Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 386 entries
Sorted by: Best Match Show Resources per page
Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus.

Frontiers in cellular neuroscience

Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K.
PMID: 28515682
Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017.

Corticotropin-releasing hormone (CRH)-synthesizing parvocellular neuroendocrine cells (PNCs) of the hypothalamic paraventricular nucleus (PVN) play a key role in the activation of the hypothalamic-pituitary-adrenocortical (HPA) axis. Several studies have demonstrated that synaptic inputs to these cells may undergo stress-related enhancement...

Cite
Kusek M, Tokarska A, Siwiec M, et al. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017;11:127doi: 10.3389/fncel.2017.00127.
Kusek, M., Tokarska, A., Siwiec, M., Gadek-Michalska, A., Szewczyk, B., Hess, G., & Tokarski, K. (2017). Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Frontiers in cellular neuroscience, 11127. https://doi.org/10.3389/fncel.2017.00127
Kusek, Magdalena, et al. "Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus." Frontiers in cellular neuroscience vol. 11 (2017): 127. doi: https://doi.org/10.3389/fncel.2017.00127
Kusek M, Tokarska A, Siwiec M, Gadek-Michalska A, Szewczyk B, Hess G, Tokarski K. Nitric Oxide Synthase Inhibitor Attenuates the Effects of Repeated Restraint Stress on Synaptic Transmission in the Paraventricular Nucleus of the Rat Hypothalamus. Front Cell Neurosci. 2017 May 03;11:127. doi: 10.3389/fncel.2017.00127. eCollection 2017. PMID: 28515682; PMCID: PMC5413825.
Copy Download .nbib Format: NLM
Impact of Adiposity and Fat Distribution on the Dynamics of Adrenocorticotropin and Cortisol Rhythms.

Current obesity reports

Roelfsema F, Pereira AM, Veldhuis JD.
PMID: 26626915
Curr Obes Rep. 2014 Dec;3(4):387-95. doi: 10.1007/s13679-014-0118-7.

Obesity impacts many hormonal systems, including pituitary hormones, as well as insulin and leptin. In this review we discuss articles which investigate the influence of obesity on the hypothalamic-pituitary-adrenal (HPA) axis. Different techniques have been used to assess the...

Cite
Roelfsema F, Pereira AM, Veldhuis JD. Impact of Adiposity and Fat Distribution on the Dynamics of Adrenocorticotropin and Cortisol Rhythms. Curr Obes Rep. 2014;3(4):387-95doi: 10.1007/s13679-014-0118-7.
Roelfsema, F., Pereira, A. M., & Veldhuis, J. D. (2014). Impact of Adiposity and Fat Distribution on the Dynamics of Adrenocorticotropin and Cortisol Rhythms. Current obesity reports, 3(4), 387-95. https://doi.org/10.1007/s13679-014-0118-7
Roelfsema, Ferdinand, et al. "Impact of Adiposity and Fat Distribution on the Dynamics of Adrenocorticotropin and Cortisol Rhythms." Current obesity reports vol. 3,4 (2014): 387-95. doi: https://doi.org/10.1007/s13679-014-0118-7
Roelfsema F, Pereira AM, Veldhuis JD. Impact of Adiposity and Fat Distribution on the Dynamics of Adrenocorticotropin and Cortisol Rhythms. Curr Obes Rep. 2014 Dec;3(4):387-95. doi: 10.1007/s13679-014-0118-7. PMID: 26626915.
Copy Download .nbib Format: NLM
Effects of the recombinant allergen rDer f 2 on neuro-endocrino-immune network in asthmatic mice.

Central-European journal of immunology

Jiang YQ, Zhou ZX, Ji YL.
PMID: 26155138
Cent Eur J Immunol. 2014;39(3):294-8. doi: 10.5114/ceji.2014.45938. Epub 2014 Oct 14.

Severe and life-threatening side effects can occur in patients receiving allergen-specific immunotherapy (SIT), and recombinant allergens made from cDNA have been used in clinical trials for ten years and appear promising for SIT. The aim of this study is...

