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Showing 1 to 10 of 10 entries
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Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca.

Circulation

Korf-Klingebiel M, Reboll MR, Polten F, Weber N, Jäckle F, Wu X, Kallikourdis M, Kunderfranco P, Condorelli G, Giannitsis E, Kustikova OS, Schambach A, Pich A, Widder JD, Bauersachs J, van den Heuvel J, Kraft T, Wang Y, Wollert KC.
PMID: 34372689
Circulation. 2021 Oct 12;144(15):1227-1240. doi: 10.1161/CIRCULATIONAHA.120.053365. Epub 2021 Aug 10.

BACKGROUND: Inflammation contributes to the pathogenesis of heart failure, but there is limited understanding of inflammation's potential benefits. Inflammatory cells secrete MYDGF (myeloid-derived growth factor) to promote tissue repair after acute myocardial infarction. We hypothesized that MYDGF has a...

Cite
Korf-Klingebiel M, Reboll MR, Polten F, et al. Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation. 2021;144(15):1227-1240doi: 10.1161/CIRCULATIONAHA.120.053365.
Korf-Klingebiel, M., Reboll, M. R., Polten, F., Weber, N., Jäckle, F., Wu, X., Kallikourdis, M., Kunderfranco, P., Condorelli, G., Giannitsis, E., Kustikova, O. S., Schambach, A., Pich, A., Widder, J. D., Bauersachs, J., van den Heuvel, J., Kraft, T., Wang, Y., & Wollert, K. C. (2021). Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation, 144(15), 1227-1240. https://doi.org/10.1161/CIRCULATIONAHA.120.053365
Korf-Klingebiel, Mortimer, et al. "Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca." Circulation vol. 144,15 (2021): 1227-1240. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.053365
Korf-Klingebiel M, Reboll MR, Polten F, Weber N, Jäckle F, Wu X, Kallikourdis M, Kunderfranco P, Condorelli G, Giannitsis E, Kustikova OS, Schambach A, Pich A, Widder JD, Bauersachs J, van den Heuvel J, Kraft T, Wang Y, Wollert KC. Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation. 2021 Oct 12;144(15):1227-1240. doi: 10.1161/CIRCULATIONAHA.120.053365. Epub 2021 Aug 10. PMID: 34372689.
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A critical review on the potential role of adipose-derived stem cells for future treatment of hypertrophic scars.

Journal of cosmetic dermatology

Putri KT, Prasetyono TOH.
PMID: 34619011
J Cosmet Dermatol. 2021 Oct 07; doi: 10.1111/jocd.14385. Epub 2021 Oct 07.

INTRODUCTION: Adipose-derived stem cells (ASCs) have recently gained researchers' interest as a solution to various diseases and conditions, including hypertrophic scar. This literature review aims to elucidate ASCs as a potential solution to alleviate hypertrophic scar in human subjects.METHODS:...

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Putri KT, Prasetyono TOH. A critical review on the potential role of adipose-derived stem cells for future treatment of hypertrophic scars. J Cosmet Dermatol. 2021;doi: 10.1111/jocd.14385.
Putri, K. T., & Prasetyono, T. O. H. (2021). A critical review on the potential role of adipose-derived stem cells for future treatment of hypertrophic scars. Journal of cosmetic dermatology, . https://doi.org/10.1111/jocd.14385
Putri, Karina Teja, and Prasetyono, Theddeus Octavianus Hari. "A critical review on the potential role of adipose-derived stem cells for future treatment of hypertrophic scars." Journal of cosmetic dermatology vol. (2021). doi: https://doi.org/10.1111/jocd.14385
Putri KT, Prasetyono TOH. A critical review on the potential role of adipose-derived stem cells for future treatment of hypertrophic scars. J Cosmet Dermatol. 2021 Oct 07; doi: 10.1111/jocd.14385. Epub 2021 Oct 07. PMID: 34619011.
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Neurotrophin expression and histomorphometric evaluation in Wistar rats subjected to neural mobilization after compression of the median nerve.

