Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 1569 entries
Sorted by: Best Match Show Resources per page
Gonadotropin-releasing hormone agonist corrects defective mini-puberty in boys with cryptorchidism: a prospective randomized study

STEM and boys

[No authors listed]
GSID: aX6I02jJJQoJ
B Vincel, G Verkauskas, V Bilius… - BioMed Research …, 2018 - hindawi.com

… Boys with isolated bilateral cryptorchidism and defective mini-puberty were randomly divided … Five boys were included in each arm. In the LH-RHa group, the median S/T increased from …

Cite
Vincel, B., Verkauskas, G., Bilius, V., Dasevicius, D., Malcius, D., Jones, B., & Hadziselimovic, F. (2018). Gonadotropin-releasing hormone agonist corrects defective mini-puberty in boys with cryptorchidism: a prospective randomized study. BioMed Research International, 2018.
Vincel, Beata, et al. "Gonadotropin-releasing hormone agonist corrects defective mini-puberty in boys with cryptorchidism: a prospective randomized study." BioMed Research International 2018 (2018).
Copy Download .nbib Format: MLA
Past, present, and future of hormonal therapy in recurrent endometrial cancer.

International journal of women's health

Carlson MJ, Thiel KW, Leslie KK.
PMID: 24833920
Int J Womens Health. 2014 May 02;6:429-35. doi: 10.2147/IJWH.S40942. eCollection 2014.

Endometrial cancer is a heterogeneous disease. Type I cancers are hormonally driven, typically present with a low grade at an early stage, and are of endometrioid histology. These cancers are often cured by surgery, and the rate of recurrence...

Cite
Carlson MJ, Thiel KW, Leslie KK. Past, present, and future of hormonal therapy in recurrent endometrial cancer. Int J Womens Health. 2014;6:429-35doi: 10.2147/IJWH.S40942.
Carlson, M. J., Thiel, K. W., & Leslie, K. K. (2014). Past, present, and future of hormonal therapy in recurrent endometrial cancer. International journal of women's health, 6429-35. https://doi.org/10.2147/IJWH.S40942
Carlson, Matthew J, et al. "Past, present, and future of hormonal therapy in recurrent endometrial cancer." International journal of women's health vol. 6 (2014): 429-35. doi: https://doi.org/10.2147/IJWH.S40942
Carlson MJ, Thiel KW, Leslie KK. Past, present, and future of hormonal therapy in recurrent endometrial cancer. Int J Womens Health. 2014 May 02;6:429-35. doi: 10.2147/IJWH.S40942. eCollection 2014. PMID: 24833920; PMCID: PMC4014387.
Copy Download .nbib Format: NLM
Erratum to: Gonadotropin-releasing hormone, kisspeptin, and gonadal steroids directly modulate nucleobindin-2/nesfatin-1 in murine hypothalamic gonadotropin-releasing hormone neurons and gonadotropes.

Biology of reproduction

Hatef A, Unniappan S.
PMID: 28863435
Biol Reprod. 2017 May 01;96(5):1118. doi: 10.1093/biolre/iox035.

No abstract available.

Cite
Hatef A, Unniappan S. Erratum to: Gonadotropin-releasing hormone, kisspeptin, and gonadal steroids directly modulate nucleobindin-2/nesfatin-1 in murine hypothalamic gonadotropin-releasing hormone neurons and gonadotropes. Biol Reprod. 2017;96(5):1118doi: 10.1093/biolre/iox035.
Hatef, A., & Unniappan, S. (2017). Erratum to: Gonadotropin-releasing hormone, kisspeptin, and gonadal steroids directly modulate nucleobindin-2/nesfatin-1 in murine hypothalamic gonadotropin-releasing hormone neurons and gonadotropes. Biology of reproduction, 96(5), 1118. https://doi.org/10.1093/biolre/iox035
Hatef, A, and Unniappan, S. "Erratum to: Gonadotropin-releasing hormone, kisspeptin, and gonadal steroids directly modulate nucleobindin-2/nesfatin-1 in murine hypothalamic gonadotropin-releasing hormone neurons and gonadotropes." Biology of reproduction vol. 96,5 (2017): 1118. doi: https://doi.org/10.1093/biolre/iox035
Hatef A, Unniappan S. Erratum to: Gonadotropin-releasing hormone, kisspeptin, and gonadal steroids directly modulate nucleobindin-2/nesfatin-1 in murine hypothalamic gonadotropin-releasing hormone neurons and gonadotropes. Biol Reprod. 2017 May 01;96(5):1118. doi: 10.1093/biolre/iox035. PMID: 28863435.
Copy Download .nbib Format: NLM
Cervical sympathectomy affects gonadotropin-releasing hormone, luteinizing hormone and testosterone in male rats.