Cite
Jiang YQ, Zhou ZX, Ji YL. Effects of the recombinant allergen rDer f 2 on neuro-endocrino-immune network in asthmatic mice. Cent Eur J Immunol. 2014;39(3):294-8doi: 10.5114/ceji.2014.45938.
Jiang, Y. Q., Zhou, Z. X., & Ji, Y. L. (2014). Effects of the recombinant allergen rDer f 2 on neuro-endocrino-immune network in asthmatic mice. Central-European journal of immunology, 39(3), 294-8. https://doi.org/10.5114/ceji.2014.45938
Jiang, Yong-Qian, et al. "Effects of the recombinant allergen rDer f 2 on neuro-endocrino-immune network in asthmatic mice." Central-European journal of immunology vol. 39,3 (2014): 294-8. doi: https://doi.org/10.5114/ceji.2014.45938
Jiang YQ, Zhou ZX, Ji YL. Effects of the recombinant allergen rDer f 2 on neuro-endocrino-immune network in asthmatic mice. Cent Eur J Immunol. 2014;39(3):294-8. doi: 10.5114/ceji.2014.45938. Epub 2014 Oct 14. PMID: 26155138; PMCID: PMC4440009.
Copy Download .nbib Format: NLM
Intermittent Access to Ethanol Induces Escalated Alcohol Consumption in Primates.

Journal of addictive behaviors, therapy & rehabilitation

Lindell SG, Schwandt ML, Suomi SJ, Rice KC, Heilig M, Barr CS.
PMID: 29082267
J Addict Behav Ther Rehabil. 2017 Apr;6(1). doi: 10.4172/2324-9005.1000163. Epub 2017 Apr 07.

BACKGROUND: Escalation of voluntary alcohol drinking is characteristic of alcohol addiction and can be induced in rodents using intermittent access to alcohol. This model has been used to evaluate candidate therapeutics, but key systems involved in the transition into...

Cite
Lindell SG, Schwandt ML, Suomi SJ, et al. Intermittent Access to Ethanol Induces Escalated Alcohol Consumption in Primates. J Addict Behav Ther Rehabil. 2017;6(1)doi: 10.4172/2324-9005.1000163.
Lindell, S. G., Schwandt, M. L., Suomi, S. J., Rice, K. C., Heilig, M., & Barr, C. S. (2017). Intermittent Access to Ethanol Induces Escalated Alcohol Consumption in Primates. Journal of addictive behaviors, therapy & rehabilitation, 6(1), . https://doi.org/10.4172/2324-9005.1000163
Lindell, S G, et al. "Intermittent Access to Ethanol Induces Escalated Alcohol Consumption in Primates." Journal of addictive behaviors, therapy & rehabilitation vol. 6,1 (2017). doi: https://doi.org/10.4172/2324-9005.1000163
Lindell SG, Schwandt ML, Suomi SJ, Rice KC, Heilig M, Barr CS. Intermittent Access to Ethanol Induces Escalated Alcohol Consumption in Primates. J Addict Behav Ther Rehabil. 2017 Apr;6(1). doi: 10.4172/2324-9005.1000163. Epub 2017 Apr 07. PMID: 29082267; PMCID: PMC5658047.
Copy Download .nbib Format: NLM
Regulation of the hypothalamic-pituitary-adrenal axis by neuropeptides.

Hormone molecular biology and clinical investigation

Aguilera G.
PMID: 25961271
Horm Mol Biol Clin Investig. 2011 Oct 01;7(2):327-36. doi: 10.1515/HMBCI.2011.123.

The major endocrine response to stress occurs via activation of the hypothalamic-pituitary-adrenal (HPA) axis, leading ultimately to increases in circulating glucocorticoids, which are essential for the metabolic adaptation to stress. The major players in the HPA axis are the...

Cite
Aguilera G. Regulation of the hypothalamic-pituitary-adrenal axis by neuropeptides. Horm Mol Biol Clin Investig. 2011;7(2):327-36doi: 10.1515/HMBCI.2011.123.
Aguilera, G. (2011). Regulation of the hypothalamic-pituitary-adrenal axis by neuropeptides. Hormone molecular biology and clinical investigation, 7(2), 327-36. https://doi.org/10.1515/HMBCI.2011.123
Aguilera, Greti. "Regulation of the hypothalamic-pituitary-adrenal axis by neuropeptides." Hormone molecular biology and clinical investigation vol. 7,2 (2011): 327-36. doi: https://doi.org/10.1515/HMBCI.2011.123
Aguilera G. Regulation of the hypothalamic-pituitary-adrenal axis by neuropeptides. Horm Mol Biol Clin Investig. 2011 Oct 01;7(2):327-36. doi: 10.1515/HMBCI.2011.123. PMID: 25961271.
Copy Download .nbib Format: NLM
Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus.

