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Showing 1 to 11 of 11 entries
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[Metabolic complications following kidney transplantation].

Harefuah

Green J, Better OS.
PMID: 6363226
Harefuah. 1983 Mar 01;104(5):187-91.

No abstract available.

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Green J, Better OS. [Metabolic complications following kidney transplantation]. Harefuah. 1983;104(5):187-91
Green, J., & Better, O. S. (1983). [Metabolic complications following kidney transplantation]. Harefuah, 104(5), 187-91.
Green, J, and Better, O S. "[Metabolic complications following kidney transplantation]." Harefuah vol. 104,5 (1983): 187-91.
Green J, Better OS. [Metabolic complications following kidney transplantation]. Harefuah. 1983 Mar 01;104(5):187-91. Hebrew. PMID: 6363226.
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Application of proteomic analysis to the study of renal diseases.

Nature reviews. Nephrology

Smith MP, Banks RE, Wood SL, Lewington AJ, Selby PJ.
PMID: 19859072
Nat Rev Nephrol. 2009 Dec;5(12):701-12. doi: 10.1038/nrneph.2009.183. Epub 2009 Oct 27.

Proteomics-based approaches are generating considerable data in clinical nephrology covering almost all aspects of the discipline. Proteomic experiments commonly involve fractionation and protein separation, followed by mass spectrometric analysis to identify proteins and peptides. Biostatistical and bioinformatical input is...

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Smith MP, Banks RE, Wood SL, et al. Application of proteomic analysis to the study of renal diseases. Nat Rev Nephrol. 2009;5(12):701-12doi: 10.1038/nrneph.2009.183.
Smith, M. P., Banks, R. E., Wood, S. L., Lewington, A. J., & Selby, P. J. (2009). Application of proteomic analysis to the study of renal diseases. Nature reviews. Nephrology, 5(12), 701-12. https://doi.org/10.1038/nrneph.2009.183
Smith, Matthew P Welberry, et al. "Application of proteomic analysis to the study of renal diseases." Nature reviews. Nephrology vol. 5,12 (2009): 701-12. doi: https://doi.org/10.1038/nrneph.2009.183
Smith MP, Banks RE, Wood SL, Lewington AJ, Selby PJ. Application of proteomic analysis to the study of renal diseases. Nat Rev Nephrol. 2009 Dec;5(12):701-12. doi: 10.1038/nrneph.2009.183. Epub 2009 Oct 27. PMID: 19859072.
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Understanding the nature of renal disease progression.

Kidney international

Remuzzi G, Ruggenenti P, Benigni A.
PMID: 8995712
Kidney Int. 1997 Jan;51(1):2-15. doi: 10.1038/ki.1997.2.
Free Article

Animal and human proteinuric glomerulopathies evolve to terminal renal failure by a process leading to progressive parenchymal damage, which appears to be relatively independent of the initial insult. Despite the fact that the mechanism(s) leading to renal disease progression...

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Remuzzi G, Ruggenenti P, Benigni A. Understanding the nature of renal disease progression. Kidney Int. 1997;51(1):2-15doi: 10.1038/ki.1997.2.
Remuzzi, G., Ruggenenti, P., & Benigni, A. (1997). Understanding the nature of renal disease progression. Kidney international, 51(1), 2-15. https://doi.org/10.1038/ki.1997.2
Remuzzi, G, et al. "Understanding the nature of renal disease progression." Kidney international vol. 51,1 (1997): 2-15. doi: https://doi.org/10.1038/ki.1997.2
Remuzzi G, Ruggenenti P, Benigni A. Understanding the nature of renal disease progression. Kidney Int. 1997 Jan;51(1):2-15. doi: 10.1038/ki.1997.2. PMID: 8995712.
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New insights into molecular mechanisms of epigenetic regulation in kidney disease.

Clinical and experimental pharmacology & physiology

Mimura I, Tanaka T, Nangaku M.
PMID: 27560313
Clin Exp Pharmacol Physiol. 2016 Dec;43(12):1159-1167. doi: 10.1111/1440-1681.12663.

The number of patients with kidney failure has increased in recent years. Different factors contribute to the progression of chronic kidney disease, including glomerular sclerosis, atherosclerosis of the renal arteries and tubulointerstitial fibrosis. Tubulointerstitial injury is induced by hypoxia...

