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Evaluation of the iodine concentration in serum and urine of hypothyroid males using an inexpensive and rapid method

STEM and males

Arain MB.
GSID: nLGa7rvLIAoJ
GA Kandhro, TG Kazi, MB Arain - Pakistan Journal of Analytical & …, 2009 - pjaec.pk

… Antioxidant properties of iodine, which stem from the series of redox reactions underlying iodination of tyrosine leading to formation of thyroid hormones, allow it to reduce the damage, …

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Kandhro, G. A., Kazi, T. G., & Arain, M. B. (2009). Evaluation of the iodine concentration in serum and urine of hypothyroid males using an inexpensive and rapid method. Pakistan Journal of Analytical & Environmental Chemistry, 10(1 & 2), 9.
Kandhro, Ghulam Abbas, Tasneem Gul Kazi, and Mohammad Balal Arain. "Evaluation of the iodine concentration in serum and urine of hypothyroid males using an inexpensive and rapid method." Pakistan Journal of Analytical & Environmental Chemistry 10.1 & 2 (2009): 9.
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Harmine treatment enhances short-term memory in old rats: Dissociation of cognition and the ability to perform the procedural requirements of maze testing

Louis Stokes Alliances for Minority Participation AND Program Evaluation

Mennenga SE.
GSID: _4EMxi7k_lYJ
SE Mennenga, JE Gerson, T Dunckley… - Physiology & …, 2015 - Elsevier

Harmine is a naturally occurring monoamine oxidase inhibitor that has recently been shown to selectively inhibit the dual-specificity tyrosine-(Y)-phosphorylation-regulated kinase 1A (DYRK1A). We investigated the cognitive effects of 1 mg (low) Harmine and 5 mg (high) Harmine using the...

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Mennenga, S. E., Gerson, J. E., Dunckley, T., & Bimonte-Nelson, H. A. (2015). Harmine treatment enhances short-term memory in old rats: Dissociation of cognition and the ability to perform the procedural requirements of maze testing. Physiology & behavior, 138, 260-265.
Mennenga, Sarah E., et al. "Harmine treatment enhances short-term memory in old rats: Dissociation of cognition and the ability to perform the procedural requirements of maze testing." Physiology & behavior 138 (2015): 260-265.
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ASL metabolically regulates tyrosine hydroxylase in the nucleus locus coeruleus

STEM and boys

[No authors listed]
GSID: xk3aFfAV8REJ
…, R Mazkereth, FD Alber, T Babikian, H Bender, C Boys… - Cell reports, 2019 - Elsevier

Patients with germline mutations in the urea-cycle enzyme argininosuccinate lyase (ASL) are at risk for developing neurobehavioral and cognitive deficits. We find that ASL is …

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Lerner, S., Anderzhanova, E., Verbitsky, S., Eilam, R., Kuperman, Y., Tsoory, M., ... & Erez, A. (2019). ASL metabolically regulates tyrosine hydroxylase in the nucleus locus coeruleus. Cell reports, 29(8), 2144-2153.
Lerner, Shaul, et al. "ASL metabolically regulates tyrosine hydroxylase in the nucleus locus coeruleus." Cell reports 29.8 (2019): 2144-2153.
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Economic benefits of adequate molecular monitoring in patients with chronic myelogenous leukemia

community settings

[No authors listed]
GSID: wFZgrPMrl1gJ
A Guerin, L Chen, K Dea, EQ Wu… - Journal of Medical …, 2014 - Taylor & Francis

Objective: Molecular monitoring of chronic myeloid leukemia (CML) has been associated with improved clinical outcomes during tyrosine kinase inhibitor therapy (TKI), yet recent …

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Guerin, A., Chen, L., Dea, K., Wu, E. Q., & Goldberg, S. L. (2014). Economic benefits of adequate molecular monitoring in patients with chronic myelogenous leukemia. Journal of Medical Economics, 17(2), 89-98.
Guerin, Annie, et al. "Economic benefits of adequate molecular monitoring in patients with chronic myelogenous leukemia." Journal of Medical Economics 17.2 (2014): 89-98.
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… : Current address of Dr. Karin M. Kirschner is at Johannes-Müller Institut fu̇r Physiologie, Charité-Universitätsmedizin Berlin, Campus Charité-Mitte, Tucholskystrasse …

science campus

[No authors listed]
GSID: 1s73hdO2Rq8J
KM Kirschner, K Baltensperger - Molecular cancer research, 2003 - AACR

