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Genome-wide Study of Atrial Fibrillation Identifies Seven Risk Loci and Highlights Biological Pathways and Regulatory Elements Involved in Cardiac Development.

American journal of human genetics

Nielsen JB, Fritsche LG, Zhou W, Teslovich TM, Holmen OL, Gustafsson S, Gabrielsen ME, Schmidt EM, Beaumont R, Wolford BN, Lin M, Brummett CM, Preuss MH, Refsgaard L, Bottinger EP, Graham SE, Surakka I, Chu Y, Skogholt AH, Dalen H, Boyle AP, Oral H, Herron TJ, Kitzman J, Jalife J, Svendsen JH, Olesen MS, Njølstad I, Løchen ML, Baras A, Gottesman O, Marcketta A, O'Dushlaine C, Ritchie MD, Wilsgaard T, Loos RJF, Frayling TM, Boehnke M, Ingelsson E, Carey DJ, Dewey FE, Kang HM, Abecasis GR, Hveem K, Willer CJ.
PMID: 29290336
Am J Hum Genet. 2018 Jan 04;102(1):103-115. doi: 10.1016/j.ajhg.2017.12.003. Epub 2017 Dec 28.

Atrial fibrillation (AF) is a common cardiac arrhythmia and a major risk factor for stroke, heart failure, and premature death. The pathogenesis of AF remains poorly understood, which contributes to the current lack of highly effective treatments. To understand...

Cite
Nielsen JB, Fritsche LG, Zhou W, et al. Genome-wide Study of Atrial Fibrillation Identifies Seven Risk Loci and Highlights Biological Pathways and Regulatory Elements Involved in Cardiac Development. Am J Hum Genet. 2017;102(1):103-115doi: 10.1016/j.ajhg.2017.12.003.
Nielsen, J. B., Fritsche, L. G., Zhou, W., Teslovich, T. M., Holmen, O. L., Gustafsson, S., Gabrielsen, M. E., Schmidt, E. M., Beaumont, R., Wolford, B. N., Lin, M., Brummett, C. M., Preuss, M. H., Refsgaard, L., Bottinger, E. P., Graham, S. E., Surakka, I., Chu, Y., Skogholt, A. H., Dalen, H., Boyle, A. P., Oral, H., Herron, T. J., Kitzman, J., Jalife, J., Svendsen, J. H., Olesen, M. S., Njølstad, I., Løchen, M. L., Baras, A., Gottesman, O., Marcketta, A., O'Dushlaine, C., Ritchie, M. D., Wilsgaard, T., Loos, R. J. F., Frayling, T. M., Boehnke, M., Ingelsson, E., Carey, D. J., Dewey, F. E., Kang, H. M., Abecasis, G. R., Hveem, K., & Willer, C. J. (2018). Genome-wide Study of Atrial Fibrillation Identifies Seven Risk Loci and Highlights Biological Pathways and Regulatory Elements Involved in Cardiac Development. American journal of human genetics, 102(1), 103-115. https://doi.org/10.1016/j.ajhg.2017.12.003
Nielsen, Jonas B, et al. "Genome-wide Study of Atrial Fibrillation Identifies Seven Risk Loci and Highlights Biological Pathways and Regulatory Elements Involved in Cardiac Development." American journal of human genetics vol. 102,1 (2018): 103-115. doi: https://doi.org/10.1016/j.ajhg.2017.12.003
Nielsen JB, Fritsche LG, Zhou W, Teslovich TM, Holmen OL, Gustafsson S, Gabrielsen ME, Schmidt EM, Beaumont R, Wolford BN, Lin M, Brummett CM, Preuss MH, Refsgaard L, Bottinger EP, Graham SE, Surakka I, Chu Y, Skogholt AH, Dalen H, Boyle AP, Oral H, Herron TJ, Kitzman J, Jalife J, Svendsen JH, Olesen MS, Njølstad I, Løchen ML, Baras A, Gottesman O, Marcketta A, O'Dushlaine C, Ritchie MD, Wilsgaard T, Loos RJF, Frayling TM, Boehnke M, Ingelsson E, Carey DJ, Dewey FE, Kang HM, Abecasis GR, Hveem K, Willer CJ. Genome-wide Study of Atrial Fibrillation Identifies Seven Risk Loci and Highlights Biological Pathways and Regulatory Elements Involved in Cardiac Development. Am J Hum Genet. 2018 Jan 04;102(1):103-115. doi: 10.1016/j.ajhg.2017.12.003. Epub 2017 Dec 28. PMID: 29290336; PMCID: PMC5777936.
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Biobank-driven genomic discovery yields new insight into atrial fibrillation biology.

