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Effects of saikosaponins on hepatic damage induced by halothane and hypoxia in phenobarbital-pretreated rats.

Journal of anesthesia

Nishiura T, Marukawa S, Ishida H, Orita M, Abe H.
PMID: 28921207
J Anesth. 1994 Mar;8(1):87-92. doi: 10.1007/BF02482762.

The effects of saikosaponins-a.-b

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Nishiura T, Marukawa S, Ishida H, et al. Effects of saikosaponins on hepatic damage induced by halothane and hypoxia in phenobarbital-pretreated rats. J Anesth. 1994;8(1):87-92doi: 10.1007/BF02482762.
Nishiura, T., Marukawa, S., Ishida, H., Orita, M., & Abe, H. (1994). Effects of saikosaponins on hepatic damage induced by halothane and hypoxia in phenobarbital-pretreated rats. Journal of anesthesia, 8(1), 87-92. https://doi.org/10.1007/BF02482762
Nishiura, Teruhiro, et al. "Effects of saikosaponins on hepatic damage induced by halothane and hypoxia in phenobarbital-pretreated rats." Journal of anesthesia vol. 8,1 (1994): 87-92. doi: https://doi.org/10.1007/BF02482762
Nishiura T, Marukawa S, Ishida H, Orita M, Abe H. Effects of saikosaponins on hepatic damage induced by halothane and hypoxia in phenobarbital-pretreated rats. J Anesth. 1994 Mar;8(1):87-92. doi: 10.1007/BF02482762. PMID: 28921207.
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Phenobarbital and Phototherapy Combination Enhances Decline of Total Serum Bilirubin and May Decrease the Need for Blood Exchange Transfusion in Newborns with Isoimmune Hemolytic Disease.

Clinical medicine insights. Pediatrics

Kaabneh MA, Salama GS, Shakkoury AG, Al-Abdallah IM, Alshamari A, Halaseh RA.
PMID: 26309423
Clin Med Insights Pediatr. 2015 Aug 09;9:67-72. doi: 10.4137/CMPed.S24909. eCollection 2015.

OBJECTIVE: The objective of this study was to evaluate the effect of phenobarbital and phototherapy combination on the total serum bilirubin of the newborn infants with isoimmune hemolytic disease (IHD) and its impact on blood exchange transfusion rates.PATIENTS AND...

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Kaabneh MA, Salama GS, Shakkoury AG, et al. Phenobarbital and Phototherapy Combination Enhances Decline of Total Serum Bilirubin and May Decrease the Need for Blood Exchange Transfusion in Newborns with Isoimmune Hemolytic Disease. Clin Med Insights Pediatr. 2015;9:67-72doi: 10.4137/CMPed.S24909.
Kaabneh, M. A., Salama, G. S., Shakkoury, A. G., Al-Abdallah, I. M., Alshamari, A., & Halaseh, R. A. (2015). Phenobarbital and Phototherapy Combination Enhances Decline of Total Serum Bilirubin and May Decrease the Need for Blood Exchange Transfusion in Newborns with Isoimmune Hemolytic Disease. Clinical medicine insights. Pediatrics, 967-72. https://doi.org/10.4137/CMPed.S24909
Kaabneh, Mahmoud Af, et al. "Phenobarbital and Phototherapy Combination Enhances Decline of Total Serum Bilirubin and May Decrease the Need for Blood Exchange Transfusion in Newborns with Isoimmune Hemolytic Disease." Clinical medicine insights. Pediatrics vol. 9 (2015): 67-72. doi: https://doi.org/10.4137/CMPed.S24909
Kaabneh MA, Salama GS, Shakkoury AG, Al-Abdallah IM, Alshamari A, Halaseh RA. Phenobarbital and Phototherapy Combination Enhances Decline of Total Serum Bilirubin and May Decrease the Need for Blood Exchange Transfusion in Newborns with Isoimmune Hemolytic Disease. Clin Med Insights Pediatr. 2015 Aug 09;9:67-72. doi: 10.4137/CMPed.S24909. eCollection 2015. PMID: 26309423; PMCID: PMC4531018.
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Clinical multifactorial analysis of early postoperative seizures in elderly patients following meningioma resection.

Molecular and clinical oncology

Zhang BO, Wang D, Guo Y, Yu J.
PMID: 26137257
Mol Clin Oncol. 2015 May;3(3):501-505. doi: 10.3892/mco.2015.493. Epub 2015 Jan 22.

