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Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 1: clinical trial design principles: A consensus document from the mitral valve academic research consortium.

European heart journal

Stone GW, Vahanian AS, Adams DH, Abraham WT, Borer JS, Bax JJ, Schofer J, Cutlip DE, Krucoff MW, Blackstone EH, Généreux P, Mack MJ, Siegel RJ, Grayburn PA, Enriquez-Sarano M, Lancellotti P, Filippatos G, Kappetein AP.
PMID: 26170467
Eur Heart J. 2015 Aug 01;36(29):1851-77. doi: 10.1093/eurheartj/ehv281. Epub 2015 Jul 13.

Mitral regurgitation (MR) is one of the most prevalent valve disorders and has numerous aetiologies, including primary (organic) MR, due to underlying degenerative/structural mitral valve (MV) pathology, and secondary (functional) MR, which is principally caused by global or regional...

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Stone GW, Vahanian AS, Adams DH, et al. Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 1: clinical trial design principles: A consensus document from the mitral valve academic research consortium. Eur Heart J. 2015;36(29):1851-77doi: 10.1093/eurheartj/ehv281.
Stone, G. W., Vahanian, A. S., Adams, D. H., Abraham, W. T., Borer, J. S., Bax, J. J., Schofer, J., Cutlip, D. E., Krucoff, M. W., Blackstone, E. H., Généreux, P., Mack, M. J., Siegel, R. J., Grayburn, P. A., Enriquez-Sarano, M., Lancellotti, P., Filippatos, G., Kappetein, A. P. (2015). Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 1: clinical trial design principles: A consensus document from the mitral valve academic research consortium. European heart journal, 36(29), 1851-77. https://doi.org/10.1093/eurheartj/ehv281
Stone, Gregg W, et al. "Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 1: clinical trial design principles: A consensus document from the mitral valve academic research consortium." European heart journal vol. 36,29 (2015): 1851-77. doi: https://doi.org/10.1093/eurheartj/ehv281
Stone GW, Vahanian AS, Adams DH, Abraham WT, Borer JS, Bax JJ, Schofer J, Cutlip DE, Krucoff MW, Blackstone EH, Généreux P, Mack MJ, Siegel RJ, Grayburn PA, Enriquez-Sarano M, Lancellotti P, Filippatos G, Kappetein AP. Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 1: clinical trial design principles: A consensus document from the mitral valve academic research consortium. Eur Heart J. 2015 Aug 01;36(29):1851-77. doi: 10.1093/eurheartj/ehv281. Epub 2015 Jul 13. PMID: 26170467.
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Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 2: endpoint definitions: A consensus document from the Mitral Valve Academic Research Consortium.

European heart journal

Stone GW, Adams DH, Abraham WT, Kappetein AP, Généreux P, Vranckx P, Mehran R, Kuck KH, Leon MB, Piazza N, Head SJ, Filippatos G, Vahanian AS.
PMID: 26170468
Eur Heart J. 2015 Aug 01;36(29):1878-91. doi: 10.1093/eurheartj/ehv333. Epub 2015 Jul 13.

Mitral regurgitation (MR) is one of the most prevalent valve disorders and has numerous aetiologies, including primary (organic) MR, due to underlying degenerative/structural mitral valve (MV) pathology, and secondary (functional) MR, which is principally caused by global or regional...

Cite
Stone GW, Adams DH, Abraham WT, et al. Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 2: endpoint definitions: A consensus document from the Mitral Valve Academic Research Consortium. Eur Heart J. 2015;36(29):1878-91doi: 10.1093/eurheartj/ehv333.
Stone, G. W., Adams, D. H., Abraham, W. T., Kappetein, A. P., Généreux, P., Vranckx, P., Mehran, R., Kuck, K. H., Leon, M. B., Piazza, N., Head, S. J., Filippatos, G., Vahanian, A. S. (2015). Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 2: endpoint definitions: A consensus document from the Mitral Valve Academic Research Consortium. European heart journal, 36(29), 1878-91. https://doi.org/10.1093/eurheartj/ehv333
Stone, Gregg W, et al. "Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 2: endpoint definitions: A consensus document from the Mitral Valve Academic Research Consortium." European heart journal vol. 36,29 (2015): 1878-91. doi: https://doi.org/10.1093/eurheartj/ehv333
Stone GW, Adams DH, Abraham WT, Kappetein AP, Généreux P, Vranckx P, Mehran R, Kuck KH, Leon MB, Piazza N, Head SJ, Filippatos G, Vahanian AS. Clinical trial design principles and endpoint definitions for transcatheter mitral valve repair and replacement: part 2: endpoint definitions: A consensus document from the Mitral Valve Academic Research Consortium. Eur Heart J. 2015 Aug 01;36(29):1878-91. doi: 10.1093/eurheartj/ehv333. Epub 2015 Jul 13. PMID: 26170468.
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Variant angina in an 11 year old boy. I. Evaluation of chest pain in children.

