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Modification of Gene Expression of Connexins in the Rat Corpus Epididymis by Estradiol Benzoate or Flutamide Exposure at the Early Neonatal Age.

Development & reproduction

Lee KH.
PMID: 27004263
Dev Reprod. 2015 Jun;19(2):69-77. doi: 10.12717/DR.2015.19.2.069.

Cell-cell direct communication through channel-forming molecules, connexin (Cx), is essential for a tissue to exchange signaling molecules between neighboring cells and establish unique functional characteristics during postnatal development. The corpus epididymis is a well-known androgen-responsive tissue and involves in...

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Lee KH. Modification of Gene Expression of Connexins in the Rat Corpus Epididymis by Estradiol Benzoate or Flutamide Exposure at the Early Neonatal Age. Dev Reprod. 2015;19(2):69-77doi: 10.12717/DR.2015.19.2.069.
Lee, K. H. (2015). Modification of Gene Expression of Connexins in the Rat Corpus Epididymis by Estradiol Benzoate or Flutamide Exposure at the Early Neonatal Age. Development & reproduction, 19(2), 69-77. https://doi.org/10.12717/DR.2015.19.2.069
Lee, Ki-Ho. "Modification of Gene Expression of Connexins in the Rat Corpus Epididymis by Estradiol Benzoate or Flutamide Exposure at the Early Neonatal Age." Development & reproduction vol. 19,2 (2015): 69-77. doi: https://doi.org/10.12717/DR.2015.19.2.069
Lee KH. Modification of Gene Expression of Connexins in the Rat Corpus Epididymis by Estradiol Benzoate or Flutamide Exposure at the Early Neonatal Age. Dev Reprod. 2015 Jun;19(2):69-77. doi: 10.12717/DR.2015.19.2.069. PMID: 27004263; PMCID: PMC4801050.
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Connexin30.2: In Vitro Interaction with Connexin36 in HeLa Cells and Expression in AII Amacrine Cells and Intrinsically Photosensitive Ganglion Cells in the Mouse Retina.

Frontiers in molecular neuroscience

Meyer A, Tetenborg S, Greb H, Segelken J, Dorgau B, Weiler R, Hormuzdi SG, Janssen-Bienhold U, Dedek K.
PMID: 27303262
Front Mol Neurosci. 2016 May 27;9:36. doi: 10.3389/fnmol.2016.00036. eCollection 2016.

Electrical coupling via gap junctions is an abundant phenomenon in the mammalian retina and occurs in all major cell types. Gap junction channels are assembled from different connexin subunits, and the connexin composition of the channel confers specific properties...

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Meyer A, Tetenborg S, Greb H, et al. Connexin30.2: In Vitro Interaction with Connexin36 in HeLa Cells and Expression in AII Amacrine Cells and Intrinsically Photosensitive Ganglion Cells in the Mouse Retina. Front Mol Neurosci. 2016;9:36doi: 10.3389/fnmol.2016.00036.
Meyer, A., Tetenborg, S., Greb, H., Segelken, J., Dorgau, B., Weiler, R., Hormuzdi, S. G., Janssen-Bienhold, U., & Dedek, K. (2016). Connexin30.2: In Vitro Interaction with Connexin36 in HeLa Cells and Expression in AII Amacrine Cells and Intrinsically Photosensitive Ganglion Cells in the Mouse Retina. Frontiers in molecular neuroscience, 936. https://doi.org/10.3389/fnmol.2016.00036
Meyer, Arndt, et al. "Connexin30.2: In Vitro Interaction with Connexin36 in HeLa Cells and Expression in AII Amacrine Cells and Intrinsically Photosensitive Ganglion Cells in the Mouse Retina." Frontiers in molecular neuroscience vol. 9 (2016): 36. doi: https://doi.org/10.3389/fnmol.2016.00036
Meyer A, Tetenborg S, Greb H, Segelken J, Dorgau B, Weiler R, Hormuzdi SG, Janssen-Bienhold U, Dedek K. Connexin30.2: In Vitro Interaction with Connexin36 in HeLa Cells and Expression in AII Amacrine Cells and Intrinsically Photosensitive Ganglion Cells in the Mouse Retina. Front Mol Neurosci. 2016 May 27;9:36. doi: 10.3389/fnmol.2016.00036. eCollection 2016. PMID: 27303262; PMCID: PMC4882342.
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On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells.

