Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 7492 entries
Sorted by: Best Match Show Resources per page
Limitation of no reflow injury by blood-free reperfusion with oxygenated perfluorochemical (Fluosol-DA 20%).

Journal of the American College of Cardiology

Kolodgie FD, Virmani R, Farb A.
PMID: 2050924
J Am Coll Cardiol. 1991 Jul;18(1):215-23. doi: 10.1016/s0735-1097(10)80242-6.
Free Article

This study was designed to assess the effects of blood-free reperfusion with oxygenated or unoxygenated intracoronary perfluorochemical (Fluosol-DA 20%) on myocardial perfusion and to determine its mechanism or mechanisms of limiting no reflow. Twenty-four dogs underwent 90 min of...

Cite
Kolodgie FD, Virmani R, Farb A. Limitation of no reflow injury by blood-free reperfusion with oxygenated perfluorochemical (Fluosol-DA 20%). J Am Coll Cardiol. 1991;18(1):215-23doi: 10.1016/s0735-1097(10)80242-6.
Kolodgie, F. D., Virmani, R., & Farb, A. (1991). Limitation of no reflow injury by blood-free reperfusion with oxygenated perfluorochemical (Fluosol-DA 20%). Journal of the American College of Cardiology, 18(1), 215-23. https://doi.org/10.1016/s0735-1097(10)80242-6
Kolodgie, F D, et al. "Limitation of no reflow injury by blood-free reperfusion with oxygenated perfluorochemical (Fluosol-DA 20%)." Journal of the American College of Cardiology vol. 18,1 (1991): 215-23. doi: https://doi.org/10.1016/s0735-1097(10)80242-6
Kolodgie FD, Virmani R, Farb A. Limitation of no reflow injury by blood-free reperfusion with oxygenated perfluorochemical (Fluosol-DA 20%). J Am Coll Cardiol. 1991 Jul;18(1):215-23. doi: 10.1016/s0735-1097(10)80242-6. PMID: 2050924.
Copy Download .nbib Format: NLM
Coronary venous noradrenaline during coronary angioplasty.

International journal of cardiology

McCance AJ, Forfar JC.
PMID: 1937987
Int J Cardiol. 1991 Oct;33(1):89-97. doi: 10.1016/0167-5273(91)90156-j.

Coronary venous and arterial noradrenaline concentrations were measured during percutaneous transluminal coronary angioplasty in 14 patients. Coronary venous noradrenaline did not increase significantly during balloon inflation but was increased during early reperfusion in patients undergoing left anterior descending angioplasty...

Cite
McCance AJ, Forfar JC. Coronary venous noradrenaline during coronary angioplasty. Int J Cardiol. 1991;33(1):89-97doi: 10.1016/0167-5273(91)90156-j.
McCance, A. J., & Forfar, J. C. (1991). Coronary venous noradrenaline during coronary angioplasty. International journal of cardiology, 33(1), 89-97. https://doi.org/10.1016/0167-5273(91)90156-j
McCance, A J, and Forfar, J C. "Coronary venous noradrenaline during coronary angioplasty." International journal of cardiology vol. 33,1 (1991): 89-97. doi: https://doi.org/10.1016/0167-5273(91)90156-j
McCance AJ, Forfar JC. Coronary venous noradrenaline during coronary angioplasty. Int J Cardiol. 1991 Oct;33(1):89-97. doi: 10.1016/0167-5273(91)90156-j. PMID: 1937987.
Copy Download .nbib Format: NLM
Effects of captopril on regional segment motion during acute coronary occlusion.

Clinical cardiology

Cosín J, Rivera M, Solaz J, Hernándiz A, Andrés F.
PMID: 1617821
Clin Cardiol. 1992 Jun;15(6):411-6. doi: 10.1002/clc.4960150604.
Free Article

The effects of captopril on myocardial segment function in different degrees of transient coronary occlusion were studied using ultrasonic dimension gauges in 15 open-chest dogs. The occlusion procedures (OP) were performed on the left anterior descending coronary artery (LAD)...

