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Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid.

Methods in molecular medicine

Dworski R, Sheller JR, Christman BW.
PMID: 21336918
Methods Mol Med. 2001;56:411-22. doi: 10.1385/1-59259-151-5:411.

Metabolism of arachidonic acid results in a host of biologically active compounds with profound effects on airway inflammation (1). After activation of cellular phospholipases and release of free arachidonic acid, catalyzed insertion of oxygen occurs enzymatically via action of...

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Dworski R, Sheller JR, Christman BW. Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid. Methods Mol Med. 2001;56:411-22doi: 10.1385/1-59259-151-5:411.
Dworski, R., Sheller, J. R., & Christman, B. W. (2001). Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid. Methods in molecular medicine, 56411-22. https://doi.org/10.1385/1-59259-151-5:411
Dworski, R, et al. "Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid." Methods in molecular medicine vol. 56 (2001): 411-22. doi: https://doi.org/10.1385/1-59259-151-5:411
Dworski R, Sheller JR, Christman BW. Quantitative analysis of cyclooxygenase metabolites of arachidonic Acid. Methods Mol Med. 2001;56:411-22. doi: 10.1385/1-59259-151-5:411. PMID: 21336918.
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Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B.

Hypertension (Dallas, Tex. : 1979)

Kriska T, Herrnreiter A, Pfister SL, Adebesin A, Falck JR, Campbell WB.
PMID: 34784723
Hypertension. 2022 Jan;79(1):104-114. doi: 10.1161/HYPERTENSIONAHA.121.17824. Epub 2021 Nov 17.

12/15-LO (12/15-lipoxygenase), encoded by

Cite
Kriska T, Herrnreiter A, Pfister SL, et al. Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension. 2021;79(1):104-114doi: 10.1161/HYPERTENSIONAHA.121.17824.
Kriska, T., Herrnreiter, A., Pfister, S. L., Adebesin, A., Falck, J. R., & Campbell, W. B. (2022). Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension (Dallas, Tex. : 1979), 79(1), 104-114. https://doi.org/10.1161/HYPERTENSIONAHA.121.17824
Kriska, Tamas, et al. "Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B." Hypertension (Dallas, Tex. : 1979) vol. 79,1 (2022): 104-114. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17824
Kriska T, Herrnreiter A, Pfister SL, Adebesin A, Falck JR, Campbell WB. Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension. 2022 Jan;79(1):104-114. doi: 10.1161/HYPERTENSIONAHA.121.17824. Epub 2021 Nov 17. PMID: 34784723.
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The prostanoids, thromboxanes, mediate hemocytic immunity to bacterial infection in the lepidopteran Spodoptera exigua.

Developmental and comparative immunology

Al Baki MA, Chandra Roy M, Lee DH, Stanley D, Kim Y.
PMID: 33737116
Dev Comp Immunol. 2021 Jul;120:104069. doi: 10.1016/j.dci.2021.104069. Epub 2021 Mar 16.

We report on a new insect prostanoid in a lepidopteran insect, Spodoptera exigua. Thromboxane B

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Al Baki MA, Chandra Roy M, Lee DH, et al. The prostanoids, thromboxanes, mediate hemocytic immunity to bacterial infection in the lepidopteran Spodoptera exigua. Dev Comp Immunol. 2021;120:104069doi: 10.1016/j.dci.2021.104069.
Al Baki, M. A., Chandra Roy, M., Lee, D. H., Stanley, D., & Kim, Y. (2021). The prostanoids, thromboxanes, mediate hemocytic immunity to bacterial infection in the lepidopteran Spodoptera exigua. Developmental and comparative immunology, 120104069. https://doi.org/10.1016/j.dci.2021.104069
Al Baki, Md Abdullah, et al. "The prostanoids, thromboxanes, mediate hemocytic immunity to bacterial infection in the lepidopteran Spodoptera exigua." Developmental and comparative immunology vol. 120 (2021): 104069. doi: https://doi.org/10.1016/j.dci.2021.104069
Al Baki MA, Chandra Roy M, Lee DH, Stanley D, Kim Y. The prostanoids, thromboxanes, mediate hemocytic immunity to bacterial infection in the lepidopteran Spodoptera exigua. Dev Comp Immunol. 2021 Jul;120:104069. doi: 10.1016/j.dci.2021.104069. Epub 2021 Mar 16. PMID: 33737116.
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Kidney damage from nonsteroidal anti-inflammatory drugs-Myth or truth? Review of selected literature.

