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Showing 1 to 12 of 1801 entries
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Urinary monocyte chemotactic protein-1 and transforming growth factor-β in systemic lupus erythematosus.

Indian journal of nephrology

Torabinejad S, Mardani R, Habibagahi Z, Roozbeh J, Khajedehi P, Pakfetrat M, Banihashemi MA, Banihashemi SJ.
PMID: 22279336
Indian J Nephrol. 2012 Jan;22(1):5-12. doi: 10.4103/0971-4065.91179.

The purpose of this investigation was to assess the correlation of two biomarkers with the occurrence of renal flares in systemic lupus erythematosus (SLE). Urine levels of monocyte chemotactic protein-1 (MCP-1) and transforming growth factor beta (TGF-β) were measured...

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Torabinejad S, Mardani R, Habibagahi Z, et al. Urinary monocyte chemotactic protein-1 and transforming growth factor-β in systemic lupus erythematosus. Indian J Nephrol. 2012;22(1):5-12doi: 10.4103/0971-4065.91179.
Torabinejad, S., Mardani, R., Habibagahi, Z., Roozbeh, J., Khajedehi, P., Pakfetrat, M., Banihashemi, M. A., & Banihashemi, S. J. (2012). Urinary monocyte chemotactic protein-1 and transforming growth factor-β in systemic lupus erythematosus. Indian journal of nephrology, 22(1), 5-12. https://doi.org/10.4103/0971-4065.91179
Torabinejad, S, et al. "Urinary monocyte chemotactic protein-1 and transforming growth factor-β in systemic lupus erythematosus." Indian journal of nephrology vol. 22,1 (2012): 5-12. doi: https://doi.org/10.4103/0971-4065.91179
Torabinejad S, Mardani R, Habibagahi Z, Roozbeh J, Khajedehi P, Pakfetrat M, Banihashemi MA, Banihashemi SJ. Urinary monocyte chemotactic protein-1 and transforming growth factor-β in systemic lupus erythematosus. Indian J Nephrol. 2012 Jan;22(1):5-12. doi: 10.4103/0971-4065.91179. PMID: 22279336; PMCID: PMC3263065.
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Severe open angle glaucoma in hereditary hemorrhagic telangiectasia.

Clinical case reports

Kuchtey RW, Naratadam GT, Kuchtey J.
PMID: 26401274
Clin Case Rep. 2015 Sep;3(9):725-7. doi: 10.1002/ccr3.324. Epub 2015 Jun 18.

Hereditary hemorrhagic telangiectasia (HHT) is an autosomal dominant disease. Conjunctival telangiectasias and retinal vascular malformations are known ocular manifestations. We report here the first case of open angle glaucoma in a patient with HHT caused by a nonsense mutation,...

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Kuchtey RW, Naratadam GT, Kuchtey J. Severe open angle glaucoma in hereditary hemorrhagic telangiectasia. Clin Case Rep. 2015;3(9):725-7doi: 10.1002/ccr3.324.
Kuchtey, R. W., Naratadam, G. T., & Kuchtey, J. (2015). Severe open angle glaucoma in hereditary hemorrhagic telangiectasia. Clinical case reports, 3(9), 725-7. https://doi.org/10.1002/ccr3.324
Kuchtey, Rachel W, et al. "Severe open angle glaucoma in hereditary hemorrhagic telangiectasia." Clinical case reports vol. 3,9 (2015): 725-7. doi: https://doi.org/10.1002/ccr3.324
Kuchtey RW, Naratadam GT, Kuchtey J. Severe open angle glaucoma in hereditary hemorrhagic telangiectasia. Clin Case Rep. 2015 Sep;3(9):725-7. doi: 10.1002/ccr3.324. Epub 2015 Jun 18. PMID: 26401274; PMCID: PMC4574785.
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An Approach to In Vitro Manufacturing of Hypertrophic Cartilage Matrix for Bone Repair.

Bioengineering (Basel, Switzerland)

Quang Le B, Van Blitterswijk C, De Boer J.
PMID: 28952514
Bioengineering (Basel). 2017 Apr 20;4(2). doi: 10.3390/bioengineering4020035.

Devitalized hypertrophic cartilage matrix (DCM) is an attractive concept for an off-the-shelf bone graft substitute. Upon implantation, DCM can trigger the natural endochondral ossification process, but only when the hypertrophic cartilage matrix has been reconstituted correctly. In vivo hypertrophic...