Revista brasileira de ortopedia

Marcioli MAR, Silva JLDC, Ribeiro LFC, Brancalhão RMC, Bertolini GRF.
PMID: 29892576
Rev Bras Ortop. 2018 Apr 04;53(3):276-280. doi: 10.1016/j.rboe.2018.03.006. eCollection 2018.

OBJECTIVE: To evaluate the neurotrophin mRNA expression and axon count in the median nerve of Wistar rats submitted to neural mobilization (NM) after nerve compression.METHODS: Eighteen animals were randomly divided into G1 (nerve compression only), G2 (NM for 1...

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Marcioli MAR, Silva JLDC, Ribeiro LFC, et al. Neurotrophin expression and histomorphometric evaluation in Wistar rats subjected to neural mobilization after compression of the median nerve. Rev Bras Ortop. 2018;53(3):276-280doi: 10.1016/j.rboe.2018.03.006.
Marcioli, M. A. R., Silva, J. L. D. C., Ribeiro, L. F. C., Brancalhão, R. M. C., & Bertolini, G. R. F. (2018). Neurotrophin expression and histomorphometric evaluation in Wistar rats subjected to neural mobilization after compression of the median nerve. Revista brasileira de ortopedia, 53(3), 276-280. https://doi.org/10.1016/j.rboe.2018.03.006
Marcioli, Marieli Araujo Rossoni, et al. "Neurotrophin expression and histomorphometric evaluation in Wistar rats subjected to neural mobilization after compression of the median nerve." Revista brasileira de ortopedia vol. 53,3 (2018): 276-280. doi: https://doi.org/10.1016/j.rboe.2018.03.006
Marcioli MAR, Silva JLDC, Ribeiro LFC, Brancalhão RMC, Bertolini GRF. Neurotrophin expression and histomorphometric evaluation in Wistar rats subjected to neural mobilization after compression of the median nerve. Rev Bras Ortop. 2018 Apr 04;53(3):276-280. doi: 10.1016/j.rboe.2018.03.006. eCollection 2018. PMID: 29892576; PMCID: PMC5993878.
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The Role of Cancer-Derived Exosomes in Tumorigenicity & Epithelial-to-Mesenchymal Transition.

Cancers

Blackwell RH, Foreman KE, Gupta GN.
PMID: 28796150
Cancers (Basel). 2017 Aug 10;9(8). doi: 10.3390/cancers9080105.

Epithelial-to-mesenchymal transition (EMT) is a process by which epithelial cells lose their basement membrane interaction and acquire a more migratory, mesenchymal phenotype. EMT has been implicated in cancer cell progression, as cells transform and increase motility and invasiveness, induce...

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Blackwell RH, Foreman KE, Gupta GN. The Role of Cancer-Derived Exosomes in Tumorigenicity & Epithelial-to-Mesenchymal Transition. Cancers (Basel). 2017;9(8)doi: 10.3390/cancers9080105.
Blackwell, R. H., Foreman, K. E., & Gupta, G. N. (2017). The Role of Cancer-Derived Exosomes in Tumorigenicity & Epithelial-to-Mesenchymal Transition. Cancers, 9(8), . https://doi.org/10.3390/cancers9080105
Blackwell, Robert H, et al. "The Role of Cancer-Derived Exosomes in Tumorigenicity & Epithelial-to-Mesenchymal Transition." Cancers vol. 9,8 (2017). doi: https://doi.org/10.3390/cancers9080105
Blackwell RH, Foreman KE, Gupta GN. The Role of Cancer-Derived Exosomes in Tumorigenicity & Epithelial-to-Mesenchymal Transition. Cancers (Basel). 2017 Aug 10;9(8). doi: 10.3390/cancers9080105. PMID: 28796150; PMCID: PMC5575608.
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Macrophage-Specific IGF-1 Overexpression Reduces CXCL12 Chemokine Levels and Suppresses Atherosclerotic Burden in Apoe-Deficient Mice.

Arteriosclerosis, thrombosis, and vascular biology

Snarski P, Sukhanov S, Yoshida T, Higashi Y, Danchuk S, Chandrasekar B, Tian D, Rivera-Lopez V, Delafontaine P.
PMID: 34852642
Arterioscler Thromb Vasc Biol. 2021 Dec 02;ATVBAHA121316090. doi: 10.1161/ATVBAHA.121.316090. Epub 2021 Dec 02.