Nature genetics

Nielsen JB, Thorolfsdottir RB, Fritsche LG, Zhou W, Skov MW, Graham SE, Herron TJ, McCarthy S, Schmidt EM, Sveinbjornsson G, Surakka I, Mathis MR, Yamazaki M, Crawford RD, Gabrielsen ME, Skogholt AH, Holmen OL, Lin M, Wolford BN, Dey R, Dalen H, Sulem P, Chung JH, Backman JD, Arnar DO, Thorsteinsdottir U, Baras A, O'Dushlaine C, Holst AG, Wen X, Hornsby W, Dewey FE, Boehnke M, Kheterpal S, Mukherjee B, Lee S, Kang HM, Holm H, Kitzman J, Shavit JA, Jalife J, Brummett CM, Teslovich TM, Carey DJ, Gudbjartsson DF, Stefansson K, Abecasis GR, Hveem K, Willer CJ.
PMID: 30061737
Nat Genet. 2018 Sep;50(9):1234-1239. doi: 10.1038/s41588-018-0171-3. Epub 2018 Jul 30.

To identify genetic variation underlying atrial fibrillation, the most common cardiac arrhythmia, we performed a genome-wide association study of >1,000,000 people, including 60,620 atrial fibrillation cases and 970,216 controls. We identified 142 independent risk variants at 111 loci and...

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Nielsen JB, Thorolfsdottir RB, Fritsche LG, et al. Biobank-driven genomic discovery yields new insight into atrial fibrillation biology. Nat Genet. 2018;50(9):1234-1239doi: 10.1038/s41588-018-0171-3.
Nielsen, J. B., Thorolfsdottir, R. B., Fritsche, L. G., Zhou, W., Skov, M. W., Graham, S. E., Herron, T. J., McCarthy, S., Schmidt, E. M., Sveinbjornsson, G., Surakka, I., Mathis, M. R., Yamazaki, M., Crawford, R. D., Gabrielsen, M. E., Skogholt, A. H., Holmen, O. L., Lin, M., Wolford, B. N., Dey, R., Dalen, H., Sulem, P., Chung, J. H., Backman, J. D., Arnar, D. O., Thorsteinsdottir, U., Baras, A., O'Dushlaine, C., Holst, A. G., Wen, X., Hornsby, W., Dewey, F. E., Boehnke, M., Kheterpal, S., Mukherjee, B., Lee, S., Kang, H. M., Holm, H., Kitzman, J., Shavit, J. A., Jalife, J., Brummett, C. M., Teslovich, T. M., Carey, D. J., Gudbjartsson, D. F., Stefansson, K., Abecasis, G. R., Hveem, K., & Willer, C. J. (2018). Biobank-driven genomic discovery yields new insight into atrial fibrillation biology. Nature genetics, 50(9), 1234-1239. https://doi.org/10.1038/s41588-018-0171-3
Nielsen, Jonas B, et al. "Biobank-driven genomic discovery yields new insight into atrial fibrillation biology." Nature genetics vol. 50,9 (2018): 1234-1239. doi: https://doi.org/10.1038/s41588-018-0171-3
Nielsen JB, Thorolfsdottir RB, Fritsche LG, Zhou W, Skov MW, Graham SE, Herron TJ, McCarthy S, Schmidt EM, Sveinbjornsson G, Surakka I, Mathis MR, Yamazaki M, Crawford RD, Gabrielsen ME, Skogholt AH, Holmen OL, Lin M, Wolford BN, Dey R, Dalen H, Sulem P, Chung JH, Backman JD, Arnar DO, Thorsteinsdottir U, Baras A, O'Dushlaine C, Holst AG, Wen X, Hornsby W, Dewey FE, Boehnke M, Kheterpal S, Mukherjee B, Lee S, Kang HM, Holm H, Kitzman J, Shavit JA, Jalife J, Brummett CM, Teslovich TM, Carey DJ, Gudbjartsson DF, Stefansson K, Abecasis GR, Hveem K, Willer CJ. Biobank-driven genomic discovery yields new insight into atrial fibrillation biology. Nat Genet. 2018 Sep;50(9):1234-1239. doi: 10.1038/s41588-018-0171-3. Epub 2018 Jul 30. PMID: 30061737; PMCID: PMC6530775.
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Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease.