Obesity research & clinical practice

Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H.
PMID: 24351449
Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002.

Under restraint stress conditions, prepro-thyrotropin releasing hormone (TRH) 178-199 suppresses adrenocorticotropic hormone (ACTH) secretion from the rat pituitary, which indicates that prepro-TRH 178-199 is a candidate endogenous corticotropin releasing inhibitory factor (CRIF). Restraint stress also activates the release of...

Cite
Fukagawa T, Fukagawa K, Gotoh K, et al. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007;1(2):I-IIdoi: 10.1016/j.orcp.2007.01.002.
Fukagawa, T., Fukagawa, K., Gotoh, K., Noguchi, H., & Yoshimatsu, H. (2007). Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obesity research & clinical practice, 1(2), I-II. https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa, Tomiyo, et al. "Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus." Obesity research & clinical practice vol. 1,2 (2007): I-II. doi: https://doi.org/10.1016/j.orcp.2007.01.002
Fukagawa T, Fukagawa K, Gotoh K, Noguchi H, Yoshimatsu H. Prepro-TRH 178-199 inhibits histamine- or restraint stress-induced activation of corticotropin releasing hormone production in rat hypothalamus. Obes Res Clin Pract. 2007 May;1(2):I-II. doi: 10.1016/j.orcp.2007.01.002. PMID: 24351449.
Copy Download .nbib Format: NLM
Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders.

Acta neuropsychiatrica

Juruena MF, Cleare AJ, Bauer ME, Pariante CM.
PMID: 26983771
Acta Neuropsychiatr. 2003 Dec;15(6):354-67. doi: 10.1046/j.1601-5215.2003.00051.x.

Changes in the hypothalamic-pituitary-adrenocortical (HPA) system are characteristic of depression, and in the majority of these patients these result in HPA axis hyperactivity. This is further supported by the reduced sensitivity to the inhibitory effects of the glucocorticoid, dexamethasone...

Cite
Juruena MF, Cleare AJ, Bauer ME, et al. Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta Neuropsychiatr. 2003;15(6):354-67doi: 10.1046/j.1601-5215.2003.00051.x.
Juruena, M. F., Cleare, A. J., Bauer, M. E., & Pariante, C. M. (2003). Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta neuropsychiatrica, 15(6), 354-67. https://doi.org/10.1046/j.1601-5215.2003.00051.x
Juruena, Mario F, et al. "Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders." Acta neuropsychiatrica vol. 15,6 (2003): 354-67. doi: https://doi.org/10.1046/j.1601-5215.2003.00051.x
Juruena MF, Cleare AJ, Bauer ME, Pariante CM. Molecular mechanisms of glucocorticoid receptor sensitivity and relevance to affective disorders. Acta Neuropsychiatr. 2003 Dec;15(6):354-67. doi: 10.1046/j.1601-5215.2003.00051.x. PMID: 26983771.
Copy Download .nbib Format: NLM
Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals.

Journal of neurogastroenterology and motility

Yamasaki T, Tomita T, Takimoto M, Kondo T, Tozawa K, Ohda Y, Oshima T, Fukui H, Watari J, Miwa H.
PMID: 28992676
J Neurogastroenterol Motil. 2017 Oct 30;23(4):526-532. doi: 10.5056/jnm17067.

BACKGROUND/AIMS: When a person is experiencing stress, corticotropin-releasing hormone (CRH) can modulate gut physiologies, such as visceral sensation or gastrointestinal motility, and its intravenous administration mimics stress-induced physiological changes. However, the influence of CRH on the esophagus is yet...