OBJECTIVE: IGF-1 (insulin-like growth factor 1) exerts pleiotropic effects including promotion of cellular growth, differentiation, survival, and anabolism. We have shown that systemic IGF-1 administration reduced atherosclerosis in ApoeCONCLUSIONS: Macrophage IGF-1 overexpression reduced atherosclerotic burden and increased features of...

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Snarski P, Sukhanov S, Yoshida T, et al. Macrophage-Specific IGF-1 Overexpression Reduces CXCL12 Chemokine Levels and Suppresses Atherosclerotic Burden in Apoe-Deficient Mice. Arterioscler Thromb Vasc Biol. 2021;ATVBAHA121316090doi: 10.1161/ATVBAHA.121.316090.
Snarski, P., Sukhanov, S., Yoshida, T., Higashi, Y., Danchuk, S., Chandrasekar, B., Tian, D., Rivera-Lopez, V., & Delafontaine, P. (2021). Macrophage-Specific IGF-1 Overexpression Reduces CXCL12 Chemokine Levels and Suppresses Atherosclerotic Burden in Apoe-Deficient Mice. Arteriosclerosis, thrombosis, and vascular biology, ATVBAHA121316090. https://doi.org/10.1161/ATVBAHA.121.316090
Snarski, Patricia, et al. "Macrophage-Specific IGF-1 Overexpression Reduces CXCL12 Chemokine Levels and Suppresses Atherosclerotic Burden in Apoe-Deficient Mice." Arteriosclerosis, thrombosis, and vascular biology vol. (2021): ATVBAHA121316090. doi: https://doi.org/10.1161/ATVBAHA.121.316090
Snarski P, Sukhanov S, Yoshida T, Higashi Y, Danchuk S, Chandrasekar B, Tian D, Rivera-Lopez V, Delafontaine P. Macrophage-Specific IGF-1 Overexpression Reduces CXCL12 Chemokine Levels and Suppresses Atherosclerotic Burden in Apoe-Deficient Mice. Arterioscler Thromb Vasc Biol. 2021 Dec 02;ATVBAHA121316090. doi: 10.1161/ATVBAHA.121.316090. Epub 2021 Dec 02. PMID: 34852642.
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Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca.

Circulation

Korf-Klingebiel M, Reboll MR, Polten F, Weber N, Jäckle F, Wu X, Kallikourdis M, Kunderfranco P, Condorelli G, Giannitsis E, Kustikova OS, Schambach A, Pich A, Widder JD, Bauersachs J, van den Heuvel J, Kraft T, Wang Y, Wollert KC.
PMID: 34372689
Circulation. 2021 Oct 12;144(15):1227-1240. doi: 10.1161/CIRCULATIONAHA.120.053365. Epub 2021 Aug 10.

BACKGROUND: Inflammation contributes to the pathogenesis of heart failure, but there is limited understanding of inflammation's potential benefits. Inflammatory cells secrete MYDGF (myeloid-derived growth factor) to promote tissue repair after acute myocardial infarction. We hypothesized that MYDGF has a...