Journal of anesthesia

Iwama H, Tase C, Tonosaki Y, Sugiura Y.
PMID: 28921287
J Anesth. 1995 Jun;9(2):166-169. doi: 10.1007/BF02479850.

To examine the effects of bilateral cervical sympathectomy on the secretion of gonadotropin-releasing hormone (GnRH), luteinizing hormone (LH) and testosterone (TS), 24 male rats were divided into four groups: control (C), light (L), sympathectomy (S), and light-sympathectomy (LS) groups....

Cite
Iwama H, Tase C, Tonosaki Y, et al. Cervical sympathectomy affects gonadotropin-releasing hormone, luteinizing hormone and testosterone in male rats. J Anesth. 1995;9(2):166-169doi: 10.1007/BF02479850.
Iwama, H., Tase, C., Tonosaki, Y., & Sugiura, Y. (1995). Cervical sympathectomy affects gonadotropin-releasing hormone, luteinizing hormone and testosterone in male rats. Journal of anesthesia, 9(2), 166-169. https://doi.org/10.1007/BF02479850
Iwama, Hiroshi, et al. "Cervical sympathectomy affects gonadotropin-releasing hormone, luteinizing hormone and testosterone in male rats." Journal of anesthesia vol. 9,2 (1995): 166-169. doi: https://doi.org/10.1007/BF02479850
Iwama H, Tase C, Tonosaki Y, Sugiura Y. Cervical sympathectomy affects gonadotropin-releasing hormone, luteinizing hormone and testosterone in male rats. J Anesth. 1995 Jun;9(2):166-169. doi: 10.1007/BF02479850. PMID: 28921287.
Copy Download .nbib Format: NLM
Leiomyomatosis peritonealis disseminata associated with endometriosis: A case report and review of the literature.

Oncology letters

Yang R, Xu T, Fu Y, Cui S, Yang S, Cui M.
PMID: 25621042
Oncol Lett. 2015 Feb;9(2):717-720. doi: 10.3892/ol.2014.2741. Epub 2014 Nov 27.

Leiomyomatosis peritonealis disseminata (LPD) is a specific type of leiomyomatosis with an unclear pathogenesis that is rarely diagnosed by clinical evaluation. To date,

Cite
Yang R, Xu T, Fu Y, et al. Leiomyomatosis peritonealis disseminata associated with endometriosis: A case report and review of the literature. Oncol Lett. 2014;9(2):717-720doi: 10.3892/ol.2014.2741.
Yang, R., Xu, T., Fu, Y., Cui, S., Yang, S., & Cui, M. (2015). Leiomyomatosis peritonealis disseminata associated with endometriosis: A case report and review of the literature. Oncology letters, 9(2), 717-720. https://doi.org/10.3892/ol.2014.2741
Yang, Rulin, et al. "Leiomyomatosis peritonealis disseminata associated with endometriosis: A case report and review of the literature." Oncology letters vol. 9,2 (2015): 717-720. doi: https://doi.org/10.3892/ol.2014.2741
Yang R, Xu T, Fu Y, Cui S, Yang S, Cui M. Leiomyomatosis peritonealis disseminata associated with endometriosis: A case report and review of the literature. Oncol Lett. 2015 Feb;9(2):717-720. doi: 10.3892/ol.2014.2741. Epub 2014 Nov 27. PMID: 25621042; PMCID: PMC4301522.
Copy Download .nbib Format: NLM
Alterations in uterine hemodynamics caused by uterine fibroids and their impact on in vitro fertilization outcomes.

Clinical and experimental reproductive medicine

Moon JW, Kim CH, Kim JB, Kim SH, Chae HD, Kang BM.
PMID: 26816875
Clin Exp Reprod Med. 2015 Dec;42(4):163-8. doi: 10.5653/cerm.2015.42.4.163. Epub 2015 Dec 31.