HGG advances

Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW.
PMID: 34888534
HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29.

Bicuspid aortic valve (BAV) with ~1%-2% prevalence is the most common congenital heart defect (CHD). It frequently results in valve disease and aorta dilation and is a major cause of adult cardiac surgery. BAV is genetically linked to rare...

Cite
Teekakirikul P, Zhu W, Gabriel GC, et al. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021;2(3)doi: 10.1016/j.xhgg.2021.100037.
Teekakirikul, P., Zhu, W., Gabriel, G. C., Young, C. B., Williams, K., Martin, L. J., Hill, J. C., Richards, T., Billaud, M., Phillippi, J. A., Wang, J., Wu, Y., Tan, T., Devine, W., Lin, J. H., Bais, A. S., Klonowski, J., de Bellaing, A. M., Saini, A., Wang, M. X., Emerel, L., Salamacha, N., Wyman, S. K., Lee, C., Sing Li, H., Miron, A., Zhang, J., Xing, J., McNamara, D. M., Fung, E., Kirshbom, P., Mahle, W., Kochilas, L. K., He, Y., Garg, V., White, P., McBride, K. L., Benson, D. W., Gleason, T. G., Mital, S., & Lo, C. W. (2021). Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG advances, 2(3), . https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul, Polakit, et al. "Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease." HGG advances vol. 2,3 (2021). doi: https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29. PMID: 34888534; PMCID: PMC8653519.
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Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas.

Applied and environmental microbiology

Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M.
PMID: 34469192
Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01.

Nordic Seas are the subarctic seas connecting the Arctic Ocean and North Atlantic Ocean with complex water masses, experiencing an abrupt climate change. Though knowledge of the marine virosphere has expanded rapidly, the diversity of viruses and their relationships...

Cite
Gao C, Xia J, Zhou X, et al. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021;87(22):e0116021doi: 10.1128/AEM.01160-21.
Gao, C., Xia, J., Zhou, X., Liang, Y., Jiang, Y., Wang, M., Shao, H., Shi, X., Guo, C., He, H., Wang, H., He, J., Hu, D., Wang, X., Zhao, J., Zhang, Y. Z., Sung, Y. Y., Mok, W. J., Wong, L. L., McMinn, A., Suttle, C. A., & Wang, M. (2021). Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Applied and environmental microbiology, 87(22), e0116021. https://doi.org/10.1128/AEM.01160-21
Gao, Chen, et al. "Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas." Applied and environmental microbiology vol. 87,22 (2021): e0116021. doi: https://doi.org/10.1128/AEM.01160-21
Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01. PMID: 34469192; PMCID: PMC8552889.
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Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease.

HGG advances

Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW.
PMID: 34888534
HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29.

Bicuspid aortic valve (BAV) with ~1%-2% prevalence is the most common congenital heart defect (CHD). It frequently results in valve disease and aorta dilation and is a major cause of adult cardiac surgery. BAV is genetically linked to rare...

