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Showing 1 to 11 of 11 entries
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The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis.

Frontiers in endocrinology

Wang HD, Zhang CS, Li MW, Lin Q, Zhang Q, Liu DF, Ma ZY, Dong J.
PMID: 34512548
Front Endocrinol (Lausanne). 2021 Aug 26;12:708139. doi: 10.3389/fendo.2021.708139. eCollection 2021.

BACKGROUND: Insulin resistance is a metabolic disorder that occurs in type 2 diabetes mellitus and obesity. Genetic factors such as β3-adrenoceptor polymorphism (Trp64Arg) may be involved in IR and insulin secretion. However, their association is controversial. Therefore, the current...

Cite
Wang HD, Zhang CS, Li MW, et al. The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Front Endocrinol (Lausanne). 2021;12:708139doi: 10.3389/fendo.2021.708139.
Wang, H. D., Zhang, C. S., Li, M. W., Lin, Q., Zhang, Q., Liu, D. F., Ma, Z. Y., & Dong, J. (2021). The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Frontiers in endocrinology, 12708139. https://doi.org/10.3389/fendo.2021.708139
Wang, Hai-Dan, et al. "The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis." Frontiers in endocrinology vol. 12 (2021): 708139. doi: https://doi.org/10.3389/fendo.2021.708139
Wang HD, Zhang CS, Li MW, Lin Q, Zhang Q, Liu DF, Ma ZY, Dong J. The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Front Endocrinol (Lausanne). 2021 Aug 26;12:708139. doi: 10.3389/fendo.2021.708139. eCollection 2021. PMID: 34512548; PMCID: PMC8426512.
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Adrenergic receptors and cardiovascular effects of catecholamines.

Annales d'endocrinologie

Motiejunaite J, Amar L, Vidal-Petiot E.
PMID: 32473788
Ann Endocrinol (Paris). 2021 Jun;82(3):193-197. doi: 10.1016/j.ando.2020.03.012. Epub 2020 Mar 18.

Activation of the sympathetic nervous system is responsible for the body's "fight or flight" reaction. The physiological responses to the activation of the sympathetic nervous system and adrenal medulla are mediated through the action of the endogenous catecholamines norepinephrine...

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Motiejunaite J, Amar L, Vidal-Petiot E. Adrenergic receptors and cardiovascular effects of catecholamines. Ann Endocrinol (Paris). 2020;82(3):193-197doi: 10.1016/j.ando.2020.03.012.
Motiejunaite, J., Amar, L., & Vidal-Petiot, E. (2021). Adrenergic receptors and cardiovascular effects of catecholamines. Annales d'endocrinologie, 82(3), 193-197. https://doi.org/10.1016/j.ando.2020.03.012
Motiejunaite, Justina, et al. "Adrenergic receptors and cardiovascular effects of catecholamines." Annales d'endocrinologie vol. 82,3 (2021): 193-197. doi: https://doi.org/10.1016/j.ando.2020.03.012
Motiejunaite J, Amar L, Vidal-Petiot E. Adrenergic receptors and cardiovascular effects of catecholamines. Ann Endocrinol (Paris). 2021 Jun;82(3):193-197. doi: 10.1016/j.ando.2020.03.012. Epub 2020 Mar 18. PMID: 32473788.
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Effect of mHCN2 gene modification on chronotropic relevant receptors in BMSCs co-cultured with atrial myocytes.

Experimental and therapeutic medicine

Ding C, Yang C, Cao Q, Zhu X, Zhang J, Zhang W, Wang Y, Li L.
PMID: 28962131
Exp Ther Med. 2017 Sep;14(3):2108-2116. doi: 10.3892/etm.2017.4789. Epub 2017 Jul 12.

Currently, the mechanism of the chronotropic ability of stem cells modified to express the hyperpolarization-activated cyclic nucleotide-gated (HCN) gene remains to be elucidated. The present study assessed the effects of mouse (m)HCN2 gene modification on the expression of chronotropic...