OBJECTIVE: To investigate the impact of fibroids on the blood flow of the uterine and subendometrial arteries and in vitro fertilization (IVF) outcomes.METHODS: In this study, we analyzed 86 IVF/intracytoplasmic sperm injection (ICSI) cycles in which a gonadotropin-releasing hormone...

Cite
Moon JW, Kim CH, Kim JB, et al. Alterations in uterine hemodynamics caused by uterine fibroids and their impact on in vitro fertilization outcomes. Clin Exp Reprod Med. 2015;42(4):163-8doi: 10.5653/cerm.2015.42.4.163.
Moon, J. W., Kim, C. H., Kim, J. B., Kim, S. H., Chae, H. D., & Kang, B. M. (2015). Alterations in uterine hemodynamics caused by uterine fibroids and their impact on in vitro fertilization outcomes. Clinical and experimental reproductive medicine, 42(4), 163-8. https://doi.org/10.5653/cerm.2015.42.4.163
Moon, Jei-Won, et al. "Alterations in uterine hemodynamics caused by uterine fibroids and their impact on in vitro fertilization outcomes." Clinical and experimental reproductive medicine vol. 42,4 (2015): 163-8. doi: https://doi.org/10.5653/cerm.2015.42.4.163
Moon JW, Kim CH, Kim JB, Kim SH, Chae HD, Kang BM. Alterations in uterine hemodynamics caused by uterine fibroids and their impact on in vitro fertilization outcomes. Clin Exp Reprod Med. 2015 Dec;42(4):163-8. doi: 10.5653/cerm.2015.42.4.163. Epub 2015 Dec 31. PMID: 26816875; PMCID: PMC4724601.
Copy Download .nbib Format: NLM
PATHOGENESIS, DIAGNOSIS AND TREATMENT OF GENITAL ENDOMETRIOSIS.

Acta obstetricia et gynecologica Scandinavica

Mettler L.
PMID: 29124724
Acta Obstet Gynecol Scand. 1989 Jan;68:31-37. doi: 10.1111/aogs.1989.68.s150.31.

The etiology of genital endometriosis remains unclear, but it is evident that persisting endometriotic foci are stimulated by ovarian steroid hormones. Many cases are treated by surgery either via laparotomy or pelviscopy. Endocrine treatments with testosterone, gestagens, danazol and...

Cite
Mettler L. PATHOGENESIS, DIAGNOSIS AND TREATMENT OF GENITAL ENDOMETRIOSIS. Acta Obstet Gynecol Scand. 1989;68:31-37doi: 10.1111/aogs.1989.68.s150.31.
Mettler, L. (1989). PATHOGENESIS, DIAGNOSIS AND TREATMENT OF GENITAL ENDOMETRIOSIS. Acta obstetricia et gynecologica Scandinavica, 6831-37. https://doi.org/10.1111/aogs.1989.68.s150.31
Mettler, L. "PATHOGENESIS, DIAGNOSIS AND TREATMENT OF GENITAL ENDOMETRIOSIS." Acta obstetricia et gynecologica Scandinavica vol. 68 (1989): 31-37. doi: https://doi.org/10.1111/aogs.1989.68.s150.31
Mettler L. PATHOGENESIS, DIAGNOSIS AND TREATMENT OF GENITAL ENDOMETRIOSIS. Acta Obstet Gynecol Scand. 1989 Jan;68:31-37. doi: 10.1111/aogs.1989.68.s150.31. PMID: 29124724.
Copy Download .nbib Format: NLM
The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron Development.

Frontiers in endocrinology

Chung WC, Linscott ML, Rodriguez KM, Stewart CE.
PMID: 27656162
Front Endocrinol (Lausanne). 2016 Sep 05;7:114. doi: 10.3389/fendo.2016.00114. eCollection 2016.

Over the last few years, numerous studies solidified the hypothesis that fibroblast growth factor (FGF) signaling regulates neuroendocrine progenitor cell proliferation, fate specification, and cell survival and, therefore, is critical for the regulation and maintenance of homeostasis of the...