The aim of the present study was to identify the major factors correlated with early postoperative seizures in elderly patients who had undergone a meningioma resection, and subsequently, to develop a logistic regression equation for assessing the seizures risk....

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Zhang BO, Wang D, Guo Y, et al. Clinical multifactorial analysis of early postoperative seizures in elderly patients following meningioma resection. Mol Clin Oncol. 2015;3(3):501-505doi: 10.3892/mco.2015.493.
Zhang, B. O., Wang, D., Guo, Y., & Yu, J. (2015). Clinical multifactorial analysis of early postoperative seizures in elderly patients following meningioma resection. Molecular and clinical oncology, 3(3), 501-505. https://doi.org/10.3892/mco.2015.493
Zhang, B O, et al. "Clinical multifactorial analysis of early postoperative seizures in elderly patients following meningioma resection." Molecular and clinical oncology vol. 3,3 (2015): 501-505. doi: https://doi.org/10.3892/mco.2015.493
Zhang BO, Wang D, Guo Y, Yu J. Clinical multifactorial analysis of early postoperative seizures in elderly patients following meningioma resection. Mol Clin Oncol. 2015 May;3(3):501-505. doi: 10.3892/mco.2015.493. Epub 2015 Jan 22. PMID: 26137257; PMCID: PMC4471514.
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An unexpected prolonged coma due to a pharmacobezoar formed from Vegetamine A® tablet ingestion.

Acute medicine & surgery

Sato C, Kamijo Y.
PMID: 29123693
Acute Med Surg. 2014 Jul 14;2(1):60-63. doi: 10.1002/ams2.58. eCollection 2015 Jan.

CASE: A 49-year-old man ingested massive quantities of Vegetamine A® tablets that contain two anticholinergic agents in addition to phenobarbital. The patient remained in an unexpectedly prolonged coma 4 days post-hospitalization. An acute gastroscopy revealed a pinkish-white pharmacobezoar on...

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Sato C, Kamijo Y. An unexpected prolonged coma due to a pharmacobezoar formed from Vegetamine A® tablet ingestion. Acute Med Surg. 2014;2(1):60-63doi: 10.1002/ams2.58.
Sato, C., & Kamijo, Y. (2015). An unexpected prolonged coma due to a pharmacobezoar formed from Vegetamine A® tablet ingestion. Acute medicine & surgery, 2(1), 60-63. https://doi.org/10.1002/ams2.58
Sato, Chie, and Kamijo, Yoshito. "An unexpected prolonged coma due to a pharmacobezoar formed from Vegetamine A® tablet ingestion." Acute medicine & surgery vol. 2,1 (2015): 60-63. doi: https://doi.org/10.1002/ams2.58
Sato C, Kamijo Y. An unexpected prolonged coma due to a pharmacobezoar formed from Vegetamine A® tablet ingestion. Acute Med Surg. 2014 Jul 14;2(1):60-63. doi: 10.1002/ams2.58. eCollection 2015 Jan. PMID: 29123693; PMCID: PMC5667188.
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Release of(14)C-adenine derivatives from cerebral cortical slices: Effect of phenytoin and phenobarbital.

Neurochemical research

Lewin E, Bleck V.
PMID: 24271574
Neurochem Res. 1976 Aug;1(4):429-35. doi: 10.1007/BF00966234.

Both ouabain, 0.1 mM, and veratridine, 0.05 mM, increased the release of(14)C-labeled compounds from rat cortical slices prelabeled with(14)C-adenine and incubated in vitro. The increment in radioactivity released by both depolarizing agents was almost entirely a result of increases...

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Lewin E, Bleck V. Release of(14)C-adenine derivatives from cerebral cortical slices: Effect of phenytoin and phenobarbital. Neurochem Res. 1976;1(4):429-35doi: 10.1007/BF00966234.
Lewin, E., & Bleck, V. (1976). Release of(14)C-adenine derivatives from cerebral cortical slices: Effect of phenytoin and phenobarbital. Neurochemical research, 1(4), 429-35. https://doi.org/10.1007/BF00966234
Lewin, E, and Bleck, V. "Release of(14)C-adenine derivatives from cerebral cortical slices: Effect of phenytoin and phenobarbital." Neurochemical research vol. 1,4 (1976): 429-35. doi: https://doi.org/10.1007/BF00966234
Lewin E, Bleck V. Release of(14)C-adenine derivatives from cerebral cortical slices: Effect of phenytoin and phenobarbital. Neurochem Res. 1976 Aug;1(4):429-35. doi: 10.1007/BF00966234. PMID: 24271574.
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Risk factors for seizures and antiepileptic drug-associated adverse effects in high-grade glioma patients: A multicentre, retrospective study in Hong Kong.