Chronic myeloid leukemia is characterized by the Philadelphia chromosome translocation that causes expression of Bcr-Abl, a deregulated tyrosine kinase. Imatinib mesylate (STI571, Gleevec), a therapeutically used inhibitor of Bcr-Abl, causes apoptosis of Bcr-Abl-positive cells…

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Kirschner, K. M., & Baltensperger, K. (2003). Erythropoietin Promotes Resistance Against the Abl Tyrosine Kinase Inhibitor Imatinib (STI571) in K562 Human Leukemia Cells11Swiss National Science Foundation grant 31-059124 and Swiss Cancer League grant SKL 778-2-1999. Note: Current address of Dr. Karin M. Kirschner is at Johannes-Müller Institut fu̇r Physiologie, Charité-Universitätsmedizin Berlin, Campus Charité-Mitte, Tucholskystrasse 2, 10117 Berlin, Germany. Molecular cancer research, 1(13), 970-980.
Kirschner, Karin M., and Kurt Baltensperger. "Erythropoietin Promotes Resistance Against the Abl Tyrosine Kinase Inhibitor Imatinib (STI571) in K562 Human Leukemia Cells11Swiss National Science Foundation grant 31-059124 and Swiss Cancer League grant SKL 778-2-1999. Note: Current address of Dr. Karin M. Kirschner is at Johannes-Müller Institut fu̇r Physiologie, Charité-Universitätsmedizin Berlin, Campus Charité-Mitte, Tucholskystrasse 2, 10117 Berlin, Germany." Molecular cancer research 1.13 (2003): 970-980.
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Exercise reverses the effects of early life stress on orexin cell reactivity in male but not female rats.

Frontiers in behavioral neuroscience

James MH, Campbell EJ, Walker FR, Smith DW, Richardson HN, Hodgson DM, Dayas CV.
PMID: 25100956
Front Behav Neurosci. 2014 Jul 23;8:244. doi: 10.3389/fnbeh.2014.00244. eCollection 2014.

Early life stress (ELS) is a known antecedent for the development of mood disorders such as depression. Orexin neurons drive arousal and motivated behaviors in response to stress. We tested the hypothesis that ELS alters orexin system function and...

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James MH, Campbell EJ, Walker FR, et al. Exercise reverses the effects of early life stress on orexin cell reactivity in male but not female rats. Front Behav Neurosci. 2014;8:244doi: 10.3389/fnbeh.2014.00244.
James, M. H., Campbell, E. J., Walker, F. R., Smith, D. W., Richardson, H. N., Hodgson, D. M., & Dayas, C. V. (2014). Exercise reverses the effects of early life stress on orexin cell reactivity in male but not female rats. Frontiers in behavioral neuroscience, 8244. https://doi.org/10.3389/fnbeh.2014.00244
James, Morgan H, et al. "Exercise reverses the effects of early life stress on orexin cell reactivity in male but not female rats." Frontiers in behavioral neuroscience vol. 8 (2014): 244. doi: https://doi.org/10.3389/fnbeh.2014.00244
James MH, Campbell EJ, Walker FR, Smith DW, Richardson HN, Hodgson DM, Dayas CV. Exercise reverses the effects of early life stress on orexin cell reactivity in male but not female rats. Front Behav Neurosci. 2014 Jul 23;8:244. doi: 10.3389/fnbeh.2014.00244. eCollection 2014. PMID: 25100956; PMCID: PMC4107856.
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Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics.

Frontiers in microbiology

Glowinski F, Holland C, Thiede B, Jungblut PR, Meyer TF.
PMID: 25101063
Front Microbiol. 2014 Jul 18;5:356. doi: 10.3389/fmicb.2014.00356. eCollection 2014.

Helicobacter pylori is a Gram-negative bacterial pathogen colonizing the human stomach. Infection with H. pylori causes chronic inflammation of the gastric mucosa and may lead to peptic ulceration and/or gastric cancer. A major virulence determinant of H. pylori is...