Cite
Chung WC, Linscott ML, Rodriguez KM, et al. The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron Development. Front Endocrinol (Lausanne). 2016;7:114doi: 10.3389/fendo.2016.00114.
Chung, W. C., Linscott, M. L., Rodriguez, K. M., & Stewart, C. E. (2016). The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron Development. Frontiers in endocrinology, 7114. https://doi.org/10.3389/fendo.2016.00114
Chung, Wilson C J, et al. "The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron Development." Frontiers in endocrinology vol. 7 (2016): 114. doi: https://doi.org/10.3389/fendo.2016.00114
Chung WC, Linscott ML, Rodriguez KM, Stewart CE. The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron Development. Front Endocrinol (Lausanne). 2016 Sep 05;7:114. doi: 10.3389/fendo.2016.00114. eCollection 2016. PMID: 27656162; PMCID: PMC5011149.
Copy Download .nbib Format: NLM
Dual trigger with gonadotropin-releasing hormone and human chorionic gonadotropin for poor responders.

Journal of the Turkish German Gynecological Association

Eser A, Devranoğlu B, Bostancı Ergen E, Yayla Abide Ç.
PMID: 29516855
J Turk Ger Gynecol Assoc. 2018 Jun 04;19(2):98-103. doi: 10.4274/jtgga.2017.0045. Epub 2018 Mar 08.

OBJECTIVE: To compare metaphase II (MII) rate, fertilization rate, and embryo quality with dual trigger gonadotropin-releasing hormone agonist (GnRH) and normal dose human chorionic gonadotropin (hCG) versus a normal dose hCG trigger in antagonist intracytoplasmic sperm injection (ICSI) cycles...

Cite
Eser A, Devranoğlu B, Bostancı Ergen E, et al. Dual trigger with gonadotropin-releasing hormone and human chorionic gonadotropin for poor responders. J Turk Ger Gynecol Assoc. 2018;19(2):98-103doi: 10.4274/jtgga.2017.0045.
Eser, A., Devranoğlu, B., Bostancı Ergen, E., & Yayla Abide, �. �. (2018). Dual trigger with gonadotropin-releasing hormone and human chorionic gonadotropin for poor responders. Journal of the Turkish German Gynecological Association, 19(2), 98-103. https://doi.org/10.4274/jtgga.2017.0045
Eser, Ahmet, et al. "Dual trigger with gonadotropin-releasing hormone and human chorionic gonadotropin for poor responders." Journal of the Turkish German Gynecological Association vol. 19,2 (2018): 98-103. doi: https://doi.org/10.4274/jtgga.2017.0045
Eser A, Devranoğlu B, Bostancı Ergen E, Yayla Abide Ç. Dual trigger with gonadotropin-releasing hormone and human chorionic gonadotropin for poor responders. J Turk Ger Gynecol Assoc. 2018 Jun 04;19(2):98-103. doi: 10.4274/jtgga.2017.0045. Epub 2018 Mar 08. PMID: 29516855; PMCID: PMC5994809.
Copy Download .nbib Format: NLM
Kisspeptin: A Potential Factor for Unexplained Infertility and Impaired Embryo Implantation.

International journal of fertility & sterility

Mumtaz A, Khalid A, Jamil Z, Fatima SS, Arif S, Rehman R.
PMID: 28670427
Int J Fertil Steril. 2017 Jul-Sep;11(2):99-104. doi: 10.22074/ijfs.2017.4957. Epub 2017 Feb 16.

BACKGROUND: Kisspeptin (KP) is a neuropeptide that causes the release of the gonadotropin releasing hormone, which controls hypothalamo pituitary ovarian axis and exerts a number of peripheral effects on reproductive organs. The primary objective of this study was to...

Cite
Mumtaz A, Khalid A, Jamil Z, et al. Kisspeptin: A Potential Factor for Unexplained Infertility and Impaired Embryo Implantation. Int J Fertil Steril. 2017;11(2):99-104doi: 10.22074/ijfs.2017.4957.
Mumtaz, A., Khalid, A., Jamil, Z., Fatima, S. S., Arif, S., & Rehman, R. (2017). Kisspeptin: A Potential Factor for Unexplained Infertility and Impaired Embryo Implantation. International journal of fertility & sterility, 11(2), 99-104. https://doi.org/10.22074/ijfs.2017.4957
Mumtaz, Aaida, et al. "Kisspeptin: A Potential Factor for Unexplained Infertility and Impaired Embryo Implantation." International journal of fertility & sterility vol. 11,2 (2017): 99-104. doi: https://doi.org/10.22074/ijfs.2017.4957
Mumtaz A, Khalid A, Jamil Z, Fatima SS, Arif S, Rehman R. Kisspeptin: A Potential Factor for Unexplained Infertility and Impaired Embryo Implantation. Int J Fertil Steril. 2017 Jul-Sep;11(2):99-104. doi: 10.22074/ijfs.2017.4957. Epub 2017 Feb 16. PMID: 28670427; PMCID: PMC5347457.
Copy Download .nbib Format: NLM
c-Src is activated by the epidermal growth factor receptor in a pathway that mediates JNK and ERK activation by gonadotropin-releasing hormone in COS7 cells.

