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The A-current: how ubiquitous a feature of excitable cells is it?

diversity

Rogawski MA.
GSID: TRQSOe9hZ40J
MA Rogawski - Trends in Neurosciences, 1985 - Elsevier

The membranes of most excitable cells contain a distinct set of potassium channels that rapidly open and close following depolarization, giving rise to a transient outward …

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Rogawski, M. A. (1985). The A-current: how ubiquitous a feature of excitable cells is it?. Trends in Neurosciences, 8, 214-219.
Rogawski, Michael A. "The A-current: how ubiquitous a feature of excitable cells is it?." Trends in Neurosciences 8 (1985): 214-219.
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Complementary and synergistic therapeutic effects of compounds found in Kampo medicine: analysis of daikenchuto.

Frontiers in pharmacology

Kono T, Shimada M, Yamamoto M, Kaneko A, Oomiya Y, Kubota K, Kase Y, Lee K, Uezono Y.
PMID: 26300774
Front Pharmacol. 2015 Aug 04;6:159. doi: 10.3389/fphar.2015.00159. eCollection 2015.

Herbal medicines have been used in Japan for more than 1500 years and traditional Japanese medicines (Kampo medicines) are now fully integrated into the modern healthcare system. In total, 148 Kampo formulae are officially approved as prescription drugs and...

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Kono T, Shimada M, Yamamoto M, et al. Complementary and synergistic therapeutic effects of compounds found in Kampo medicine: analysis of daikenchuto. Front Pharmacol. 2015;6:159doi: 10.3389/fphar.2015.00159.
Kono, T., Shimada, M., Yamamoto, M., Kaneko, A., Oomiya, Y., Kubota, K., Kase, Y., Lee, K., & Uezono, Y. (2015). Complementary and synergistic therapeutic effects of compounds found in Kampo medicine: analysis of daikenchuto. Frontiers in pharmacology, 6159. https://doi.org/10.3389/fphar.2015.00159
Kono, Toru, et al. "Complementary and synergistic therapeutic effects of compounds found in Kampo medicine: analysis of daikenchuto." Frontiers in pharmacology vol. 6 (2015): 159. doi: https://doi.org/10.3389/fphar.2015.00159
Kono T, Shimada M, Yamamoto M, Kaneko A, Oomiya Y, Kubota K, Kase Y, Lee K, Uezono Y. Complementary and synergistic therapeutic effects of compounds found in Kampo medicine: analysis of daikenchuto. Front Pharmacol. 2015 Aug 04;6:159. doi: 10.3389/fphar.2015.00159. eCollection 2015. PMID: 26300774; PMCID: PMC4523940.
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The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga.

Biology open

Jovanovic ZD, Stanojevic MB, Nedeljkov VB.
PMID: 26935393
Biol Open. 2016 Apr 15;5(4):381-8. doi: 10.1242/bio.014936.

Oxidative stress and the generation of reactive oxygen species (ROS) play an important role in cellular damage. Electrophysiological analyses have shown that membrane transport proteins are susceptible to ROS. In the present study, oxidative stress was induced in Retzius...

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Jovanovic ZD, Stanojevic MB, Nedeljkov VB. The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga. Biol Open. 2016;5(4):381-8doi: 10.1242/bio.014936.
Jovanovic, Z. D., Stanojevic, M. B., & Nedeljkov, V. B. (2016). The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga. Biology open, 5(4), 381-8. https://doi.org/10.1242/bio.014936
Jovanovic, Zorica D, et al. "The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga." Biology open vol. 5,4 (2016): 381-8. doi: https://doi.org/10.1242/bio.014936
Jovanovic ZD, Stanojevic MB, Nedeljkov VB. The neurotoxic effects of hydrogen peroxide and copper in Retzius nerve cells of the leech Haemopis sanguisuga. Biol Open. 2016 Apr 15;5(4):381-8. doi: 10.1242/bio.014936. PMID: 26935393; PMCID: PMC4890660.
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M channels and stress response.

Oncotarget

Wang S, Yuan F, Li DP.
PMID: 28423349
Oncotarget. 2017 May 23;8(21):34026-34027. doi: 10.18632/oncotarget.17110.

No abstract available.