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Ding C, Yang C, Cao Q, et al. Effect of mHCN2 gene modification on chronotropic relevant receptors in BMSCs co-cultured with atrial myocytes. Exp Ther Med. 2017;14(3):2108-2116doi: 10.3892/etm.2017.4789.
Ding, C., Yang, C., Cao, Q., Zhu, X., Zhang, J., Zhang, W., Wang, Y., & Li, L. (2017). Effect of mHCN2 gene modification on chronotropic relevant receptors in BMSCs co-cultured with atrial myocytes. Experimental and therapeutic medicine, 14(3), 2108-2116. https://doi.org/10.3892/etm.2017.4789
Ding, Chandong, et al. "Effect of mHCN2 gene modification on chronotropic relevant receptors in BMSCs co-cultured with atrial myocytes." Experimental and therapeutic medicine vol. 14,3 (2017): 2108-2116. doi: https://doi.org/10.3892/etm.2017.4789
Ding C, Yang C, Cao Q, Zhu X, Zhang J, Zhang W, Wang Y, Li L. Effect of mHCN2 gene modification on chronotropic relevant receptors in BMSCs co-cultured with atrial myocytes. Exp Ther Med. 2017 Sep;14(3):2108-2116. doi: 10.3892/etm.2017.4789. Epub 2017 Jul 12. PMID: 28962131; PMCID: PMC5609193.
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Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity.

Circulation

Payumo AY, Chen X, Hirose K, Chen X, Hoang A, Khyeam S, Yu H, Wang J, Chen Q, Powers N, Chen L, Bigley RB, Lovas J, Hu G, Huang GN.
PMID: 34543065
Circulation. 2021 Sep 21;144(12):1000-1003. doi: 10.1161/CIRCULATIONAHA.121.054846. Epub 2021 Sep 20.

No abstract available.

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Payumo AY, Chen X, Hirose K, et al. Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation. 2021;144(12):1000-1003doi: 10.1161/CIRCULATIONAHA.121.054846.
Payumo, A. Y., Chen, X., Hirose, K., Chen, X., Hoang, A., Khyeam, S., Yu, H., Wang, J., Chen, Q., Powers, N., Chen, L., Bigley, R. B., Lovas, J., Hu, G., & Huang, G. N. (2021). Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation, 144(12), 1000-1003. https://doi.org/10.1161/CIRCULATIONAHA.121.054846
Payumo, Alexander Y, et al. "Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity." Circulation vol. 144,12 (2021): 1000-1003. doi: https://doi.org/10.1161/CIRCULATIONAHA.121.054846
Payumo AY, Chen X, Hirose K, Chen X, Hoang A, Khyeam S, Yu H, Wang J, Chen Q, Powers N, Chen L, Bigley RB, Lovas J, Hu G, Huang GN. Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation. 2021 Sep 21;144(12):1000-1003. doi: 10.1161/CIRCULATIONAHA.121.054846. Epub 2021 Sep 20. PMID: 34543065; PMCID: PMC8457303.
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Vasopressor Therapy in the Intensive Care Unit.

Seminars in respiratory and critical care medicine

Russell JA, Gordon AC, Williams MD, Boyd JH, Walley KR, Kissoon N.
PMID: 32820475
Semin Respir Crit Care Med. 2021 Feb;42(1):59-77. doi: 10.1055/s-0040-1710320. Epub 2020 Aug 20.

After fluid administration for vasodilatory shock, vasopressors are commonly infused. Causes of vasodilatory shock include septic shock, post-cardiovascular surgery, post-acute myocardial infarction, postsurgery, other causes of an intense systemic inflammatory response, and drug -associated anaphylaxis. Therapeutic vasopressors are hormones...

