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External validity in the assessment of intellectual development in adulthood.

Journal of gerontology

Schaie KW.
PMID: 400894
J Gerontol. 1978 Sep;33(5):695-701. doi: 10.1093/geronj/33.5.695.

The relation of intelligence and competence is discussed and external validity issues are examined for the dimensions of settings, measurement variables, treatment variables and experimental units. It is argued that external validity across situations and life stages cannot be...

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Schaie KW. External validity in the assessment of intellectual development in adulthood. J Gerontol. 1978;33(5):695-701doi: 10.1093/geronj/33.5.695.
Schaie, K. W. (1978). External validity in the assessment of intellectual development in adulthood. Journal of gerontology, 33(5), 695-701. https://doi.org/10.1093/geronj/33.5.695
Schaie, K W. "External validity in the assessment of intellectual development in adulthood." Journal of gerontology vol. 33,5 (1978): 695-701. doi: https://doi.org/10.1093/geronj/33.5.695
Schaie KW. External validity in the assessment of intellectual development in adulthood. J Gerontol. 1978 Sep;33(5):695-701. doi: 10.1093/geronj/33.5.695. PMID: 400894.
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Role of genetic factors in atherosclerotic disease.

The American journal of clinical nutrition

Berg K.
PMID: 2655412
Am J Clin Nutr. 1989 May;49(5):1025-9. doi: 10.1093/ajcn/49.5.1025.

No abstract available.

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Berg K. Role of genetic factors in atherosclerotic disease. Am J Clin Nutr. 1989;49(5):1025-9doi: 10.1093/ajcn/49.5.1025.
Berg, K. (1989). Role of genetic factors in atherosclerotic disease. The American journal of clinical nutrition, 49(5), 1025-9. https://doi.org/10.1093/ajcn/49.5.1025
Berg, K. "Role of genetic factors in atherosclerotic disease." The American journal of clinical nutrition vol. 49,5 (1989): 1025-9. doi: https://doi.org/10.1093/ajcn/49.5.1025
Berg K. Role of genetic factors in atherosclerotic disease. Am J Clin Nutr. 1989 May;49(5):1025-9. doi: 10.1093/ajcn/49.5.1025. PMID: 2655412.
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SNPTransformer: a lightweight toolkit for genome-wide association studies.

Genomics, proteomics & bioinformatics

Dong C.
PMID: 21382596
Genomics Proteomics Bioinformatics. 2010 Dec;8(4):268-73. doi: 10.1016/S1672-0229(10)60029-0.

High-throughput genotyping chips have produced huge datasets for genome-wide association studies (GWAS) that have contributed greatly to discovering susceptibility genes for complex diseases. There are two strategies for performing data analysis for GWAS. One strategy is to use open-source...

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Dong C. SNPTransformer: a lightweight toolkit for genome-wide association studies. Genomics Proteomics Bioinformatics. 2010;8(4):268-73doi: 10.1016/S1672-0229(10)60029-0.
Dong, C. (2010). SNPTransformer: a lightweight toolkit for genome-wide association studies. Genomics, proteomics & bioinformatics, 8(4), 268-73. https://doi.org/10.1016/S1672-0229(10)60029-0
Dong, Changzheng. "SNPTransformer: a lightweight toolkit for genome-wide association studies." Genomics, proteomics & bioinformatics vol. 8,4 (2010): 268-73. doi: https://doi.org/10.1016/S1672-0229(10)60029-0
Dong C. SNPTransformer: a lightweight toolkit for genome-wide association studies. Genomics Proteomics Bioinformatics. 2010 Dec;8(4):268-73. doi: 10.1016/S1672-0229(10)60029-0. PMID: 21382596; PMCID: PMC5054149.
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FAPI: Fast and accurate P-value Imputation for genome-wide association study.

European journal of human genetics : EJHG

Kwan JS, Li MX, Deng JE, Sham PC.
PMID: 26306642
Eur J Hum Genet. 2016 May;24(5):761-6. doi: 10.1038/ejhg.2015.190. Epub 2015 Aug 26.