Surgical practice

Woo PY, Chan DT, Chan KY, Wong WK, Po YC, Kwok JC, Poon WS.
PMID: 26097495
Surg Pract. 2015 Feb;19(1):2-8. doi: 10.1111/1744-1633.12102. Epub 2015 Jan 11.

AIM: The aim of this present study was to determine the frequency, as well as risk factors, for seizures and antiepileptic drug (AED)-associated adverse effects among high-grade glioma (HGG) patients.PATIENTS AND METHODS: A multicentre, retrospective study of adult Chinese...

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Woo PY, Chan DT, Chan KY, et al. Risk factors for seizures and antiepileptic drug-associated adverse effects in high-grade glioma patients: A multicentre, retrospective study in Hong Kong. Surg Pract. 2015;19(1):2-8doi: 10.1111/1744-1633.12102.
Woo, P. Y., Chan, D. T., Chan, K. Y., Wong, W. K., Po, Y. C., Kwok, J. C., & Poon, W. S. (2015). Risk factors for seizures and antiepileptic drug-associated adverse effects in high-grade glioma patients: A multicentre, retrospective study in Hong Kong. Surgical practice, 19(1), 2-8. https://doi.org/10.1111/1744-1633.12102
Woo, Peter Yat-Ming, et al. "Risk factors for seizures and antiepileptic drug-associated adverse effects in high-grade glioma patients: A multicentre, retrospective study in Hong Kong." Surgical practice vol. 19,1 (2015): 2-8. doi: https://doi.org/10.1111/1744-1633.12102
Woo PY, Chan DT, Chan KY, Wong WK, Po YC, Kwok JC, Poon WS. Risk factors for seizures and antiepileptic drug-associated adverse effects in high-grade glioma patients: A multicentre, retrospective study in Hong Kong. Surg Pract. 2015 Feb;19(1):2-8. doi: 10.1111/1744-1633.12102. Epub 2015 Jan 11. PMID: 26097495; PMCID: PMC4467242.
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Effect of Tissue-Culture Substratum and Extracellular Matrix Overlay on Liver-Selective and Xenobiotic Inducible Gene Expression in Primary Rat Hepatocytes.

In vitro toxicology

Sidhu JS, Farin FM, Kavanagh TJ, Omiecinski CJ.
PMID: 24817786
In Vitro Toxicol. 1994;7(3):225-242.

In a previous study (Sidhu et al., 1993), we demonstrated that a combination of certain cell culture media, hormone addition, and extracellular matrix (ECM) overlay coordinately modulated the expression of certain liver-selective genes in primary rat hepatocyte cultures, including...

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Sidhu JS, Farin FM, Kavanagh TJ, et al. Effect of Tissue-Culture Substratum and Extracellular Matrix Overlay on Liver-Selective and Xenobiotic Inducible Gene Expression in Primary Rat Hepatocytes. In Vitro Toxicol. 1994;7(3):225-242
Sidhu, J. S., Farin, F. M., Kavanagh, T. J., & Omiecinski, C. J. (1994). Effect of Tissue-Culture Substratum and Extracellular Matrix Overlay on Liver-Selective and Xenobiotic Inducible Gene Expression in Primary Rat Hepatocytes. In vitro toxicology, 7(3), 225-242.
Sidhu, J S, et al. "Effect of Tissue-Culture Substratum and Extracellular Matrix Overlay on Liver-Selective and Xenobiotic Inducible Gene Expression in Primary Rat Hepatocytes." In vitro toxicology vol. 7,3 (1994): 225-242.
Sidhu JS, Farin FM, Kavanagh TJ, Omiecinski CJ. Effect of Tissue-Culture Substratum and Extracellular Matrix Overlay on Liver-Selective and Xenobiotic Inducible Gene Expression in Primary Rat Hepatocytes. In Vitro Toxicol. 1994;7(3):225-242. PMID: 24817786; PMCID: PMC4012392.
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Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transporters.

Frontiers in cellular neuroscience

Kang SK, Markowitz GJ, Kim ST, Johnston MV, Kadam SD.
PMID: 26029047
Front Cell Neurosci. 2015 May 12;9:173. doi: 10.3389/fncel.2015.00173. eCollection 2015.

Ischemia in the immature brain is an important cause of neonatal seizures. Temporal evolution of acquired neonatal seizures and their response to anticonvulsants are of great interest, given the unreliability of the clinical correlates and poor efficacy of first-line...