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Quang Le B, Van Blitterswijk C, De Boer J. An Approach to In Vitro Manufacturing of Hypertrophic Cartilage Matrix for Bone Repair. Bioengineering (Basel). 2017;4(2)doi: 10.3390/bioengineering4020035.
Quang Le, B., Van Blitterswijk, C., & De Boer, J. (2017). An Approach to In Vitro Manufacturing of Hypertrophic Cartilage Matrix for Bone Repair. Bioengineering (Basel, Switzerland), 4(2), . https://doi.org/10.3390/bioengineering4020035
Quang Le, Bach, et al. "An Approach to In Vitro Manufacturing of Hypertrophic Cartilage Matrix for Bone Repair." Bioengineering (Basel, Switzerland) vol. 4,2 (2017). doi: https://doi.org/10.3390/bioengineering4020035
Quang Le B, Van Blitterswijk C, De Boer J. An Approach to In Vitro Manufacturing of Hypertrophic Cartilage Matrix for Bone Repair. Bioengineering (Basel). 2017 Apr 20;4(2). doi: 10.3390/bioengineering4020035. PMID: 28952514; PMCID: PMC5590482.
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Conjunctival TGF-B Level in Primary Augmented Trabeculectomy.

The open ophthalmology journal

Ng GF, Raihan IS, Azhany Y, Maraina CH, Banumathi KG, Liza-Sharmini T.
PMID: 26401171
Open Ophthalmol J. 2015 Jul 31;9:136-44. doi: 10.2174/1874364101509010136. eCollection 2015.

PURPOSE: To compare the levels of conjunctival transforming growth factor beta (TGF-β) between glaucoma and control patients and to determine conjunctival TGF-β levels before and 3 months after augmented primary trabeculectomy.METHODS: Patients with primary open angle glaucoma (POAG) or...

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Ng GF, Raihan IS, Azhany Y, et al. Conjunctival TGF-B Level in Primary Augmented Trabeculectomy. Open Ophthalmol J. 2015;9:136-44doi: 10.2174/1874364101509010136.
Ng, G. F., Raihan, I. S., Azhany, Y., Maraina, C. H., Banumathi, K. G., & Liza-Sharmini, T. (2015). Conjunctival TGF-B Level in Primary Augmented Trabeculectomy. The open ophthalmology journal, 9136-44. https://doi.org/10.2174/1874364101509010136
Ng, Guan Fook, et al. "Conjunctival TGF-B Level in Primary Augmented Trabeculectomy." The open ophthalmology journal vol. 9 (2015): 136-44. doi: https://doi.org/10.2174/1874364101509010136
Ng GF, Raihan IS, Azhany Y, Maraina CH, Banumathi KG, Liza-Sharmini T. Conjunctival TGF-B Level in Primary Augmented Trabeculectomy. Open Ophthalmol J. 2015 Jul 31;9:136-44. doi: 10.2174/1874364101509010136. eCollection 2015. PMID: 26401171; PMCID: PMC4578138.
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Inhibitory effects of Doenjang, Korean traditional fermented soybean paste, on oxidative stress and inflammation in adipose tissue of mice fed a high-fat diet.

Nutrition research and practice

Nam YR, Won SB, Chung YS, Kwak CS, Kwon YH.
PMID: 26060534
Nutr Res Pract. 2015 Jun;9(3):235-41. doi: 10.4162/nrp.2015.9.3.235. Epub 2015 May 06.

BACKGROUND/OBJECTIVES: Doenjang, Korean traditional fermented soybean paste has been reported to have an anti-obesity effect. Because adipose tissue is considered a major source of inflammatory signals, we investigated the protective effects of Doenjang and steamed soybean on oxidative stress...

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Nam YR, Won SB, Chung YS, et al. Inhibitory effects of Doenjang, Korean traditional fermented soybean paste, on oxidative stress and inflammation in adipose tissue of mice fed a high-fat diet. Nutr Res Pract. 2015;9(3):235-41doi: 10.4162/nrp.2015.9.3.235.
Nam, Y. R., Won, S. B., Chung, Y. S., Kwak, C. S., & Kwon, Y. H. (2015). Inhibitory effects of Doenjang, Korean traditional fermented soybean paste, on oxidative stress and inflammation in adipose tissue of mice fed a high-fat diet. Nutrition research and practice, 9(3), 235-41. https://doi.org/10.4162/nrp.2015.9.3.235
Nam, Ye Rim, et al. "Inhibitory effects of Doenjang, Korean traditional fermented soybean paste, on oxidative stress and inflammation in adipose tissue of mice fed a high-fat diet." Nutrition research and practice vol. 9,3 (2015): 235-41. doi: https://doi.org/10.4162/nrp.2015.9.3.235
Nam YR, Won SB, Chung YS, Kwak CS, Kwon YH. Inhibitory effects of Doenjang, Korean traditional fermented soybean paste, on oxidative stress and inflammation in adipose tissue of mice fed a high-fat diet. Nutr Res Pract. 2015 Jun;9(3):235-41. doi: 10.4162/nrp.2015.9.3.235. Epub 2015 May 06. PMID: 26060534; PMCID: PMC4460054.
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Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses.