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Glowinski F, Holland C, Thiede B, et al. Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics. Front Microbiol. 2014;5:356doi: 10.3389/fmicb.2014.00356.
Glowinski, F., Holland, C., Thiede, B., Jungblut, P. R., & Meyer, T. F. (2014). Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics. Frontiers in microbiology, 5356. https://doi.org/10.3389/fmicb.2014.00356
Glowinski, Frithjof, et al. "Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics." Frontiers in microbiology vol. 5 (2014): 356. doi: https://doi.org/10.3389/fmicb.2014.00356
Glowinski F, Holland C, Thiede B, Jungblut PR, Meyer TF. Analysis of T4SS-induced signaling by H. pylori using quantitative phosphoproteomics. Front Microbiol. 2014 Jul 18;5:356. doi: 10.3389/fmicb.2014.00356. eCollection 2014. PMID: 25101063; PMCID: PMC4102909.
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Receptor tyrosine kinases and schistosome reproduction: new targets for chemotherapy.

Frontiers in genetics

Morel M, Vanderstraete M, Hahnel S, Grevelding CG, Dissous C.
PMID: 25101117
Front Genet. 2014 Jul 18;5:238. doi: 10.3389/fgene.2014.00238. eCollection 2014.

Schistosome parasites still represent a serious public health concern and a major economic problem in developing countries. Pathology of schistosomiasis is mainly due to massive egg production by these parasites and to inflammatory responses raised against the eggs which...

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Morel M, Vanderstraete M, Hahnel S, et al. Receptor tyrosine kinases and schistosome reproduction: new targets for chemotherapy. Front Genet. 2014;5:238doi: 10.3389/fgene.2014.00238.
Morel, M., Vanderstraete, M., Hahnel, S., Grevelding, C. G., & Dissous, C. (2014). Receptor tyrosine kinases and schistosome reproduction: new targets for chemotherapy. Frontiers in genetics, 5238. https://doi.org/10.3389/fgene.2014.00238
Morel, Marion, et al. "Receptor tyrosine kinases and schistosome reproduction: new targets for chemotherapy." Frontiers in genetics vol. 5 (2014): 238. doi: https://doi.org/10.3389/fgene.2014.00238
Morel M, Vanderstraete M, Hahnel S, Grevelding CG, Dissous C. Receptor tyrosine kinases and schistosome reproduction: new targets for chemotherapy. Front Genet. 2014 Jul 18;5:238. doi: 10.3389/fgene.2014.00238. eCollection 2014. PMID: 25101117; PMCID: PMC4102852.
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Discovery of BRM Targeted Therapies: Novel Reactivation of an Anti-cancer Gene.

Letters in drug design & discovery

Gramling S, Reisman D.
PMID: 23565070
Lett Drug Des Discov. 2011 Jan 01;8(1):93-99. doi: 10.2174/157018011793663840.

Drug discovery in the field of oncology has been advanced mainly through the targeting of receptor tyrosine kinases. Both antibodies and small molecule inhibitors have been found to have successful applications in blocking the proliferative functions of these cell...

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Gramling S, Reisman D. Discovery of BRM Targeted Therapies: Novel Reactivation of an Anti-cancer Gene. Lett Drug Des Discov. 2011;8(1):93-99doi: 10.2174/157018011793663840.
Gramling, S., & Reisman, D. (2011). Discovery of BRM Targeted Therapies: Novel Reactivation of an Anti-cancer Gene. Letters in drug design & discovery, 8(1), 93-99. https://doi.org/10.2174/157018011793663840
Gramling, Sarah, and Reisman, David. "Discovery of BRM Targeted Therapies: Novel Reactivation of an Anti-cancer Gene." Letters in drug design & discovery vol. 8,1 (2011): 93-99. doi: https://doi.org/10.2174/157018011793663840
Gramling S, Reisman D. Discovery of BRM Targeted Therapies: Novel Reactivation of an Anti-cancer Gene. Lett Drug Des Discov. 2011 Jan 01;8(1):93-99. doi: 10.2174/157018011793663840. PMID: 23565070; PMCID: PMC3615482.
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Targeting the HGF-cMET Axis in Hepatocellular Carcinoma.

International journal of hepatology

Venepalli NK, Goff L.
PMID: 23606971
Int J Hepatol. 2013;2013:341636. doi: 10.1155/2013/341636. Epub 2013 Mar 31.