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Kang SK, Markowitz GJ, Kim ST, et al. Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transporters. Front Cell Neurosci. 2015;9:173doi: 10.3389/fncel.2015.00173.
Kang, S. K., Markowitz, G. J., Kim, S. T., Johnston, M. V., & Kadam, S. D. (2015). Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transporters. Frontiers in cellular neuroscience, 9173. https://doi.org/10.3389/fncel.2015.00173
Kang, Seok Kyu, et al. "Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transporters." Frontiers in cellular neuroscience vol. 9 (2015): 173. doi: https://doi.org/10.3389/fncel.2015.00173
Kang SK, Markowitz GJ, Kim ST, Johnston MV, Kadam SD. Age- and sex-dependent susceptibility to phenobarbital-resistant neonatal seizures: role of chloride co-transporters. Front Cell Neurosci. 2015 May 12;9:173. doi: 10.3389/fncel.2015.00173. eCollection 2015. PMID: 26029047; PMCID: PMC4429249.
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Update on the Genetic Polymorphisms of Drug-Metabolizing Enzymes in Antiepileptic Drug Therapy.

Pharmaceuticals (Basel, Switzerland)

Saruwatari J, Ishitsu T, Nakagawa K.
PMID: 27713373
Pharmaceuticals (Basel). 2010 Aug 20;3(8):2709-2732. doi: 10.3390/ph3082709.

Genetic polymorphisms in the genes that encode drug-metabolizing enzymes are implicated in the inter-individual variability in the pharmacokinetics and pharmaco-dynamics of antiepileptic drugs (AEDs). However, the clinical impact of these polymorphisms on AED therapy still remains controversial. The defective...

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Saruwatari J, Ishitsu T, Nakagawa K. Update on the Genetic Polymorphisms of Drug-Metabolizing Enzymes in Antiepileptic Drug Therapy. Pharmaceuticals (Basel). 2010;3(8):2709-2732doi: 10.3390/ph3082709.
Saruwatari, J., Ishitsu, T., & Nakagawa, K. (2010). Update on the Genetic Polymorphisms of Drug-Metabolizing Enzymes in Antiepileptic Drug Therapy. Pharmaceuticals (Basel, Switzerland), 3(8), 2709-2732. https://doi.org/10.3390/ph3082709
Saruwatari, Junji, et al. "Update on the Genetic Polymorphisms of Drug-Metabolizing Enzymes in Antiepileptic Drug Therapy." Pharmaceuticals (Basel, Switzerland) vol. 3,8 (2010): 2709-2732. doi: https://doi.org/10.3390/ph3082709
Saruwatari J, Ishitsu T, Nakagawa K. Update on the Genetic Polymorphisms of Drug-Metabolizing Enzymes in Antiepileptic Drug Therapy. Pharmaceuticals (Basel). 2010 Aug 20;3(8):2709-2732. doi: 10.3390/ph3082709. PMID: 27713373; PMCID: PMC4033946.
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A Facile Electrochemical Preparation of Reduced Graphene Oxide@Polydopamine Composite: A Novel Electrochemical Sensing Platform for Amperometric Detection of Chlorpromazine.

Scientific reports

Palanisamy S, Thirumalraj B, Chen SM, Wang YT, Velusamy V, Ramaraj SK.
PMID: 27650697
Sci Rep. 2016 Sep 21;6:33599. doi: 10.1038/srep33599.

We report a novel and sensitive amperometric sensor for chlorpromazine (CPZ) based on reduced graphene oxide (RGO) and polydopamine (PDA) composite modified glassy carbon electrode. The RGO@PDA composite was prepared by electrochemical reduction of graphene oxide (GO) with PDA....

