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Piroxicam confer neuroprotection in Cerebral Ischemia by inhibiting Cyclooxygenases, Acid- Sensing Ion Channel-1a and Aquaporin-4: an in silico comparison with Aspirin and Nimesulide.

Bioinformation

Mazumder MK, Borah A.
PMID: 26124563
Bioinformation. 2015 Apr 30;11(4):217-22. doi: 10.6026/97320630011217. eCollection 2015.

Cerebral ischemia (CI), caused by the deprivation of oxygen and glucose to the brain, is the leading cause of permanent disability. Neuronal demise in CI has been linked to several pathways which include cyclooxygenases (COX) - mediated production of...

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Mazumder MK, Borah A. Piroxicam confer neuroprotection in Cerebral Ischemia by inhibiting Cyclooxygenases, Acid- Sensing Ion Channel-1a and Aquaporin-4: an in silico comparison with Aspirin and Nimesulide. Bioinformation. 2015;11(4):217-22doi: 10.6026/97320630011217.
Mazumder, M. K., & Borah, A. (2015). Piroxicam confer neuroprotection in Cerebral Ischemia by inhibiting Cyclooxygenases, Acid- Sensing Ion Channel-1a and Aquaporin-4: an in silico comparison with Aspirin and Nimesulide. Bioinformation, 11(4), 217-22. https://doi.org/10.6026/97320630011217
Mazumder, Muhammed Khairujjaman, and Borah, Anupom. "Piroxicam confer neuroprotection in Cerebral Ischemia by inhibiting Cyclooxygenases, Acid- Sensing Ion Channel-1a and Aquaporin-4: an in silico comparison with Aspirin and Nimesulide." Bioinformation vol. 11,4 (2015): 217-22. doi: https://doi.org/10.6026/97320630011217
Mazumder MK, Borah A. Piroxicam confer neuroprotection in Cerebral Ischemia by inhibiting Cyclooxygenases, Acid- Sensing Ion Channel-1a and Aquaporin-4: an in silico comparison with Aspirin and Nimesulide. Bioinformation. 2015 Apr 30;11(4):217-22. doi: 10.6026/97320630011217. eCollection 2015. PMID: 26124563; PMCID: PMC4479049.
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Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension.

Hepatology international

Rizvi MR, Tauseef M, Shahid M, Babbar R, Fahim M, Sakhuja P, Sarin SK.
PMID: 26201795
Hepatol Int. 2013 Jun;7(2):622-35. doi: 10.1007/s12072-012-9397-9. Epub 2012 Sep 02.

AIMS: To evaluate the participation of nitric oxide (NO) and prostaglandin (PGI2) on hyperdynamic state in endotoxemia-induced portal hypertension (EIP) induced by chronic endotoxemia.METHODS: The portal pressure (PP) and mean arterial pressure (MAP) were recorded, in vivo before and...

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Rizvi MR, Tauseef M, Shahid M, et al. Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatol Int. 2012;7(2):622-35doi: 10.1007/s12072-012-9397-9.
Rizvi, M. R., Tauseef, M., Shahid, M., Babbar, R., Fahim, M., Sakhuja, P., & Sarin, S. K. (2013). Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatology international, 7(2), 622-35. https://doi.org/10.1007/s12072-012-9397-9
Rizvi, Moattar Raza, et al. "Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension." Hepatology international vol. 7,2 (2013): 622-35. doi: https://doi.org/10.1007/s12072-012-9397-9
Rizvi MR, Tauseef M, Shahid M, Babbar R, Fahim M, Sakhuja P, Sarin SK. Nitric oxide and prostaglandin as mediators in the pathogenesis of hyperkinetic circulatory state in a model of endotoxemia-induced portal hypertension. Hepatol Int. 2013 Jun;7(2):622-35. doi: 10.1007/s12072-012-9397-9. Epub 2012 Sep 02. PMID: 26201795.
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Maternal age effects on myometrial expression of contractile proteins, uterine gene expression, and contractile activity during labor in the rat.

Physiological reports

Elmes M, Szyszka A, Pauliat C, Clifford B, Daniel Z, Cheng Z, Wathes C, McMullen S.
PMID: 25876907
Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12305.

Advanced maternal age of first time pregnant mothers is associated with prolonged and dysfunctional labor and significant risk of emergency cesarean section. We investigated the influence of maternal age on myometrial contractility, expression of contractile associated proteins (CAPs), and...

