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Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model.

JACC. Basic to translational science

McCarthy JC, Aronovitz M, DuPont JJ, Calamaras TD, Jaffe IZ, Blanton RM.
PMID: 30062150
JACC Basic Transl Sci. 2017 Jun 26;2(3):285-296. doi: 10.1016/j.jacbts.2017.03.011. eCollection 2017 Jun.

In patients hospitalized with acute heart failure, temporary serelaxin infusion reduced 6-month mortality through unknown mechanisms. This study therefore explored the cardiovascular effects of temporary serelaxin administration in mice subjected to the angiotensin II (AngII)/L-NG-nitroarginine methyl ester (L-NAME) heart...

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McCarthy JC, Aronovitz M, DuPont JJ, et al. Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model. JACC Basic Transl Sci. 2017;2(3):285-296doi: 10.1016/j.jacbts.2017.03.011.
McCarthy, J. C., Aronovitz, M., DuPont, J. J., Calamaras, T. D., Jaffe, I. Z., & Blanton, R. M. (2017). Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model. JACC. Basic to translational science, 2(3), 285-296. https://doi.org/10.1016/j.jacbts.2017.03.011
McCarthy, Joseph C, et al. "Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model." JACC. Basic to translational science vol. 2,3 (2017): 285-296. doi: https://doi.org/10.1016/j.jacbts.2017.03.011
McCarthy JC, Aronovitz M, DuPont JJ, Calamaras TD, Jaffe IZ, Blanton RM. Short-Term Administration of Serelaxin Produces Predominantly Vascular Benefits in the Angiotensin II/L-NAME Chronic Heart Failure Model. JACC Basic Transl Sci. 2017 Jun 26;2(3):285-296. doi: 10.1016/j.jacbts.2017.03.011. eCollection 2017 Jun. PMID: 30062150; PMCID: PMC6034497.
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Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model.

World neurosurgery

Nishida S, Kawauchi S, Toyooka T, Kumagai K, Takeuchi S, Sato S, Kondo A, Wada K, Mori K.
PMID: 34500101
World Neurosurg. 2021 Nov;155:e704-e715. doi: 10.1016/j.wneu.2021.08.130. Epub 2021 Sep 06.

OBJECTIVE: Cortical spreading depolarization (CSD), cortical spreading ischemia (CSI), and early brain injury are involved in the occurrence of delayed brain ischemia after subarachnoid hemorrhage (SAH). We tested whether local application of magnesium (Mg) sulfate solution suppressed CSD and...

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Nishida S, Kawauchi S, Toyooka T, et al. Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World Neurosurg. 2021;155:e704-e715doi: 10.1016/j.wneu.2021.08.130.
Nishida, S., Kawauchi, S., Toyooka, T., Kumagai, K., Takeuchi, S., Sato, S., Kondo, A., Wada, K., & Mori, K. (2021). Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World neurosurgery, 155e704-e715. https://doi.org/10.1016/j.wneu.2021.08.130
Nishida, Sho, et al. "Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model." World neurosurgery vol. 155 (2021): e704-e715. doi: https://doi.org/10.1016/j.wneu.2021.08.130
Nishida S, Kawauchi S, Toyooka T, Kumagai K, Takeuchi S, Sato S, Kondo A, Wada K, Mori K. Local Application of Magnesium Sulfate Solution Suppressed Cortical Spreading Ischemia and Reduced Brain Damage in a Rat Subarachnoid Hemorrhage-Mimicking Model. World Neurosurg. 2021 Nov;155:e704-e715. doi: 10.1016/j.wneu.2021.08.130. Epub 2021 Sep 06. PMID: 34500101.
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Nitric Oxide Synthase Is Involved in Follicular Development via the PI3K/AKT/FoxO3a Pathway in Neonatal and Immature Rats.

Animals : an open access journal from MDPI

Li J, Zhang W, Zhu S, Shi F.
PMID: 32033275
Animals (Basel). 2020 Feb 05;10(2). doi: 10.3390/ani10020248.