World journal of cardiology

Sun L, Sucosky P.
PMID: 26131338
World J Cardiol. 2015 Jun 26;7(6):331-43. doi: 10.4330/wjc.v7.i6.331.

AIM: To explore ex vivo the role of bone morphogenetic protein-4 (BMP-4) and transforming growth factor-beta1 (TGF-β1) in acute valvular response to fluid shear stress (FSS) abnormalities.METHODS: Porcine valve leaflets were subjected ex vivo to physiologic FSS, supra-physiologic FSS...

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Sun L, Sucosky P. Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World J Cardiol. 2015;7(6):331-43doi: 10.4330/wjc.v7.i6.331.
Sun, L., & Sucosky, P. (2015). Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World journal of cardiology, 7(6), 331-43. https://doi.org/10.4330/wjc.v7.i6.331
Sun, Ling, and Sucosky, Philippe. "Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses." World journal of cardiology vol. 7,6 (2015): 331-43. doi: https://doi.org/10.4330/wjc.v7.i6.331
Sun L, Sucosky P. Bone morphogenetic protein-4 and transforming growth factor-beta1 mechanisms in acute valvular response to supra-physiologic hemodynamic stresses. World J Cardiol. 2015 Jun 26;7(6):331-43. doi: 10.4330/wjc.v7.i6.331. PMID: 26131338; PMCID: PMC4478568.
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Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis.

Chinese medicine

Duval F, Moreno-Cuevas JE, González-Garza MT, Rodríguez-Montalvo C, Cruz-Vega DE.
PMID: 25606051
Chin Med. 2014 Dec 24;9(1):27. doi: 10.1186/s13020-014-0027-4. eCollection 2014.

During chronic liver injury, hepatic stellate cells (HSC) are activated and proliferate, which causes excessive extracellular matrix (ECM) deposition, leading to scar formation and fibrosis. Medicinal plants are gaining popularity as antifibrotic agents, and are often safe, cost-effective, and...

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Duval F, Moreno-Cuevas JE, González-Garza MT, et al. Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis. Chin Med. 2014;9(1):27doi: 10.1186/s13020-014-0027-4.
Duval, F., Moreno-Cuevas, J. E., González-Garza, M. T., Rodríguez-Montalvo, C., & Cruz-Vega, D. E. (2014). Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis. Chinese medicine, 9(1), 27. https://doi.org/10.1186/s13020-014-0027-4
Duval, Florent, et al. "Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis." Chinese medicine vol. 9,1 (2014): 27. doi: https://doi.org/10.1186/s13020-014-0027-4
Duval F, Moreno-Cuevas JE, González-Garza MT, Rodríguez-Montalvo C, Cruz-Vega DE. Protective mechanisms of medicinal plants targeting hepatic stellate cell activation and extracellular matrix deposition in liver fibrosis. Chin Med. 2014 Dec 24;9(1):27. doi: 10.1186/s13020-014-0027-4. eCollection 2014. PMID: 25606051; PMCID: PMC4299307.
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Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ.

Skeletal muscle

Lamarche É, Lala-Tabbert N, Gunanayagam A, St-Louis C, Wiper-Bergeron N.
PMID: 25878769
Skelet Muscle. 2015 Mar 18;5:8. doi: 10.1186/s13395-015-0032-z. eCollection 2015.

BACKGROUND: The effects of transforming growth factor-beta (TGFβ) are mediated by the transcription factors Smad2 and Smad3. During adult skeletal myogenesis, TGFβ signaling inhibits the differentiation of myoblasts, and this can be reversed by treatment with retinoic acid (RA)....

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Lamarche É, Lala-Tabbert N, Gunanayagam A, et al. Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ. Skelet Muscle. 2015;5:8doi: 10.1186/s13395-015-0032-z.
Lamarche, �. �., Lala-Tabbert, N., Gunanayagam, A., St-Louis, C., & Wiper-Bergeron, N. (2015). Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ. Skeletal muscle, 58. https://doi.org/10.1186/s13395-015-0032-z
Lamarche, Émilie, et al. "Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ." Skeletal muscle vol. 5 (2015): 8. doi: https://doi.org/10.1186/s13395-015-0032-z
Lamarche É, Lala-Tabbert N, Gunanayagam A, St-Louis C, Wiper-Bergeron N. Retinoic acid promotes myogenesis in myoblasts by antagonizing transforming growth factor-beta signaling via C/EBPβ. Skelet Muscle. 2015 Mar 18;5:8. doi: 10.1186/s13395-015-0032-z. eCollection 2015. PMID: 25878769; PMCID: PMC4397812.
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Serum Markers of Epithelial Mesenchymal Transition as Predictors of HCV-induced Liver Fibrosis, Cirrhosis and Hepatocellular Carcinoma.