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Mimura I, Tanaka T, Nangaku M. New insights into molecular mechanisms of epigenetic regulation in kidney disease. Clin Exp Pharmacol Physiol. 2016;43(12):1159-1167doi: 10.1111/1440-1681.12663.
Mimura, I., Tanaka, T., & Nangaku, M. (2016). New insights into molecular mechanisms of epigenetic regulation in kidney disease. Clinical and experimental pharmacology & physiology, 43(12), 1159-1167. https://doi.org/10.1111/1440-1681.12663
Mimura, Imari, et al. "New insights into molecular mechanisms of epigenetic regulation in kidney disease." Clinical and experimental pharmacology & physiology vol. 43,12 (2016): 1159-1167. doi: https://doi.org/10.1111/1440-1681.12663
Mimura I, Tanaka T, Nangaku M. New insights into molecular mechanisms of epigenetic regulation in kidney disease. Clin Exp Pharmacol Physiol. 2016 Dec;43(12):1159-1167. doi: 10.1111/1440-1681.12663. PMID: 27560313.
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Current world literature. Clinical nephrology.

Current opinion in nephrology and hypertension

[No authors listed]
PMID: 17293690
Curr Opin Nephrol Hypertens. 2007 Mar;16(2):143-58. doi: 10.1097/MNH.0b013e32809cc9eb.

No abstract available.

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Current world literature. Clinical nephrology. Curr Opin Nephrol Hypertens. 2007;16(2):143-58doi: 10.1097/MNH.0b013e32809cc9eb.
(2007). Current world literature. Clinical nephrology. Current opinion in nephrology and hypertension, 16(2), 143-58. https://doi.org/10.1097/MNH.0b013e32809cc9eb
"Current world literature. Clinical nephrology." Current opinion in nephrology and hypertension vol. 16,2 (2007): 143-58. doi: https://doi.org/10.1097/MNH.0b013e32809cc9eb
Current world literature. Clinical nephrology. Curr Opin Nephrol Hypertens. 2007 Mar;16(2):143-58. doi: 10.1097/MNH.0b013e32809cc9eb. PMID: 17293690.
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Bibliography. Current world literature. Renal pathophysiology.

Current opinion in nephrology and hypertension

[No authors listed]
PMID: 16869063
Curr Opin Nephrol Hypertens. 2006 Jul;15(4):467-71.

No abstract available.

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Bibliography. Current world literature. Renal pathophysiology. Curr Opin Nephrol Hypertens. 2006;15(4):467-71
(2006). Bibliography. Current world literature. Renal pathophysiology. Current opinion in nephrology and hypertension, 15(4), 467-71.
"Bibliography. Current world literature. Renal pathophysiology." Current opinion in nephrology and hypertension vol. 15,4 (2006): 467-71.
Bibliography. Current world literature. Renal pathophysiology. Curr Opin Nephrol Hypertens. 2006 Jul;15(4):467-71. PMID: 16869063.
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Essential hypertension in blacks: is it a metabolic disorder?.

American journal of kidney diseases : the official journal of the National Kidney Foundation

Weir MR, Tuck ML.
PMID: 8465837
Am J Kidney Dis. 1993 Apr;21(4):58-67. doi: 10.1016/s0272-6386(12)80863-8.

No abstract available.

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Weir MR, Tuck ML. Essential hypertension in blacks: is it a metabolic disorder?. Am J Kidney Dis. 1993;21(4):58-67doi: 10.1016/s0272-6386(12)80863-8.
Weir, M. R., & Tuck, M. L. (1993). Essential hypertension in blacks: is it a metabolic disorder?. American journal of kidney diseases : the official journal of the National Kidney Foundation, 21(4), 58-67. https://doi.org/10.1016/s0272-6386(12)80863-8
Weir, M R, and Tuck, M L. "Essential hypertension in blacks: is it a metabolic disorder?." American journal of kidney diseases : the official journal of the National Kidney Foundation vol. 21,4 (1993): 58-67. doi: https://doi.org/10.1016/s0272-6386(12)80863-8
Weir MR, Tuck ML. Essential hypertension in blacks: is it a metabolic disorder?. Am J Kidney Dis. 1993 Apr;21(4):58-67. doi: 10.1016/s0272-6386(12)80863-8. PMID: 8465837.
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[Prevention of progression in nephropathies].

Bratislavske lekarske listy

Dzúrik R, Dzúriková V, Oksa A, Fedelesová V, Gajdos M, Spustová V.
PMID: 7812847
Bratisl Lek Listy. 1994 Sep;95(9):408-14.

Intermittent dialysis is excessively expensive and pretentious psychologically and socially. As a result the research is concentrated on the prevention of kidney disease progression. Preventive measures: a) Protein restriction forms the basis of nonpharmacologic measures. b) Consequent antihypertensive therapy,...