Cite
Cosín J, Rivera M, Solaz J, et al. Effects of captopril on regional segment motion during acute coronary occlusion. Clin Cardiol. 1992;15(6):411-6doi: 10.1002/clc.4960150604.
Cosín, J., Rivera, M., Solaz, J., Hernándiz, A., & Andrés, F. (1992). Effects of captopril on regional segment motion during acute coronary occlusion. Clinical cardiology, 15(6), 411-6. https://doi.org/10.1002/clc.4960150604
Cosín, J, et al. "Effects of captopril on regional segment motion during acute coronary occlusion." Clinical cardiology vol. 15,6 (1992): 411-6. doi: https://doi.org/10.1002/clc.4960150604
Cosín J, Rivera M, Solaz J, Hernándiz A, Andrés F. Effects of captopril on regional segment motion during acute coronary occlusion. Clin Cardiol. 1992 Jun;15(6):411-6. doi: 10.1002/clc.4960150604. PMID: 1617821.
Copy Download .nbib Format: NLM
Reversal of rest asynergy during exercise in patients with coronary artery disease.

Japanese heart journal

Konishi T, Koyama T, Aoki T, Futagami Y, Nakano T, Yamamuro M, Watanabe K.
PMID: 2810764
Jpn Heart J. 1989 Jul;30(4):459-70. doi: 10.1536/ihj.30.459.

The diagnosis of ischemic heart disease by radionuclide ventriculography (RNV) is performed on the basis of an abnormal response of the left ventricular ejection fraction and the occurrence, or aggravation, of regional wall motion abnormality during exercise. However, the...

Cite
Konishi T, Koyama T, Aoki T, et al. Reversal of rest asynergy during exercise in patients with coronary artery disease. Jpn Heart J. 1989;30(4):459-70doi: 10.1536/ihj.30.459.
Konishi, T., Koyama, T., Aoki, T., Futagami, Y., Nakano, T., Yamamuro, M., & Watanabe, K. (1989). Reversal of rest asynergy during exercise in patients with coronary artery disease. Japanese heart journal, 30(4), 459-70. https://doi.org/10.1536/ihj.30.459
Konishi, T, et al. "Reversal of rest asynergy during exercise in patients with coronary artery disease." Japanese heart journal vol. 30,4 (1989): 459-70. doi: https://doi.org/10.1536/ihj.30.459
Konishi T, Koyama T, Aoki T, Futagami Y, Nakano T, Yamamuro M, Watanabe K. Reversal of rest asynergy during exercise in patients with coronary artery disease. Jpn Heart J. 1989 Jul;30(4):459-70. doi: 10.1536/ihj.30.459. PMID: 2810764.
Copy Download .nbib Format: NLM
The coronary stress of skiing at high altitude.

Archives of internal medicine

Grover RF, Tucker CE, McGroarty SR, Travis RR.
PMID: 2353853
Arch Intern Med. 1990 Jun;150(6):1205-8.

Skiing, which may involve strenuous exercise in the cold at high altitude, could place considerable stress on the coronary circulation. To explore this possibility, we obtained by telemetry electrocardiograms on 149 men during recreational skiing at altitudes above 3100...

Cite
Grover RF, Tucker CE, McGroarty SR, et al. The coronary stress of skiing at high altitude. Arch Intern Med. 1990;150(6):1205-8
Grover, R. F., Tucker, C. E., McGroarty, S. R., & Travis, R. R. (1990). The coronary stress of skiing at high altitude. Archives of internal medicine, 150(6), 1205-8.
Grover, R F, et al. "The coronary stress of skiing at high altitude." Archives of internal medicine vol. 150,6 (1990): 1205-8.
Grover RF, Tucker CE, McGroarty SR, Travis RR. The coronary stress of skiing at high altitude. Arch Intern Med. 1990 Jun;150(6):1205-8. PMID: 2353853.
Copy Download .nbib Format: NLM
Interpretation of cardiac pathophysiology from pressure waveform analysis: aortic regurgitation.

Catheterization and cardiovascular diagnosis

Kern MJ, Aguirre FV.
PMID: 1617718
Cathet Cardiovasc Diagn. 1992 Jul;26(3):232-40. doi: 10.1002/ccd.1810260314.

No abstract available.

Cite
Kern MJ, Aguirre FV. Interpretation of cardiac pathophysiology from pressure waveform analysis: aortic regurgitation. Cathet Cardiovasc Diagn. 1992;26(3):232-40doi: 10.1002/ccd.1810260314.
Kern, M. J., & Aguirre, F. V. (1992). Interpretation of cardiac pathophysiology from pressure waveform analysis: aortic regurgitation. Catheterization and cardiovascular diagnosis, 26(3), 232-40. https://doi.org/10.1002/ccd.1810260314
Kern, M J, and Aguirre, F V. "Interpretation of cardiac pathophysiology from pressure waveform analysis: aortic regurgitation." Catheterization and cardiovascular diagnosis vol. 26,3 (1992): 232-40. doi: https://doi.org/10.1002/ccd.1810260314
Kern MJ, Aguirre FV. Interpretation of cardiac pathophysiology from pressure waveform analysis: aortic regurgitation. Cathet Cardiovasc Diagn. 1992 Jul;26(3):232-40. doi: 10.1002/ccd.1810260314. PMID: 1617718.
Copy Download .nbib Format: NLM
Structure and function of ion channels in the heart and the vascular system. May 8-11, 1990, Chicago, IL. Abstracts.

