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Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease.

HGG advances

Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW.
PMID: 34888534
HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29.

Bicuspid aortic valve (BAV) with ~1%-2% prevalence is the most common congenital heart defect (CHD). It frequently results in valve disease and aorta dilation and is a major cause of adult cardiac surgery. BAV is genetically linked to rare...

Cite
Teekakirikul P, Zhu W, Gabriel GC, et al. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021;2(3)doi: 10.1016/j.xhgg.2021.100037.
Teekakirikul, P., Zhu, W., Gabriel, G. C., Young, C. B., Williams, K., Martin, L. J., Hill, J. C., Richards, T., Billaud, M., Phillippi, J. A., Wang, J., Wu, Y., Tan, T., Devine, W., Lin, J. H., Bais, A. S., Klonowski, J., de Bellaing, A. M., Saini, A., Wang, M. X., Emerel, L., Salamacha, N., Wyman, S. K., Lee, C., Sing Li, H., Miron, A., Zhang, J., Xing, J., McNamara, D. M., Fung, E., Kirshbom, P., Mahle, W., Kochilas, L. K., He, Y., Garg, V., White, P., McBride, K. L., Benson, D. W., Gleason, T. G., Mital, S., & Lo, C. W. (2021). Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG advances, 2(3), . https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul, Polakit, et al. "Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease." HGG advances vol. 2,3 (2021). doi: https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29. PMID: 34888534; PMCID: PMC8653519.
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Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas.

Applied and environmental microbiology

Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M.
PMID: 34469192
Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01.

Nordic Seas are the subarctic seas connecting the Arctic Ocean and North Atlantic Ocean with complex water masses, experiencing an abrupt climate change. Though knowledge of the marine virosphere has expanded rapidly, the diversity of viruses and their relationships...

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Gao C, Xia J, Zhou X, et al. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021;87(22):e0116021doi: 10.1128/AEM.01160-21.
Gao, C., Xia, J., Zhou, X., Liang, Y., Jiang, Y., Wang, M., Shao, H., Shi, X., Guo, C., He, H., Wang, H., He, J., Hu, D., Wang, X., Zhao, J., Zhang, Y. Z., Sung, Y. Y., Mok, W. J., Wong, L. L., McMinn, A., Suttle, C. A., & Wang, M. (2021). Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Applied and environmental microbiology, 87(22), e0116021. https://doi.org/10.1128/AEM.01160-21
Gao, Chen, et al. "Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas." Applied and environmental microbiology vol. 87,22 (2021): e0116021. doi: https://doi.org/10.1128/AEM.01160-21
Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01. PMID: 34469192; PMCID: PMC8552889.
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Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease.

HGG advances

Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW.
PMID: 34888534
HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29.

Bicuspid aortic valve (BAV) with ~1%-2% prevalence is the most common congenital heart defect (CHD). It frequently results in valve disease and aorta dilation and is a major cause of adult cardiac surgery. BAV is genetically linked to rare...

Cite
Teekakirikul P, Zhu W, Gabriel GC, et al. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021;2(3)doi: 10.1016/j.xhgg.2021.100037.
Teekakirikul, P., Zhu, W., Gabriel, G. C., Young, C. B., Williams, K., Martin, L. J., Hill, J. C., Richards, T., Billaud, M., Phillippi, J. A., Wang, J., Wu, Y., Tan, T., Devine, W., Lin, J. H., Bais, A. S., Klonowski, J., de Bellaing, A. M., Saini, A., Wang, M. X., Emerel, L., Salamacha, N., Wyman, S. K., Lee, C., Sing Li, H., Miron, A., Zhang, J., Xing, J., McNamara, D. M., Fung, E., Kirshbom, P., Mahle, W., Kochilas, L. K., He, Y., Garg, V., White, P., McBride, K. L., Benson, D. W., Gleason, T. G., Mital, S., & Lo, C. W. (2021). Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG advances, 2(3), . https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul, Polakit, et al. "Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease." HGG advances vol. 2,3 (2021). doi: https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29. PMID: 34888534; PMCID: PMC8653519.
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THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS.