Frontiers in physiology

Vargas AA, Cisterna BA, Saavedra-Leiva F, Urrutia C, Cea LA, Vielma AH, Gutierrez-Maldonado SE, Martin AJ, Pareja-Barrueto C, Escalona Y, Schmachtenberg O, Lagos CF, Perez-Acle T, Sáez JC.
PMID: 28232803
Front Physiol. 2017 Feb 09;8:38. doi: 10.3389/fphys.2017.00038. eCollection 2017.

Although connexins (Cxs) are broadly expressed by cells of mammalian organisms, Cx39 has a very restricted pattern of expression and the biophysical properties of Cx39-based channels [hemichannels (HCs) and gap junction channels (GJCs)] remain largely unknown. Here, we used...

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Vargas AA, Cisterna BA, Saavedra-Leiva F, et al. On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells. Front Physiol. 2017;8:38doi: 10.3389/fphys.2017.00038.
Vargas, A. A., Cisterna, B. A., Saavedra-Leiva, F., Urrutia, C., Cea, L. A., Vielma, A. H., Gutierrez-Maldonado, S. E., Martin, A. J., Pareja-Barrueto, C., Escalona, Y., Schmachtenberg, O., Lagos, C. F., Perez-Acle, T., & Sáez, J. C. (2017). On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells. Frontiers in physiology, 838. https://doi.org/10.3389/fphys.2017.00038
Vargas, Anibal A, et al. "On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells." Frontiers in physiology vol. 8 (2017): 38. doi: https://doi.org/10.3389/fphys.2017.00038
Vargas AA, Cisterna BA, Saavedra-Leiva F, Urrutia C, Cea LA, Vielma AH, Gutierrez-Maldonado SE, Martin AJ, Pareja-Barrueto C, Escalona Y, Schmachtenberg O, Lagos CF, Perez-Acle T, Sáez JC. On Biophysical Properties and Sensitivity to Gap Junction Blockers of Connexin 39 Hemichannels Expressed in HeLa Cells. Front Physiol. 2017 Feb 09;8:38. doi: 10.3389/fphys.2017.00038. eCollection 2017. PMID: 28232803; PMCID: PMC5298994.
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Prenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet.

Frontiers in cellular neuroscience

Orellana JA, Busso D, Ramírez G, Campos M, Rigotti A, Eugenín J, von Bernhardi R.
PMID: 25520621
Front Cell Neurosci. 2014 Dec 02;8:403. doi: 10.3389/fncel.2014.00403. eCollection 2014.

Nicotine, the most important neuroteratogen of tobacco smoke, can reproduce brain and cognitive disturbances per se when administered prenatally. However, it is still unknown if paracrine signaling among brain cells participates in prenatal nicotine-induced brain impairment of adult offspring....

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Orellana JA, Busso D, Ramírez G, et al. Prenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet. Front Cell Neurosci. 2014;8:403doi: 10.3389/fncel.2014.00403.
Orellana, J. A., Busso, D., Ramírez, G., Campos, M., Rigotti, A., Eugenín, J., & von Bernhardi, R. (2014). Prenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet. Frontiers in cellular neuroscience, 8403. https://doi.org/10.3389/fncel.2014.00403
Orellana, Juan A, et al. "Prenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet." Frontiers in cellular neuroscience vol. 8 (2014): 403. doi: https://doi.org/10.3389/fncel.2014.00403
Orellana JA, Busso D, Ramírez G, Campos M, Rigotti A, Eugenín J, von Bernhardi R. Prenatal nicotine exposure enhances Cx43 and Panx1 unopposed channel activity in brain cells of adult offspring mice fed a high-fat/cholesterol diet. Front Cell Neurosci. 2014 Dec 02;8:403. doi: 10.3389/fncel.2014.00403. eCollection 2014. PMID: 25520621; PMCID: PMC4251442.
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Roles of gap junctions, connexins, and pannexins in epilepsy.

Frontiers in physiology

Mylvaganam S, Ramani M, Krawczyk M, Carlen PL.
PMID: 24847276
Front Physiol. 2014 May 07;5:172. doi: 10.3389/fphys.2014.00172. eCollection 2014.

Enhanced gap junctional communication (GJC) between neurons is considered a major factor underlying the neuronal synchrony driving seizure activity. In addition, the hippocampal sharp wave ripple complexes, associated with learning and seizures, are diminished by GJC blocking agents. Although...