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Palanisamy S, Thirumalraj B, Chen SM, et al. A Facile Electrochemical Preparation of Reduced Graphene Oxide@Polydopamine Composite: A Novel Electrochemical Sensing Platform for Amperometric Detection of Chlorpromazine. Sci Rep. 2016;6:33599doi: 10.1038/srep33599.
Palanisamy, S., Thirumalraj, B., Chen, S. M., Wang, Y. T., Velusamy, V., & Ramaraj, S. K. (2016). A Facile Electrochemical Preparation of Reduced Graphene Oxide@Polydopamine Composite: A Novel Electrochemical Sensing Platform for Amperometric Detection of Chlorpromazine. Scientific reports, 633599. https://doi.org/10.1038/srep33599
Palanisamy, Selvakumar, et al. "A Facile Electrochemical Preparation of Reduced Graphene Oxide@Polydopamine Composite: A Novel Electrochemical Sensing Platform for Amperometric Detection of Chlorpromazine." Scientific reports vol. 6 (2016): 33599. doi: https://doi.org/10.1038/srep33599
Palanisamy S, Thirumalraj B, Chen SM, Wang YT, Velusamy V, Ramaraj SK. A Facile Electrochemical Preparation of Reduced Graphene Oxide@Polydopamine Composite: A Novel Electrochemical Sensing Platform for Amperometric Detection of Chlorpromazine. Sci Rep. 2016 Sep 21;6:33599. doi: 10.1038/srep33599. PMID: 27650697; PMCID: PMC5030524.
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Propylene Glycol Toxicity in Adolescent with Refractory Myoclonic Status Epilepticus.

Case reports in pediatrics

Bjur KA, Cannon BC, Fine AL, Ritter MJ, Schueler KE, Nemergut ME.
PMID: 28331645
Case Rep Pediatr. 2017;2017:2979486. doi: 10.1155/2017/2979486. Epub 2017 Feb 26.

Propylene glycol (PG) is a solvent commonly used in medications that, while benign at low doses, may cause toxicity in adults and children at high doses. We describe a case and the physiologic sequelae of propylene glycol toxicity manifested...

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Bjur KA, Cannon BC, Fine AL, et al. Propylene Glycol Toxicity in Adolescent with Refractory Myoclonic Status Epilepticus. Case Rep Pediatr. 2017;2017:2979486doi: 10.1155/2017/2979486.
Bjur, K. A., Cannon, B. C., Fine, A. L., Ritter, M. J., Schueler, K. E., & Nemergut, M. E. (2017). Propylene Glycol Toxicity in Adolescent with Refractory Myoclonic Status Epilepticus. Case reports in pediatrics, 20172979486. https://doi.org/10.1155/2017/2979486
Bjur, Kara A, et al. "Propylene Glycol Toxicity in Adolescent with Refractory Myoclonic Status Epilepticus." Case reports in pediatrics vol. 2017 (2017): 2979486. doi: https://doi.org/10.1155/2017/2979486
Bjur KA, Cannon BC, Fine AL, Ritter MJ, Schueler KE, Nemergut ME. Propylene Glycol Toxicity in Adolescent with Refractory Myoclonic Status Epilepticus. Case Rep Pediatr. 2017;2017:2979486. doi: 10.1155/2017/2979486. Epub 2017 Feb 26. PMID: 28331645; PMCID: PMC5346383.
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Does cytochrome P450 liver isoenzyme induction increase the risk of liver toxicity after paracetamol overdose?.

Open access emergency medicine : OAEM

Kalsi SS, Wood DM, Waring WS, Dargan PI.
PMID: 27147854
Open Access Emerg Med. 2011 Oct 13;3:69-76. doi: 10.2147/OAEM.S24962. eCollection 2011.

Paracetamol (acetaminophen, N-acetyl-p-aminophenol, 4-hydroxyacetanilide) is the most common cause of acute liver failure in developed countries. There are a number of factors which potentially impact on the risk of an individual developing hepatotoxicity following an acute paracetamol overdose. These...

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Kalsi SS, Wood DM, Waring WS, et al. Does cytochrome P450 liver isoenzyme induction increase the risk of liver toxicity after paracetamol overdose?. Open Access Emerg Med. 2011;3:69-76doi: 10.2147/OAEM.S24962.
Kalsi, S. S., Wood, D. M., Waring, W. S., & Dargan, P. I. (2011). Does cytochrome P450 liver isoenzyme induction increase the risk of liver toxicity after paracetamol overdose?. Open access emergency medicine : OAEM, 369-76. https://doi.org/10.2147/OAEM.S24962
Kalsi, Sarbjeet S, et al. "Does cytochrome P450 liver isoenzyme induction increase the risk of liver toxicity after paracetamol overdose?." Open access emergency medicine : OAEM vol. 3 (2011): 69-76. doi: https://doi.org/10.2147/OAEM.S24962
Kalsi SS, Wood DM, Waring WS, Dargan PI. Does cytochrome P450 liver isoenzyme induction increase the risk of liver toxicity after paracetamol overdose?. Open Access Emerg Med. 2011 Oct 13;3:69-76. doi: 10.2147/OAEM.S24962. eCollection 2011. PMID: 27147854; PMCID: PMC4753969.
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Showing 1 to 12 of 563 entries