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Russell JA, Gordon AC, Williams MD, et al. Vasopressor Therapy in the Intensive Care Unit. Semin Respir Crit Care Med. 2020;42(1):59-77doi: 10.1055/s-0040-1710320.
Russell, J. A., Gordon, A. C., Williams, M. D., Boyd, J. H., Walley, K. R., & Kissoon, N. (2021). Vasopressor Therapy in the Intensive Care Unit. Seminars in respiratory and critical care medicine, 42(1), 59-77. https://doi.org/10.1055/s-0040-1710320
Russell, James A, et al. "Vasopressor Therapy in the Intensive Care Unit." Seminars in respiratory and critical care medicine vol. 42,1 (2021): 59-77. doi: https://doi.org/10.1055/s-0040-1710320
Russell JA, Gordon AC, Williams MD, Boyd JH, Walley KR, Kissoon N. Vasopressor Therapy in the Intensive Care Unit. Semin Respir Crit Care Med. 2021 Feb;42(1):59-77. doi: 10.1055/s-0040-1710320. Epub 2020 Aug 20. PMID: 32820475.
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Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity.

Circulation

Payumo AY, Chen X, Hirose K, Chen X, Hoang A, Khyeam S, Yu H, Wang J, Chen Q, Powers N, Chen L, Bigley RB, Lovas J, Hu G, Huang GN.
PMID: 34543065
Circulation. 2021 Sep 21;144(12):1000-1003. doi: 10.1161/CIRCULATIONAHA.121.054846. Epub 2021 Sep 20.

No abstract available.

Cite
Payumo AY, Chen X, Hirose K, et al. Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation. 2021;144(12):1000-1003doi: 10.1161/CIRCULATIONAHA.121.054846.
Payumo, A. Y., Chen, X., Hirose, K., Chen, X., Hoang, A., Khyeam, S., Yu, H., Wang, J., Chen, Q., Powers, N., Chen, L., Bigley, R. B., Lovas, J., Hu, G., & Huang, G. N. (2021). Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation, 144(12), 1000-1003. https://doi.org/10.1161/CIRCULATIONAHA.121.054846
Payumo, Alexander Y, et al. "Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity." Circulation vol. 144,12 (2021): 1000-1003. doi: https://doi.org/10.1161/CIRCULATIONAHA.121.054846
Payumo AY, Chen X, Hirose K, Chen X, Hoang A, Khyeam S, Yu H, Wang J, Chen Q, Powers N, Chen L, Bigley RB, Lovas J, Hu G, Huang GN. Adrenergic-Thyroid Hormone Interactions Drive Postnatal Thermogenesis and Loss of Mammalian Heart Regenerative Capacity. Circulation. 2021 Sep 21;144(12):1000-1003. doi: 10.1161/CIRCULATIONAHA.121.054846. Epub 2021 Sep 20. PMID: 34543065; PMCID: PMC8457303.
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Network Pharmacology-Based Strategy for Predicting Active Ingredients and Potential Targets of Gegen Qinlian Decoction for Rotavirus Enteritis.

Evidence-based complementary and alternative medicine : eCAM

Zhong P, Song L, Gao M, Wang X, Tan W, Lu H, Lan Q, Zhao Z, Zhao W.
PMID: 32802121
Evid Based Complement Alternat Med. 2020 Jul 29;2020:2957567. doi: 10.1155/2020/2957567. eCollection 2020.

MATERIALS AND METHODS: In this study, a network pharmacology-based strategy was used to elucidate the mechanism of GGQLD for the treatment of RVE. Oral bioavailability and drug-likeness were taken as the judgment criteria to search the active ingredients of...

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Zhong P, Song L, Gao M, et al. Network Pharmacology-Based Strategy for Predicting Active Ingredients and Potential Targets of Gegen Qinlian Decoction for Rotavirus Enteritis. Evid Based Complement Alternat Med. 2020;2020:2957567doi: 10.1155/2020/2957567.
Zhong, P., Song, L., Gao, M., Wang, X., Tan, W., Lu, H., Lan, Q., Zhao, Z., & Zhao, W. (2020). Network Pharmacology-Based Strategy for Predicting Active Ingredients and Potential Targets of Gegen Qinlian Decoction for Rotavirus Enteritis. Evidence-based complementary and alternative medicine : eCAM, 20202957567. https://doi.org/10.1155/2020/2957567
Zhong, Peicheng, et al. "Network Pharmacology-Based Strategy for Predicting Active Ingredients and Potential Targets of Gegen Qinlian Decoction for Rotavirus Enteritis." Evidence-based complementary and alternative medicine : eCAM vol. 2020 (2020): 2957567. doi: https://doi.org/10.1155/2020/2957567
Zhong P, Song L, Gao M, Wang X, Tan W, Lu H, Lan Q, Zhao Z, Zhao W. Network Pharmacology-Based Strategy for Predicting Active Ingredients and Potential Targets of Gegen Qinlian Decoction for Rotavirus Enteritis. Evid Based Complement Alternat Med. 2020 Jul 29;2020:2957567. doi: 10.1155/2020/2957567. eCollection 2020. PMID: 32802121; PMCID: PMC7414372.
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The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis.