It is assumed that nitric oxide synthase and nitric oxide are involved in the regulation of female reproduction. This study aimed to assess the roles of nitric oxide synthase (NOS) in follicular development. The endothelial NOS (eNOS) inhibitor L-NAME,...

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Li J, Zhang W, Zhu S, et al. Nitric Oxide Synthase Is Involved in Follicular Development via the PI3K/AKT/FoxO3a Pathway in Neonatal and Immature Rats. Animals (Basel). 2020;10(2)doi: 10.3390/ani10020248.
Li, J., Zhang, W., Zhu, S., & Shi, F. (2020). Nitric Oxide Synthase Is Involved in Follicular Development via the PI3K/AKT/FoxO3a Pathway in Neonatal and Immature Rats. Animals : an open access journal from MDPI, 10(2), . https://doi.org/10.3390/ani10020248
Li, Junrong, et al. "Nitric Oxide Synthase Is Involved in Follicular Development via the PI3K/AKT/FoxO3a Pathway in Neonatal and Immature Rats." Animals : an open access journal from MDPI vol. 10,2 (2020). doi: https://doi.org/10.3390/ani10020248
Li J, Zhang W, Zhu S, Shi F. Nitric Oxide Synthase Is Involved in Follicular Development via the PI3K/AKT/FoxO3a Pathway in Neonatal and Immature Rats. Animals (Basel). 2020 Feb 05;10(2). doi: 10.3390/ani10020248. PMID: 32033275; PMCID: PMC7070647.
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Effects of L-citrulline diet on stress-induced cold hypersensitivity in mice.

Pharmacognosy research

Kobayashi Y, Narita K, Chiba K, Takemoto H, Morita M, Morishita K.
PMID: 25276066
Pharmacognosy Res. 2014 Oct;6(4):297-302. doi: 10.4103/0974-8490.138269.

BACKGROUND: L-citrulline is an amino acid discovered in watermelon (Citrullus lanatus, Cucurbitaceae) and is a known component of the nitric oxide (NO) cycle that plays an important role in adjusting blood circulation and supplying NO and a key component...

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Kobayashi Y, Narita K, Chiba K, et al. Effects of L-citrulline diet on stress-induced cold hypersensitivity in mice. Pharmacognosy Res. 2014;6(4):297-302doi: 10.4103/0974-8490.138269.
Kobayashi, Y., Narita, K., Chiba, K., Takemoto, H., Morita, M., & Morishita, K. (2014). Effects of L-citrulline diet on stress-induced cold hypersensitivity in mice. Pharmacognosy research, 6(4), 297-302. https://doi.org/10.4103/0974-8490.138269
Kobayashi, Yoshinori, et al. "Effects of L-citrulline diet on stress-induced cold hypersensitivity in mice." Pharmacognosy research vol. 6,4 (2014): 297-302. doi: https://doi.org/10.4103/0974-8490.138269
Kobayashi Y, Narita K, Chiba K, Takemoto H, Morita M, Morishita K. Effects of L-citrulline diet on stress-induced cold hypersensitivity in mice. Pharmacognosy Res. 2014 Oct;6(4):297-302. doi: 10.4103/0974-8490.138269. PMID: 25276066; PMCID: PMC4166817.
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Ameliorating of Memory Impairment and Apoptosis in Amyloid β-Injected Rats Via Inhibition of Nitric Oxide Synthase: Possible Participation of Autophagy.

Iranian journal of pharmaceutical research : IJPR

Shariatpanahi M, Khodagholi F, Ashabi G, Aghazadeh Khasraghi A, Azimi L, Abdollahi M, Ghahremani MH, Ostad SN, Noorbakhsh F, Sharifzadeh M.
PMID: 26330869
Iran J Pharm Res. 2015;14(3):811-24.