Imputing individual-level genotypes (or genotype imputation) is now a standard procedure in genome-wide association studies (GWAS) to examine disease associations at untyped common genetic variants. Meta-analysis of publicly available GWAS summary statistics can allow more disease-associated loci to be...

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Kwan JS, Li MX, Deng JE, et al. FAPI: Fast and accurate P-value Imputation for genome-wide association study. Eur J Hum Genet. 2015;24(5):761-6doi: 10.1038/ejhg.2015.190.
Kwan, J. S., Li, M. X., Deng, J. E., & Sham, P. C. (2016). FAPI: Fast and accurate P-value Imputation for genome-wide association study. European journal of human genetics : EJHG, 24(5), 761-6. https://doi.org/10.1038/ejhg.2015.190
Kwan, Johnny S H, et al. "FAPI: Fast and accurate P-value Imputation for genome-wide association study." European journal of human genetics : EJHG vol. 24,5 (2016): 761-6. doi: https://doi.org/10.1038/ejhg.2015.190
Kwan JS, Li MX, Deng JE, Sham PC. FAPI: Fast and accurate P-value Imputation for genome-wide association study. Eur J Hum Genet. 2016 May;24(5):761-6. doi: 10.1038/ejhg.2015.190. Epub 2015 Aug 26. PMID: 26306642; PMCID: PMC4930094.
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VCF-Explorer: filtering and analysing whole genome VCF files.

Bioinformatics (Oxford, England)

Akgün M, Demirci H.
PMID: 29036499
Bioinformatics. 2017 Nov 01;33(21):3468-3470. doi: 10.1093/bioinformatics/btx422.

SUMMARY: The decreasing cost in high-throughput technologies led to a number of sequencing projects consisting of thousands of whole genomes. The paradigm shift from exome to whole genome brings a significant increase in the size of output files. Most...

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Akgün M, Demirci H. VCF-Explorer: filtering and analysing whole genome VCF files. Bioinformatics. 2017;33(21):3468-3470doi: 10.1093/bioinformatics/btx422.
Akgün, M., & Demirci, H. (2017). VCF-Explorer: filtering and analysing whole genome VCF files. Bioinformatics (Oxford, England), 33(21), 3468-3470. https://doi.org/10.1093/bioinformatics/btx422
Akgün, Mete, and Demirci, Hüseyin. "VCF-Explorer: filtering and analysing whole genome VCF files." Bioinformatics (Oxford, England) vol. 33,21 (2017): 3468-3470. doi: https://doi.org/10.1093/bioinformatics/btx422
Akgün M, Demirci H. VCF-Explorer: filtering and analysing whole genome VCF files. Bioinformatics. 2017 Nov 01;33(21):3468-3470. doi: 10.1093/bioinformatics/btx422. PMID: 29036499.
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[Double lesions of GIST with different morphology and genotypes: report of a case].

Zhonghua bing li xue za zhi = Chinese journal of pathology

Tian Z, Liu Y, Sun X, Li M, Duan X.
PMID: 26702536
Zhonghua Bing Li Xue Za Zhi. 2015 Oct;44(10):747-8.

No abstract available.

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Tian Z, Liu Y, Sun X, et al. [Double lesions of GIST with different morphology and genotypes: report of a case]. Zhonghua Bing Li Xue Za Zhi. 2015;44(10):747-8
Tian, Z., Liu, Y., Sun, X., Li, M., & Duan, X. (2015). [Double lesions of GIST with different morphology and genotypes: report of a case]. Zhonghua bing li xue za zhi = Chinese journal of pathology, 44(10), 747-8.
Tian, Zhuang, et al. "[Double lesions of GIST with different morphology and genotypes: report of a case]." Zhonghua bing li xue za zhi = Chinese journal of pathology vol. 44,10 (2015): 747-8.
Tian Z, Liu Y, Sun X, Li M, Duan X. [Double lesions of GIST with different morphology and genotypes: report of a case]. Zhonghua Bing Li Xue Za Zhi. 2015 Oct;44(10):747-8. Chinese. PMID: 26702536.
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A general approach for combining diverse rare variant association tests provides improved robustness across a wider range of genetic architectures.