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Elmes M, Szyszka A, Pauliat C, et al. Maternal age effects on myometrial expression of contractile proteins, uterine gene expression, and contractile activity during labor in the rat. Physiol Rep. 2015;3(4)doi: 10.14814/phy2.12305.
Elmes, M., Szyszka, A., Pauliat, C., Clifford, B., Daniel, Z., Cheng, Z., Wathes, C., & McMullen, S. (2015). Maternal age effects on myometrial expression of contractile proteins, uterine gene expression, and contractile activity during labor in the rat. Physiological reports, 3(4), . https://doi.org/10.14814/phy2.12305
Elmes, Matthew, et al. "Maternal age effects on myometrial expression of contractile proteins, uterine gene expression, and contractile activity during labor in the rat." Physiological reports vol. 3,4 (2015). doi: https://doi.org/10.14814/phy2.12305
Elmes M, Szyszka A, Pauliat C, Clifford B, Daniel Z, Cheng Z, Wathes C, McMullen S. Maternal age effects on myometrial expression of contractile proteins, uterine gene expression, and contractile activity during labor in the rat. Physiol Rep. 2015 Apr;3(4). doi: 10.14814/phy2.12305. PMID: 25876907; PMCID: PMC4425948.
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Perinatal circulatory physiology.

Indian journal of pediatrics

Syamasundar Rao P.
PMID: 28326498
Indian J Pediatr. 1991 Jul;58(4):441-451. doi: 10.1007/BF02750928.

CONCLUSION: Fetal circulation is designed to utilize placenta for gas exchange whereas postnatal circulation uses lungs for gas exchange. Fetal circulatory pathways, namely, umbilical vessels, ductus venosus, foramen ovale and ductus arteriosus facilitate placental gas exchange and promote distribution...

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Syamasundar Rao P. Perinatal circulatory physiology. Indian J Pediatr. 1991;58(4):441-451doi: 10.1007/BF02750928.
Syamasundar Rao, P. (1991). Perinatal circulatory physiology. Indian journal of pediatrics, 58(4), 441-451. https://doi.org/10.1007/BF02750928
Syamasundar Rao, P. "Perinatal circulatory physiology." Indian journal of pediatrics vol. 58,4 (1991): 441-451. doi: https://doi.org/10.1007/BF02750928
Syamasundar Rao P. Perinatal circulatory physiology. Indian J Pediatr. 1991 Jul;58(4):441-451. doi: 10.1007/BF02750928. PMID: 28326498.
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Osamu Hayaishi-from the discovery of oxygenases in soil microorganisms to unraveling the enigma of sleep in mammals.

Temperature (Austin, Tex.)

Lazarus M, Huang ZL, Urade Y.
PMID: 27227031
Temperature (Austin). 2015 Jul 25;2(3):303-7. doi: 10.1080/23328940.2015.1072658. eCollection 2015.

No abstract available.

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Lazarus M, Huang ZL, Urade Y. Osamu Hayaishi-from the discovery of oxygenases in soil microorganisms to unraveling the enigma of sleep in mammals. Temperature (Austin). 2015;2(3):303-7doi: 10.1080/23328940.2015.1072658.
Lazarus, M., Huang, Z. L., & Urade, Y. (2015). Osamu Hayaishi-from the discovery of oxygenases in soil microorganisms to unraveling the enigma of sleep in mammals. Temperature (Austin, Tex.), 2(3), 303-7. https://doi.org/10.1080/23328940.2015.1072658
Lazarus, Michael, et al. "Osamu Hayaishi-from the discovery of oxygenases in soil microorganisms to unraveling the enigma of sleep in mammals." Temperature (Austin, Tex.) vol. 2,3 (2015): 303-7. doi: https://doi.org/10.1080/23328940.2015.1072658
Lazarus M, Huang ZL, Urade Y. Osamu Hayaishi-from the discovery of oxygenases in soil microorganisms to unraveling the enigma of sleep in mammals. Temperature (Austin). 2015 Jul 25;2(3):303-7. doi: 10.1080/23328940.2015.1072658. eCollection 2015. PMID: 27227031; PMCID: PMC4843939.
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Combination therapy in pulmonary arterial hypertension: recent accomplishments and future challenges.

Pulmonary circulation

Lajoie AC, Bonnet S, Provencher S.
PMID: 28597774
Pulm Circ. 2017 Apr-Jun;7(2):312-325. doi: 10.1177/2045893217710639. Epub 2017 May 30.