It has been proposed that appearance of amyloid beta (Aβ) in hippocampus is one of the characteristic features of Alzheimer's disease (AD). The role of Nitric oxide (NO) in neurodegenerative disorders is controversy in different contexts. Here, we examined...

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Shariatpanahi M, Khodagholi F, Ashabi G, et al. Ameliorating of Memory Impairment and Apoptosis in Amyloid β-Injected Rats Via Inhibition of Nitric Oxide Synthase: Possible Participation of Autophagy. Iran J Pharm Res. 2015;14(3):811-24
Shariatpanahi, M., Khodagholi, F., Ashabi, G., Aghazadeh Khasraghi, A., Azimi, L., Abdollahi, M., Ghahremani, M. H., Ostad, S. N., Noorbakhsh, F., & Sharifzadeh, M. (2015). Ameliorating of Memory Impairment and Apoptosis in Amyloid β-Injected Rats Via Inhibition of Nitric Oxide Synthase: Possible Participation of Autophagy. Iranian journal of pharmaceutical research : IJPR, 14(3), 811-24.
Shariatpanahi, Marjan, et al. "Ameliorating of Memory Impairment and Apoptosis in Amyloid β-Injected Rats Via Inhibition of Nitric Oxide Synthase: Possible Participation of Autophagy." Iranian journal of pharmaceutical research : IJPR vol. 14,3 (2015): 811-24.
Shariatpanahi M, Khodagholi F, Ashabi G, Aghazadeh Khasraghi A, Azimi L, Abdollahi M, Ghahremani MH, Ostad SN, Noorbakhsh F, Sharifzadeh M. Ameliorating of Memory Impairment and Apoptosis in Amyloid β-Injected Rats Via Inhibition of Nitric Oxide Synthase: Possible Participation of Autophagy. Iran J Pharm Res. 2015;14(3):811-24. PMID: 26330869; PMCID: PMC4518109.
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The protective role of nitric oxide-dependent innate immunosuppression in the early stage of cartilage damage in rats: Role of nitric oxide in cartilage damage.

Bone & joint research

Hsu CC, Lin CL, Jou IM, Wang PH, Lee JS.
PMID: 28450318
Bone Joint Res. 2017 Apr;6(4):253-258. doi: 10.1302/2046-3758.64.BJJ-2016-0161.R1.

OBJECTIVES: Osteoarthritis (OA) is the most common form of arthritis, affecting approximately 15% of the human population. Recently, increased concentration of nitric oxide in serum and synovial fluid in patients with OA has been observed. However, the exact role...

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Hsu CC, Lin CL, Jou IM, et al. The protective role of nitric oxide-dependent innate immunosuppression in the early stage of cartilage damage in rats: Role of nitric oxide in cartilage damage. Bone Joint Res. 2017;6(4):253-258doi: 10.1302/2046-3758.64.BJJ-2016-0161.R1.
Hsu, C. C., Lin, C. L., Jou, I. M., Wang, P. H., & Lee, J. S. (2017). The protective role of nitric oxide-dependent innate immunosuppression in the early stage of cartilage damage in rats: Role of nitric oxide in cartilage damage. Bone & joint research, 6(4), 253-258. https://doi.org/10.1302/2046-3758.64.BJJ-2016-0161.R1
Hsu, C-C, et al. "The protective role of nitric oxide-dependent innate immunosuppression in the early stage of cartilage damage in rats: Role of nitric oxide in cartilage damage." Bone & joint research vol. 6,4 (2017): 253-258. doi: https://doi.org/10.1302/2046-3758.64.BJJ-2016-0161.R1
Hsu CC, Lin CL, Jou IM, Wang PH, Lee JS. The protective role of nitric oxide-dependent innate immunosuppression in the early stage of cartilage damage in rats: Role of nitric oxide in cartilage damage. Bone Joint Res. 2017 Apr;6(4):253-258. doi: 10.1302/2046-3758.64.BJJ-2016-0161.R1. PMID: 28450318; PMCID: PMC5426177.
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Cerebrovascular damage after midlife transient hypertension in non-transgenic and Alzheimer's disease rats.