Cite
Teekakirikul P, Zhu W, Gabriel GC, et al. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021;2(3)doi: 10.1016/j.xhgg.2021.100037.
Teekakirikul, P., Zhu, W., Gabriel, G. C., Young, C. B., Williams, K., Martin, L. J., Hill, J. C., Richards, T., Billaud, M., Phillippi, J. A., Wang, J., Wu, Y., Tan, T., Devine, W., Lin, J. H., Bais, A. S., Klonowski, J., de Bellaing, A. M., Saini, A., Wang, M. X., Emerel, L., Salamacha, N., Wyman, S. K., Lee, C., Sing Li, H., Miron, A., Zhang, J., Xing, J., McNamara, D. M., Fung, E., Kirshbom, P., Mahle, W., Kochilas, L. K., He, Y., Garg, V., White, P., McBride, K. L., Benson, D. W., Gleason, T. G., Mital, S., & Lo, C. W. (2021). Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG advances, 2(3), . https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul, Polakit, et al. "Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease." HGG advances vol. 2,3 (2021). doi: https://doi.org/10.1016/j.xhgg.2021.100037
Teekakirikul P, Zhu W, Gabriel GC, Young CB, Williams K, Martin LJ, Hill JC, Richards T, Billaud M, Phillippi JA, Wang J, Wu Y, Tan T, Devine W, Lin JH, Bais AS, Klonowski J, de Bellaing AM, Saini A, Wang MX, Emerel L, Salamacha N, Wyman SK, Lee C, Sing Li H, Miron A, Zhang J, Xing J, McNamara DM, Fung E, Kirshbom P, Mahle W, Kochilas LK, He Y, Garg V, White P, McBride KL, Benson DW, Gleason TG, Mital S, Lo CW. Common deletion variants causing protocadherin-α deficiency contribute to the complex genetics of BAV and left-sided congenital heart disease. HGG Adv. 2021 Jul 08;2(3). doi: 10.1016/j.xhgg.2021.100037. Epub 2021 Jul 29. PMID: 34888534; PMCID: PMC8653519.
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Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas.

Applied and environmental microbiology

Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M.
PMID: 34469192
Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01.

Nordic Seas are the subarctic seas connecting the Arctic Ocean and North Atlantic Ocean with complex water masses, experiencing an abrupt climate change. Though knowledge of the marine virosphere has expanded rapidly, the diversity of viruses and their relationships...

Cite
Gao C, Xia J, Zhou X, et al. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021;87(22):e0116021doi: 10.1128/AEM.01160-21.
Gao, C., Xia, J., Zhou, X., Liang, Y., Jiang, Y., Wang, M., Shao, H., Shi, X., Guo, C., He, H., Wang, H., He, J., Hu, D., Wang, X., Zhao, J., Zhang, Y. Z., Sung, Y. Y., Mok, W. J., Wong, L. L., McMinn, A., Suttle, C. A., & Wang, M. (2021). Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Applied and environmental microbiology, 87(22), e0116021. https://doi.org/10.1128/AEM.01160-21
Gao, Chen, et al. "Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas." Applied and environmental microbiology vol. 87,22 (2021): e0116021. doi: https://doi.org/10.1128/AEM.01160-21
Gao C, Xia J, Zhou X, Liang Y, Jiang Y, Wang M, Shao H, Shi X, Guo C, He H, Wang H, He J, Hu D, Wang X, Zhao J, Zhang YZ, Sung YY, Mok WJ, Wong LL, McMinn A, Suttle CA, Wang M. Viral Characteristics of the Warm Atlantic and Cold Arctic Water Masses in the Nordic Seas. Appl Environ Microbiol. 2021 Oct 28;87(22):e0116021. doi: 10.1128/AEM.01160-21. Epub 2021 Sep 01. PMID: 34469192; PMCID: PMC8552889.
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THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS.

The Journal of experimental medicine

Lurie MB.
PMID: 19869977
J Exp Med. 1932 Jan 01;55(1):31-54. doi: 10.1084/jem.55.1.31.

It has been found that although there is some parallelism between the quantity of tubercle bacilli demonstrable histologically and the number of colonies that can be isolated from a given tissue, the culture method is far the more efficient...