Pulmonary arterial hypertension (PAH) is a life-threatening disease characterized by a progressive increase in pulmonary vascular resistance, ultimately leading to right heart failure and death. Throughout the past 20 years, numerous specific pharmacologic agents, including phosphodiesterase-5 inhibitors, endothelin receptor...

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Lajoie AC, Bonnet S, Provencher S. Combination therapy in pulmonary arterial hypertension: recent accomplishments and future challenges. Pulm Circ. 2017;7(2):312-325doi: 10.1177/2045893217710639.
Lajoie, A. C., Bonnet, S., & Provencher, S. (2017). Combination therapy in pulmonary arterial hypertension: recent accomplishments and future challenges. Pulmonary circulation, 7(2), 312-325. https://doi.org/10.1177/2045893217710639
Lajoie, Annie-Christine, et al. "Combination therapy in pulmonary arterial hypertension: recent accomplishments and future challenges." Pulmonary circulation vol. 7,2 (2017): 312-325. doi: https://doi.org/10.1177/2045893217710639
Lajoie AC, Bonnet S, Provencher S. Combination therapy in pulmonary arterial hypertension: recent accomplishments and future challenges. Pulm Circ. 2017 Apr-Jun;7(2):312-325. doi: 10.1177/2045893217710639. Epub 2017 May 30. PMID: 28597774; PMCID: PMC5467950.
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Central mediators involved in the febrile response: effects of antipyretic drugs.

Temperature (Austin, Tex.)

Zampronio AR, Soares DM, Souza GE.
PMID: 27227071
Temperature (Austin). 2015 Oct 13;2(4):506-21. doi: 10.1080/23328940.2015.1102802. eCollection 2015.

Fever is a complex signal of inflammatory and infectious diseases. It is generally initiated when peripherally produced endogenous pyrogens reach areas that surround the hypothalamus. These peripheral endogenous pyrogens are cytokines that are produced by leukocytes and other cells,...

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Zampronio AR, Soares DM, Souza GE. Central mediators involved in the febrile response: effects of antipyretic drugs. Temperature (Austin). 2015;2(4):506-21doi: 10.1080/23328940.2015.1102802.
Zampronio, A. R., Soares, D. M., & Souza, G. E. (2015). Central mediators involved in the febrile response: effects of antipyretic drugs. Temperature (Austin, Tex.), 2(4), 506-21. https://doi.org/10.1080/23328940.2015.1102802
Zampronio, Aleksander R, et al. "Central mediators involved in the febrile response: effects of antipyretic drugs." Temperature (Austin, Tex.) vol. 2,4 (2015): 506-21. doi: https://doi.org/10.1080/23328940.2015.1102802
Zampronio AR, Soares DM, Souza GE. Central mediators involved in the febrile response: effects of antipyretic drugs. Temperature (Austin). 2015 Oct 13;2(4):506-21. doi: 10.1080/23328940.2015.1102802. eCollection 2015. PMID: 27227071; PMCID: PMC4843933.
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Re: Magierowski M, Jasnos K, Pawlik M, Krzysiek-Maczka G, Ptak-Belowska A, Olszanecki R, Kwiecien S, Korbut R, and Brzozowski T (2013) Role of Angiotensin-(1-7) in Gastroprotection against Stress-Induced Ulcerogenesis. The Involvement of Mas Receptor, Nitric Oxide, Prostaglandins, and Sensory Neuropeptides. J Pharmacol Exp Ther 347:717-726; doi:10.1124/jpet.113.207233.

The Journal of pharmacology and experimental therapeutics

[No authors listed]
PMID: 25934764
J Pharmacol Exp Ther. 2015 Jun;353(3):563. doi: 10.1124/jpet.115.06er15.

No abstract available.