Brain research

Lai AY, Joo IL, Trivedi AU, Dorr A, Hill ME, Stefanovic B, McLaurin J.
PMID: 33582120
Brain Res. 2021 May 01;1758:147369. doi: 10.1016/j.brainres.2021.147369. Epub 2021 Feb 12.

Hypertension, including transient events, is a major risk factor for developing late-onset dementia and Alzheimer's disease (AD). Anti-hypertensive drugs facilitate restoration of normotension without amelioration of increased dementia risk suggesting that transient hypertensive insults cause irreversible damage. This study...

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Lai AY, Joo IL, Trivedi AU, et al. Cerebrovascular damage after midlife transient hypertension in non-transgenic and Alzheimer's disease rats. Brain Res. 2021;1758:147369doi: 10.1016/j.brainres.2021.147369.
Lai, A. Y., Joo, I. L., Trivedi, A. U., Dorr, A., Hill, M. E., Stefanovic, B., & McLaurin, J. (2021). Cerebrovascular damage after midlife transient hypertension in non-transgenic and Alzheimer's disease rats. Brain research, 1758147369. https://doi.org/10.1016/j.brainres.2021.147369
Lai, Aaron Y, et al. "Cerebrovascular damage after midlife transient hypertension in non-transgenic and Alzheimer's disease rats." Brain research vol. 1758 (2021): 147369. doi: https://doi.org/10.1016/j.brainres.2021.147369
Lai AY, Joo IL, Trivedi AU, Dorr A, Hill ME, Stefanovic B, McLaurin J. Cerebrovascular damage after midlife transient hypertension in non-transgenic and Alzheimer's disease rats. Brain Res. 2021 May 01;1758:147369. doi: 10.1016/j.brainres.2021.147369. Epub 2021 Feb 12. PMID: 33582120; PMCID: PMC8005286.
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AMP-activated protein kinase regulates L-arginine mediated cellular responses.

Nutrition & metabolism

Mohan S, Patel H, Bolinaga J, Soekamto N.
PMID: 23718875
Nutr Metab (Lond). 2013 May 29;10(1):40. doi: 10.1186/1743-7075-10-40.

BACKGROUND: Our prior study revealed the loss in short-term L-Arginine (ARG) therapeutic efficacy after continuous exposure; resulting in tolerance development, mediated by endothelial nitric oxide synthase (eNOS) down-regulation, secondary to oxidative stress and induced glucose accumulation. However, the potential...

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Mohan S, Patel H, Bolinaga J, et al. AMP-activated protein kinase regulates L-arginine mediated cellular responses. Nutr Metab (Lond). 2013;10(1):40doi: 10.1186/1743-7075-10-40.
Mohan, S., Patel, H., Bolinaga, J., & Soekamto, N. (2013). AMP-activated protein kinase regulates L-arginine mediated cellular responses. Nutrition & metabolism, 10(1), 40. https://doi.org/10.1186/1743-7075-10-40
Mohan, Srinidi, et al. "AMP-activated protein kinase regulates L-arginine mediated cellular responses." Nutrition & metabolism vol. 10,1 (2013): 40. doi: https://doi.org/10.1186/1743-7075-10-40
Mohan S, Patel H, Bolinaga J, Soekamto N. AMP-activated protein kinase regulates L-arginine mediated cellular responses. Nutr Metab (Lond). 2013 May 29;10(1):40. doi: 10.1186/1743-7075-10-40. PMID: 23718875; PMCID: PMC3680329.
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Relaxing effect of acetylcholine on phenylephrine-induced contraction of isolated rabbit prostate strips is mediated by neuronal nitric oxide synthase.

Korean journal of urology

Nguyen HB, Lee SY, Park SH, Lee MY, Chang IH, Myung SC.
PMID: 23700500
Korean J Urol. 2013 May;54(5):333-8. doi: 10.4111/kju.2013.54.5.333. Epub 2013 May 14.

