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Cardiovascular effects of nitric oxide in the brain stem nuclei of rats

Stem education interventions

[No authors listed]
GSID: C-lr0sDhVUkJ
CJ Tseng, HY Liu, HC Lin, LP Ger, CS Tung… - Hypertension, 1996 - Am Heart Assoc

Nitric oxide, synthesized from the semiessential amino acid l-arginine by nitric oxide synthase, is a remarkable regulatory molecule and plays an important role in physiological functions. However, the physiological role of nitric oxide in cardiovascular regulation by the central...

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Tseng, C. J., Liu, H. Y., Lin, H. C., Ger, L. P., Tung, C. S., & Yen, M. H. (1996). Cardiovascular effects of nitric oxide in the brain stem nuclei of rats. Hypertension, 27(1), 36-42.
Tseng, Ching-Jiunn, et al. "Cardiovascular effects of nitric oxide in the brain stem nuclei of rats." Hypertension 27.1 (1996): 36-42.
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Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells.

Biology of sex differences

Addis R, Campesi I, Fois M, Capobianco G, Dessole S, Fenu G, Montella A, Cattaneo MG, Vicentini LM, Franconi F.
PMID: 25535548
Biol Sex Differ. 2014 Dec 14;5(1):18. doi: 10.1186/s13293-014-0018-2. eCollection 2014.

BACKGROUND: Human umbilical endothelial cells (HUVECs) are widely used to study the endothelial physiology and pathology that might be involved in sex and gender differences detected at the cardiovascular level. This study evaluated whether HUVECs are sexually dimorphic in...

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Addis R, Campesi I, Fois M, et al. Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biol Sex Differ. 2014;5(1):18doi: 10.1186/s13293-014-0018-2.
Addis, R., Campesi, I., Fois, M., Capobianco, G., Dessole, S., Fenu, G., Montella, A., Cattaneo, M. G., Vicentini, L. M., & Franconi, F. (2014). Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biology of sex differences, 5(1), 18. https://doi.org/10.1186/s13293-014-0018-2
Addis, Roberta, et al. "Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells." Biology of sex differences vol. 5,1 (2014): 18. doi: https://doi.org/10.1186/s13293-014-0018-2
Addis R, Campesi I, Fois M, Capobianco G, Dessole S, Fenu G, Montella A, Cattaneo MG, Vicentini LM, Franconi F. Human umbilical endothelial cells (HUVECs) have a sex: characterisation of the phenotype of male and female cells. Biol Sex Differ. 2014 Dec 14;5(1):18. doi: 10.1186/s13293-014-0018-2. eCollection 2014. PMID: 25535548; PMCID: PMC4273493.
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Mechanism of endothelial nitric oxide synthase phosphorylation and activation by tentacle extract from the jellyfish .

PeerJ

Wang B, Liu D, Wang C, Wang Q, Zhang H, Liu G, Tao X, Zhang L.
PMID: 28413728
PeerJ. 2017 Apr 11;5:e3172. doi: 10.7717/peerj.3172. eCollection 2017.

Our previous study demonstrated that tentacle extract (TE) from the jellyfish

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Wang B, Liu D, Wang C, et al. Mechanism of endothelial nitric oxide synthase phosphorylation and activation by tentacle extract from the jellyfish . PeerJ. 2017;5:e3172doi: 10.7717/peerj.3172.
Wang, B., Liu, D., Wang, C., Wang, Q., Zhang, H., Liu, G., Tao, X., & Zhang, L. (2017). Mechanism of endothelial nitric oxide synthase phosphorylation and activation by tentacle extract from the jellyfish . PeerJ, 5e3172. https://doi.org/10.7717/peerj.3172
Wang, Beilei, et al. "Mechanism of endothelial nitric oxide synthase phosphorylation and activation by tentacle extract from the jellyfish ." PeerJ vol. 5 (2017): e3172. doi: https://doi.org/10.7717/peerj.3172
Wang B, Liu D, Wang C, Wang Q, Zhang H, Liu G, Tao X, Zhang L. Mechanism of endothelial nitric oxide synthase phosphorylation and activation by tentacle extract from the jellyfish . PeerJ. 2017 Apr 11;5:e3172. doi: 10.7717/peerj.3172. eCollection 2017. PMID: 28413728; PMCID: PMC5390764.
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Expression of Concern: Induction of nitric oxide synthase-2 proceeds with the concomitant downregulation of the endogenous caveolin levels. J. Cell Sci. doi: 10.1242/jcs.01002.