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Dzúrik R, Dzúriková V, Oksa A, et al. Prevencia progresie nefropatií. [Prevention of progression in nephropathies]. Bratisl Lek Listy. 1994;95(9):408-14
Dzúrik, R., Dzúriková, V., Oksa, A., Fedelesová, V., Gajdos, M., & Spustová, V. (1994). Prevencia progresie nefropatií. [Prevention of progression in nephropathies]. Bratislavske lekarske listy, 95(9), 408-14.
Dzúrik, R, et al. "Prevencia progresie nefropatií." [Prevention of progression in nephropathies]. Bratislavske lekarske listy vol. 95,9 (1994): 408-14.
Dzúrik R, Dzúriková V, Oksa A, Fedelesová V, Gajdos M, Spustová V. Prevencia progresie nefropatií. [Prevention of progression in nephropathies]. Bratisl Lek Listy. 1994 Sep;95(9):408-14. Slovak. PMID: 7812847.
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[Nutrition and metabolism in kidney disease].

Revue medicale suisse

Martin PY, Teta D, Burnier M.
PMID: 20344991
Rev Med Suisse. 2010 Mar 03;6(238):427-8.

No abstract available.

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Martin PY, Teta D, Burnier M. La nutrition et le métabolisme dans les maladies rénales. [Nutrition and metabolism in kidney disease]. Rev Med Suisse. 2010;6(238):427-8
Martin, P. Y., Teta, D., & Burnier, M. (2010). La nutrition et le métabolisme dans les maladies rénales. [Nutrition and metabolism in kidney disease]. Revue medicale suisse, 6(238), 427-8.
Martin, Pierre-Yves, et al. "La nutrition et le métabolisme dans les maladies rénales." [Nutrition and metabolism in kidney disease]. Revue medicale suisse vol. 6,238 (2010): 427-8.
Martin PY, Teta D, Burnier M. La nutrition et le métabolisme dans les maladies rénales. [Nutrition and metabolism in kidney disease]. Rev Med Suisse. 2010 Mar 03;6(238):427-8. French. PMID: 20344991.
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Proteomics for Clinical Assessment of Kidney Disease.

Proteomics. Clinical applications

Ortiz A.
PMID: 30768767
Proteomics Clin Appl. 2019 Mar;13(2):e1900004. doi: 10.1002/prca.201900004. Epub 2019 Feb 25.

Kidney disease is one of the fastest growing causes of death worldwide, disclosing an unmet clinical need for early diagnosis and optimized risk stratification that allows high risk patient selection for clinical trials and for more intensive nephroprotective interventions...

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Ortiz A. Proteomics for Clinical Assessment of Kidney Disease. Proteomics Clin Appl. 2019;13(2):e1900004doi: 10.1002/prca.201900004.
Ortiz, A. (2019). Proteomics for Clinical Assessment of Kidney Disease. Proteomics. Clinical applications, 13(2), e1900004. https://doi.org/10.1002/prca.201900004
Ortiz, Alberto. "Proteomics for Clinical Assessment of Kidney Disease." Proteomics. Clinical applications vol. 13,2 (2019): e1900004. doi: https://doi.org/10.1002/prca.201900004
Ortiz A. Proteomics for Clinical Assessment of Kidney Disease. Proteomics Clin Appl. 2019 Mar;13(2):e1900004. doi: 10.1002/prca.201900004. Epub 2019 Feb 25. PMID: 30768767.
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Ferroptosis and kidney diseases.

International urology and nephrology

Tang S, Xiao X.
PMID: 31768804
Int Urol Nephrol. 2020 Mar;52(3):497-503. doi: 10.1007/s11255-019-02335-7. Epub 2019 Nov 25.

Ferroptosis is a form of iron-dependent, non-apoptotic regulated cell death, which is characterized by the accumulation of lipid hydroperoxides to lethal levels. Ferroptosis recently has been shown to have implications in diverse kidney diseases, such as acute kidney injury,...

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Tang S, Xiao X. Ferroptosis and kidney diseases. Int Urol Nephrol. 2019;52(3):497-503doi: 10.1007/s11255-019-02335-7.
Tang, S., & Xiao, X. (2020). Ferroptosis and kidney diseases. International urology and nephrology, 52(3), 497-503. https://doi.org/10.1007/s11255-019-02335-7
Tang, Shumei, and Xiao, Xiangcheng. "Ferroptosis and kidney diseases." International urology and nephrology vol. 52,3 (2020): 497-503. doi: https://doi.org/10.1007/s11255-019-02335-7
Tang S, Xiao X. Ferroptosis and kidney diseases. Int Urol Nephrol. 2020 Mar;52(3):497-503. doi: 10.1007/s11255-019-02335-7. Epub 2019 Nov 25. PMID: 31768804.
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Showing 1 to 11 of 11 entries