The Journal of biological chemistry

Kraus S, Benard O, Naor Z, Seger R.
PMID: 28550137
J Biol Chem. 2017 May 26;292(21):8851. doi: 10.1074/jbc.A117.303886.

No abstract available.

Cite
Kraus S, Benard O, Naor Z, et al. c-Src is activated by the epidermal growth factor receptor in a pathway that mediates JNK and ERK activation by gonadotropin-releasing hormone in COS7 cells. J Biol Chem. 2017;292(21):8851doi: 10.1074/jbc.A117.303886.
Kraus, S., Benard, O., Naor, Z., & Seger, R. (2017). c-Src is activated by the epidermal growth factor receptor in a pathway that mediates JNK and ERK activation by gonadotropin-releasing hormone in COS7 cells. The Journal of biological chemistry, 292(21), 8851. https://doi.org/10.1074/jbc.A117.303886
Kraus, Sarah, et al. "c-Src is activated by the epidermal growth factor receptor in a pathway that mediates JNK and ERK activation by gonadotropin-releasing hormone in COS7 cells." The Journal of biological chemistry vol. 292,21 (2017): 8851. doi: https://doi.org/10.1074/jbc.A117.303886
Kraus S, Benard O, Naor Z, Seger R. c-Src is activated by the epidermal growth factor receptor in a pathway that mediates JNK and ERK activation by gonadotropin-releasing hormone in COS7 cells. J Biol Chem. 2017 May 26;292(21):8851. doi: 10.1074/jbc.A117.303886. PMID: 28550137; PMCID: PMC5448115.
Copy Download .nbib Format: NLM
Ovarian transcriptomic analysis of Shan Ma ducks at peak and late stages of egg production.

Asian-Australasian journal of animal sciences

Zhu Z, Miao Z, Chen H, Xin Q, Li L, Lin R, Huang Q, Zheng N.
PMID: 28111447
Asian-Australas J Anim Sci. 2017 Sep;30(9):1215-1224. doi: 10.5713/ajas.16.0470. Epub 2016 Dec 27.

OBJECTIVE: To assess the differences in ovarian transcriptomes in Shan Ma ducks between their peak and late stages of egg production, and to obtain new transcriptomic data of these egg-producing ducks.METHODS: The Illumina HiSeq 2000 system was used for...

Cite
Zhu Z, Miao Z, Chen H, et al. Ovarian transcriptomic analysis of Shan Ma ducks at peak and late stages of egg production. Asian-Australas J Anim Sci. 2016;30(9):1215-1224doi: 10.5713/ajas.16.0470.
Zhu, Z., Miao, Z., Chen, H., Xin, Q., Li, L., Lin, R., Huang, Q., & Zheng, N. (2017). Ovarian transcriptomic analysis of Shan Ma ducks at peak and late stages of egg production. Asian-Australasian journal of animal sciences, 30(9), 1215-1224. https://doi.org/10.5713/ajas.16.0470
Zhu, ZhiMing, et al. "Ovarian transcriptomic analysis of Shan Ma ducks at peak and late stages of egg production." Asian-Australasian journal of animal sciences vol. 30,9 (2017): 1215-1224. doi: https://doi.org/10.5713/ajas.16.0470
Zhu Z, Miao Z, Chen H, Xin Q, Li L, Lin R, Huang Q, Zheng N. Ovarian transcriptomic analysis of Shan Ma ducks at peak and late stages of egg production. Asian-Australas J Anim Sci. 2017 Sep;30(9):1215-1224. doi: 10.5713/ajas.16.0470. Epub 2016 Dec 27. PMID: 28111447; PMCID: PMC5582276.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 1569 entries