Cite
Lurie MB. THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. J Exp Med. 1932;55(1):31-54doi: 10.1084/jem.55.1.31.
Lurie, M. B. (1932). THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. The Journal of experimental medicine, 55(1), 31-54. https://doi.org/10.1084/jem.55.1.31
Lurie, M B. "THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS." The Journal of experimental medicine vol. 55,1 (1932): 31-54. doi: https://doi.org/10.1084/jem.55.1.31
Lurie MB. THE CORRELATION BETWEEN THE HISTOLOGICAL CHANGES AND THE FATE OF LIVING TUBERCLE BACILLI IN THE ORGANS OF TUBERCULOUS RABBITS. J Exp Med. 1932 Jan 01;55(1):31-54. doi: 10.1084/jem.55.1.31. PMID: 19869977; PMCID: PMC2132067.
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Effect of Genetically Low 25-Hydroxyvitamin D on Mortality Risk: Mendelian Randomization Analysis in 3 Large European Cohorts.

Nutrients

Aspelund T, Grübler MR, Smith AV, Gudmundsson EF, Keppel M, Cotch MF, Harris TB, Jorde R, Grimnes G, Joakimsen R, Schirmer H, Wilsgaard T, Mathiesen EB, Njølstad I, Løchen ML, März W, Kleber ME, Tomaschitz A, Grove-Laugesen D, Rejnmark L, Swart KMA, Brouwer IA, Lips P, van Schoor NM, Sempos CT, Durazo-Arvizu RA, Škrabáková Z, Dowling KG, Cashman KD, Kiely M, Pilz S, Gudnason V, Eiriksdottir G.
PMID: 30609725
Nutrients. 2019 Jan 02;11(1). doi: 10.3390/nu11010074.

The aim of this study was to determine if increased mortality associated with low levels of serum 25-hydroxyvitamin D (25(OH)D) reflects a causal relationship by using a Mendelian randomisation (MR) approach with genetic variants in the vitamin D synthesis...

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Aspelund T, Grübler MR, Smith AV, et al. Effect of Genetically Low 25-Hydroxyvitamin D on Mortality Risk: Mendelian Randomization Analysis in 3 Large European Cohorts. Nutrients. 2019;11(1)doi: 10.3390/nu11010074.
Aspelund, T., Grübler, M. R., Smith, A. V., Gudmundsson, E. F., Keppel, M., Cotch, M. F., Harris, T. B., Jorde, R., Grimnes, G., Joakimsen, R., Schirmer, H., Wilsgaard, T., Mathiesen, E. B., Njølstad, I., Løchen, M. L., März, W., Kleber, M. E., Tomaschitz, A., Grove-Laugesen, D., Rejnmark, L., Swart, K. M. A., Brouwer, I. A., Lips, P., van Schoor, N. M., Sempos, C. T., Durazo-Arvizu, R. A., Škrabáková, Z., Dowling, K. G., Cashman, K. D., Kiely, M., Pilz, S., Gudnason, V., & Eiriksdottir, G. (2019). Effect of Genetically Low 25-Hydroxyvitamin D on Mortality Risk: Mendelian Randomization Analysis in 3 Large European Cohorts. Nutrients, 11(1), . https://doi.org/10.3390/nu11010074
Aspelund, Thor, et al. "Effect of Genetically Low 25-Hydroxyvitamin D on Mortality Risk: Mendelian Randomization Analysis in 3 Large European Cohorts." Nutrients vol. 11,1 (2019). doi: https://doi.org/10.3390/nu11010074
Aspelund T, Grübler MR, Smith AV, Gudmundsson EF, Keppel M, Cotch MF, Harris TB, Jorde R, Grimnes G, Joakimsen R, Schirmer H, Wilsgaard T, Mathiesen EB, Njølstad I, Løchen ML, März W, Kleber ME, Tomaschitz A, Grove-Laugesen D, Rejnmark L, Swart KMA, Brouwer IA, Lips P, van Schoor NM, Sempos CT, Durazo-Arvizu RA, Škrabáková Z, Dowling KG, Cashman KD, Kiely M, Pilz S, Gudnason V, Eiriksdottir G. Effect of Genetically Low 25-Hydroxyvitamin D on Mortality Risk: Mendelian Randomization Analysis in 3 Large European Cohorts. Nutrients. 2019 Jan 02;11(1). doi: 10.3390/nu11010074. PMID: 30609725; PMCID: PMC6356674.
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Global outbreak of severe Mycobacterium chimaera disease after cardiac surgery: a molecular epidemiological study.