Journal of the American College of Cardiology

Brenner JI.
PMID: 4045044
J Am Coll Cardiol. 1985 Nov;6(5):1178. doi: 10.1016/s0735-1097(85)80330-2.

No abstract available.

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Brenner JI. Variant angina in an 11 year old boy. I. Evaluation of chest pain in children. J Am Coll Cardiol. 1985;6(5):1178doi: 10.1016/s0735-1097(85)80330-2.
Brenner, J. I. (1985). Variant angina in an 11 year old boy. I. Evaluation of chest pain in children. Journal of the American College of Cardiology, 6(5), 1178. https://doi.org/10.1016/s0735-1097(85)80330-2
Brenner, J I. "Variant angina in an 11 year old boy. I. Evaluation of chest pain in children." Journal of the American College of Cardiology vol. 6,5 (1985): 1178. doi: https://doi.org/10.1016/s0735-1097(85)80330-2
Brenner JI. Variant angina in an 11 year old boy. I. Evaluation of chest pain in children. J Am Coll Cardiol. 1985 Nov;6(5):1178. doi: 10.1016/s0735-1097(85)80330-2. PMID: 4045044.
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Variant angina in an 11 year old boy. II. Sickle cell trait.

Journal of the American College of Cardiology

Zoneraich S, Rosner F.
PMID: 4045045
J Am Coll Cardiol. 1985 Nov;6(5):1178-9. doi: 10.1016/s0735-1097(85)80331-4.

No abstract available.

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Zoneraich S, Rosner F. Variant angina in an 11 year old boy. II. Sickle cell trait. J Am Coll Cardiol. 1985;6(5):1178-9doi: 10.1016/s0735-1097(85)80331-4.
Zoneraich, S., & Rosner, F. (1985). Variant angina in an 11 year old boy. II. Sickle cell trait. Journal of the American College of Cardiology, 6(5), 1178-9. https://doi.org/10.1016/s0735-1097(85)80331-4
Zoneraich, S, and Rosner, F. "Variant angina in an 11 year old boy. II. Sickle cell trait." Journal of the American College of Cardiology vol. 6,5 (1985): 1178-9. doi: https://doi.org/10.1016/s0735-1097(85)80331-4
Zoneraich S, Rosner F. Variant angina in an 11 year old boy. II. Sickle cell trait. J Am Coll Cardiol. 1985 Nov;6(5):1178-9. doi: 10.1016/s0735-1097(85)80331-4. PMID: 4045045.
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Abstracts of papers. 38th annual scientific session of the American College of Cardiology. Anaheim, California, March 19-23, 1989.

Journal of the American College of Cardiology

[No authors listed]
PMID: 2930618
J Am Coll Cardiol. 1989 Feb;13(2):1A-305A.

No abstract available.

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Abstracts of papers. 38th annual scientific session of the American College of Cardiology. Anaheim, California, March 19-23, 1989. J Am Coll Cardiol. 1989;13(2):1A-305A
(1989). Abstracts of papers. 38th annual scientific session of the American College of Cardiology. Anaheim, California, March 19-23, 1989. Journal of the American College of Cardiology, 13(2), 1A-305A.
"Abstracts of papers. 38th annual scientific session of the American College of Cardiology. Anaheim, California, March 19-23, 1989." Journal of the American College of Cardiology vol. 13,2 (1989): 1A-305A.
Abstracts of papers. 38th annual scientific session of the American College of Cardiology. Anaheim, California, March 19-23, 1989. J Am Coll Cardiol. 1989 Feb;13(2):1A-305A. PMID: 2930618.
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Why do beta-receptor blockers decrease mortality after myocardial infarction?.

Journal of the American College of Cardiology

Lichstein E.
PMID: 2864366
J Am Coll Cardiol. 1985 Nov;6(5):973-5. doi: 10.1016/s0735-1097(85)80296-5.
Free Article

No abstract available.

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Lichstein E. Why do beta-receptor blockers decrease mortality after myocardial infarction?. J Am Coll Cardiol. 1985;6(5):973-5doi: 10.1016/s0735-1097(85)80296-5.
Lichstein, E. (1985). Why do beta-receptor blockers decrease mortality after myocardial infarction?. Journal of the American College of Cardiology, 6(5), 973-5. https://doi.org/10.1016/s0735-1097(85)80296-5
Lichstein, E. "Why do beta-receptor blockers decrease mortality after myocardial infarction?." Journal of the American College of Cardiology vol. 6,5 (1985): 973-5. doi: https://doi.org/10.1016/s0735-1097(85)80296-5
Lichstein E. Why do beta-receptor blockers decrease mortality after myocardial infarction?. J Am Coll Cardiol. 1985 Nov;6(5):973-5. doi: 10.1016/s0735-1097(85)80296-5. PMID: 2864366.
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Significance of hyperventilation-induced ST segment depression in patients with coronary artery disease.