Cite
Yamasaki T, Tomita T, Takimoto M, et al. Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. J Neurogastroenterol Motil. 2017;23(4):526-532doi: 10.5056/jnm17067.
Yamasaki, T., Tomita, T., Takimoto, M., Kondo, T., Tozawa, K., Ohda, Y., Oshima, T., Fukui, H., Watari, J., & Miwa, H. (2017). Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. Journal of neurogastroenterology and motility, 23(4), 526-532. https://doi.org/10.5056/jnm17067
Yamasaki, Takahisa, et al. "Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals." Journal of neurogastroenterology and motility vol. 23,4 (2017): 526-532. doi: https://doi.org/10.5056/jnm17067
Yamasaki T, Tomita T, Takimoto M, Kondo T, Tozawa K, Ohda Y, Oshima T, Fukui H, Watari J, Miwa H. Intravenous Corticotropin-releasing Hormone Administration Increases Esophageal Electrical Sensitivity in Healthy Individuals. J Neurogastroenterol Motil. 2017 Oct 30;23(4):526-532. doi: 10.5056/jnm17067. PMID: 28992676; PMCID: PMC5628984.
Copy Download .nbib Format: NLM
Peripheral endotoxin causes long-lasting changes in locus coeruleus activity via IL-1 in the brain.

Acta neuropsychiatrica

Borsody MK, Weiss JM.
PMID: 26984577
Acta Neuropsychiatr. 2002 Dec;14(6):303-21. doi: 10.1034/j.1601-5215.2002.140605.x.

Activity of locus coeruleus (LC) neurons, the major noradrenergic cell-body group in the brain whose axons give rise to approximately 70% of norepinephrine (NE) in the brain, is believed to play an important role in attention/vigilance, cognitive functions and...

Cite
Borsody MK, Weiss JM. Peripheral endotoxin causes long-lasting changes in locus coeruleus activity via IL-1 in the brain. Acta Neuropsychiatr. 2002;14(6):303-21doi: 10.1034/j.1601-5215.2002.140605.x.
Borsody, M. K., & Weiss, J. M. (2002). Peripheral endotoxin causes long-lasting changes in locus coeruleus activity via IL-1 in the brain. Acta neuropsychiatrica, 14(6), 303-21. https://doi.org/10.1034/j.1601-5215.2002.140605.x
Borsody, Mark K, and Weiss, Jay M. "Peripheral endotoxin causes long-lasting changes in locus coeruleus activity via IL-1 in the brain." Acta neuropsychiatrica vol. 14,6 (2002): 303-21. doi: https://doi.org/10.1034/j.1601-5215.2002.140605.x
Borsody MK, Weiss JM. Peripheral endotoxin causes long-lasting changes in locus coeruleus activity via IL-1 in the brain. Acta Neuropsychiatr. 2002 Dec;14(6):303-21. doi: 10.1034/j.1601-5215.2002.140605.x. PMID: 26984577.
Copy Download .nbib Format: NLM
Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case.

Obstetric medicine

Trinh A, Chan I, Alexiadis M, Pell M, Kumar B, Fuller PJ.
PMID: 27512490
Obstet Med. 2016 Mar;9(1):43-5. doi: 10.1177/1753495X15618542. Epub 2015 Dec 09.

BACKGROUND: Cushing's syndrome is rare during pregnancy and more commonly due to adrenal pathology, in contrast to the non-pregnant population. Increased levels of cortisol-binding globulin and placental production of corticotropin-releasing hormone and adrenocorticotropic hormone complicate the diagnostic strategies usually...

Cite
Trinh A, Chan I, Alexiadis M, et al. Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstet Med. 2015;9(1):43-5doi: 10.1177/1753495X15618542.
Trinh, A., Chan, I., Alexiadis, M., Pell, M., Kumar, B., & Fuller, P. J. (2016). Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstetric medicine, 9(1), 43-5. https://doi.org/10.1177/1753495X15618542
Trinh, Anne, et al. "Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case." Obstetric medicine vol. 9,1 (2016): 43-5. doi: https://doi.org/10.1177/1753495X15618542
Trinh A, Chan I, Alexiadis M, Pell M, Kumar B, Fuller PJ. Adrenal Cushing's syndrome in pregnancy: Clinical and molecular characterisation of a case. Obstet Med. 2016 Mar;9(1):43-5. doi: 10.1177/1753495X15618542. Epub 2015 Dec 09. PMID: 27512490; PMCID: PMC4950436.
Copy Download .nbib Format: NLM
Mid-gut ACTH-secreting neuroendocrine tumor unmasked with (18)F-dihydroxyphenylalanine-positron emission tomography.

Endocrinology, diabetes & metabolism case reports

Ducry J, Gomez F, Prior JO, Boubaker A, Matter M, Monti M, Pu Y, Pitteloud N, Portmann L.
PMID: 25861450
Endocrinol Diabetes Metab Case Rep. 2015;2015:140104. doi: 10.1530/EDM-14-0104. Epub 2015 Mar 01.