Journal of cell science

Navarro-Lérida I, Portolés MT, Barrientos AÁ, Gavilanes F, Boscá L, Rodríguez-Crespo I.
PMID: 28855381
J Cell Sci. 2017 Oct 01;130(19):3415. doi: 10.1242/jcs.209981. Epub 2017 Aug 30.

No abstract available.

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Navarro-Lérida I, Portolés MT, Barrientos AÁ, et al. Expression of Concern: Induction of nitric oxide synthase-2 proceeds with the concomitant downregulation of the endogenous caveolin levels. J. Cell Sci. doi: 10.1242/jcs.01002. J Cell Sci. 2017;130(19):3415doi: 10.1242/jcs.209981.
Navarro-Lérida, I., Portolés, M. T., Barrientos, A. �. �., Gavilanes, F., Boscá, L., & Rodríguez-Crespo, I. (2017). Expression of Concern: Induction of nitric oxide synthase-2 proceeds with the concomitant downregulation of the endogenous caveolin levels. J. Cell Sci. doi: 10.1242/jcs.01002. Journal of cell science, 130(19), 3415. https://doi.org/10.1242/jcs.209981
Navarro-Lérida, Inmaculada, et al. "Expression of Concern: Induction of nitric oxide synthase-2 proceeds with the concomitant downregulation of the endogenous caveolin levels. J. Cell Sci. doi: 10.1242/jcs.01002." Journal of cell science vol. 130,19 (2017): 3415. doi: https://doi.org/10.1242/jcs.209981
Navarro-Lérida I, Portolés MT, Barrientos AÁ, Gavilanes F, Boscá L, Rodríguez-Crespo I. Expression of Concern: Induction of nitric oxide synthase-2 proceeds with the concomitant downregulation of the endogenous caveolin levels. J. Cell Sci. doi: 10.1242/jcs.01002. J Cell Sci. 2017 Oct 01;130(19):3415. doi: 10.1242/jcs.209981. Epub 2017 Aug 30. PMID: 28855381.
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Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy.

Pulmonary circulation

Mohamed NA, Davies RP, Lickiss PD, Ahmetaj-Shala B, Reed DM, Gashaw HH, Saleem H, Freeman GR, George PM, Wort SJ, Morales-Cano D, Barreira B, Tetley TD, Chester AH, Yacoub MH, Kirkby NS, Moreno L, Mitchell JA.
PMID: 28447910
Pulm Circ. 2017 Jul-Sep;7(3):643-653. doi: 10.1177/2045893217710224. Epub 2017 Jun 27.

Pulmonary arterial hypertension (PAH) is a progressive and debilitating condition. Despite promoting vasodilation, current drugs have a therapeutic window within which they are limited by systemic side effects. Nanomedicine uses nanoparticles to improve drug delivery and/or reduce side effects....

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Mohamed NA, Davies RP, Lickiss PD, et al. Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulm Circ. 2017;7(3):643-653doi: 10.1177/2045893217710224.
Mohamed, N. A., Davies, R. P., Lickiss, P. D., Ahmetaj-Shala, B., Reed, D. M., Gashaw, H. H., Saleem, H., Freeman, G. R., George, P. M., Wort, S. J., Morales-Cano, D., Barreira, B., Tetley, T. D., Chester, A. H., Yacoub, M. H., Kirkby, N. S., Moreno, L., & Mitchell, J. A. (2017). Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulmonary circulation, 7(3), 643-653. https://doi.org/10.1177/2045893217710224
Mohamed, Nura A, et al. "Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy." Pulmonary circulation vol. 7,3 (2017): 643-653. doi: https://doi.org/10.1177/2045893217710224
Mohamed NA, Davies RP, Lickiss PD, Ahmetaj-Shala B, Reed DM, Gashaw HH, Saleem H, Freeman GR, George PM, Wort SJ, Morales-Cano D, Barreira B, Tetley TD, Chester AH, Yacoub MH, Kirkby NS, Moreno L, Mitchell JA. Chemical and biological assessment of metal organic frameworks (MOFs) in pulmonary cells and in an acute in vivo model: relevance to pulmonary arterial hypertension therapy. Pulm Circ. 2017 Jul-Sep;7(3):643-653. doi: 10.1177/2045893217710224. Epub 2017 Jun 27. PMID: 28447910; PMCID: PMC5841901.
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Safety, pharmacokinetics, and pharmacodynamics of oral omaveloxolone (RTA 408), a synthetic triterpenoid, in a first-in-human trial of patients with advanced solid tumors.