Journal of the American College of Cardiology

Ardissino D, De Servi S, Barberis P, Demicheli G, Falcone C, Ochan M, Specchia G, Montemartini C.
PMID: 2926035
J Am Coll Cardiol. 1989 Mar 15;13(4):804-10. doi: 10.1016/0735-1097(89)90220-9.
Free Article

To investigate the significance of hyperventilation-induced ST segment depression, 329 consecutive patients with angina and documented coronary artery disease who underwent hyperventilation and exercise tests during pharmacologic washout were studied. The hyperventilation test induced ST segment depression in 79...

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Ardissino D, De Servi S, Barberis P, et al. Significance of hyperventilation-induced ST segment depression in patients with coronary artery disease. J Am Coll Cardiol. 1989;13(4):804-10doi: 10.1016/0735-1097(89)90220-9.
Ardissino, D., De Servi, S., Barberis, P., Demicheli, G., Falcone, C., Ochan, M., Specchia, G., & Montemartini, C. (1989). Significance of hyperventilation-induced ST segment depression in patients with coronary artery disease. Journal of the American College of Cardiology, 13(4), 804-10. https://doi.org/10.1016/0735-1097(89)90220-9
Ardissino, D, et al. "Significance of hyperventilation-induced ST segment depression in patients with coronary artery disease." Journal of the American College of Cardiology vol. 13,4 (1989): 804-10. doi: https://doi.org/10.1016/0735-1097(89)90220-9
Ardissino D, De Servi S, Barberis P, Demicheli G, Falcone C, Ochan M, Specchia G, Montemartini C. Significance of hyperventilation-induced ST segment depression in patients with coronary artery disease. J Am Coll Cardiol. 1989 Mar 15;13(4):804-10. doi: 10.1016/0735-1097(89)90220-9. PMID: 2926035.
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Flow characteristics in the dilated left ventricle with thrombus: qualitative and quantitative Doppler analysis.

Journal of the American College of Cardiology

Maze SS, Kotler MN, Parry WR.
PMID: 2926040
J Am Coll Cardiol. 1989 Mar 15;13(4):873-81. doi: 10.1016/0735-1097(89)90230-1.
Free Article

In an attempt to determine whether mural thrombus in a dilated left ventricle is associated with specific flow patterns, a study was undertaken to assess qualitatively and quantitatively the flow characteristics by conventional pulsed and two-dimensional Doppler color flow...

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Maze SS, Kotler MN, Parry WR. Flow characteristics in the dilated left ventricle with thrombus: qualitative and quantitative Doppler analysis. J Am Coll Cardiol. 1989;13(4):873-81doi: 10.1016/0735-1097(89)90230-1.
Maze, S. S., Kotler, M. N., & Parry, W. R. (1989). Flow characteristics in the dilated left ventricle with thrombus: qualitative and quantitative Doppler analysis. Journal of the American College of Cardiology, 13(4), 873-81. https://doi.org/10.1016/0735-1097(89)90230-1
Maze, S S, et al. "Flow characteristics in the dilated left ventricle with thrombus: qualitative and quantitative Doppler analysis." Journal of the American College of Cardiology vol. 13,4 (1989): 873-81. doi: https://doi.org/10.1016/0735-1097(89)90230-1
Maze SS, Kotler MN, Parry WR. Flow characteristics in the dilated left ventricle with thrombus: qualitative and quantitative Doppler analysis. J Am Coll Cardiol. 1989 Mar 15;13(4):873-81. doi: 10.1016/0735-1097(89)90230-1. PMID: 2926040.
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Ischemia-induced diastolic dysfunction: new observations, new questions.

Journal of the American College of Cardiology

Greenberg MA, Menegus MA.
PMID: 2926057
J Am Coll Cardiol. 1989 Apr;13(5):1071-2. doi: 10.1016/0735-1097(89)90262-3.
Free Article

No abstract available.

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Greenberg MA, Menegus MA. Ischemia-induced diastolic dysfunction: new observations, new questions. J Am Coll Cardiol. 1989;13(5):1071-2doi: 10.1016/0735-1097(89)90262-3.
Greenberg, M. A., & Menegus, M. A. (1989). Ischemia-induced diastolic dysfunction: new observations, new questions. Journal of the American College of Cardiology, 13(5), 1071-2. https://doi.org/10.1016/0735-1097(89)90262-3
Greenberg, M A, and Menegus, M A. "Ischemia-induced diastolic dysfunction: new observations, new questions." Journal of the American College of Cardiology vol. 13,5 (1989): 1071-2. doi: https://doi.org/10.1016/0735-1097(89)90262-3
Greenberg MA, Menegus MA. Ischemia-induced diastolic dysfunction: new observations, new questions. J Am Coll Cardiol. 1989 Apr;13(5):1071-2. doi: 10.1016/0735-1097(89)90262-3. PMID: 2926057.
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Regression in thromboembolic type of primary pulmonary hypertension during 2 1/2 years of antithrombotic therapy.