Electronic physician

Zoheiry MM, Hasan SA, El-Ahwany E, Nagy FM, Taleb HA, Nosseir M, Magdy M, Meshaal S, El-Talkawy MD, Raafat I.
PMID: 26816590
Electron Physician. 2015 Dec 20;7(8):1626-37. doi: 10.19082/1626. eCollection 2015 Dec.

INTRODUCTION: Hepatitis C virus (HCV) is a major cause of chronic liver disease in Egypt, leading to hepatic fibrosis, liver cirrhosis (LC), and hepatocellular carcinoma (HCC). Liver fibrosis is characterized by excessive deposition of extracellular matrix (ECM). Newly-recognized pathogenic...

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Zoheiry MM, Hasan SA, El-Ahwany E, et al. Serum Markers of Epithelial Mesenchymal Transition as Predictors of HCV-induced Liver Fibrosis, Cirrhosis and Hepatocellular Carcinoma. Electron Physician. 2015;7(8):1626-37doi: 10.19082/1626.
Zoheiry, M. M., Hasan, S. A., El-Ahwany, E., Nagy, F. M., Taleb, H. A., Nosseir, M., Magdy, M., Meshaal, S., El-Talkawy, M. D., & Raafat, I. (2015). Serum Markers of Epithelial Mesenchymal Transition as Predictors of HCV-induced Liver Fibrosis, Cirrhosis and Hepatocellular Carcinoma. Electronic physician, 7(8), 1626-37. https://doi.org/10.19082/1626
Zoheiry, Mona M, et al. "Serum Markers of Epithelial Mesenchymal Transition as Predictors of HCV-induced Liver Fibrosis, Cirrhosis and Hepatocellular Carcinoma." Electronic physician vol. 7,8 (2015): 1626-37. doi: https://doi.org/10.19082/1626
Zoheiry MM, Hasan SA, El-Ahwany E, Nagy FM, Taleb HA, Nosseir M, Magdy M, Meshaal S, El-Talkawy MD, Raafat I. Serum Markers of Epithelial Mesenchymal Transition as Predictors of HCV-induced Liver Fibrosis, Cirrhosis and Hepatocellular Carcinoma. Electron Physician. 2015 Dec 20;7(8):1626-37. doi: 10.19082/1626. eCollection 2015 Dec. PMID: 26816590; PMCID: PMC4725417.
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The role of tumor necrosis factor-alpha -308 G/A and transforming growth factor-beta 1 -915 G/C polymorphisms in childhood idiopathic thrombocytopenic purpura.

Turkish journal of haematology : official journal of Turkish Society of Haematology

Okulu E, İleri T, Koşan Çulha V, Azık FM, Eğin Y, Uysal Z, Akar N.
PMID: 27264363
Turk J Haematol. 2011 Sep 05;28(3):170-5. doi: 10.5152/tjh.2011.50.

OBJECTIVE: To increase our understanding of the etiology of idiopathic thrombocytopenic purpura (ITP) some cytokine gene polymorphisms were analyzed for susceptibility to the disease. The aim of this study was to investigate the role of tumor necrosis factor-alpha (TNF-α)...