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Wang S, Yuan F, Li DP. M channels and stress response. Oncotarget. 2017;8(21):34026-34027doi: 10.18632/oncotarget.17110.
Wang, S., Yuan, F., & Li, D. P. (2017). M channels and stress response. Oncotarget, 8(21), 34026-34027. https://doi.org/10.18632/oncotarget.17110
Wang, Sheng, et al. "M channels and stress response." Oncotarget vol. 8,21 (2017): 34026-34027. doi: https://doi.org/10.18632/oncotarget.17110
Wang S, Yuan F, Li DP. M channels and stress response. Oncotarget. 2017 May 23;8(21):34026-34027. doi: 10.18632/oncotarget.17110. PMID: 28423349; PMCID: PMC5470946.
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The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location.

Frontiers in cellular neuroscience

Bock T, Stuart GJ.
PMID: 27630543
Front Cell Neurosci. 2016 Aug 31;10:206. doi: 10.3389/fncel.2016.00206. eCollection 2016.

Large conductance calcium-activated potassium channels (or BK channels) fulfil a multitude of roles in the central nervous system. At the soma of many neuronal cell types they control the speed of action potential (AP) repolarization and therefore they can...

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Bock T, Stuart GJ. The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Front Cell Neurosci. 2016;10:206doi: 10.3389/fncel.2016.00206.
Bock, T., & Stuart, G. J. (2016). The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Frontiers in cellular neuroscience, 10206. https://doi.org/10.3389/fncel.2016.00206
Bock, Tobias, and Stuart, Greg J. "The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location." Frontiers in cellular neuroscience vol. 10 (2016): 206. doi: https://doi.org/10.3389/fncel.2016.00206
Bock T, Stuart GJ. The Impact of BK Channels on Cellular Excitability Depends on their Subcellular Location. Front Cell Neurosci. 2016 Aug 31;10:206. doi: 10.3389/fncel.2016.00206. eCollection 2016. PMID: 27630543; PMCID: PMC5006691.
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Molecular dynamics simulations of membrane proteins.

Biophysical reviews

Baştuğ T, Kuyucak S.
PMID: 28510077
Biophys Rev. 2012 Sep;4(3):271-282. doi: 10.1007/s12551-012-0084-9. Epub 2012 Sep 01.

Membrane proteins control the traffic across cell membranes and thereby play an essential role in cell function from transport of various solutes to immune response via molecular recognition. Because it is very difficult to determine the structures of membrane...

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Baştuğ T, Kuyucak S. Molecular dynamics simulations of membrane proteins. Biophys Rev. 2012;4(3):271-282doi: 10.1007/s12551-012-0084-9.
Baştuğ, T., & Kuyucak, S. (2012). Molecular dynamics simulations of membrane proteins. Biophysical reviews, 4(3), 271-282. https://doi.org/10.1007/s12551-012-0084-9
Baştuğ, Turgut, and Kuyucak, Serdar. "Molecular dynamics simulations of membrane proteins." Biophysical reviews vol. 4,3 (2012): 271-282. doi: https://doi.org/10.1007/s12551-012-0084-9
Baştuğ T, Kuyucak S. Molecular dynamics simulations of membrane proteins. Biophys Rev. 2012 Sep;4(3):271-282. doi: 10.1007/s12551-012-0084-9. Epub 2012 Sep 01. PMID: 28510077; PMCID: PMC5418402.
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Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy.

Scientific reports

Sicca F, Ambrosini E, Marchese M, Sforna L, Servettini I, Valvo G, Brignone MS, Lanciotti A, Moro F, Grottesi A, Catacuzzeno L, Baldini S, Hasan S, D'Adamo MC, Franciolini F, Molinari P, Santorelli FM, Pessia M.
PMID: 27677466
Sci Rep. 2016 Sep 28;6:34325. doi: 10.1038/srep34325.

Dysfunction of the inwardly-rectifying potassium channels Kir4.1 (KCNJ10) represents a pathogenic mechanism contributing to Autism-Epilepsy comorbidity. To define the role of Kir4.1 variants in the disorder, we sequenced KCNJ10 in a sample of affected individuals, and performed genotype-phenotype correlations....