The Lancet. Infectious diseases

van Ingen J, Kohl TA, Kranzer K, Hasse B, Keller PM, Katarzyna Szafrańska A, Hillemann D, Chand M, Schreiber PW, Sommerstein R, Berger C, Genoni M, Rüegg C, Troillet N, Widmer AF, Becker SL, Herrmann M, Eckmanns T, Haller S, Höller C, Debast SB, Wolfhagen MJ, Hopman J, Kluytmans J, Langelaar M, Notermans DW, Ten Oever J, van den Barselaar P, Vonk ABA, Vos MC, Ahmed N, Brown T, Crook D, Lamagni T, Phin N, Smith EG, Zambon M, Serr A, Götting T, Ebner W, Thürmer A, Utpatel C, Spröer C, Bunk B, Nübel U, Bloemberg GV, Böttger EC, Niemann S, Wagner D, Sax H.
PMID: 28711585
Lancet Infect Dis. 2017 Oct;17(10):1033-1041. doi: 10.1016/S1473-3099(17)30324-9. Epub 2017 Jul 12.

BACKGROUND: Since 2013, over 100 cases of Mycobacterium chimaera prosthetic valve endocarditis and disseminated disease were notified in Europe and the USA, linked to contaminated heater-cooler units (HCUs) used during cardiac surgery. We did a molecular epidemiological investigation to...

Cite
van Ingen J, Kohl TA, Kranzer K, et al. Global outbreak of severe Mycobacterium chimaera disease after cardiac surgery: a molecular epidemiological study. Lancet Infect Dis. 2017;17(10):1033-1041doi: 10.1016/S1473-3099(17)30324-9.
van Ingen, J., Kohl, T. A., Kranzer, K., Hasse, B., Keller, P. M., Katarzyna Szafrańska, A., Hillemann, D., Chand, M., Schreiber, P. W., Sommerstein, R., Berger, C., Genoni, M., Rüegg, C., Troillet, N., Widmer, A. F., Becker, S. L., Herrmann, M., Eckmanns, T., Haller, S., Höller, C., Debast, S. B., Wolfhagen, M. J., Hopman, J., Kluytmans, J., Langelaar, M., Notermans, D. W., Ten Oever, J., van den Barselaar, P., Vonk, A. B. A., Vos, M. C., Ahmed, N., Brown, T., Crook, D., Lamagni, T., Phin, N., Smith, E. G., Zambon, M., Serr, A., Götting, T., Ebner, W., Thürmer, A., Utpatel, C., Spröer, C., Bunk, B., Nübel, U., Bloemberg, G. V., Böttger, E. C., Niemann, S., Wagner, D., & Sax, H. (2017). Global outbreak of severe Mycobacterium chimaera disease after cardiac surgery: a molecular epidemiological study. The Lancet. Infectious diseases, 17(10), 1033-1041. https://doi.org/10.1016/S1473-3099(17)30324-9
van Ingen, Jakko, et al. "Global outbreak of severe Mycobacterium chimaera disease after cardiac surgery: a molecular epidemiological study." The Lancet. Infectious diseases vol. 17,10 (2017): 1033-1041. doi: https://doi.org/10.1016/S1473-3099(17)30324-9
van Ingen J, Kohl TA, Kranzer K, Hasse B, Keller PM, Katarzyna Szafrańska A, Hillemann D, Chand M, Schreiber PW, Sommerstein R, Berger C, Genoni M, Rüegg C, Troillet N, Widmer AF, Becker SL, Herrmann M, Eckmanns T, Haller S, Höller C, Debast SB, Wolfhagen MJ, Hopman J, Kluytmans J, Langelaar M, Notermans DW, Ten Oever J, van den Barselaar P, Vonk ABA, Vos MC, Ahmed N, Brown T, Crook D, Lamagni T, Phin N, Smith EG, Zambon M, Serr A, Götting T, Ebner W, Thürmer A, Utpatel C, Spröer C, Bunk B, Nübel U, Bloemberg GV, Böttger EC, Niemann S, Wagner D, Sax H. Global outbreak of severe Mycobacterium chimaera disease after cardiac surgery: a molecular epidemiological study. Lancet Infect Dis. 2017 Oct;17(10):1033-1041. doi: 10.1016/S1473-3099(17)30324-9. Epub 2017 Jul 12. PMID: 28711585.
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Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction.