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Mylvaganam S, Ramani M, Krawczyk M, et al. Roles of gap junctions, connexins, and pannexins in epilepsy. Front Physiol. 2014;5:172doi: 10.3389/fphys.2014.00172.
Mylvaganam, S., Ramani, M., Krawczyk, M., & Carlen, P. L. (2014). Roles of gap junctions, connexins, and pannexins in epilepsy. Frontiers in physiology, 5172. https://doi.org/10.3389/fphys.2014.00172
Mylvaganam, Shanthini, et al. "Roles of gap junctions, connexins, and pannexins in epilepsy." Frontiers in physiology vol. 5 (2014): 172. doi: https://doi.org/10.3389/fphys.2014.00172
Mylvaganam S, Ramani M, Krawczyk M, Carlen PL. Roles of gap junctions, connexins, and pannexins in epilepsy. Front Physiol. 2014 May 07;5:172. doi: 10.3389/fphys.2014.00172. eCollection 2014. PMID: 24847276; PMCID: PMC4019879.
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Connexins and pannexins in liver damage.

EXCLI journal

Crespo Yanguas S, Willebrords J, Maes M, da Silva TC, Veloso Alves Pereira I, Cogliati B, Zaidan Dagli ML, Vinken M.
PMID: 27065778
EXCLI J. 2016 Feb 25;15:177-86. doi: 10.17179/excli2016-119. eCollection 2016.

Connexins and pannexins are key players in the control of cellular communication and thus in the maintenance of tissue homeostasis. Inherent to this function these proteins are frequently involved in pathological processes. The present paper reviews the role of...

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Crespo Yanguas S, Willebrords J, Maes M, et al. Connexins and pannexins in liver damage. EXCLI J. 2016;15:177-86doi: 10.17179/excli2016-119.
Crespo Yanguas, S., Willebrords, J., Maes, M., da Silva, T. C., Veloso Alves Pereira, I., Cogliati, B., Zaidan Dagli, M. L., & Vinken, M. (2016). Connexins and pannexins in liver damage. EXCLI journal, 15177-86. https://doi.org/10.17179/excli2016-119
Crespo Yanguas, Sara, et al. "Connexins and pannexins in liver damage." EXCLI journal vol. 15 (2016): 177-86. doi: https://doi.org/10.17179/excli2016-119
Crespo Yanguas S, Willebrords J, Maes M, da Silva TC, Veloso Alves Pereira I, Cogliati B, Zaidan Dagli ML, Vinken M. Connexins and pannexins in liver damage. EXCLI J. 2016 Feb 25;15:177-86. doi: 10.17179/excli2016-119. eCollection 2016. PMID: 27065778; PMCID: PMC4822047.
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The Expression of Connexins and SOX2 Reflects the Plasticity of Glioma Stem-Like Cells.

Translational oncology

Balça-Silva J, Matias D, Dubois LG, Carneiro B, do Carmo A, Girão H, Ferreira F, Ferrer VP, Chimelli L, Filho PN, Tão H, Rebelo O, Barbosa M, Sarmento-Ribeiro AB, Lopes MC, Moura-Neto V.
PMID: 28654819
Transl Oncol. 2017 Aug;10(4):555-569. doi: 10.1016/j.tranon.2017.04.005. Epub 2017 Jun 24.

Glioblastoma (GBM) is the most malignant primary brain tumor, with an average survival rate of 15 months. GBM is highly refractory to therapy, and such unresponsiveness is due, primarily, but not exclusively, to the glioma stem-like cells (GSCs). This...