Under normal physiological conditions, the hepatocyte growth factor (HGF) and its receptor, the MET transmembrane tyrosine kinase (cMET), are involved in embryogenesis, morphogenesis, and wound healing. The HGF-cMET axis promotes cell survival, proliferation, migration, and invasion via modulation of...

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Venepalli NK, Goff L. Targeting the HGF-cMET Axis in Hepatocellular Carcinoma. Int J Hepatol. 2013;2013:341636doi: 10.1155/2013/341636.
Venepalli, N. K., & Goff, L. (2013). Targeting the HGF-cMET Axis in Hepatocellular Carcinoma. International journal of hepatology, 2013341636. https://doi.org/10.1155/2013/341636
Venepalli, Neeta K, and Goff, Laura. "Targeting the HGF-cMET Axis in Hepatocellular Carcinoma." International journal of hepatology vol. 2013 (2013): 341636. doi: https://doi.org/10.1155/2013/341636
Venepalli NK, Goff L. Targeting the HGF-cMET Axis in Hepatocellular Carcinoma. Int J Hepatol. 2013;2013:341636. doi: 10.1155/2013/341636. Epub 2013 Mar 31. PMID: 23606971; PMCID: PMC3626399.
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Evolution of the FGF Gene Family.

International journal of evolutionary biology

Oulion S, Bertrand S, Escriva H.
PMID: 22919541
Int J Evol Biol. 2012;2012:298147. doi: 10.1155/2012/298147. Epub 2012 Aug 07.

Fibroblast Growth Factors (FGFs) are small proteins generally secreted, acting through binding to transmembrane tyrosine kinase receptors (FGFRs). Activation of FGFRs triggers several cytoplasmic cascades leading to the modification of cell behavior. FGFs play critical roles in a variety...

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Oulion S, Bertrand S, Escriva H. Evolution of the FGF Gene Family. Int J Evol Biol. 2012;2012:298147doi: 10.1155/2012/298147.
Oulion, S., Bertrand, S., & Escriva, H. (2012). Evolution of the FGF Gene Family. International journal of evolutionary biology, 2012298147. https://doi.org/10.1155/2012/298147
Oulion, Silvan, et al. "Evolution of the FGF Gene Family." International journal of evolutionary biology vol. 2012 (2012): 298147. doi: https://doi.org/10.1155/2012/298147
Oulion S, Bertrand S, Escriva H. Evolution of the FGF Gene Family. Int J Evol Biol. 2012;2012:298147. doi: 10.1155/2012/298147. Epub 2012 Aug 07. PMID: 22919541; PMCID: PMC3420111.
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Profile of pacritinib and its potential in the treatment of hematologic disorders.

Journal of blood medicine

Hatzimichael E, Tsolas E, Briasoulis E.
PMID: 25170285
J Blood Med. 2014 Aug 19;5:143-52. doi: 10.2147/JBM.S51253. eCollection 2014.

Pacritinib (previously known as SB-1518) is an innovative selective inhibitor of Janus kinase 2 and FMS-related tyrosine kinase 3 providing potential in the treatment of hematological malignancies such as myeloproliferative neoplasias, acute myeloid leukemia, and various lymphomas. Pacritinib has...

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Hatzimichael E, Tsolas E, Briasoulis E. Profile of pacritinib and its potential in the treatment of hematologic disorders. J Blood Med. 2014;5:143-52doi: 10.2147/JBM.S51253.
Hatzimichael, E., Tsolas, E., & Briasoulis, E. (2014). Profile of pacritinib and its potential in the treatment of hematologic disorders. Journal of blood medicine, 5143-52. https://doi.org/10.2147/JBM.S51253
Hatzimichael, Eleftheria, et al. "Profile of pacritinib and its potential in the treatment of hematologic disorders." Journal of blood medicine vol. 5 (2014): 143-52. doi: https://doi.org/10.2147/JBM.S51253
Hatzimichael E, Tsolas E, Briasoulis E. Profile of pacritinib and its potential in the treatment of hematologic disorders. J Blood Med. 2014 Aug 19;5:143-52. doi: 10.2147/JBM.S51253. eCollection 2014. PMID: 25170285; PMCID: PMC4145824.
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Showing 1 to 12 of 16586 entries