The Journal of experimental medicine

Lurie MB.
PMID: 19869977
J Exp Med. 1932 Jan 01;55(1):31-54. doi: 10.1084/jem.55.1.31.

It has been found that although there is some parallelism between the quantity of tubercle bacilli demonstrable histologically and the number of colonies that can be isolated from a given tissue, the culture method is far the more efficient...

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Lurie MB. THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. J Exp Med. 1932;55(1):31-54doi: 10.1084/jem.55.1.31.
Lurie, M. B. (1932). THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. The Journal of experimental medicine, 55(1), 31-54. https://doi.org/10.1084/jem.55.1.31
Lurie, M B. "THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS." The Journal of experimental medicine vol. 55,1 (1932): 31-54. doi: https://doi.org/10.1084/jem.55.1.31
Lurie MB. THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. J Exp Med. 1932 Jan 01;55(1):31-54. doi: 10.1084/jem.55.1.31. PMID: 19869977; PMCID: PMC2132067.
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Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction.

Cardiovascular research

Xu JY, Xiong YY, Tang RJ, Jiang WY, Ning Y, Gong ZT, Huang PS, Chen GH, Xu J, Wu CX, Hu MJ, Xu J, Xu Y, Huang CR, Jin C, Lu XT, Qian HY, Li XD, Yang YJ.
PMID: 34259869
Cardiovasc Res. 2021 Jul 14; doi: 10.1093/cvr/cvab237. Epub 2021 Jul 14.

AIMS: Interleukin (IL)-5 mediates the development of eosinophils (EOS) that are essential for tissue post-injury repair. It remains unknown whether IL-5 plays a role in heart repair after myocardial infarction (MI). This study aims to test whether IL-5-induced EOS...

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Xu JY, Xiong YY, Tang RJ, et al. Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovasc Res. 2021;doi: 10.1093/cvr/cvab237.
Xu, J. Y., Xiong, Y. Y., Tang, R. J., Jiang, W. Y., Ning, Y., Gong, Z. T., Huang, P. S., Chen, G. H., Xu, J., Wu, C. X., Hu, M. J., Xu, J., Xu, Y., Huang, C. R., Jin, C., Lu, X. T., Qian, H. Y., Li, X. D., & Yang, Y. J. (2021). Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovascular research, . https://doi.org/10.1093/cvr/cvab237
Xu, Jun-Yan, et al. "Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction." Cardiovascular research vol. (2021). doi: https://doi.org/10.1093/cvr/cvab237
Xu JY, Xiong YY, Tang RJ, Jiang WY, Ning Y, Gong ZT, Huang PS, Chen GH, Xu J, Wu CX, Hu MJ, Xu J, Xu Y, Huang CR, Jin C, Lu XT, Qian HY, Li XD, Yang YJ. Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovasc Res. 2021 Jul 14; doi: 10.1093/cvr/cvab237. Epub 2021 Jul 14. PMID: 34259869.
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Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074392
Ont Health Technol Assess Ser. 2010;10(15):1-45. Epub 2010 Jul 01.

UNLABELLED: In July 2009, the Medical Advisory Secretariat (MAS) began work on Non-Invasive Cardiac Imaging Technologies for the Assessment of Myocardial Viability, an evidence-based review of the literature surrounding different cardiac imaging modalities to ensure that appropriate technologies are...

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Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(15):1-45
(2010). Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ontario health technology assessment series, 10(15), 1-45.
"Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis." Ontario health technology assessment series vol. 10,15 (2010): 1-45.
Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(15):1-45. Epub 2010 Jul 01. PMID: 23074392; PMCID: PMC3426228.
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Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074534
Ont Health Technol Assess Ser. 2009;9(4):1-51. Epub 2009 Jul 01.