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Sicca F, Ambrosini E, Marchese M, et al. Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy. Sci Rep. 2016;6:34325doi: 10.1038/srep34325.
Sicca, F., Ambrosini, E., Marchese, M., Sforna, L., Servettini, I., Valvo, G., Brignone, M. S., Lanciotti, A., Moro, F., Grottesi, A., Catacuzzeno, L., Baldini, S., Hasan, S., D'Adamo, M. C., Franciolini, F., Molinari, P., Santorelli, F. M., & Pessia, M. (2016). Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy. Scientific reports, 634325. https://doi.org/10.1038/srep34325
Sicca, Federico, et al. "Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy." Scientific reports vol. 6 (2016): 34325. doi: https://doi.org/10.1038/srep34325
Sicca F, Ambrosini E, Marchese M, Sforna L, Servettini I, Valvo G, Brignone MS, Lanciotti A, Moro F, Grottesi A, Catacuzzeno L, Baldini S, Hasan S, D'Adamo MC, Franciolini F, Molinari P, Santorelli FM, Pessia M. Gain-of-function defects of astrocytic Kir4.1 channels in children with autism spectrum disorders and epilepsy. Sci Rep. 2016 Sep 28;6:34325. doi: 10.1038/srep34325. PMID: 27677466; PMCID: PMC5039625.
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KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing.

Archives of plastic surgery

Shim JH, Lim JW, Kim BK, Park SJ, Kim SW, Choi TH.
PMID: 25606484
Arch Plast Surg. 2015 Jan;42(1):11-9. doi: 10.5999/aps.2015.42.1.11. Epub 2015 Jan 14.

BACKGROUND: Wound healing is an interaction of a complex signaling cascade of cellular events, including inflammation, proliferation, and maturation. K(+) channels modulate the mitogen-activated protein kinase (MAPK) signaling pathway. Here, we investigated whether K(+) channel-activated MAPK signaling directs collagen...

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Shim JH, Lim JW, Kim BK, et al. KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Arch Plast Surg. 2015;42(1):11-9doi: 10.5999/aps.2015.42.1.11.
Shim, J. H., Lim, J. W., Kim, B. K., Park, S. J., Kim, S. W., & Choi, T. H. (2015). KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Archives of plastic surgery, 42(1), 11-9. https://doi.org/10.5999/aps.2015.42.1.11
Shim, Jung Hee, et al. "KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing." Archives of plastic surgery vol. 42,1 (2015): 11-9. doi: https://doi.org/10.5999/aps.2015.42.1.11
Shim JH, Lim JW, Kim BK, Park SJ, Kim SW, Choi TH. KCl Mediates K(+) Channel-Activated Mitogen-Activated Protein Kinases Signaling in Wound Healing. Arch Plast Surg. 2015 Jan;42(1):11-9. doi: 10.5999/aps.2015.42.1.11. Epub 2015 Jan 14. PMID: 25606484; PMCID: PMC4297800.
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Autoimmune encephalopathy and drug refractory seizures with the presence of two autoantibodies specific for the neuronal cell surface.

Epilepsy & behavior case reports

Al-Ajlan FS, Althobiti A, Baz S, Al-Attas A.
PMID: 25667907
Epilepsy Behav Case Rep. 2014 Nov 27;2:199-202. doi: 10.1016/j.ebcr.2014.06.002. eCollection 2014.

BACKGROUND: An increasing number of autoantibodies are being described in epilepsy and other seizure-related disorders. A pathogenic role of autoantibodies in epilepsy has been suggested based on observations of the efficacy of immunotherapy.OBJECTIVE: This study aimed to report a...

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Al-Ajlan FS, Althobiti A, Baz S, et al. Autoimmune encephalopathy and drug refractory seizures with the presence of two autoantibodies specific for the neuronal cell surface. Epilepsy Behav Case Rep. 2014;2:199-202doi: 10.1016/j.ebcr.2014.06.002.
Al-Ajlan, F. S., Althobiti, A., Baz, S., & Al-Attas, A. (2014). Autoimmune encephalopathy and drug refractory seizures with the presence of two autoantibodies specific for the neuronal cell surface. Epilepsy & behavior case reports, 2199-202. https://doi.org/10.1016/j.ebcr.2014.06.002
Al-Ajlan, Fahad S, et al. "Autoimmune encephalopathy and drug refractory seizures with the presence of two autoantibodies specific for the neuronal cell surface." Epilepsy & behavior case reports vol. 2 (2014): 199-202. doi: https://doi.org/10.1016/j.ebcr.2014.06.002
Al-Ajlan FS, Althobiti A, Baz S, Al-Attas A. Autoimmune encephalopathy and drug refractory seizures with the presence of two autoantibodies specific for the neuronal cell surface. Epilepsy Behav Case Rep. 2014 Nov 27;2:199-202. doi: 10.1016/j.ebcr.2014.06.002. eCollection 2014. PMID: 25667907; PMCID: PMC4307881.
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Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels.