Pharmacology research & perspectives

Drożdżal S, Lechowicz K, Szostak B, Rosik J, Kotfis K, Machoy-Mokrzyńska A, Białecka M, Ciechanowski K, Gawrońska-Szklarz B.
PMID: 34310861
Pharmacol Res Perspect. 2021 Aug;9(4):e00817. doi: 10.1002/prp2.817.

Nonsteroidal anti-inflammatory drugs (NSAIDs) are widely available drugs with anti-inflammatory and analgesic properties. Their mechanism of action is associated with the enzymes of the arachidonic acid cycle (cyclooxygenases: COX-1 and COX-2). The cyclooxygenase pathway results in the formation of...

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Drożdżal S, Lechowicz K, Szostak B, et al. Kidney damage from nonsteroidal anti-inflammatory drugs-Myth or truth? Review of selected literature. Pharmacol Res Perspect. 2021;9(4):e00817doi: 10.1002/prp2.817.
Drożdżal, S., Lechowicz, K., Szostak, B., Rosik, J., Kotfis, K., Machoy-Mokrzyńska, A., Białecka, M., Ciechanowski, K., & Gawrońska-Szklarz, B. (2021). Kidney damage from nonsteroidal anti-inflammatory drugs-Myth or truth? Review of selected literature. Pharmacology research & perspectives, 9(4), e00817. https://doi.org/10.1002/prp2.817
Drożdżal, Sylwester, et al. "Kidney damage from nonsteroidal anti-inflammatory drugs-Myth or truth? Review of selected literature." Pharmacology research & perspectives vol. 9,4 (2021): e00817. doi: https://doi.org/10.1002/prp2.817
Drożdżal S, Lechowicz K, Szostak B, Rosik J, Kotfis K, Machoy-Mokrzyńska A, Białecka M, Ciechanowski K, Gawrońska-Szklarz B. Kidney damage from nonsteroidal anti-inflammatory drugs-Myth or truth? Review of selected literature. Pharmacol Res Perspect. 2021 Aug;9(4):e00817. doi: 10.1002/prp2.817. PMID: 34310861; PMCID: PMC8313037.
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Molecular evolution of enzymes involved in the arachidonic acid cascade.

Mediators of inflammation

Toh H, Urade Y, Tanabe T.
PMID: 18475465
Mediators Inflamm. 1992;1(4):223-33. doi: 10.1155/S0962935192000346.

The metabolic pathway called the arachidonic acid cascade produces a wide range of eicosanoids, such as prostaglandins, thromboxanes and leukotrienes with potent biological activities. Recombinant DNA techniques have made it possible to determine the nucleotide sequences of cDNAs and/or...

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Toh H, Urade Y, Tanabe T. Molecular evolution of enzymes involved in the arachidonic acid cascade. Mediators Inflamm. 1992;1(4):223-33doi: 10.1155/S0962935192000346.
Toh, H., Urade, Y., & Tanabe, T. (1992). Molecular evolution of enzymes involved in the arachidonic acid cascade. Mediators of inflammation, 1(4), 223-33. https://doi.org/10.1155/S0962935192000346
Toh, H, et al. "Molecular evolution of enzymes involved in the arachidonic acid cascade." Mediators of inflammation vol. 1,4 (1992): 223-33. doi: https://doi.org/10.1155/S0962935192000346
Toh H, Urade Y, Tanabe T. Molecular evolution of enzymes involved in the arachidonic acid cascade. Mediators Inflamm. 1992;1(4):223-33. doi: 10.1155/S0962935192000346. PMID: 18475465; PMCID: PMC2365353.
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New insights into eicosanoid biosynthetic pathways: implications for arthritis.