Cite
Korf-Klingebiel M, Reboll MR, Polten F, et al. Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation. 2021;144(15):1227-1240doi: 10.1161/CIRCULATIONAHA.120.053365.
Korf-Klingebiel, M., Reboll, M. R., Polten, F., Weber, N., Jäckle, F., Wu, X., Kallikourdis, M., Kunderfranco, P., Condorelli, G., Giannitsis, E., Kustikova, O. S., Schambach, A., Pich, A., Widder, J. D., Bauersachs, J., van den Heuvel, J., Kraft, T., Wang, Y., & Wollert, K. C. (2021). Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation, 144(15), 1227-1240. https://doi.org/10.1161/CIRCULATIONAHA.120.053365
Korf-Klingebiel, Mortimer, et al. "Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca." Circulation vol. 144,15 (2021): 1227-1240. doi: https://doi.org/10.1161/CIRCULATIONAHA.120.053365
Korf-Klingebiel M, Reboll MR, Polten F, Weber N, Jäckle F, Wu X, Kallikourdis M, Kunderfranco P, Condorelli G, Giannitsis E, Kustikova OS, Schambach A, Pich A, Widder JD, Bauersachs J, van den Heuvel J, Kraft T, Wang Y, Wollert KC. Myeloid-Derived Growth Factor Protects Against Pressure Overload-Induced Heart Failure by Preserving Sarco/Endoplasmic Reticulum Ca. Circulation. 2021 Oct 12;144(15):1227-1240. doi: 10.1161/CIRCULATIONAHA.120.053365. Epub 2021 Aug 10. PMID: 34372689.
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Does Adjunctive Use of Growth Factors Improve Clinical Outcomes of Soft Tissue Grafting at Miller Class III Recession Defects? A Review of Current Evidence.

Clinical advances in periodontics

Geisinger ML, Trammell K, Holmes CM, Kaur M, Geurs NC.
PMID: 31535488
Clin Adv Periodontics. 2016 May;6(2):99-103. doi: 10.1902/cap.2015.150048.

Focused Clinical Question In patients with gingival recession that is classified as Miller Class III, does adjunctive use of local growth factors with gingival grafting procedures improve clinical outcomes? Clinical Scenario A 31-year-old female presents with a chief complaint...

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Geisinger ML, Trammell K, Holmes CM, et al. Does Adjunctive Use of Growth Factors Improve Clinical Outcomes of Soft Tissue Grafting at Miller Class III Recession Defects? A Review of Current Evidence. Clin Adv Periodontics. 2016;6(2):99-103doi: 10.1902/cap.2015.150048.
Geisinger, M. L., Trammell, K., Holmes, C. M., Kaur, M., & Geurs, N. C. (2016). Does Adjunctive Use of Growth Factors Improve Clinical Outcomes of Soft Tissue Grafting at Miller Class III Recession Defects? A Review of Current Evidence. Clinical advances in periodontics, 6(2), 99-103. https://doi.org/10.1902/cap.2015.150048
Geisinger, Maria L, et al. "Does Adjunctive Use of Growth Factors Improve Clinical Outcomes of Soft Tissue Grafting at Miller Class III Recession Defects? A Review of Current Evidence." Clinical advances in periodontics vol. 6,2 (2016): 99-103. doi: https://doi.org/10.1902/cap.2015.150048
Geisinger ML, Trammell K, Holmes CM, Kaur M, Geurs NC. Does Adjunctive Use of Growth Factors Improve Clinical Outcomes of Soft Tissue Grafting at Miller Class III Recession Defects? A Review of Current Evidence. Clin Adv Periodontics. 2016 May;6(2):99-103. doi: 10.1902/cap.2015.150048. PMID: 31535488.
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Biologic Agents to Promote Periodontal Regeneration and Bone Augmentation.

Clinical advances in periodontics

Bashutski JD, Wang HL.
PMID: 32698555
Clin Adv Periodontics. 2011 Aug;1(2):80-87. doi: 10.1902/cap.2011.110044.

Periodontal regeneration is preferred over tissue repair and is accomplished through the exclusion of epithelial tissues, which allows cementum, bone, and connective tissue to repopulate the wound. Recently, biologic materials have emerged as adjuncts to aid in regeneration by...

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Bashutski JD, Wang HL. Biologic Agents to Promote Periodontal Regeneration and Bone Augmentation. Clin Adv Periodontics. 2011;1(2):80-87doi: 10.1902/cap.2011.110044.
Bashutski, J. D., & Wang, H. L. (2011). Biologic Agents to Promote Periodontal Regeneration and Bone Augmentation. Clinical advances in periodontics, 1(2), 80-87. https://doi.org/10.1902/cap.2011.110044
Bashutski, Jill D, and Wang, Hom-Lay. "Biologic Agents to Promote Periodontal Regeneration and Bone Augmentation." Clinical advances in periodontics vol. 1,2 (2011): 80-87. doi: https://doi.org/10.1902/cap.2011.110044
Bashutski JD, Wang HL. Biologic Agents to Promote Periodontal Regeneration and Bone Augmentation. Clin Adv Periodontics. 2011 Aug;1(2):80-87. doi: 10.1902/cap.2011.110044. PMID: 32698555.
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Changes in Communication between Muscle Stem Cells and their Environment with Aging.