UNLABELLED: Ectopic ACTH Cushing's syndrome (EAS) is often caused by neuroendocrine tumors (NETs) of lungs, pancreas, thymus, and other less frequent locations. Localizing the source of ACTH can be challenging. A 64-year-old man presented with rapidly progressing fatigue, muscular...

Cite
Ducry J, Gomez F, Prior JO, et al. Mid-gut ACTH-secreting neuroendocrine tumor unmasked with (18)F-dihydroxyphenylalanine-positron emission tomography. Endocrinol Diabetes Metab Case Rep. 2015;2015:140104doi: 10.1530/EDM-14-0104.
Ducry, J., Gomez, F., Prior, J. O., Boubaker, A., Matter, M., Monti, M., Pu, Y., Pitteloud, N., & Portmann, L. (2015). Mid-gut ACTH-secreting neuroendocrine tumor unmasked with (18)F-dihydroxyphenylalanine-positron emission tomography. Endocrinology, diabetes & metabolism case reports, 2015140104. https://doi.org/10.1530/EDM-14-0104
Ducry, Julien, et al. "Mid-gut ACTH-secreting neuroendocrine tumor unmasked with (18)F-dihydroxyphenylalanine-positron emission tomography." Endocrinology, diabetes & metabolism case reports vol. 2015 (2015): 140104. doi: https://doi.org/10.1530/EDM-14-0104
Ducry J, Gomez F, Prior JO, Boubaker A, Matter M, Monti M, Pu Y, Pitteloud N, Portmann L. Mid-gut ACTH-secreting neuroendocrine tumor unmasked with (18)F-dihydroxyphenylalanine-positron emission tomography. Endocrinol Diabetes Metab Case Rep. 2015;2015:140104. doi: 10.1530/EDM-14-0104. Epub 2015 Mar 01. PMID: 25861450; PMCID: PMC4361871.
Copy Download .nbib Format: NLM
An Atraumatic Symphysiolysis with a Unilateral Injured Sacroiliac Joint in a Patient with Cushing's Disease: A Loss of Pelvic Stability Related to Ligamentous Insufficiency?.

Case reports in orthopedics

Höch A, Pieroh P, Dehghani F, Josten C, Böhme J.
PMID: 26904337
Case Rep Orthop. 2016;2016:9250938. doi: 10.1155/2016/9250938. Epub 2016 Jan 20.

Glucocorticoids are well known for altering bone structure and elevating fracture risk. Nevertheless, there are very few reports on pelvic ring fractures, compared to other bones, especially with a predominantly ligamentous insufficiency, resulting in a rotationally unstable pelvic girdle....

Cite
Höch A, Pieroh P, Dehghani F, et al. An Atraumatic Symphysiolysis with a Unilateral Injured Sacroiliac Joint in a Patient with Cushing's Disease: A Loss of Pelvic Stability Related to Ligamentous Insufficiency?. Case Rep Orthop. 2016;2016:9250938doi: 10.1155/2016/9250938.
Höch, A., Pieroh, P., Dehghani, F., Josten, C., & Böhme, J. (2016). An Atraumatic Symphysiolysis with a Unilateral Injured Sacroiliac Joint in a Patient with Cushing's Disease: A Loss of Pelvic Stability Related to Ligamentous Insufficiency?. Case reports in orthopedics, 20169250938. https://doi.org/10.1155/2016/9250938
Höch, Andreas, et al. "An Atraumatic Symphysiolysis with a Unilateral Injured Sacroiliac Joint in a Patient with Cushing's Disease: A Loss of Pelvic Stability Related to Ligamentous Insufficiency?." Case reports in orthopedics vol. 2016 (2016): 9250938. doi: https://doi.org/10.1155/2016/9250938
Höch A, Pieroh P, Dehghani F, Josten C, Böhme J. An Atraumatic Symphysiolysis with a Unilateral Injured Sacroiliac Joint in a Patient with Cushing's Disease: A Loss of Pelvic Stability Related to Ligamentous Insufficiency?. Case Rep Orthop. 2016;2016:9250938. doi: 10.1155/2016/9250938. Epub 2016 Jan 20. PMID: 26904337; PMCID: PMC4745920.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 386 entries