OncoTargets and therapy

Creelan BC, Gabrilovich DI, Gray JE, Williams CC, Tanvetyanon T, Haura EB, Weber JS, Gibney GT, Markowitz J, Proksch JW, Reisman SA, McKee MD, Chin MP, Meyer CJ, Antonia SJ.
PMID: 28919776
Onco Targets Ther. 2017 Aug 29;10:4239-4250. doi: 10.2147/OTT.S136992. eCollection 2017.

BACKGROUND: Omaveloxolone is a semisynthetic oleanane triterpenoid that potently activates Nrf2 with subsequent antioxidant function. We conducted a first-in-human Phase I clinical trial (NCT02029729) with the primary objectives to determine the appropriate dose for Phase II studies, characterize pharmacokinetic...

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Creelan BC, Gabrilovich DI, Gray JE, et al. Safety, pharmacokinetics, and pharmacodynamics of oral omaveloxolone (RTA 408), a synthetic triterpenoid, in a first-in-human trial of patients with advanced solid tumors. Onco Targets Ther. 2017;10:4239-4250doi: 10.2147/OTT.S136992.
Creelan, B. C., Gabrilovich, D. I., Gray, J. E., Williams, C. C., Tanvetyanon, T., Haura, E. B., Weber, J. S., Gibney, G. T., Markowitz, J., Proksch, J. W., Reisman, S. A., McKee, M. D., Chin, M. P., Meyer, C. J., & Antonia, S. J. (2017). Safety, pharmacokinetics, and pharmacodynamics of oral omaveloxolone (RTA 408), a synthetic triterpenoid, in a first-in-human trial of patients with advanced solid tumors. OncoTargets and therapy, 104239-4250. https://doi.org/10.2147/OTT.S136992
Creelan, Ben C, et al. "Safety, pharmacokinetics, and pharmacodynamics of oral omaveloxolone (RTA 408), a synthetic triterpenoid, in a first-in-human trial of patients with advanced solid tumors." OncoTargets and therapy vol. 10 (2017): 4239-4250. doi: https://doi.org/10.2147/OTT.S136992
Creelan BC, Gabrilovich DI, Gray JE, Williams CC, Tanvetyanon T, Haura EB, Weber JS, Gibney GT, Markowitz J, Proksch JW, Reisman SA, McKee MD, Chin MP, Meyer CJ, Antonia SJ. Safety, pharmacokinetics, and pharmacodynamics of oral omaveloxolone (RTA 408), a synthetic triterpenoid, in a first-in-human trial of patients with advanced solid tumors. Onco Targets Ther. 2017 Aug 29;10:4239-4250. doi: 10.2147/OTT.S136992. eCollection 2017. PMID: 28919776; PMCID: PMC5587199.
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Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus.

Frontiers in cellular neuroscience

Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K.
PMID: 28515682
Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017.

Corticotropin-releasing hormone (CRH)-synthesizing parvocellular neuroendocrine cells (PNCs) of the hypothalamic paraventricular nucleus (PVN) play a key role in the activation of the hypothalamic-pituitary-adrenocortical (HPA) axis. Several studies have demonstrated that synaptic inputs to these cells may undergo stress-related enhancement...

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Kusek M, Tokarska A, Siwiec M, et al. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017;11:127doi: 10.3389/fncel.2017.00127.
Kusek, M., Tokarska, A., Siwiec, M., Gadek-Michalska, A., Szewczyk, B., Hess, G., & Tokarski, K. (2017). Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Frontiers in cellular neuroscience, 11127. https://doi.org/10.3389/fncel.2017.00127
Kusek, Magdalena, et al. "Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus." Frontiers in cellular neuroscience vol. 11 (2017): 127. doi: https://doi.org/10.3389/fncel.2017.00127
Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017. PMID: 28515682; PMCID: PMC5413825.
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Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide.