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Re: Magierowski M, Jasnos K, Pawlik M, Krzysiek-Maczka G, Ptak-Belowska A, Olszanecki R, Kwiecien S, Korbut R, and Brzozowski T (2013) Role of Angiotensin-(1-7) in Gastroprotection against Stress-Induced Ulcerogenesis. The Involvement of Mas Receptor, Nitric Oxide, Prostaglandins, and Sensory Neuropeptides. J Pharmacol Exp Ther 347:717-726; doi:10.1124/jpet.113.207233. J Pharmacol Exp Ther. 2015;353(3):563doi: 10.1124/jpet.115.06er15.
(2015). Re: Magierowski M, Jasnos K, Pawlik M, Krzysiek-Maczka G, Ptak-Belowska A, Olszanecki R, Kwiecien S, Korbut R, and Brzozowski T (2013) Role of Angiotensin-(1-7) in Gastroprotection against Stress-Induced Ulcerogenesis. The Involvement of Mas Receptor, Nitric Oxide, Prostaglandins, and Sensory Neuropeptides. J Pharmacol Exp Ther 347:717-726; doi:10.1124/jpet.113.207233. The Journal of pharmacology and experimental therapeutics, 353(3), 563. https://doi.org/10.1124/jpet.115.06er15
"Re: Magierowski M, Jasnos K, Pawlik M, Krzysiek-Maczka G, Ptak-Belowska A, Olszanecki R, Kwiecien S, Korbut R, and Brzozowski T (2013) Role of Angiotensin-(1-7) in Gastroprotection against Stress-Induced Ulcerogenesis. The Involvement of Mas Receptor, Nitric Oxide, Prostaglandins, and Sensory Neuropeptides. J Pharmacol Exp Ther 347:717-726; doi:10.1124/jpet.113.207233." The Journal of pharmacology and experimental therapeutics vol. 353,3 (2015): 563. doi: https://doi.org/10.1124/jpet.115.06er15
Re: Magierowski M, Jasnos K, Pawlik M, Krzysiek-Maczka G, Ptak-Belowska A, Olszanecki R, Kwiecien S, Korbut R, and Brzozowski T (2013) Role of Angiotensin-(1-7) in Gastroprotection against Stress-Induced Ulcerogenesis. The Involvement of Mas Receptor, Nitric Oxide, Prostaglandins, and Sensory Neuropeptides. J Pharmacol Exp Ther 347:717-726; doi:10.1124/jpet.113.207233. J Pharmacol Exp Ther. 2015 Jun;353(3):563. doi: 10.1124/jpet.115.06er15. PMID: 25934764.
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A critical appraisal of the misoprostol removable, controlled-release vaginal delivery system of labor induction.

International journal of women's health

Patte C, Deruelle P.
PMID: 26648758
Int J Womens Health. 2015 Nov 12;7:889-99. doi: 10.2147/IJWH.S62372. eCollection 2015.

BACKGROUND: Induction of labor is a major issue in pregnancy management. Finding strategies to increase rate and decrease time to vaginal delivery is an important goal, but maternal or neonatal safety must remain the primary objective. Misoprostol is a...

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Patte C, Deruelle P. A critical appraisal of the misoprostol removable, controlled-release vaginal delivery system of labor induction. Int J Womens Health. 2015;7:889-99doi: 10.2147/IJWH.S62372.
Patte, C., & Deruelle, P. (2015). A critical appraisal of the misoprostol removable, controlled-release vaginal delivery system of labor induction. International journal of women's health, 7889-99. https://doi.org/10.2147/IJWH.S62372
Patte, Charlotte, and Deruelle, Philippe. "A critical appraisal of the misoprostol removable, controlled-release vaginal delivery system of labor induction." International journal of women's health vol. 7 (2015): 889-99. doi: https://doi.org/10.2147/IJWH.S62372
Patte C, Deruelle P. A critical appraisal of the misoprostol removable, controlled-release vaginal delivery system of labor induction. Int J Womens Health. 2015 Nov 12;7:889-99. doi: 10.2147/IJWH.S62372. eCollection 2015. PMID: 26648758; PMCID: PMC4648618.
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Anti-inflammatory effect of Pycnocycla spinosa extract and its component isoacetovanillone on acetic acid induced colitis in rats.

Research in pharmaceutical sciences

Minaiyan M, Asghari G, Sadraei H, Feili E.
PMID: 26600861
Res Pharm Sci. 2015 Jul-Aug;10(4):345-55.

Colitis is an inflammatory disease of the intestine with unknown etiology involving multiple immune, genetic and environmental factors. We were interested to examine the effect of total extract from Pycnocycla spinosa Boiss. on the treatment of experimental colitis. Mediators...