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Balça-Silva J, Matias D, Dubois LG, et al. The Expression of Connexins and SOX2 Reflects the Plasticity of Glioma Stem-Like Cells. Transl Oncol. 2017;10(4):555-569doi: 10.1016/j.tranon.2017.04.005.
Balça-Silva, J., Matias, D., Dubois, L. G., Carneiro, B., do Carmo, A., Girão, H., Ferreira, F., Ferrer, V. P., Chimelli, L., Filho, P. N., Tão, H., Rebelo, O., Barbosa, M., Sarmento-Ribeiro, A. B., Lopes, M. C., & Moura-Neto, V. (2017). The Expression of Connexins and SOX2 Reflects the Plasticity of Glioma Stem-Like Cells. Translational oncology, 10(4), 555-569. https://doi.org/10.1016/j.tranon.2017.04.005
Balça-Silva, Joana, et al. "The Expression of Connexins and SOX2 Reflects the Plasticity of Glioma Stem-Like Cells." Translational oncology vol. 10,4 (2017): 555-569. doi: https://doi.org/10.1016/j.tranon.2017.04.005
Balça-Silva J, Matias D, Dubois LG, Carneiro B, do Carmo A, Girão H, Ferreira F, Ferrer VP, Chimelli L, Filho PN, Tão H, Rebelo O, Barbosa M, Sarmento-Ribeiro AB, Lopes MC, Moura-Neto V. The Expression of Connexins and SOX2 Reflects the Plasticity of Glioma Stem-Like Cells. Transl Oncol. 2017 Aug;10(4):555-569. doi: 10.1016/j.tranon.2017.04.005. Epub 2017 Jun 24. PMID: 28654819; PMCID: PMC5487246.
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Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells.

Cell biology international reports

Wan CK, O'Carroll SJ, Kim SL, Green CR, Nicholson LF.
PMID: 25505515
Cell Biol Int Rep (2010). 2013 Dec;20(2):13-23. doi: 10.1002/cbi3.10005. Epub 2013 Aug 30.

While connexins (Cxs) are thought to be involved in differentiation, their expression and role has yet to be fully elucidated. We investigated the temporal expression of Cx30, Cx36 and Cx43 in two in vitro models of neuronal differentiation: human...

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Wan CK, O'Carroll SJ, Kim SL, et al. Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells. Cell Biol Int Rep (2010). 2013;20(2):13-23doi: 10.1002/cbi3.10005.
Wan, C. K., O'Carroll, S. J., Kim, S. L., Green, C. R., & Nicholson, L. F. (2013). Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells. Cell biology international reports, 20(2), 13-23. https://doi.org/10.1002/cbi3.10005
Wan, Carthur K, et al. "Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells." Cell biology international reports vol. 20,2 (2013): 13-23. doi: https://doi.org/10.1002/cbi3.10005
Wan CK, O'Carroll SJ, Kim SL, Green CR, Nicholson LF. Spatiotemporal changes in Cx30 and Cx43 expression during neuronal differentiation of P19 EC and NT2/D1 cells. Cell Biol Int Rep (2010). 2013 Dec;20(2):13-23. doi: 10.1002/cbi3.10005. Epub 2013 Aug 30. PMID: 25505515; PMCID: PMC4255794.
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Effect and mechanism of the metastasis suppressor gene BRMS1 on the migration of breast cancer cells.

International journal of clinical and experimental medicine

Yang YL, Chen CZ, Jin LP, Ji QQ, Chen YZ, Li Q, Zhang XH, Qu JM.
PMID: 24260596
Int J Clin Exp Med. 2013 Oct 25;6(10):908-16. eCollection 2013.

OBJECTIVE: To study the effect of the transfected Breast cancer metastasis suppressor 1 (BRMS1) gene on the migration of breast cancer cells and the possible mechanisms involved.METHODS: MDA-MB-231HM cells which have a high propensity of metastasize to lung was...

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Yang YL, Chen CZ, Jin LP, et al. Effect and mechanism of the metastasis suppressor gene BRMS1 on the migration of breast cancer cells. Int J Clin Exp Med. 2013;6(10):908-16
Yang, Y. L., Chen, C. Z., Jin, L. P., Ji, Q. Q., Chen, Y. Z., Li, Q., Zhang, X. H., & Qu, J. M. (2013). Effect and mechanism of the metastasis suppressor gene BRMS1 on the migration of breast cancer cells. International journal of clinical and experimental medicine, 6(10), 908-16.
Yang, Yin-Long, et al. "Effect and mechanism of the metastasis suppressor gene BRMS1 on the migration of breast cancer cells." International journal of clinical and experimental medicine vol. 6,10 (2013): 908-16.
Yang YL, Chen CZ, Jin LP, Ji QQ, Chen YZ, Li Q, Zhang XH, Qu JM. Effect and mechanism of the metastasis suppressor gene BRMS1 on the migration of breast cancer cells. Int J Clin Exp Med. 2013 Oct 25;6(10):908-16. eCollection 2013. PMID: 24260596; PMCID: PMC3832327.
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MEK inhibitor can reverse the resistance to bevacizumab in A549 cells harboring Kirsten rat sarcoma oncogene homolog mutation.