UNLABELLED: Endovascular repair of abdominal aortic aneurysm (AAA) allows the exclusion of the dilated aneurismal segment of the aorta from the systematic circulation. The procedure requires, however, that the endograft extends to the healthy parts of the aorta above...

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Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ont Health Technol Assess Ser. 2009;9(4):1-51
(2009). Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ontario health technology assessment series, 9(4), 1-51.
"Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis." Ontario health technology assessment series vol. 9,4 (2009): 1-51.
Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ont Health Technol Assess Ser. 2009;9(4):1-51. Epub 2009 Jul 01. PMID: 23074534; PMCID: PMC3377528.
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Positron emission tomography for the assessment of myocardial viability: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074393
Ont Health Technol Assess Ser. 2010;10(16):1-80. Epub 2010 Jul 01.

UNLABELLED: In July 2009, the Medical Advisory Secretariat (MAS) began work on Non-Invasive Cardiac Imaging Technologies for the Assessment of Myocardial Viability, an evidence-based review of the literature surrounding different cardiac imaging modalities to ensure that appropriate technologies are...

Cite
Positron emission tomography for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(16):1-80
(2010). Positron emission tomography for the assessment of myocardial viability: an evidence-based analysis. Ontario health technology assessment series, 10(16), 1-80.
"Positron emission tomography for the assessment of myocardial viability: an evidence-based analysis." Ontario health technology assessment series vol. 10,16 (2010): 1-80.
Positron emission tomography for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(16):1-80. Epub 2010 Jul 01. PMID: 23074393; PMCID: PMC3377573.
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Catalyzing Transcriptomics Research in Cardiovascular Disease: The CardioRNA COST Action CA17129.

Non-coding RNA

Gomes CPC, Ágg B, Andova A, Arslan S, Baker A, Barteková M, Beis D, Betsou F, Wettinger SB, Bugarski B, Condorelli G, Silva GJJD, Danilin S, de Gonzalo-Calvo D, Buil A, Carmo-Fonseca M, Enguita FJ, Felekkis K, Ferdinandy P, Gyöngyösi M, Hackl M, Karaduzovic-Hadziabdic K, Hellemans J, Heymans S, Hlavackova M, Hoydal MA, Jankovic A, Jusic A, Kardassis D, Kerkelä R, Kuster GM, Lakkisto P, Leszek P, Lustrek M, Maegdefessel L, Martelli F, Novella S, O'Brien T, Papaneophytou C, Pedrazzini T, Pinet F, Popescu O, Potočnjak I, Robinson E, Sasson S, Scholz M, Simionescu M, Stoll M, Varga ZV, Vinciguerra M, Xuereb A, Yilmaz MB, Emanueli C, Devaux Y, on behalf of the EU-CardioRNA COST Action (CA17129).
PMID: 30934986
Noncoding RNA. 2019 Mar 29;5(2). doi: 10.3390/ncrna5020031.

Cardiovascular disease (CVD) remains the leading cause of death worldwide and, despite continuous advances, better diagnostic and prognostic tools, as well as therapy, are needed. The human transcriptome, which is the set of all RNA produced in a cell,...