Cardiovascular research

Xu JY, Xiong YY, Tang RJ, Jiang WY, Ning Y, Gong ZT, Huang PS, Chen GH, Xu J, Wu CX, Hu MJ, Xu J, Xu Y, Huang CR, Jin C, Lu XT, Qian HY, Li XD, Yang YJ.
PMID: 34259869
Cardiovasc Res. 2021 Jul 14; doi: 10.1093/cvr/cvab237. Epub 2021 Jul 14.

AIMS: Interleukin (IL)-5 mediates the development of eosinophils (EOS) that are essential for tissue post-injury repair. It remains unknown whether IL-5 plays a role in heart repair after myocardial infarction (MI). This study aims to test whether IL-5-induced EOS...

Cite
Xu JY, Xiong YY, Tang RJ, et al. Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovasc Res. 2021;doi: 10.1093/cvr/cvab237.
Xu, J. Y., Xiong, Y. Y., Tang, R. J., Jiang, W. Y., Ning, Y., Gong, Z. T., Huang, P. S., Chen, G. H., Xu, J., Wu, C. X., Hu, M. J., Xu, J., Xu, Y., Huang, C. R., Jin, C., Lu, X. T., Qian, H. Y., Li, X. D., & Yang, Y. J. (2021). Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovascular research, . https://doi.org/10.1093/cvr/cvab237
Xu, Jun-Yan, et al. "Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction." Cardiovascular research vol. (2021). doi: https://doi.org/10.1093/cvr/cvab237
Xu JY, Xiong YY, Tang RJ, Jiang WY, Ning Y, Gong ZT, Huang PS, Chen GH, Xu J, Wu CX, Hu MJ, Xu J, Xu Y, Huang CR, Jin C, Lu XT, Qian HY, Li XD, Yang YJ. Interleukin-5-induced eosinophil population improves cardiac function after myocardial infarction. Cardiovasc Res. 2021 Jul 14; doi: 10.1093/cvr/cvab237. Epub 2021 Jul 14. PMID: 34259869.
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Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074534
Ont Health Technol Assess Ser. 2009;9(4):1-51. Epub 2009 Jul 01.

UNLABELLED: Endovascular repair of abdominal aortic aneurysm (AAA) allows the exclusion of the dilated aneurismal segment of the aorta from the systematic circulation. The procedure requires, however, that the endograft extends to the healthy parts of the aorta above...

Cite
Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ont Health Technol Assess Ser. 2009;9(4):1-51
(2009). Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ontario health technology assessment series, 9(4), 1-51.
"Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis." Ontario health technology assessment series vol. 9,4 (2009): 1-51.
Fenestrated endovascular grafts for the repair of juxtarenal aortic aneurysms: an evidence-based analysis. Ont Health Technol Assess Ser. 2009;9(4):1-51. Epub 2009 Jul 01. PMID: 23074534; PMCID: PMC3377528.
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Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis.

Ontario health technology assessment series

[No authors listed]
PMID: 23074392
Ont Health Technol Assess Ser. 2010;10(15):1-45. Epub 2010 Jul 01.

UNLABELLED: In July 2009, the Medical Advisory Secretariat (MAS) began work on Non-Invasive Cardiac Imaging Technologies for the Assessment of Myocardial Viability, an evidence-based review of the literature surrounding different cardiac imaging modalities to ensure that appropriate technologies are...

Cite
Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(15):1-45
(2010). Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ontario health technology assessment series, 10(15), 1-45.
"Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis." Ontario health technology assessment series vol. 10,15 (2010): 1-45.
Magnetic resonance imaging (MRI) for the assessment of myocardial viability: an evidence-based analysis. Ont Health Technol Assess Ser. 2010;10(15):1-45. Epub 2010 Jul 01. PMID: 23074392; PMCID: PMC3426228.
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