Expert review of clinical immunology

Kojima F, Kapoor M, Kawai S, Crofford LJ.
PMID: 20477078
Expert Rev Clin Immunol. 2006 Mar;2(2):277-91. doi: 10.1586/1744666X.2.2.277.

Eicosanoid is a collective term for the family of lipid mediators derived from arachidonic acid (AA) metabolism. This complex family of lipids is comprised of prostaglandins, thromboxanes, leukotrienes, hydroxyeicosatetraenoic acids (HETEs) and lipoxins (LXs). The most studied enzymatic pathway...

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Kojima F, Kapoor M, Kawai S, et al. New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert Rev Clin Immunol. 2006;2(2):277-91doi: 10.1586/1744666X.2.2.277.
Kojima, F., Kapoor, M., Kawai, S., & Crofford, L. J. (2006). New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert review of clinical immunology, 2(2), 277-91. https://doi.org/10.1586/1744666X.2.2.277
Kojima, Fumiaki, et al. "New insights into eicosanoid biosynthetic pathways: implications for arthritis." Expert review of clinical immunology vol. 2,2 (2006): 277-91. doi: https://doi.org/10.1586/1744666X.2.2.277
Kojima F, Kapoor M, Kawai S, Crofford LJ. New insights into eicosanoid biosynthetic pathways: implications for arthritis. Expert Rev Clin Immunol. 2006 Mar;2(2):277-91. doi: 10.1586/1744666X.2.2.277. PMID: 20477078.
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Expression of thromboxane synthase, TBXAS1 and the thromboxane A2 receptor, TBXA2R, in human breast cancer.

International seminars in surgical oncology : ISSO

Watkins G, Douglas-Jones A, Mansel RE, Jiang WG.
PMID: 16250911
Int Semin Surg Oncol. 2005 Oct 26;2:23. doi: 10.1186/1477-7800-2-23.

BACKGROUND: Thromboxane synthase (TxS) metabolizes the cyclooxygenase product, prostaglandin H(2), into thromboxanes. Some of the thromboxanes are known to be biologically active on cancer cells. The aim of the study was to investigate the expression of thromboxane synthases, TBXAS1...

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Watkins G, Douglas-Jones A, Mansel RE, et al. Expression of thromboxane synthase, TBXAS1 and the thromboxane A2 receptor, TBXA2R, in human breast cancer. Int Semin Surg Oncol. 2005;2:23doi: 10.1186/1477-7800-2-23.
Watkins, G., Douglas-Jones, A., Mansel, R. E., & Jiang, W. G. (2005). Expression of thromboxane synthase, TBXAS1 and the thromboxane A2 receptor, TBXA2R, in human breast cancer. International seminars in surgical oncology : ISSO, 223. https://doi.org/10.1186/1477-7800-2-23
Watkins, Gareth, et al. "Expression of thromboxane synthase, TBXAS1 and the thromboxane A2 receptor, TBXA2R, in human breast cancer." International seminars in surgical oncology : ISSO vol. 2 (2005): 23. doi: https://doi.org/10.1186/1477-7800-2-23
Watkins G, Douglas-Jones A, Mansel RE, Jiang WG. Expression of thromboxane synthase, TBXAS1 and the thromboxane A2 receptor, TBXA2R, in human breast cancer. Int Semin Surg Oncol. 2005 Oct 26;2:23. doi: 10.1186/1477-7800-2-23. PMID: 16250911; PMCID: PMC1289289.
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Proinflammatory and immunoregulatory roles of eicosanoids in T cells.

Frontiers in immunology

Lone AM, Taskén K.
PMID: 23760108
Front Immunol. 2013 Jun 04;4:130. doi: 10.3389/fimmu.2013.00130. eCollection 2013.