Journal of neuromuscular diseases

Thorley M, Malatras A, Duddy W, Le Gall L, Mouly V, Butler Browne G, Duguez S.
PMID: 27858742
J Neuromuscul Dis. 2015 Sep 02;2(3):205-217. doi: 10.3233/JND-150097.

Aging is associated with both muscle weakness and a loss of muscle mass, contributing towards overall frailty in the elderly. Aging skeletal muscle is also characterised by a decreasing efficiency in repair and regeneration, together with a decline in...

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Thorley M, Malatras A, Duddy W, et al. Changes in Communication between Muscle Stem Cells and their Environment with Aging. J Neuromuscul Dis. 2015;2(3):205-217doi: 10.3233/JND-150097.
Thorley, M., Malatras, A., Duddy, W., Le Gall, L., Mouly, V., Butler Browne, G., & Duguez, S. (2015). Changes in Communication between Muscle Stem Cells and their Environment with Aging. Journal of neuromuscular diseases, 2(3), 205-217. https://doi.org/10.3233/JND-150097
Thorley, Matthew, et al. "Changes in Communication between Muscle Stem Cells and their Environment with Aging." Journal of neuromuscular diseases vol. 2,3 (2015): 205-217. doi: https://doi.org/10.3233/JND-150097
Thorley M, Malatras A, Duddy W, Le Gall L, Mouly V, Butler Browne G, Duguez S. Changes in Communication between Muscle Stem Cells and their Environment with Aging. J Neuromuscul Dis. 2015 Sep 02;2(3):205-217. doi: 10.3233/JND-150097. PMID: 27858742; PMCID: PMC5240546.
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Recombinant human bone morphogenetic protein type 2 in the reconstruction of atrophic maxilla: Case report with long-term follow-up.

Journal of Indian Society of Periodontology

Zétola AL, Verbicaro T, Littieri S, Larson R, Giovanini AF, Deliberador TM.
PMID: 25624638
J Indian Soc Periodontol. 2014 Nov-Dec;18(6):781-5. doi: 10.4103/0972-124X.147437.

Autologous bone is reported by scientific literature as the gold standard for the replacement of the bone loss in maxillary atrophic area. Notwithstanding, this grafting type shows several disadvantages as: The procedure morbidity, limited size of the graft and...

Cite
Zétola AL, Verbicaro T, Littieri S, et al. Recombinant human bone morphogenetic protein type 2 in the reconstruction of atrophic maxilla: Case report with long-term follow-up. J Indian Soc Periodontol. 2014;18(6):781-5doi: 10.4103/0972-124X.147437.
Zétola, A. L., Verbicaro, T., Littieri, S., Larson, R., Giovanini, A. F., & Deliberador, T. M. (2014). Recombinant human bone morphogenetic protein type 2 in the reconstruction of atrophic maxilla: Case report with long-term follow-up. Journal of Indian Society of Periodontology, 18(6), 781-5. https://doi.org/10.4103/0972-124X.147437
Zétola, André Luiz, et al. "Recombinant human bone morphogenetic protein type 2 in the reconstruction of atrophic maxilla: Case report with long-term follow-up." Journal of Indian Society of Periodontology vol. 18,6 (2014): 781-5. doi: https://doi.org/10.4103/0972-124X.147437
Zétola AL, Verbicaro T, Littieri S, Larson R, Giovanini AF, Deliberador TM. Recombinant human bone morphogenetic protein type 2 in the reconstruction of atrophic maxilla: Case report with long-term follow-up. J Indian Soc Periodontol. 2014 Nov-Dec;18(6):781-5. doi: 10.4103/0972-124X.147437. PMID: 25624638; PMCID: PMC4296466.
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Showing 1 to 10 of 10 entries