PURPOSE: The location of acetylcholinesterase-containing nerve fibers suggests a role for acetylcholine in both contractility and secretion in the prostate gland. The colocalization of nitrergic nerves with cholinergic nerves, and the cotransmission of nitric oxide with acetylcholine in cholinergic...

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Nguyen HB, Lee SY, Park SH, et al. Relaxing effect of acetylcholine on phenylephrine-induced contraction of isolated rabbit prostate strips is mediated by neuronal nitric oxide synthase. Korean J Urol. 2013;54(5):333-8doi: 10.4111/kju.2013.54.5.333.
Nguyen, H. B., Lee, S. Y., Park, S. H., Lee, M. Y., Chang, I. H., & Myung, S. C. (2013). Relaxing effect of acetylcholine on phenylephrine-induced contraction of isolated rabbit prostate strips is mediated by neuronal nitric oxide synthase. Korean journal of urology, 54(5), 333-8. https://doi.org/10.4111/kju.2013.54.5.333
Nguyen, Hoai Bac, et al. "Relaxing effect of acetylcholine on phenylephrine-induced contraction of isolated rabbit prostate strips is mediated by neuronal nitric oxide synthase." Korean journal of urology vol. 54,5 (2013): 333-8. doi: https://doi.org/10.4111/kju.2013.54.5.333
Nguyen HB, Lee SY, Park SH, Lee MY, Chang IH, Myung SC. Relaxing effect of acetylcholine on phenylephrine-induced contraction of isolated rabbit prostate strips is mediated by neuronal nitric oxide synthase. Korean J Urol. 2013 May;54(5):333-8. doi: 10.4111/kju.2013.54.5.333. Epub 2013 May 14. PMID: 23700500; PMCID: PMC3659228.
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The effect of L-arginine and L-NAME on myocardial capillary density in normal rats.

Advanced biomedical research

Khazaei M, Moshayedi MA, Jervekani MT, Aghili S, Montazer S, Dastjerdi RM, Hashemzehi F, Jazi HH.
PMID: 24223382
Adv Biomed Res. 2013 Jul 30;2:67. doi: 10.4103/2277-9175.115819. eCollection 2013.

BACKGROUND: This study evaluated the effect of L-arginine (Nitric Oxide (NO) precursor) and L-NG-Nitroarginine Methyl Ester (L-NAME) (NO synthase inhibitor) on myocardial capillary density in normal rats.MATERIALS AND METHODS: EIGHTEEN MALE RATS WERE DIVIDED INTO THREE GROUPS: Group 1:...

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Khazaei M, Moshayedi MA, Jervekani MT, et al. The effect of L-arginine and L-NAME on myocardial capillary density in normal rats. Adv Biomed Res. 2013;2:67doi: 10.4103/2277-9175.115819.
Khazaei, M., Moshayedi, M. A., Jervekani, M. T., Aghili, S., Montazer, S., Dastjerdi, R. M., Hashemzehi, F., & Jazi, H. H. (2013). The effect of L-arginine and L-NAME on myocardial capillary density in normal rats. Advanced biomedical research, 267. https://doi.org/10.4103/2277-9175.115819
Khazaei, Majid, et al. "The effect of L-arginine and L-NAME on myocardial capillary density in normal rats." Advanced biomedical research vol. 2 (2013): 67. doi: https://doi.org/10.4103/2277-9175.115819
Khazaei M, Moshayedi MA, Jervekani MT, Aghili S, Montazer S, Dastjerdi RM, Hashemzehi F, Jazi HH. The effect of L-arginine and L-NAME on myocardial capillary density in normal rats. Adv Biomed Res. 2013 Jul 30;2:67. doi: 10.4103/2277-9175.115819. eCollection 2013. PMID: 24223382; PMCID: PMC3814566.
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The Protective Effect of .