Journal of the American College of Cardiology

Cohen M, Edwards WD, Fuster V.
PMID: 3941208
J Am Coll Cardiol. 1986 Jan;7(1):172-5. doi: 10.1016/s0735-1097(86)80277-7.

Primary pulmonary hypertension carries a poor prognosis, with a 5 year survival rate of less than 25%. However, a previous study of more than 100 patients with tissue-proved primary pulmonary hypertension suggested that antithrombotic therapy may have a beneficial...

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Cohen M, Edwards WD, Fuster V. Regression in thromboembolic type of primary pulmonary hypertension during 2 1/2 years of antithrombotic therapy. J Am Coll Cardiol. 1986;7(1):172-5doi: 10.1016/s0735-1097(86)80277-7.
Cohen, M., Edwards, W. D., & Fuster, V. (1986). Regression in thromboembolic type of primary pulmonary hypertension during 2 1/2 years of antithrombotic therapy. Journal of the American College of Cardiology, 7(1), 172-5. https://doi.org/10.1016/s0735-1097(86)80277-7
Cohen, M, et al. "Regression in thromboembolic type of primary pulmonary hypertension during 2 1/2 years of antithrombotic therapy." Journal of the American College of Cardiology vol. 7,1 (1986): 172-5. doi: https://doi.org/10.1016/s0735-1097(86)80277-7
Cohen M, Edwards WD, Fuster V. Regression in thromboembolic type of primary pulmonary hypertension during 2 1/2 years of antithrombotic therapy. J Am Coll Cardiol. 1986 Jan;7(1):172-5. doi: 10.1016/s0735-1097(86)80277-7. PMID: 3941208.
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The electrocardiogram in the acute coronary syndromes.

Journal of the American College of Cardiology

Ayres SM.
PMID: 2212354
J Am Coll Cardiol. 1990 Oct;16(4):1026-8. doi: 10.1016/s0735-1097(10)80359-6.
Free Article

No abstract available.

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Ayres SM. The electrocardiogram in the acute coronary syndromes. J Am Coll Cardiol. 1990;16(4):1026-8doi: 10.1016/s0735-1097(10)80359-6.
Ayres, S. M. (1990). The electrocardiogram in the acute coronary syndromes. Journal of the American College of Cardiology, 16(4), 1026-8. https://doi.org/10.1016/s0735-1097(10)80359-6
Ayres, S M. "The electrocardiogram in the acute coronary syndromes." Journal of the American College of Cardiology vol. 16,4 (1990): 1026-8. doi: https://doi.org/10.1016/s0735-1097(10)80359-6
Ayres SM. The electrocardiogram in the acute coronary syndromes. J Am Coll Cardiol. 1990 Oct;16(4):1026-8. doi: 10.1016/s0735-1097(10)80359-6. PMID: 2212354.
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Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape.

Journal of the American College of Cardiology

Mitchell GF, Lamas GA, Vaughan DE, Pfeffer MA.
PMID: 1532970
J Am Coll Cardiol. 1992 May;19(6):1136-44. doi: 10.1016/0735-1097(92)90314-d.
Free Article

Infarct expansion after myocardial infarction results in early ventricular enlargement and distortion of ventricular geometry. To characterize the components of late volume enlargement, biplane left ventriculography was performed in 52 patients 3 weeks and 1 year after a first...

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Mitchell GF, Lamas GA, Vaughan DE, et al. Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape. J Am Coll Cardiol. 1992;19(6):1136-44doi: 10.1016/0735-1097(92)90314-d.
Mitchell, G. F., Lamas, G. A., Vaughan, D. E., & Pfeffer, M. A. (1992). Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape. Journal of the American College of Cardiology, 19(6), 1136-44. https://doi.org/10.1016/0735-1097(92)90314-d
Mitchell, G F, et al. "Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape." Journal of the American College of Cardiology vol. 19,6 (1992): 1136-44. doi: https://doi.org/10.1016/0735-1097(92)90314-d
Mitchell GF, Lamas GA, Vaughan DE, Pfeffer MA. Left ventricular remodeling in the year after first anterior myocardial infarction: a quantitative analysis of contractile segment lengths and ventricular shape. J Am Coll Cardiol. 1992 May;19(6):1136-44. doi: 10.1016/0735-1097(92)90314-d. PMID: 1532970.
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Showing 1 to 12 of 28097 entries