Journal of gastroenterology and hepatology

Kawada N, Seki S, Kuroki T, Inoue M.
PMID: 28976688
J Gastroenterol Hepatol. 1998 Sep;13:S6-S13. doi: 10.1111/jgh.1998.13.s1.6.

We studied the effect of lipopolysaccharide (LPS) on the proliferation of culture-stimulated rat stellate cells. DNA synthesis as determined by [

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Kawada N, Seki S, Kuroki T, et al. Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. J Gastroenterol Hepatol. 1998;13:S6-S13doi: 10.1111/jgh.1998.13.s1.6.
Kawada, N., Seki, S., Kuroki, T., & Inoue, M. (1998). Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. Journal of gastroenterology and hepatology, 13S6-S13. https://doi.org/10.1111/jgh.1998.13.s1.6
Kawada, Norifumi, et al. "Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide." Journal of gastroenterology and hepatology vol. 13 (1998): S6-S13. doi: https://doi.org/10.1111/jgh.1998.13.s1.6
Kawada N, Seki S, Kuroki T, Inoue M. Regulation of stellate cell proliferation by lipopolysaccharide: Role of endogenous nitric oxide. J Gastroenterol Hepatol. 1998 Sep;13:S6-S13. doi: 10.1111/jgh.1998.13.s1.6. PMID: 28976688.
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The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis.

Experimental and therapeutic medicine

Shin SJ, Lee KH, Chung KS, Cheon SY, An HJ.
PMID: 28450936
Exp Ther Med. 2017 Mar;13(3):1025-1031. doi: 10.3892/etm.2017.4088. Epub 2017 Jan 25.

Benign prostatic hyperplasia (BPH) is a pathological condition that affects the majority of men above the age of 50 years. Pharmacological agents are typically used to treat BPH; however, there are currently no pharmacological agents that are able to...

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Shin SJ, Lee KH, Chung KS, et al. The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Exp Ther Med. 2017;13(3):1025-1031doi: 10.3892/etm.2017.4088.
Shin, S. J., Lee, K. H., Chung, K. S., Cheon, S. Y., & An, H. J. (2017). The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Experimental and therapeutic medicine, 13(3), 1025-1031. https://doi.org/10.3892/etm.2017.4088
Shin, Su-Jin, et al. "The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis." Experimental and therapeutic medicine vol. 13,3 (2017): 1025-1031. doi: https://doi.org/10.3892/etm.2017.4088
Shin SJ, Lee KH, Chung KS, Cheon SY, An HJ. The traditional Korean herbal medicine Ga-Gam-Nai-Go-Hyan suppresses testosterone-induced benign prostatic hyperplasia by regulating inflammatory responses and apoptosis. Exp Ther Med. 2017 Mar;13(3):1025-1031. doi: 10.3892/etm.2017.4088. Epub 2017 Jan 25. PMID: 28450936; PMCID: PMC5403478.
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Inhibitory effects of preimplantation exposure to bisphenol-A on blastocyst development and implantation.

International journal of clinical and experimental medicine

Pan X, Wang X, Sun Y, Dou Z, Li Z.
PMID: 26309523
Int J Clin Exp Med. 2015 Jun 15;8(6):8720-9. eCollection 2015.

The effect of preimplantation exposure to bisphenol-A (BPA) on blastocyst development and implantation is investigated. Mice were orally administered with BPA (200, 400, 600, and 800 mg/kg/day) from Day 0.5 to Day 3.5 of their pregnancy. Blastocyst development was...