Journal of neurogastroenterology and motility

Dolatabadi F, Abdolghaffari AH, Farzaei MH, Baeeri M, Ziarani FS, Eslami M, Abdollahi M, Rahimi R.
PMID: 29879761
J Neurogastroenterol Motil. 2018 Jul 30;24(3):490-501. doi: 10.5056/jnm17035.

BACKGROUND/AIMS: The aim of present study is to estimate the effects of METHODS: Two individual models of IBS were induced in male Wistar-albino rats. In the acetic acid model, the animals were exposed to rectal distension and abdominal withdrawal...

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Dolatabadi F, Abdolghaffari AH, Farzaei MH, et al. The Protective Effect of . J Neurogastroenterol Motil. 2018;24(3):490-501doi: 10.5056/jnm17035.
Dolatabadi, F., Abdolghaffari, A. H., Farzaei, M. H., Baeeri, M., Ziarani, F. S., Eslami, M., Abdollahi, M., & Rahimi, R. (2018). The Protective Effect of . Journal of neurogastroenterology and motility, 24(3), 490-501. https://doi.org/10.5056/jnm17035
Dolatabadi, Fatemeh, et al. "The Protective Effect of ." Journal of neurogastroenterology and motility vol. 24,3 (2018): 490-501. doi: https://doi.org/10.5056/jnm17035
Dolatabadi F, Abdolghaffari AH, Farzaei MH, Baeeri M, Ziarani FS, Eslami M, Abdollahi M, Rahimi R. The Protective Effect of . J Neurogastroenterol Motil. 2018 Jul 30;24(3):490-501. doi: 10.5056/jnm17035. PMID: 29879761; PMCID: PMC6034661.
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Exploring the role of dimethylarginine dimethylaminohydrolase-mediated reduction in tissue asymmetrical dimethylarginine levels in cardio-protective mechanism of ischaemic postconditioning in rats.

Iranian journal of basic medical sciences

Kaur K, Singh N, Dhawan RK.
PMID: 32133059
Iran J Basic Med Sci. 2019 Dec;22(12):1415-1423. doi: 10.22038/IJBMS.2019.14067.

OBJECTIVES: Reperfusion of ischaemic myocardium results in reduced nitric oxide (NO) biosynthesis by endothelial nitric oxide synthase (eNOS) leading to endothelial dysfunction and subsequent tissue damage. Impaired NO biosynthesis may be partly due to increased levels of asymmetrical dimethylarginine...

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Kaur K, Singh N, Dhawan RK. Exploring the role of dimethylarginine dimethylaminohydrolase-mediated reduction in tissue asymmetrical dimethylarginine levels in cardio-protective mechanism of ischaemic postconditioning in rats. Iran J Basic Med Sci. 2019;22(12):1415-1423doi: 10.22038/IJBMS.2019.14067.
Kaur, K., Singh, N., & Dhawan, R. K. (2019). Exploring the role of dimethylarginine dimethylaminohydrolase-mediated reduction in tissue asymmetrical dimethylarginine levels in cardio-protective mechanism of ischaemic postconditioning in rats. Iranian journal of basic medical sciences, 22(12), 1415-1423. https://doi.org/10.22038/IJBMS.2019.14067
Kaur, Kamaldeep, et al. "Exploring the role of dimethylarginine dimethylaminohydrolase-mediated reduction in tissue asymmetrical dimethylarginine levels in cardio-protective mechanism of ischaemic postconditioning in rats." Iranian journal of basic medical sciences vol. 22,12 (2019): 1415-1423. doi: https://doi.org/10.22038/IJBMS.2019.14067
Kaur K, Singh N, Dhawan RK. Exploring the role of dimethylarginine dimethylaminohydrolase-mediated reduction in tissue asymmetrical dimethylarginine levels in cardio-protective mechanism of ischaemic postconditioning in rats. Iran J Basic Med Sci. 2019 Dec;22(12):1415-1423. doi: 10.22038/IJBMS.2019.14067. PMID: 32133059; PMCID: PMC7043882.
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