Thoracic cancer

Wang Y, Yan P, Liu Z, Yang X, Wang Y, Shen Z, Bai H, Wang J, Wang Z.
PMID: 27148412
Thorac Cancer. 2016 Apr 26;7(3):279-87. doi: 10.1111/1759-7714.12325. Epub 2015 Dec 16.

BACKGROUND: Bevacizumab (BV) is broadly used to treat a number of cancers; however, BV resistance mechanisms and strategies to overcome this resistance are yet to be determined.METHODS: We established xenograft mice models harboring Kirsten rat sarcoma oncogene homolog (KRAS)...

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Wang Y, Yan P, Liu Z, et al. MEK inhibitor can reverse the resistance to bevacizumab in A549 cells harboring Kirsten rat sarcoma oncogene homolog mutation. Thorac Cancer. 2015;7(3):279-87doi: 10.1111/1759-7714.12325.
Wang, Y., Yan, P., Liu, Z., Yang, X., Wang, Y., Shen, Z., Bai, H., Wang, J., & Wang, Z. (2016). MEK inhibitor can reverse the resistance to bevacizumab in A549 cells harboring Kirsten rat sarcoma oncogene homolog mutation. Thoracic cancer, 7(3), 279-87. https://doi.org/10.1111/1759-7714.12325
Wang, Yuan, et al. "MEK inhibitor can reverse the resistance to bevacizumab in A549 cells harboring Kirsten rat sarcoma oncogene homolog mutation." Thoracic cancer vol. 7,3 (2016): 279-87. doi: https://doi.org/10.1111/1759-7714.12325
Wang Y, Yan P, Liu Z, Yang X, Wang Y, Shen Z, Bai H, Wang J, Wang Z. MEK inhibitor can reverse the resistance to bevacizumab in A549 cells harboring Kirsten rat sarcoma oncogene homolog mutation. Thorac Cancer. 2016 Apr 26;7(3):279-87. doi: 10.1111/1759-7714.12325. Epub 2015 Dec 16. PMID: 27148412; PMCID: PMC4846615.
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Targeting motifs and functional parameters governing the assembly of connexins into gap junctions.

The Biochemical journal

Martin PE, Steggles J, Wilson C, Ahmad S, Evans WH.
PMID: 10970812
Biochem J. 2000 Sep 15;350:943.

No abstract available.

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Martin PE, Steggles J, Wilson C, et al. Targeting motifs and functional parameters governing the assembly of connexins into gap junctions. Biochem J. 2000;350:943
Martin, P. E., Steggles, J., Wilson, C., Ahmad, S., & Evans, W. H. (2000). Targeting motifs and functional parameters governing the assembly of connexins into gap junctions. The Biochemical journal, 350943.
Martin, et al. "Targeting motifs and functional parameters governing the assembly of connexins into gap junctions." The Biochemical journal vol. 350 (2000): 943.
Martin PE, Steggles J, Wilson C, Ahmad S, Evans WH. Targeting motifs and functional parameters governing the assembly of connexins into gap junctions. Biochem J. 2000 Sep 15;350:943. PMID: 10970812; PMCID: PMC1221330.
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Connexins and the heart.

Trends in cardiovascular medicine

Fishman GI.
PMID: 21239274
Trends Cardiovasc Med. 1992 Mar-Apr;2(2):50-5. doi: 10.1016/1050-1738(92)90004-C.

Gap junctions are specialized regions of adjoining cell membranes composed of numerous intercellular low-resistance channels. In the heart, these channels electrotonically couple adjacent myocytes and synchronize the cardiac action potential. Signaling through gap junction channels may also influence embryogenesis...

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Fishman GI. Connexins and the heart. Trends Cardiovasc Med. 1992;2(2):50-5doi: 10.1016/1050-1738(92)90004-C.
Fishman, G. I. (1992). Connexins and the heart. Trends in cardiovascular medicine, 2(2), 50-5. https://doi.org/10.1016/1050-1738(92)90004-C
Fishman, G I. "Connexins and the heart." Trends in cardiovascular medicine vol. 2,2 (1992): 50-5. doi: https://doi.org/10.1016/1050-1738(92)90004-C
Fishman GI. Connexins and the heart. Trends Cardiovasc Med. 1992 Mar-Apr;2(2):50-5. doi: 10.1016/1050-1738(92)90004-C. PMID: 21239274.
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Showing 1 to 12 of 313 entries