Basic research in cardiology

[No authors listed]
PMID: 27519302
Basic Res Cardiol. 2004 May;99(3):239. doi: 10.1007/s00395-004-0464-9.

No abstract available.

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Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels. Basic Res Cardiol. 2004;99(3):239doi: 10.1007/s00395-004-0464-9.
(2004). Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels. Basic research in cardiology, 99(3), 239. https://doi.org/10.1007/s00395-004-0464-9
"Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels." Basic research in cardiology vol. 99,3 (2004): 239. doi: https://doi.org/10.1007/s00395-004-0464-9
Acute cardioprotective effects of erythropoietin in infant rabbits are mediated by activation of protein kinases and potassium channels. Basic Res Cardiol. 2004 May;99(3):239. doi: 10.1007/s00395-004-0464-9. PMID: 27519302.
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Computational Analysis of the Mode of Action of Disopyramide and Quinidine on hERG-Linked Short QT Syndrome in Human Ventricles.

Frontiers in physiology

Whittaker DG, Ni H, Benson AP, Hancox JC, Zhang H.
PMID: 29085299
Front Physiol. 2017 Oct 04;8:759. doi: 10.3389/fphys.2017.00759. eCollection 2017.

The short QT syndrome (SQTS) is a rare cardiac disorder associated with arrhythmias and sudden death. Gain-of-function mutations to potassium channels mediating the rapid delayed rectifier current,

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Whittaker DG, Ni H, Benson AP, et al. Computational Analysis of the Mode of Action of Disopyramide and Quinidine on hERG-Linked Short QT Syndrome in Human Ventricles. Front Physiol. 2017;8:759doi: 10.3389/fphys.2017.00759.
Whittaker, D. G., Ni, H., Benson, A. P., Hancox, J. C., & Zhang, H. (2017). Computational Analysis of the Mode of Action of Disopyramide and Quinidine on hERG-Linked Short QT Syndrome in Human Ventricles. Frontiers in physiology, 8759. https://doi.org/10.3389/fphys.2017.00759
Whittaker, Dominic G, et al. "Computational Analysis of the Mode of Action of Disopyramide and Quinidine on hERG-Linked Short QT Syndrome in Human Ventricles." Frontiers in physiology vol. 8 (2017): 759. doi: https://doi.org/10.3389/fphys.2017.00759
Whittaker DG, Ni H, Benson AP, Hancox JC, Zhang H. Computational Analysis of the Mode of Action of Disopyramide and Quinidine on hERG-Linked Short QT Syndrome in Human Ventricles. Front Physiol. 2017 Oct 04;8:759. doi: 10.3389/fphys.2017.00759. eCollection 2017. PMID: 29085299; PMCID: PMC5649182.
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Presynaptic mechanisms of neuronal plasticity and their role in epilepsy.

Frontiers in cellular neuroscience

Meier JC, Meier J, Semtner M, Winkelmann A, Wolfart J.
PMID: 24987332
Front Cell Neurosci. 2014 Jun 17;8:164. doi: 10.3389/fncel.2014.00164. eCollection 2014.

Synaptic communication requires constant adjustments of pre- and postsynaptic efficacies. In addition to synaptic long term plasticity, the presynaptic machinery underlies homeostatic regulations which prevent out of range transmitter release. In this minireview we will discuss the relevance of...

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Meier JC, Meier J, Semtner M, et al. Presynaptic mechanisms of neuronal plasticity and their role in epilepsy. Front Cell Neurosci. 2014;8:164doi: 10.3389/fncel.2014.00164.
Meier, J. C., Meier, J., Semtner, M., Winkelmann, A., & Wolfart, J. (2014). Presynaptic mechanisms of neuronal plasticity and their role in epilepsy. Frontiers in cellular neuroscience, 8164. https://doi.org/10.3389/fncel.2014.00164
Meier, Jochen C, et al. "Presynaptic mechanisms of neuronal plasticity and their role in epilepsy." Frontiers in cellular neuroscience vol. 8 (2014): 164. doi: https://doi.org/10.3389/fncel.2014.00164
Meier JC, Meier J, Semtner M, Winkelmann A, Wolfart J. Presynaptic mechanisms of neuronal plasticity and their role in epilepsy. Front Cell Neurosci. 2014 Jun 17;8:164. doi: 10.3389/fncel.2014.00164. eCollection 2014. PMID: 24987332; PMCID: PMC4060558.
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Showing 1 to 12 of 852 entries