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Gomes CPC, Ágg B, Andova A, et al. Catalyzing Transcriptomics Research in Cardiovascular Disease: The CardioRNA COST Action CA17129. Noncoding RNA. 2019;5(2)doi: 10.3390/ncrna5020031.
Gomes, C. P. C., Ágg, B., Andova, A., Arslan, S., Baker, A., Barteková, M., Beis, D., Betsou, F., Wettinger, S. B., Bugarski, B., Condorelli, G., Silva, G. J. J. D., Danilin, S., de Gonzalo-Calvo, D., Buil, A., Carmo-Fonseca, M., Enguita, F. J., Felekkis, K., Ferdinandy, P., Gyöngyösi, M., Hackl, M., Karaduzovic-Hadziabdic, K., Hellemans, J., Heymans, S., Hlavackova, M., Hoydal, M. A., Jankovic, A., Jusic, A., Kardassis, D., Kerkelä, R., Kuster, G. M., Lakkisto, P., Leszek, P., Lustrek, M., Maegdefessel, L., Martelli, F., Novella, S., O'Brien, T., Papaneophytou, C., Pedrazzini, T., Pinet, F., Popescu, O., Potočnjak, I., Robinson, E., Sasson, S., Scholz, M., Simionescu, M., Stoll, M., Varga, Z. V., Vinciguerra, M., Xuereb, A., Yilmaz, M. B., Emanueli, C., Devaux, Y., & on behalf of the EU-CardioRNA COST Action (CA17129). (2019). Catalyzing Transcriptomics Research in Cardiovascular Disease: The CardioRNA COST Action CA17129. Non-coding RNA, 5(2), . https://doi.org/10.3390/ncrna5020031
Gomes, Clarissa Pedrosa da C, et al. "Catalyzing Transcriptomics Research in Cardiovascular Disease: The CardioRNA COST Action CA17129." Non-coding RNA vol. 5,2 (2019). doi: https://doi.org/10.3390/ncrna5020031
Gomes CPC, Ágg B, Andova A, Arslan S, Baker A, Barteková M, Beis D, Betsou F, Wettinger SB, Bugarski B, Condorelli G, Silva GJJD, Danilin S, de Gonzalo-Calvo D, Buil A, Carmo-Fonseca M, Enguita FJ, Felekkis K, Ferdinandy P, Gyöngyösi M, Hackl M, Karaduzovic-Hadziabdic K, Hellemans J, Heymans S, Hlavackova M, Hoydal MA, Jankovic A, Jusic A, Kardassis D, Kerkelä R, Kuster GM, Lakkisto P, Leszek P, Lustrek M, Maegdefessel L, Martelli F, Novella S, O'Brien T, Papaneophytou C, Pedrazzini T, Pinet F, Popescu O, Potočnjak I, Robinson E, Sasson S, Scholz M, Simionescu M, Stoll M, Varga ZV, Vinciguerra M, Xuereb A, Yilmaz MB, Emanueli C, Devaux Y, on behalf of the EU-CardioRNA COST Action (CA17129). Catalyzing Transcriptomics Research in Cardiovascular Disease: The CardioRNA COST Action CA17129. Noncoding RNA. 2019 Mar 29;5(2). doi: 10.3390/ncrna5020031. PMID: 30934986; PMCID: PMC6630366.
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Left ventricular assist devices: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074453
Ont Health Technol Assess Ser. 2004;4(3):1-69. Epub 2004 Mar 01.

OBJECTIVE: The objective of this health technology policy assessment was to determine the effectiveness and cost-effectiveness of using implantable ventricular assist devices in the treatment of end-stage heart failure.HEART FAILURE: Heart failure is a complex syndrome that impairs the...

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Left ventricular assist devices: an evidence-based analysis. Ont Health Technol Assess Ser. 2004;4(3):1-69
(2004). Left ventricular assist devices: an evidence-based analysis. Ontario health technology assessment series, 4(3), 1-69.
"Left ventricular assist devices: an evidence-based analysis." Ontario health technology assessment series vol. 4,3 (2004): 1-69.
Left ventricular assist devices: an evidence-based analysis. Ont Health Technol Assess Ser. 2004;4(3):1-69. Epub 2004 Mar 01. PMID: 23074453; PMCID: PMC3387736.
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Periodontal Disease as a Risk Factor for Rheumatoid Arthritis: A Systematic Review.

JBI library of systematic reviews

Kaur S, White S, Bartold M.
PMID: 27820156
JBI Libr Syst Rev. 2012;10(42):1-12. doi: 10.11124/jbisrir-2012-288.