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Pan X, Wang X, Sun Y, et al. Inhibitory effects of preimplantation exposure to bisphenol-A on blastocyst development and implantation. Int J Clin Exp Med. 2015;8(6):8720-9
Pan, X., Wang, X., Sun, Y., Dou, Z., & Li, Z. (2015). Inhibitory effects of preimplantation exposure to bisphenol-A on blastocyst development and implantation. International journal of clinical and experimental medicine, 8(6), 8720-9.
Pan, Xiaoyan, et al. "Inhibitory effects of preimplantation exposure to bisphenol-A on blastocyst development and implantation." International journal of clinical and experimental medicine vol. 8,6 (2015): 8720-9.
Pan X, Wang X, Sun Y, Dou Z, Li Z. Inhibitory effects of preimplantation exposure to bisphenol-A on blastocyst development and implantation. Int J Clin Exp Med. 2015 Jun 15;8(6):8720-9. eCollection 2015. PMID: 26309523; PMCID: PMC4538079.
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The role of reactive oxygen species in myocardial redox signaling and regulation.

Annals of translational medicine

Moris D, Spartalis M, Tzatzaki E, Spartalis E, Karachaliou GS, Triantafyllis AS, Karaolanis GI, Tsilimigras DI, Theocharis S.
PMID: 28861421
Ann Transl Med. 2017 Aug;5(16):324. doi: 10.21037/atm.2017.06.17.

Reactive oxygen species (ROS) are subcellular messengers in gene regulatory and signal transduction pathways. In pathological situations, ROS accumulate due to excessive production or insufficient degradation, leading to oxidative stress (OS). OS causes oxidation of DNA, membranes, cellular lipids,...

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Moris D, Spartalis M, Tzatzaki E, et al. The role of reactive oxygen species in myocardial redox signaling and regulation. Ann Transl Med. 2017;5(16):324doi: 10.21037/atm.2017.06.17.
Moris, D., Spartalis, M., Tzatzaki, E., Spartalis, E., Karachaliou, G. S., Triantafyllis, A. S., Karaolanis, G. I., Tsilimigras, D. I., & Theocharis, S. (2017). The role of reactive oxygen species in myocardial redox signaling and regulation. Annals of translational medicine, 5(16), 324. https://doi.org/10.21037/atm.2017.06.17
Moris, Demetrios, et al. "The role of reactive oxygen species in myocardial redox signaling and regulation." Annals of translational medicine vol. 5,16 (2017): 324. doi: https://doi.org/10.21037/atm.2017.06.17
Moris D, Spartalis M, Tzatzaki E, Spartalis E, Karachaliou GS, Triantafyllis AS, Karaolanis GI, Tsilimigras DI, Theocharis S. The role of reactive oxygen species in myocardial redox signaling and regulation. Ann Transl Med. 2017 Aug;5(16):324. doi: 10.21037/atm.2017.06.17. PMID: 28861421; PMCID: PMC5566737.
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Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosis.

OncoTargets and therapy

Lien CY, Chuang TY, Hsu CH, Lin CL, Wang SE, Sheu SJ, Chien CT, Wu CH.
PMID: 26060405
Onco Targets Ther. 2015 May 26;8:1193-210. doi: 10.2147/OTT.S82936. eCollection 2015.

OBJECTIVE: This study aimed to investigate whether the herbal formula B307 could alleviate doxorubicin (DOX)-induced acute cardiotoxicity. If so, we further unraveled possible molecular mechanisms of cardiac protection under treatment with the herbal formula B307.METHODS: Before the animal experiment,...

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Lien CY, Chuang TY, Hsu CH, et al. Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosis. Onco Targets Ther. 2015;8:1193-210doi: 10.2147/OTT.S82936.
Lien, C. Y., Chuang, T. Y., Hsu, C. H., Lin, C. L., Wang, S. E., Sheu, S. J., Chien, C. T., & Wu, C. H. (2015). Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosis. OncoTargets and therapy, 81193-210. https://doi.org/10.2147/OTT.S82936
Lien, Chia-Ying, et al. "Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosis." OncoTargets and therapy vol. 8 (2015): 1193-210. doi: https://doi.org/10.2147/OTT.S82936
Lien CY, Chuang TY, Hsu CH, Lin CL, Wang SE, Sheu SJ, Chien CT, Wu CH. Oral treatment with the herbal formula B307 alleviates cardiac toxicity in doxorubicin-treated mice via suppressing oxidative stress, inflammation, and apoptosis. Onco Targets Ther. 2015 May 26;8:1193-210. doi: 10.2147/OTT.S82936. eCollection 2015. PMID: 26060405; PMCID: PMC4454207.
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Showing 1 to 12 of 3476 entries