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Minaiyan M, Asghari G, Sadraei H, et al. Anti-inflammatory effect of Pycnocycla spinosa extract and its component isoacetovanillone on acetic acid induced colitis in rats. Res Pharm Sci. 2015;10(4):345-55
Minaiyan, M., Asghari, G., Sadraei, H., & Feili, E. (2015). Anti-inflammatory effect of Pycnocycla spinosa extract and its component isoacetovanillone on acetic acid induced colitis in rats. Research in pharmaceutical sciences, 10(4), 345-55.
Minaiyan, Mohsen, et al. "Anti-inflammatory effect of Pycnocycla spinosa extract and its component isoacetovanillone on acetic acid induced colitis in rats." Research in pharmaceutical sciences vol. 10,4 (2015): 345-55.
Minaiyan M, Asghari G, Sadraei H, Feili E. Anti-inflammatory effect of Pycnocycla spinosa extract and its component isoacetovanillone on acetic acid induced colitis in rats. Res Pharm Sci. 2015 Jul-Aug;10(4):345-55. PMID: 26600861; PMCID: PMC4623623.
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Update on the Angiotensin converting enzyme 2-Angiotensin (1-7)-MAS receptor axis: fetal programing, sex differences, and intracellular pathways.

Frontiers in endocrinology

Chappell MC, Marshall AC, Alzayadneh EM, Shaltout HA, Diz DI.
PMID: 24409169
Front Endocrinol (Lausanne). 2014 Jan 09;4:201. doi: 10.3389/fendo.2013.00201.

The renin-angiotensin-system (RAS) constitutes an important hormonal system in the physiological regulation of blood pressure. Indeed, dysregulation of the RAS may lead to the development of cardiovascular pathologies including kidney injury. Moreover, the blockade of this system by the...

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Chappell MC, Marshall AC, Alzayadneh EM, et al. Update on the Angiotensin converting enzyme 2-Angiotensin (1-7)-MAS receptor axis: fetal programing, sex differences, and intracellular pathways. Front Endocrinol (Lausanne). 2014;4:201doi: 10.3389/fendo.2013.00201.
Chappell, M. C., Marshall, A. C., Alzayadneh, E. M., Shaltout, H. A., & Diz, D. I. (2014). Update on the Angiotensin converting enzyme 2-Angiotensin (1-7)-MAS receptor axis: fetal programing, sex differences, and intracellular pathways. Frontiers in endocrinology, 4201. https://doi.org/10.3389/fendo.2013.00201
Chappell, Mark C, et al. "Update on the Angiotensin converting enzyme 2-Angiotensin (1-7)-MAS receptor axis: fetal programing, sex differences, and intracellular pathways." Frontiers in endocrinology vol. 4 (2014): 201. doi: https://doi.org/10.3389/fendo.2013.00201
Chappell MC, Marshall AC, Alzayadneh EM, Shaltout HA, Diz DI. Update on the Angiotensin converting enzyme 2-Angiotensin (1-7)-MAS receptor axis: fetal programing, sex differences, and intracellular pathways. Front Endocrinol (Lausanne). 2014 Jan 09;4:201. doi: 10.3389/fendo.2013.00201. PMID: 24409169; PMCID: PMC3886117.
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Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1.

Frontiers in pharmacology

Tuure L, Hämäläinen M, Whittle BJ, Moilanen E.
PMID: 28983247
Front Pharmacol. 2017 Sep 21;8:646. doi: 10.3389/fphar.2017.00646. eCollection 2017.

Microsomal prostaglandin E synthase-1 (mPGES-1) is an inducible enzyme situated downstream of cyclo-oxygenase-2, promoting the excessive PGE

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Tuure L, Hämäläinen M, Whittle BJ, et al. Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1. Front Pharmacol. 2017;8:646doi: 10.3389/fphar.2017.00646.
Tuure, L., Hämäläinen, M., Whittle, B. J., & Moilanen, E. (2017). Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1. Frontiers in pharmacology, 8646. https://doi.org/10.3389/fphar.2017.00646
Tuure, Lauri, et al. "Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1." Frontiers in pharmacology vol. 8 (2017): 646. doi: https://doi.org/10.3389/fphar.2017.00646
Tuure L, Hämäläinen M, Whittle BJ, Moilanen E. Microsomal Prostaglandin E Synthase-1 Expression in Inflammatory Conditions Is Downregulated by Dexamethasone: Seminal Role of the Regulatory Phosphatase MKP-1. Front Pharmacol. 2017 Sep 21;8:646. doi: 10.3389/fphar.2017.00646. eCollection 2017. PMID: 28983247; PMCID: PMC5613146.
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Showing 1 to 12 of 1392 entries