European journal of human genetics : EJHG

Greco B, Hainline A, Arbet J, Grinde K, Benitez A, Tintle N.
PMID: 26508571
Eur J Hum Genet. 2016 May;24(5):767-73. doi: 10.1038/ejhg.2015.194. Epub 2015 Oct 28.

The widespread availability of genome sequencing data made possible by way of next-generation technologies has yielded a flood of different gene-based rare variant association tests. Most of these tests have been published because they have superior power for particular...

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Greco B, Hainline A, Arbet J, et al. A general approach for combining diverse rare variant association tests provides improved robustness across a wider range of genetic architectures. Eur J Hum Genet. 2015;24(5):767-73doi: 10.1038/ejhg.2015.194.
Greco, B., Hainline, A., Arbet, J., Grinde, K., Benitez, A., & Tintle, N. (2016). A general approach for combining diverse rare variant association tests provides improved robustness across a wider range of genetic architectures. European journal of human genetics : EJHG, 24(5), 767-73. https://doi.org/10.1038/ejhg.2015.194
Greco, Brian, et al. "A general approach for combining diverse rare variant association tests provides improved robustness across a wider range of genetic architectures." European journal of human genetics : EJHG vol. 24,5 (2016): 767-73. doi: https://doi.org/10.1038/ejhg.2015.194
Greco B, Hainline A, Arbet J, Grinde K, Benitez A, Tintle N. A general approach for combining diverse rare variant association tests provides improved robustness across a wider range of genetic architectures. Eur J Hum Genet. 2016 May;24(5):767-73. doi: 10.1038/ejhg.2015.194. Epub 2015 Oct 28. PMID: 26508571; PMCID: PMC4930097.
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Genetic conditions associated with a predisposition to kidney cancer.

Clinical advances in hematology & oncology : H&O

[No authors listed]
PMID: 28591096
Clin Adv Hematol Oncol. 2017 Apr;15(4):239-241.

No abstract available.

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Genetic conditions associated with a predisposition to kidney cancer. Clin Adv Hematol Oncol. 2017;15(4):239-241
(2017). Genetic conditions associated with a predisposition to kidney cancer. Clinical advances in hematology & oncology : H&O, 15(4), 239-241.
"Genetic conditions associated with a predisposition to kidney cancer." Clinical advances in hematology & oncology : H&O vol. 15,4 (2017): 239-241.
Genetic conditions associated with a predisposition to kidney cancer. Clin Adv Hematol Oncol. 2017 Apr;15(4):239-241. PMID: 28591096.
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Autosomal dominant spinocerebellar ataxias ad infinitum?.

Neurology

Subramony SH, Filla A.
PMID: 11171890
Neurology. 2001 Feb 13;56(3):287-9. doi: 10.1212/wnl.56.3.287.

No abstract available.

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Subramony SH, Filla A. Autosomal dominant spinocerebellar ataxias ad infinitum?. Neurology. 2001;56(3):287-9doi: 10.1212/wnl.56.3.287.
Subramony, S. H., & Filla, A. (2001). Autosomal dominant spinocerebellar ataxias ad infinitum?. Neurology, 56(3), 287-9. https://doi.org/10.1212/wnl.56.3.287
Subramony, S H, and Filla, A. "Autosomal dominant spinocerebellar ataxias ad infinitum?." Neurology vol. 56,3 (2001): 287-9. doi: https://doi.org/10.1212/wnl.56.3.287
Subramony SH, Filla A. Autosomal dominant spinocerebellar ataxias ad infinitum?. Neurology. 2001 Feb 13;56(3):287-9. doi: 10.1212/wnl.56.3.287. PMID: 11171890.
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[Distribution of six STR loci in Bai ethnic group in Yunnan province of China].

Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics

Zou P, Yang Y, Li D, Zou L, Shen B.
PMID: 10359866
Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 1999 Jun;16(3):160-3.

OBJECTIVE: This study was conducted to make known the distribution of six STR loci in Bai ethnic group in Yunnan province.METHODS: DAN extraction from blood samples (126 in number ) and multiplex amplification of CSF1PO,TPOX, TH01,F13A01, FESFPS and vWA...

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Zou P, Yang Y, Li D, et al. [Distribution of six STR loci in Bai ethnic group in Yunnan province of China]. Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 1999;16(3):160-3
Zou, P., Yang, Y., Li, D., Zou, L., & Shen, B. (1999). [Distribution of six STR loci in Bai ethnic group in Yunnan province of China]. Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics, 16(3), 160-3.
Zou, P, et al. "[Distribution of six STR loci in Bai ethnic group in Yunnan province of China]." Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics vol. 16,3 (1999): 160-3.
Zou P, Yang Y, Li D, Zou L, Shen B. [Distribution of six STR loci in Bai ethnic group in Yunnan province of China]. Zhonghua Yi Xue Yi Chuan Xue Za Zhi. 1999 Jun;16(3):160-3. Chinese. PMID: 10359866.
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Gene-environment interactions--the BioBank UK study.

The pharmacogenomics journal

Wright AF, Carothers AD, Campbell H.
PMID: 12049178
Pharmacogenomics J. 2002;2(2):75-82. doi: 10.1038/sj.tpj.6500085.

No abstract available.

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Wright AF, Carothers AD, Campbell H. Gene-environment interactions--the BioBank UK study. Pharmacogenomics J. 2002;2(2):75-82doi: 10.1038/sj.tpj.6500085.
Wright, A. F., Carothers, A. D., & Campbell, H. (2002). Gene-environment interactions--the BioBank UK study. The pharmacogenomics journal, 2(2), 75-82. https://doi.org/10.1038/sj.tpj.6500085
Wright, A F, et al. "Gene-environment interactions--the BioBank UK study." The pharmacogenomics journal vol. 2,2 (2002): 75-82. doi: https://doi.org/10.1038/sj.tpj.6500085
Wright AF, Carothers AD, Campbell H. Gene-environment interactions--the BioBank UK study. Pharmacogenomics J. 2002;2(2):75-82. doi: 10.1038/sj.tpj.6500085. PMID: 12049178.
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A mixed strategy model for the emergence and intensification of social learning in a periodically changing natural environment.

Theoretical population biology

Wakano JY, Aoki K.
PMID: 16730768
Theor Popul Biol. 2006 Dec;70(4):486-97. doi: 10.1016/j.tpb.2006.04.003. Epub 2006 Apr 25.

Based on a population genetic model of mixed strategies determined by alleles of small effect, we derive conditions for the evolution of social learning in an infinite-state environment that changes periodically over time. Each mixed strategy is defined by...

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Wakano JY, Aoki K. A mixed strategy model for the emergence and intensification of social learning in a periodically changing natural environment. Theor Popul Biol. 2006;70(4):486-97doi: 10.1016/j.tpb.2006.04.003.
Wakano, J. Y., & Aoki, K. (2006). A mixed strategy model for the emergence and intensification of social learning in a periodically changing natural environment. Theoretical population biology, 70(4), 486-97. https://doi.org/10.1016/j.tpb.2006.04.003
Wakano, Joe Yuichiro, and Aoki, Kenichi. "A mixed strategy model for the emergence and intensification of social learning in a periodically changing natural environment." Theoretical population biology vol. 70,4 (2006): 486-97. doi: https://doi.org/10.1016/j.tpb.2006.04.003
Wakano JY, Aoki K. A mixed strategy model for the emergence and intensification of social learning in a periodically changing natural environment. Theor Popul Biol. 2006 Dec;70(4):486-97. doi: 10.1016/j.tpb.2006.04.003. Epub 2006 Apr 25. PMID: 16730768.
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Showing 1 to 12 of 131 entries