Journal of molecular and cellular cardiology

[No authors listed]
PMID: 1693404
J Mol Cell Cardiol. 1990 Mar;22:S1-49.

No abstract available.

Cite
Structure and function of ion channels in the heart and the vascular system. May 8-11, 1990, Chicago, IL. Abstracts. J Mol Cell Cardiol. 1990;22:S1-49
(1990). Structure and function of ion channels in the heart and the vascular system. May 8-11, 1990, Chicago, IL. Abstracts. Journal of molecular and cellular cardiology, 22S1-49.
"Structure and function of ion channels in the heart and the vascular system. May 8-11, 1990, Chicago, IL. Abstracts." Journal of molecular and cellular cardiology vol. 22 (1990): S1-49.
Structure and function of ion channels in the heart and the vascular system. May 8-11, 1990, Chicago, IL. Abstracts. J Mol Cell Cardiol. 1990 Mar;22:S1-49. PMID: 1693404.
Copy Download .nbib Format: NLM
Investigation of impaired coronary vasodilator reserve in the guinea pig heart with pressure induced hypertrophy.

European heart journal

O'Gorman DJ, Thomas P, Turner MA, Sheridan DJ.
PMID: 1535590
Eur Heart J. 1992 May;13(5):697-703. doi: 10.1093/oxfordjournals.eurheartj.a060237.

Impaired coronary reserve and increased minimal coronary resistance have been documented in several animal models of left ventricular hypertrophy. There is controversy whether the increased minimal coronary resistance is due to vascular or extravascular causes. To test the hypothesis...

Cite
O'Gorman DJ, Thomas P, Turner MA, et al. Investigation of impaired coronary vasodilator reserve in the guinea pig heart with pressure induced hypertrophy. Eur Heart J. 1992;13(5):697-703doi: 10.1093/oxfordjournals.eurheartj.a060237.
O'Gorman, D. J., Thomas, P., Turner, M. A., & Sheridan, D. J. (1992). Investigation of impaired coronary vasodilator reserve in the guinea pig heart with pressure induced hypertrophy. European heart journal, 13(5), 697-703. https://doi.org/10.1093/oxfordjournals.eurheartj.a060237
O'Gorman, D J, et al. "Investigation of impaired coronary vasodilator reserve in the guinea pig heart with pressure induced hypertrophy." European heart journal vol. 13,5 (1992): 697-703. doi: https://doi.org/10.1093/oxfordjournals.eurheartj.a060237
O'Gorman DJ, Thomas P, Turner MA, Sheridan DJ. Investigation of impaired coronary vasodilator reserve in the guinea pig heart with pressure induced hypertrophy. Eur Heart J. 1992 May;13(5):697-703. doi: 10.1093/oxfordjournals.eurheartj.a060237. PMID: 1535590.
Copy Download .nbib Format: NLM
Early and late angiographic findings of the "no-reflow" phenomenon following direct angioplasty as primary treatment for acute myocardial infarction.

American heart journal

Feld H, Lichstein E, Schachter J, Shani J.
PMID: 1539531
Am Heart J. 1992 Mar;123(3):782-4. doi: 10.1016/0002-8703(92)90520-6.

No abstract available.

Cite
Feld H, Lichstein E, Schachter J, et al. Early and late angiographic findings of the "no-reflow" phenomenon following direct angioplasty as primary treatment for acute myocardial infarction. Am Heart J. 1992;123(3):782-4doi: 10.1016/0002-8703(92)90520-6.
Feld, H., Lichstein, E., Schachter, J., & Shani, J. (1992). Early and late angiographic findings of the "no-reflow" phenomenon following direct angioplasty as primary treatment for acute myocardial infarction. American heart journal, 123(3), 782-4. https://doi.org/10.1016/0002-8703(92)90520-6
Feld, H, et al. "Early and late angiographic findings of the "no-reflow" phenomenon following direct angioplasty as primary treatment for acute myocardial infarction." American heart journal vol. 123,3 (1992): 782-4. doi: https://doi.org/10.1016/0002-8703(92)90520-6
Feld H, Lichstein E, Schachter J, Shani J. Early and late angiographic findings of the "no-reflow" phenomenon following direct angioplasty as primary treatment for acute myocardial infarction. Am Heart J. 1992 Mar;123(3):782-4. doi: 10.1016/0002-8703(92)90520-6. PMID: 1539531.
Copy Download .nbib Format: NLM
Simulation of coronary circulation with special regard to the venous bed and coronary sinus occlusion.