Eicosanoids are inflammatory mediators primarily generated by hydrolysis of membrane phospholipids by phospholipase A2 to ω-3 and ω-6 C20 fatty acids that next are converted to leukotrienes (LTs), prostaglandins (PGs), prostacyclins (PCs), and thromboxanes (TXAs). The rate-limiting and tightly...

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Lone AM, Taskén K. Proinflammatory and immunoregulatory roles of eicosanoids in T cells. Front Immunol. 2013;4:130doi: 10.3389/fimmu.2013.00130.
Lone, A. M., & Taskén, K. (2013). Proinflammatory and immunoregulatory roles of eicosanoids in T cells. Frontiers in immunology, 4130. https://doi.org/10.3389/fimmu.2013.00130
Lone, Anna Mari, and Taskén, Kjetil. "Proinflammatory and immunoregulatory roles of eicosanoids in T cells." Frontiers in immunology vol. 4 (2013): 130. doi: https://doi.org/10.3389/fimmu.2013.00130
Lone AM, Taskén K. Proinflammatory and immunoregulatory roles of eicosanoids in T cells. Front Immunol. 2013 Jun 04;4:130. doi: 10.3389/fimmu.2013.00130. eCollection 2013. PMID: 23760108; PMCID: PMC3671288.
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Quantitative analysis of lipids: a higher-throughput LC-MS/MS-based method and its comparison to ELISA.

Future science OA

Gandhi AS, Budac D, Khayrullina T, Staal R, Chandrasena G.
PMID: 28344822
Future Sci OA. 2017 Jan 16;3(1):FSO157. doi: 10.4155/fsoa-2016-0067. eCollection 2017 Mar.

AIM: Lipids such as prostaglandins, leukotrienes and thromboxanes are released as a result of an inflammatory episode in pain (central and peripheral).METHODOLOGY & RESULTS: To measure these lipids as potential mechanistic biomarkers in neuropathic pain models, we developed a...

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Gandhi AS, Budac D, Khayrullina T, et al. Quantitative analysis of lipids: a higher-throughput LC-MS/MS-based method and its comparison to ELISA. Future Sci OA. 2017;3(1):FSO157doi: 10.4155/fsoa-2016-0067.
Gandhi, A. S., Budac, D., Khayrullina, T., Staal, R., & Chandrasena, G. (2017). Quantitative analysis of lipids: a higher-throughput LC-MS/MS-based method and its comparison to ELISA. Future science OA, 3(1), FSO157. https://doi.org/10.4155/fsoa-2016-0067
Gandhi, Adarsh S, et al. "Quantitative analysis of lipids: a higher-throughput LC-MS/MS-based method and its comparison to ELISA." Future science OA vol. 3,1 (2017): FSO157. doi: https://doi.org/10.4155/fsoa-2016-0067
Gandhi AS, Budac D, Khayrullina T, Staal R, Chandrasena G. Quantitative analysis of lipids: a higher-throughput LC-MS/MS-based method and its comparison to ELISA. Future Sci OA. 2017 Jan 16;3(1):FSO157. doi: 10.4155/fsoa-2016-0067. eCollection 2017 Mar. PMID: 28344822; PMCID: PMC5351511.
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Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B.

Hypertension (Dallas, Tex. : 1979)

Kriska T, Herrnreiter A, Pfister SL, Adebesin A, Falck JR, Campbell WB.
PMID: 34784723
Hypertension. 2022 Jan;79(1):104-114. doi: 10.1161/HYPERTENSIONAHA.121.17824. Epub 2021 Nov 17.