REVIEW OBJECTIVE: The objective of this systematic review is to identify and synthesise the best available evidence to examine whether periodontal disease is a risk factor for rheumatoid arthritis.BACKGROUND: Rheumatoid arthritis (RA) is a chronic inflammatory disease of unknown...

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Kaur S, White S, Bartold M. Periodontal Disease as a Risk Factor for Rheumatoid Arthritis: A Systematic Review. JBI Libr Syst Rev. 2012;10(42):1-12doi: 10.11124/jbisrir-2012-288.
Kaur, S., White, S., & Bartold, M. (2012). Periodontal Disease as a Risk Factor for Rheumatoid Arthritis: A Systematic Review. JBI library of systematic reviews, 10(42), 1-12. https://doi.org/10.11124/jbisrir-2012-288
Kaur, Sushil, et al. "Periodontal Disease as a Risk Factor for Rheumatoid Arthritis: A Systematic Review." JBI library of systematic reviews vol. 10,42 (2012): 1-12. doi: https://doi.org/10.11124/jbisrir-2012-288
Kaur S, White S, Bartold M. Periodontal Disease as a Risk Factor for Rheumatoid Arthritis: A Systematic Review. JBI Libr Syst Rev. 2012;10(42):1-12. doi: 10.11124/jbisrir-2012-288. PMID: 27820156.
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STUDIES ON EXPERIMENTAL HYPERTENSION : IX. THE EFFECT ON BLOOD PRESSURE OF CONSTRICTION OF THE ABDOMINAL AORTA ABOVE AND BELOW THE SITE OF ORIGIN OF BOTH MAIN RENAL ARTERIES.

The Journal of experimental medicine

Goldblatt H, Kahn JR, Hanzal RF.
PMID: 19870869
J Exp Med. 1939 Apr 30;69(5):649-74. doi: 10.1084/jem.69.5.649.

Constriction of the aorta just above the origin of both main renal arteries invariably resulted in elevation of the carotid systolic and carotid mean pressure. The hypertension was not immediate, but developed in about the same time as after...

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Goldblatt H, Kahn JR, Hanzal RF. STUDIES ON EXPERIMENTAL HYPERTENSION : IX. THE EFFECT ON BLOOD PRESSURE OF CONSTRICTION OF THE ABDOMINAL AORTA ABOVE AND BELOW THE SITE OF ORIGIN OF BOTH MAIN RENAL ARTERIES. J Exp Med. 1939;69(5):649-74doi: 10.1084/jem.69.5.649.
Goldblatt, H., Kahn, J. R., & Hanzal, R. F. (1939). STUDIES ON EXPERIMENTAL HYPERTENSION : IX. THE EFFECT ON BLOOD PRESSURE OF CONSTRICTION OF THE ABDOMINAL AORTA ABOVE AND BELOW THE SITE OF ORIGIN OF BOTH MAIN RENAL ARTERIES. The Journal of experimental medicine, 69(5), 649-74. https://doi.org/10.1084/jem.69.5.649
Goldblatt, H, et al. "STUDIES ON EXPERIMENTAL HYPERTENSION : IX. THE EFFECT ON BLOOD PRESSURE OF CONSTRICTION OF THE ABDOMINAL AORTA ABOVE AND BELOW THE SITE OF ORIGIN OF BOTH MAIN RENAL ARTERIES." The Journal of experimental medicine vol. 69,5 (1939): 649-74. doi: https://doi.org/10.1084/jem.69.5.649
Goldblatt H, Kahn JR, Hanzal RF. STUDIES ON EXPERIMENTAL HYPERTENSION : IX. THE EFFECT ON BLOOD PRESSURE OF CONSTRICTION OF THE ABDOMINAL AORTA ABOVE AND BELOW THE SITE OF ORIGIN OF BOTH MAIN RENAL ARTERIES. J Exp Med. 1939 Apr 30;69(5):649-74. doi: 10.1084/jem.69.5.649. PMID: 19870869; PMCID: PMC2133753.
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Showing 1 to 12 of 27737 entries