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Chronic inflammation, Adverse Outcome Pathways, and risk assessment: A diagrammatic exposition.

Regulatory toxicology and pharmacology : RTP

Cox LA, Goodman JE, Engel AM.
PMID: 32330641
Regul Toxicol Pharmacol. 2020 Jul;114:104663. doi: 10.1016/j.yrtph.2020.104663. Epub 2020 Apr 21.

Inflammasomes are a family of pro-inflammatory signaling complexes that orchestrate inflammatory responses in many tissues. The NLRP3 inflammasome has been implicated in several diseases associated with chronic inflammation. In this paper, we present an Adverse Outcome Pathway (AOP) for...

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Cox LA, Goodman JE, Engel AM. Chronic inflammation, Adverse Outcome Pathways, and risk assessment: A diagrammatic exposition. Regul Toxicol Pharmacol. 2020;114:104663doi: 10.1016/j.yrtph.2020.104663.
Cox, L. A., Goodman, J. E., & Engel, A. M. (2020). Chronic inflammation, Adverse Outcome Pathways, and risk assessment: A diagrammatic exposition. Regulatory toxicology and pharmacology : RTP, 114104663. https://doi.org/10.1016/j.yrtph.2020.104663
Cox, Louis Anthony, et al. "Chronic inflammation, Adverse Outcome Pathways, and risk assessment: A diagrammatic exposition." Regulatory toxicology and pharmacology : RTP vol. 114 (2020): 104663. doi: https://doi.org/10.1016/j.yrtph.2020.104663
Cox LA, Goodman JE, Engel AM. Chronic inflammation, Adverse Outcome Pathways, and risk assessment: A diagrammatic exposition. Regul Toxicol Pharmacol. 2020 Jul;114:104663. doi: 10.1016/j.yrtph.2020.104663. Epub 2020 Apr 21. PMID: 32330641.
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Potential metabolic and inflammatory pathways between COVID-19 and new-onset diabetes.

Diabetes & metabolism

Sathish T, Tapp RJ, Cooper ME, Zimmet P.
PMID: 33129968
Diabetes Metab. 2021 Mar;47(2):101204. doi: 10.1016/j.diabet.2020.10.002. Epub 2020 Oct 28.

No abstract available.

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Sathish T, Tapp RJ, Cooper ME, et al. Potential metabolic and inflammatory pathways between COVID-19 and new-onset diabetes. Diabetes Metab. 2020;47(2):101204doi: 10.1016/j.diabet.2020.10.002.
Sathish, T., Tapp, R. J., Cooper, M. E., & Zimmet, P. (2021). Potential metabolic and inflammatory pathways between COVID-19 and new-onset diabetes. Diabetes & metabolism, 47(2), 101204. https://doi.org/10.1016/j.diabet.2020.10.002
Sathish, Thirunavukkarasu, et al. "Potential metabolic and inflammatory pathways between COVID-19 and new-onset diabetes." Diabetes & metabolism vol. 47,2 (2021): 101204. doi: https://doi.org/10.1016/j.diabet.2020.10.002
Sathish T, Tapp RJ, Cooper ME, Zimmet P. Potential metabolic and inflammatory pathways between COVID-19 and new-onset diabetes. Diabetes Metab. 2021 Mar;47(2):101204. doi: 10.1016/j.diabet.2020.10.002. Epub 2020 Oct 28. PMID: 33129968; PMCID: PMC7591867.
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Gut microbiota, inflammation, and molecular signatures of host response to infection.

Journal of genetics and genomics = Yi chuan xue bao

Gou W, Fu Y, Yue L, Chen GD, Cai X, Shuai M, Xu F, Yi X, Chen H, Zhu Y, Xiao ML, Jiang Z, Miao Z, Xiao C, Shen B, Wu X, Zhao H, Ling W, Wang J, Chen YM, Guo T, Zheng JS.
PMID: 34257044
J Genet Genomics. 2021 Sep 20;48(9):792-802. doi: 10.1016/j.jgg.2021.04.002. Epub 2021 May 03.

Gut microbial dysbiosis has been linked to many noncommunicable diseases. However, little is known about specific gut microbiota composition and its correlated metabolites associated with molecular signatures underlying host response to infection. Here, we describe the construction of a...