12/15-LO (12/15-lipoxygenase), encoded by

Cite
Kriska T, Herrnreiter A, Pfister SL, et al. Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension. 2021;79(1):104-114doi: 10.1161/HYPERTENSIONAHA.121.17824.
Kriska, T., Herrnreiter, A., Pfister, S. L., Adebesin, A., Falck, J. R., & Campbell, W. B. (2022). Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension (Dallas, Tex. : 1979), 79(1), 104-114. https://doi.org/10.1161/HYPERTENSIONAHA.121.17824
Kriska, Tamas, et al. "Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B." Hypertension (Dallas, Tex. : 1979) vol. 79,1 (2022): 104-114. doi: https://doi.org/10.1161/HYPERTENSIONAHA.121.17824
Kriska T, Herrnreiter A, Pfister SL, Adebesin A, Falck JR, Campbell WB. Macrophage 12(S)-HETE Enhances Angiotensin II-Induced Contraction by a BLT2 (Leukotriene B. Hypertension. 2022 Jan;79(1):104-114. doi: 10.1161/HYPERTENSIONAHA.121.17824. Epub 2021 Nov 17. PMID: 34784723.
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Physiology and pathophysiology of prostanoid receptors.

Proceedings of the Japan Academy. Series B, Physical and biological sciences

Narumiya S.
PMID: 24367153
Proc Jpn Acad Ser B Phys Biol Sci. 2007 Dec;83(9):296-319. doi: 10.2183/pjab/83.296.

Prostanoids, consisting of prostaglandins (PGs) and thromboxanes (TXs), are oxygenated products of C20 unsaturated fatty acids. They include PGD2, PGE2, PGF2 α , PGI2, and TXA2. Given that aspirin-like nonsteroidal anti-inflammatory drugs exert their actions by suppressing prostanoid production,...

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Narumiya S. Physiology and pathophysiology of prostanoid receptors. Proc Jpn Acad Ser B Phys Biol Sci. 2007;83(9):296-319doi: 10.2183/pjab/83.296.
Narumiya, S. (2007). Physiology and pathophysiology of prostanoid receptors. Proceedings of the Japan Academy. Series B, Physical and biological sciences, 83(9), 296-319. https://doi.org/10.2183/pjab/83.296
Narumiya, Shuh. "Physiology and pathophysiology of prostanoid receptors." Proceedings of the Japan Academy. Series B, Physical and biological sciences vol. 83,9 (2007): 296-319. doi: https://doi.org/10.2183/pjab/83.296
Narumiya S. Physiology and pathophysiology of prostanoid receptors. Proc Jpn Acad Ser B Phys Biol Sci. 2007 Dec;83(9):296-319. doi: 10.2183/pjab/83.296. PMID: 24367153; PMCID: PMC3859365.
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The Role of Fish Oil in Inflammatory Eye Diseases.

Biomedicine hub

Wang H, Daggy BP.
PMID: 31988905
Biomed Hub. 2017 Feb 21;2(1):1-12. doi: 10.1159/000455818. eCollection 2017.

Consumption of fish oil is associated with reduced morbidity and mortality of cardiovascular diseases and also reduces the severity of many other inflammatory diseases and autoimmune disorders. The beneficial effects are attributed to the anti-inflammatory effects of the omega-3...

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Wang H, Daggy BP. The Role of Fish Oil in Inflammatory Eye Diseases. Biomed Hub. 2017;2(1):1-12doi: 10.1159/000455818.
Wang, H., & Daggy, B. P. (2017). The Role of Fish Oil in Inflammatory Eye Diseases. Biomedicine hub, 2(1), 1-12. https://doi.org/10.1159/000455818
Wang, Hong, and Daggy, Bruce P. "The Role of Fish Oil in Inflammatory Eye Diseases." Biomedicine hub vol. 2,1 (2017): 1-12. doi: https://doi.org/10.1159/000455818
Wang H, Daggy BP. The Role of Fish Oil in Inflammatory Eye Diseases. Biomed Hub. 2017 Feb 21;2(1):1-12. doi: 10.1159/000455818. eCollection 2017. PMID: 31988905; PMCID: PMC6945974.
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Showing 1 to 12 of 23 entries