Journal of biomedical engineering

Schreiner W, Neumann F, Mohl W.
PMID: 2214733
J Biomed Eng. 1990 Sep;12(5):429-43. doi: 10.1016/0141-5425(90)90029-m.

The vascular beds of the left circumflex and the left anterior descending coronary arteries are modelled by means of coupled differential equations that consider an arterial, a capillary and a venous section. In a stepwise procedure, experimental data from...

Cite
Schreiner W, Neumann F, Mohl W. Simulation of coronary circulation with special regard to the venous bed and coronary sinus occlusion. J Biomed Eng. 1990;12(5):429-43doi: 10.1016/0141-5425(90)90029-m.
Schreiner, W., Neumann, F., & Mohl, W. (1990). Simulation of coronary circulation with special regard to the venous bed and coronary sinus occlusion. Journal of biomedical engineering, 12(5), 429-43. https://doi.org/10.1016/0141-5425(90)90029-m
Schreiner, W, et al. "Simulation of coronary circulation with special regard to the venous bed and coronary sinus occlusion." Journal of biomedical engineering vol. 12,5 (1990): 429-43. doi: https://doi.org/10.1016/0141-5425(90)90029-m
Schreiner W, Neumann F, Mohl W. Simulation of coronary circulation with special regard to the venous bed and coronary sinus occlusion. J Biomed Eng. 1990 Sep;12(5):429-43. doi: 10.1016/0141-5425(90)90029-m. PMID: 2214733.
Copy Download .nbib Format: NLM
Reperfusion injury in ischemic myocardium: protective effect of controlled reperfusion.

The American journal of cardiovascular pathology

Mrak RE, Carry MM, Murphy ML, Peng CF, Straub KD.
PMID: 2095828
Am J Cardiovasc Pathol. 1990;3(3):217-24.

Restoration of coronary artery flow following a period of ischemia often results in further ultrastructural damage to cardiac fibers, a phenomenon known as reperfusion injury. We have compared the ultrastructural effects of uncontrolled reperfusion in vivo of ischemic pig...

Cite
Mrak RE, Carry MM, Murphy ML, et al. Reperfusion injury in ischemic myocardium: protective effect of controlled reperfusion. Am J Cardiovasc Pathol. 1990;3(3):217-24
Mrak, R. E., Carry, M. M., Murphy, M. L., Peng, C. F., & Straub, K. D. (1990). Reperfusion injury in ischemic myocardium: protective effect of controlled reperfusion. The American journal of cardiovascular pathology, 3(3), 217-24.
Mrak, R E, et al. "Reperfusion injury in ischemic myocardium: protective effect of controlled reperfusion." The American journal of cardiovascular pathology vol. 3,3 (1990): 217-24.
Mrak RE, Carry MM, Murphy ML, Peng CF, Straub KD. Reperfusion injury in ischemic myocardium: protective effect of controlled reperfusion. Am J Cardiovasc Pathol. 1990;3(3):217-24. PMID: 2095828.
Copy Download .nbib Format: NLM
[Pattern of the coronary endothelium in the human].

Zeitschrift fur Kardiologie

Bürrig KF, Hort W.
PMID: 2694658
Z Kardiol. 1989;78:100-4.

The ultrastructural morphology of the endothelium of the normal human coronary arteries is equal to the endothelial cells of other locations in the macrovascular system. However, the patterns of the endothelial nuclei and the endothelial cell shape are consistent...

Cite
Bürrig KF, Hort W. Das koronare Endothelmuster beim Menschen. [Pattern of the coronary endothelium in the human]. Z Kardiol. 1989;78:100-4
Bürrig, K. F., & Hort, W. (1989). Das koronare Endothelmuster beim Menschen. [Pattern of the coronary endothelium in the human]. Zeitschrift fur Kardiologie, 78100-4.
Bürrig, K F, and Hort, W. "Das koronare Endothelmuster beim Menschen." [Pattern of the coronary endothelium in the human]. Zeitschrift fur Kardiologie vol. 78 (1989): 100-4.
Bürrig KF, Hort W. Das koronare Endothelmuster beim Menschen. [Pattern of the coronary endothelium in the human]. Z Kardiol. 1989;78:100-4. German. PMID: 2694658.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 7492 entries