of drugs according to their mechanisms of action in HL-1 cardiomyocytes. Toxicol Appl Pharmacol. 2011;252(2):183-91doi: 10.1016/j.taap.2011.02.008.
Strigun, A., Wahrheit, J., Beckers, S., Heinzle, E., & Noor, F. (2011). Metabolic profiling using HPLC allows classification of drugs according to their mechanisms of action in HL-1 cardiomyocytes. Toxicology and applied pharmacology, 252(2), 183-91. https://doi.org/10.1016/j.taap.2011.02.008
Strigun, Alexander, et al. "Metabolic profiling using HPLC allows classification of drugs according to their mechanisms of action in HL-1 cardiomyocytes." Toxicology and applied pharmacology vol. 252,2 (2011): 183-91. doi: https://doi.org/10.1016/j.taap.2011.02.008
Strigun A, Wahrheit J, Beckers S, Heinzle E, Noor F. Metabolic profiling using HPLC allows classification of drugs according to their mechanisms of action in HL-1 cardiomyocytes. Toxicol Appl Pharmacol. 2011 Apr 15;252(2):183-91. doi: 10.1016/j.taap.2011.02.008. Epub 2011 Feb 12. PMID: 21320520.
Copy Download .nbib Format: NLM
9th Ascona International Workshop on Cardiomyocyte Biology.

Biochimica et biophysica acta. Molecular cell research

Brink M, Zuppinger C.
PMID: 31837983
Biochim Biophys Acta Mol Cell Res. 2020 Mar;1867(3):118618. doi: 10.1016/j.bbamcr.2019.118618. Epub 2019 Dec 12.

No abstract available.

Cite
Brink M, Zuppinger C. 9th Ascona International Workshop on Cardiomyocyte Biology. Biochim Biophys Acta Mol Cell Res. 2019;1867(3):118618doi: 10.1016/j.bbamcr.2019.118618.
Brink, M., & Zuppinger, C. (2020). 9th Ascona International Workshop on Cardiomyocyte Biology. Biochimica et biophysica acta. Molecular cell research, 1867(3), 118618. https://doi.org/10.1016/j.bbamcr.2019.118618
Brink, Marijke, and Zuppinger, Christian. "9th Ascona International Workshop on Cardiomyocyte Biology." Biochimica et biophysica acta. Molecular cell research vol. 1867,3 (2020): 118618. doi: https://doi.org/10.1016/j.bbamcr.2019.118618
Brink M, Zuppinger C. 9th Ascona International Workshop on Cardiomyocyte Biology. Biochim Biophys Acta Mol Cell Res. 2020 Mar;1867(3):118618. doi: 10.1016/j.bbamcr.2019.118618. Epub 2019 Dec 12. PMID: 31837983.
Copy Download .nbib Format: NLM
Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic intervention.

Trends in cardiovascular medicine

Xiao S, Shaw RM.
PMID: 25649302
Trends Cardiovasc Med. 2015 Jul;25(5):379-89. doi: 10.1016/j.tcm.2014.12.012. Epub 2014 Dec 29.

Cardiomyocytes, the individual contractile units of heart muscle, are long-lived and robust. Given the longevity of these cells, it can be easy to overlook their dynamic intracellular environment that contain rapid protein movements and frequent protein turnover. Critical gene...

Cite
Xiao S, Shaw RM. Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic intervention. Trends Cardiovasc Med. 2014;25(5):379-89doi: 10.1016/j.tcm.2014.12.012.
Xiao, S., & Shaw, R. M. (2015). Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic intervention. Trends in cardiovascular medicine, 25(5), 379-89. https://doi.org/10.1016/j.tcm.2014.12.012
Xiao, Shaohua, and Shaw, Robin M. "Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic intervention." Trends in cardiovascular medicine vol. 25,5 (2015): 379-89. doi: https://doi.org/10.1016/j.tcm.2014.12.012
Xiao S, Shaw RM. Cardiomyocyte protein trafficking: Relevance to heart disease and opportunities for therapeutic intervention. Trends Cardiovasc Med. 2015 Jul;25(5):379-89. doi: 10.1016/j.tcm.2014.12.012. Epub 2014 Dec 29. PMID: 25649302; PMCID: PMC4468031.
Copy Download .nbib Format: NLM
Showing 1 to 5 of 5 entries