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Gou W, Fu Y, Yue L, et al. Gut microbiota, inflammation, and molecular signatures of host response to infection. J Genet Genomics. 2021;48(9):792-802doi: 10.1016/j.jgg.2021.04.002.
Gou, W., Fu, Y., Yue, L., Chen, G. D., Cai, X., Shuai, M., Xu, F., Yi, X., Chen, H., Zhu, Y., Xiao, M. L., Jiang, Z., Miao, Z., Xiao, C., Shen, B., Wu, X., Zhao, H., Ling, W., Wang, J., Chen, Y. M., Guo, T., & Zheng, J. S. (2021). Gut microbiota, inflammation, and molecular signatures of host response to infection. Journal of genetics and genomics = Yi chuan xue bao, 48(9), 792-802. https://doi.org/10.1016/j.jgg.2021.04.002
Gou, Wanglong, et al. "Gut microbiota, inflammation, and molecular signatures of host response to infection." Journal of genetics and genomics = Yi chuan xue bao vol. 48,9 (2021): 792-802. doi: https://doi.org/10.1016/j.jgg.2021.04.002
Gou W, Fu Y, Yue L, Chen GD, Cai X, Shuai M, Xu F, Yi X, Chen H, Zhu Y, Xiao ML, Jiang Z, Miao Z, Xiao C, Shen B, Wu X, Zhao H, Ling W, Wang J, Chen YM, Guo T, Zheng JS. Gut microbiota, inflammation, and molecular signatures of host response to infection. J Genet Genomics. 2021 Sep 20;48(9):792-802. doi: 10.1016/j.jgg.2021.04.002. Epub 2021 May 03. PMID: 34257044.
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[Inflammation].

Concours medical

Delaunay A.
PMID: 5838917
Concours Med. 1965 May 22;87(21):3598-614.

No abstract available.

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Delaunay A. L'inflammation. [Inflammation]. Concours Med. 1965;87(21):3598-614
Delaunay, A. (1965). L'inflammation. [Inflammation]. Concours medical, 87(21), 3598-614.
Delaunay, A. "L'inflammation." [Inflammation]. Concours medical vol. 87,21 (1965): 3598-614.
Delaunay A. L'inflammation. [Inflammation]. Concours Med. 1965 May 22;87(21):3598-614. French. PMID: 5838917.
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The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation.

Sub-cellular biochemistry

Monie TP.
PMID: 28271472
Subcell Biochem. 2017;83:43-73. doi: 10.1007/978-3-319-46503-6_2.

The inflammasome is a multi-molecular platform crucial to the induction of an inflammatory response to cellular danger. Recognition in the cytoplasm of endogenously and exogenously derived ligands initiates conformational change in sensor proteins, such as NLRP3, that permits the...

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Monie TP. The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation. Subcell Biochem. 2017;83:43-73doi: 10.1007/978-3-319-46503-6_2.
Monie, T. P. (2017). The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation. Sub-cellular biochemistry, 8343-73. https://doi.org/10.1007/978-3-319-46503-6_2
Monie, Tom P. "The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation." Sub-cellular biochemistry vol. 83 (2017): 43-73. doi: https://doi.org/10.1007/978-3-319-46503-6_2
Monie TP. The Canonical Inflammasome: A Macromolecular Complex Driving Inflammation. Subcell Biochem. 2017;83:43-73. doi: 10.1007/978-3-319-46503-6_2. PMID: 28271472.
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Human malarial disease: a consequence of inflammatory cytokine release.

Malaria journal

Clark IA, Budd AC, Alleva LM, Cowden WB.
PMID: 17029647
Malar J. 2006 Oct 10;5:85. doi: 10.1186/1475-2875-5-85.

Malaria causes an acute systemic human disease that bears many similarities, both clinically and mechanistically, to those caused by bacteria, rickettsia, and viruses. Over the past few decades, a literature has emerged that argues for most of the pathology...

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Clark IA, Budd AC, Alleva LM, et al. Human malarial disease: a consequence of inflammatory cytokine release. Malar J. 2006;5:85doi: 10.1186/1475-2875-5-85.
Clark, I. A., Budd, A. C., Alleva, L. M., & Cowden, W. B. (2006). Human malarial disease: a consequence of inflammatory cytokine release. Malaria journal, 585. https://doi.org/10.1186/1475-2875-5-85
Clark, Ian A, et al. "Human malarial disease: a consequence of inflammatory cytokine release." Malaria journal vol. 5 (2006): 85. doi: https://doi.org/10.1186/1475-2875-5-85
Clark IA, Budd AC, Alleva LM, Cowden WB. Human malarial disease: a consequence of inflammatory cytokine release. Malar J. 2006 Oct 10;5:85. doi: 10.1186/1475-2875-5-85. PMID: 17029647; PMCID: PMC1629020.
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Showing 1 to 6 of 6 entries