Frontiers in endocrinology

Wang HD, Zhang CS, Li MW, Lin Q, Zhang Q, Liu DF, Ma ZY, Dong J.
PMID: 34512548
Front Endocrinol (Lausanne). 2021 Aug 26;12:708139. doi: 10.3389/fendo.2021.708139. eCollection 2021.

BACKGROUND: Insulin resistance is a metabolic disorder that occurs in type 2 diabetes mellitus and obesity. Genetic factors such as β3-adrenoceptor polymorphism (Trp64Arg) may be involved in IR and insulin secretion. However, their association is controversial. Therefore, the current...

Cite
Wang HD, Zhang CS, Li MW, et al. The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Front Endocrinol (Lausanne). 2021;12:708139doi: 10.3389/fendo.2021.708139.
Wang, H. D., Zhang, C. S., Li, M. W., Lin, Q., Zhang, Q., Liu, D. F., Ma, Z. Y., & Dong, J. (2021). The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Frontiers in endocrinology, 12708139. https://doi.org/10.3389/fendo.2021.708139
Wang, Hai-Dan, et al. "The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis." Frontiers in endocrinology vol. 12 (2021): 708139. doi: https://doi.org/10.3389/fendo.2021.708139
Wang HD, Zhang CS, Li MW, Lin Q, Zhang Q, Liu DF, Ma ZY, Dong J. The Association of Trp64Arg Polymorphism in the Beta-Adrenergic Receptor With Insulin Resistance: Meta-Analysis. Front Endocrinol (Lausanne). 2021 Aug 26;12:708139. doi: 10.3389/fendo.2021.708139. eCollection 2021. PMID: 34512548; PMCID: PMC8426512.
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The Role of Visceral Hypersensitivity in Irritable Bowel Syndrome: Pharmacological Targets and Novel Treatments.

Journal of neurogastroenterology and motility

Farzaei MH, Bahramsoltani R, Abdollahi M, Rahimi R.
PMID: 27431236
J Neurogastroenterol Motil. 2016 Oct 30;22(4):558-574. doi: 10.5056/jnm16001.

Irritable bowel syndrome (IBS) is the most common disorder referred to gastroenterologists and is characterized by altered bowel habits, abdominal pain, and bloating. Visceral hypersensitivity (VH) is a multifactorial process that may occur within the peripheral or central nervous...

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Farzaei MH, Bahramsoltani R, Abdollahi M, et al. The Role of Visceral Hypersensitivity in Irritable Bowel Syndrome: Pharmacological Targets and Novel Treatments. J Neurogastroenterol Motil. 2016;22(4):558-574doi: 10.5056/jnm16001.
Farzaei, M. H., Bahramsoltani, R., Abdollahi, M., & Rahimi, R. (2016). The Role of Visceral Hypersensitivity in Irritable Bowel Syndrome: Pharmacological Targets and Novel Treatments. Journal of neurogastroenterology and motility, 22(4), 558-574. https://doi.org/10.5056/jnm16001
Farzaei, Mohammad H, et al. "The Role of Visceral Hypersensitivity in Irritable Bowel Syndrome: Pharmacological Targets and Novel Treatments." Journal of neurogastroenterology and motility vol. 22,4 (2016): 558-574. doi: https://doi.org/10.5056/jnm16001
Farzaei MH, Bahramsoltani R, Abdollahi M, Rahimi R. The Role of Visceral Hypersensitivity in Irritable Bowel Syndrome: Pharmacological Targets and Novel Treatments. J Neurogastroenterol Motil. 2016 Oct 30;22(4):558-574. doi: 10.5056/jnm16001. PMID: 27431236; PMCID: PMC5056566.
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Arg389Gly β1-adrenergic receptor polymorphism and susceptibility to syncope during tilt test.