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Okulu E, İleri T, Koşan Çulha V, et al. Çocukluk çağı idiopatik trombositopenik purpura'da TNF-α-308G/A ve TGF-β1-915G/C polimorfizmlerinin rolü. The role of tumor necrosis factor-alpha -308 G/A and transforming growth factor-beta 1 -915 G/C polymorphisms in childhood idiopathic thrombocytopenic purpura. Turk J Haematol. 2011;28(3):170-5doi: 10.5152/tjh.2011.50.
Okulu, E., İleri, T., Koşan Çulha, V., Azık, F. M., Eğin, Y., Uysal, Z., & Akar, N. (2011). Çocukluk çağı idiopatik trombositopenik purpura'da TNF-α-308G/A ve TGF-β1-915G/C polimorfizmlerinin rolü. The role of tumor necrosis factor-alpha -308 G/A and transforming growth factor-beta 1 -915 G/C polymorphisms in childhood idiopathic thrombocytopenic purpura. Turkish journal of haematology : official journal of Turkish Society of Haematology, 28(3), 170-5. https://doi.org/10.5152/tjh.2011.50
Okulu, Emel, et al. "Çocukluk çağı idiopatik trombositopenik purpura'da TNF-α-308G/A ve TGF-β1-915G/C polimorfizmlerinin rolü." The role of tumor necrosis factor-alpha -308 G/A and transforming growth factor-beta 1 -915 G/C polymorphisms in childhood idiopathic thrombocytopenic purpura. Turkish journal of haematology : official journal of Turkish Society of Haematology vol. 28,3 (2011): 170-5. doi: https://doi.org/10.5152/tjh.2011.50
Okulu E, İleri T, Koşan Çulha V, Azık FM, Eğin Y, Uysal Z, Akar N. Çocukluk çağı idiopatik trombositopenik purpura'da TNF-α-308G/A ve TGF-β1-915G/C polimorfizmlerinin rolü. The role of tumor necrosis factor-alpha -308 G/A and transforming growth factor-beta 1 -915 G/C polymorphisms in childhood idiopathic thrombocytopenic purpura. Turk J Haematol. 2011 Sep 05;28(3):170-5. doi: 10.5152/tjh.2011.50. PMID: 27264363.
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BAMBI inhibits skin fibrosis in keloid through suppressing TGF-β1-induced hypernomic fibroblast cell proliferation and excessive accumulation of collagen I.

International journal of clinical and experimental medicine

Lin L, Wang Y, Liu W, Huang Y.
PMID: 26550247
Int J Clin Exp Med. 2015 Aug 15;8(8):13227-34. eCollection 2015.

Keloids are scars characterized by pathologically excessive dermal fibrosis and aberrant wound healing. Hypernomic growth of fibroblast cells and excessive accumulation of collagens, especially collagen I, made important contribution to keloid formation. Transforming growth factor-beta 1 (TGF-β1) was a...

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Lin L, Wang Y, Liu W, et al. BAMBI inhibits skin fibrosis in keloid through suppressing TGF-β1-induced hypernomic fibroblast cell proliferation and excessive accumulation of collagen I. Int J Clin Exp Med. 2015;8(8):13227-34
Lin, L., Wang, Y., Liu, W., & Huang, Y. (2015). BAMBI inhibits skin fibrosis in keloid through suppressing TGF-β1-induced hypernomic fibroblast cell proliferation and excessive accumulation of collagen I. International journal of clinical and experimental medicine, 8(8), 13227-34.
Lin, Lixin, et al. "BAMBI inhibits skin fibrosis in keloid through suppressing TGF-β1-induced hypernomic fibroblast cell proliferation and excessive accumulation of collagen I." International journal of clinical and experimental medicine vol. 8,8 (2015): 13227-34.
Lin L, Wang Y, Liu W, Huang Y. BAMBI inhibits skin fibrosis in keloid through suppressing TGF-β1-induced hypernomic fibroblast cell proliferation and excessive accumulation of collagen I. Int J Clin Exp Med. 2015 Aug 15;8(8):13227-34. eCollection 2015. PMID: 26550247; PMCID: PMC4612932.
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Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome.

Journal of neurogastroenterology and motility

Ratanasirintrawoot S, Israsena N.
PMID: 27184041
J Neurogastroenterol Motil. 2016 Jul 30;22(3):367-82. doi: 10.5056/jnm16023.

Irritable bowel syndrome is one of the most common functional gastrointestinal (GI) disorders that significantly impair quality of life in patients. Current available treatments are still not effective and the pathophysiology of this condition remains unclearly defined. Recently, research...

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Ratanasirintrawoot S, Israsena N. Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome. J Neurogastroenterol Motil. 2016;22(3):367-82doi: 10.5056/jnm16023.
Ratanasirintrawoot, S., & Israsena, N. (2016). Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome. Journal of neurogastroenterology and motility, 22(3), 367-82. https://doi.org/10.5056/jnm16023
Ratanasirintrawoot, Sutheera, and Israsena, Nipan. "Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome." Journal of neurogastroenterology and motility vol. 22,3 (2016): 367-82. doi: https://doi.org/10.5056/jnm16023
Ratanasirintrawoot S, Israsena N. Stem Cells in the Intestine: Possible Roles in Pathogenesis of Irritable Bowel Syndrome. J Neurogastroenterol Motil. 2016 Jul 30;22(3):367-82. doi: 10.5056/jnm16023. PMID: 27184041; PMCID: PMC4930294.
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