Archives of medical science : AMS

Zelazowska M, Lelonek M, Fendler W, Pietrucha T.
PMID: 24904655
Arch Med Sci. 2014 May 12;10(2):240-5. doi: 10.5114/aoms.2014.42576. Epub 2014 May 13.

INTRODUCTION: Numerous hormones, neurotransmitters, and other stimuli exert their biological effect on cellular functioning through heptahelical receptors coupled to G proteins (GPCR - G protein-coupled receptors). Adrenergic receptors that belong to this superfamily of receptors are components of the...

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Zelazowska M, Lelonek M, Fendler W, et al. Arg389Gly β1-adrenergic receptor polymorphism and susceptibility to syncope during tilt test. Arch Med Sci. 2014;10(2):240-5doi: 10.5114/aoms.2014.42576.
Zelazowska, M., Lelonek, M., Fendler, W., & Pietrucha, T. (2014). Arg389Gly β1-adrenergic receptor polymorphism and susceptibility to syncope during tilt test. Archives of medical science : AMS, 10(2), 240-5. https://doi.org/10.5114/aoms.2014.42576
Zelazowska, Monika, et al. "Arg389Gly β1-adrenergic receptor polymorphism and susceptibility to syncope during tilt test." Archives of medical science : AMS vol. 10,2 (2014): 240-5. doi: https://doi.org/10.5114/aoms.2014.42576
Zelazowska M, Lelonek M, Fendler W, Pietrucha T. Arg389Gly β1-adrenergic receptor polymorphism and susceptibility to syncope during tilt test. Arch Med Sci. 2014 May 12;10(2):240-5. doi: 10.5114/aoms.2014.42576. Epub 2014 May 13. PMID: 24904655; PMCID: PMC4042044.
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Role of G Protein-Coupled Receptors in Microglial Activation: Implication in Parkinson's Disease.

Frontiers in aging neuroscience

Gu C, Chen Y, Chen Y, Liu CF, Zhu Z, Wang M.
PMID: 34867296
Front Aging Neurosci. 2021 Nov 16;13:768156. doi: 10.3389/fnagi.2021.768156. eCollection 2021.

Parkinson's disease (PD) is one of the prevalent neurodegenerative diseases associated with preferential loss of dopaminergic (DA) neurons in the substantia nigra compacta (SNc) and accumulation of α-synuclein in DA neurons. Even though the precise pathogenesis of PD is...

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Gu C, Chen Y, Chen Y, et al. Role of G Protein-Coupled Receptors in Microglial Activation: Implication in Parkinson's Disease. Front Aging Neurosci. 2021;13:768156doi: 10.3389/fnagi.2021.768156.
Gu, C., Chen, Y., Chen, Y., Liu, C. F., Zhu, Z., & Wang, M. (2021). Role of G Protein-Coupled Receptors in Microglial Activation: Implication in Parkinson's Disease. Frontiers in aging neuroscience, 13768156. https://doi.org/10.3389/fnagi.2021.768156
Gu, Chao, et al. "Role of G Protein-Coupled Receptors in Microglial Activation: Implication in Parkinson's Disease." Frontiers in aging neuroscience vol. 13 (2021): 768156. doi: https://doi.org/10.3389/fnagi.2021.768156
Gu C, Chen Y, Chen Y, Liu CF, Zhu Z, Wang M. Role of G Protein-Coupled Receptors in Microglial Activation: Implication in Parkinson's Disease. Front Aging Neurosci. 2021 Nov 16;13:768156. doi: 10.3389/fnagi.2021.768156. eCollection 2021. PMID: 34867296; PMCID: PMC8635063.
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