Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 1334 entries
Sorted by: Best Match Show Resources per page
Degradation of organic dyes in the presence of activated titanium-based nanoparticles

Louis Stokes Alliances for Minority Participation AND Program Evaluation

Bailón-Ruiz SJ.
GSID: BFxIg8e-kG0J
AA Lugo-Ruiz, MJ Paz-Ruiz, SJ Bailón-Ruiz - MRS Advances, 2022 - Springer

There has been a high discharge of non-biodegradable dyes into natural streams and water bodies, thus, contaminating them. Many of these dyes are synthesized by carcinogens and can be water soluble, therefore, being more problematic to remove. One effective...

Cite
Lugo-Ruiz, A. A., Paz-Ruiz, M. J., & Bailón-Ruiz, S. J. (2022). Degradation of organic dyes in the presence of activated titanium-based nanoparticles. MRS Advances, 7(13), 289-294.
Lugo-Ruiz, Alondra A., María J. Paz-Ruiz, and Sonia J. Bailón-Ruiz. "Degradation of organic dyes in the presence of activated titanium-based nanoparticles." MRS Advances 7.13 (2022): 289-294.
Copy Download .nbib Format: MLA
Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line.

Toxicology reports

Simon BR, Wilson MJ, Blake DA, Yu H, Wickliffe JK.
PMID: 25170436
Toxicol Rep. 2014 Jul 14;1:391-400. doi: 10.1016/j.toxrep.2014.07.003.

Previously, we demonstrated the sensitivity of RPTEC/TERT1 cells, an immortalized human renal proximal tubule epithelial cell line, to two common environmental carcinogens, cadmium (Cd) and benzo[a]pyrene (B[a]P). Here, we measured BPDE-DNA adducts using a competitive ELISA method after cells...

Cite
Simon BR, Wilson MJ, Blake DA, et al. Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicol Rep. 2014;1:391-400doi: 10.1016/j.toxrep.2014.07.003.
Simon, B. R., Wilson, M. J., Blake, D. A., Yu, H., & Wickliffe, J. K. (2014). Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicology reports, 1391-400. https://doi.org/10.1016/j.toxrep.2014.07.003
Simon, Bridget R, et al. "Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line." Toxicology reports vol. 1 (2014): 391-400. doi: https://doi.org/10.1016/j.toxrep.2014.07.003
Simon BR, Wilson MJ, Blake DA, Yu H, Wickliffe JK. Cadmium alters the formation of benzo[a]pyrene DNA adducts in the RPTEC/TERT1 human renal proximal tubule epithelial cell line. Toxicol Rep. 2014 Jul 14;1:391-400. doi: 10.1016/j.toxrep.2014.07.003. PMID: 25170436; PMCID: PMC4142648.
Copy Download .nbib Format: NLM
Occupational risk of bladder cancer among Iranian male workers.

Urology annals

Aminian O, Saburi A, Mohseni H, Akbari H, Chavoshi F, Akbari H.
PMID: 24833825
Urol Ann. 2014 Apr;6(2):135-8. doi: 10.4103/0974-7796.130643.

BACKGROUND: Approximately 5-10% of human cancers are thought to be caused by occupational exposure to carcinogens. Compare to other cancers, bladder cancer is most strongly linked to occupational exposure to chemical toxins. This study has been performed to understand...

Cite
Aminian O, Saburi A, Mohseni H, et al. Occupational risk of bladder cancer among Iranian male workers. Urol Ann. 2014;6(2):135-8doi: 10.4103/0974-7796.130643.
Aminian, O., Saburi, A., Mohseni, H., Akbari, H., Chavoshi, F., & Akbari, H. (2014). Occupational risk of bladder cancer among Iranian male workers. Urology annals, 6(2), 135-8. https://doi.org/10.4103/0974-7796.130643
Aminian, Omid, et al. "Occupational risk of bladder cancer among Iranian male workers." Urology annals vol. 6,2 (2014): 135-8. doi: https://doi.org/10.4103/0974-7796.130643
Aminian O, Saburi A, Mohseni H, Akbari H, Chavoshi F, Akbari H. Occupational risk of bladder cancer among Iranian male workers. Urol Ann. 2014 Apr;6(2):135-8. doi: 10.4103/0974-7796.130643. PMID: 24833825; PMCID: PMC4021653.
Copy Download .nbib Format: NLM
Inhibition of Carcinogen-Activating Cytochrome P450 Enzymes by Xenobiotic Chemicals in Relation to Antimutagenicity and Anticarcinogenicity.

Toxicological research

Shimada T.
PMID: 28443179
Toxicol Res. 2017 Apr;33(2):79-96. doi: 10.5487/TR.2017.33.2.079. Epub 2017 Apr 15.

A variety of xenobiotic chemicals, such as polycyclic aromatic hydrocarbons (PAHs), aryl- and heterocyclic amines and tobacco related nitrosamines, are ubiquitous environmental carcinogens and are required to be activated to chemically reactive metabolites by xenobiotic-metabolizing enzymes, including cytochrome P450...

Cite
Shimada T. Inhibition of Carcinogen-Activating Cytochrome P450 Enzymes by Xenobiotic Chemicals in Relation to Antimutagenicity and Anticarcinogenicity. Toxicol Res. 2017;33(2):79-96doi: 10.5487/TR.2017.33.2.079.
Shimada, T. (2017). Inhibition of Carcinogen-Activating Cytochrome P450 Enzymes by Xenobiotic Chemicals in Relation to Antimutagenicity and Anticarcinogenicity. Toxicological research, 33(2), 79-96. https://doi.org/10.5487/TR.2017.33.2.079
Shimada, Tsutomu. "Inhibition of Carcinogen-Activating Cytochrome P450 Enzymes by Xenobiotic Chemicals in Relation to Antimutagenicity and Anticarcinogenicity." Toxicological research vol. 33,2 (2017): 79-96. doi: https://doi.org/10.5487/TR.2017.33.2.079
Shimada T. Inhibition of Carcinogen-Activating Cytochrome P450 Enzymes by Xenobiotic Chemicals in Relation to Antimutagenicity and Anticarcinogenicity. Toxicol Res. 2017 Apr;33(2):79-96. doi: 10.5487/TR.2017.33.2.079. Epub 2017 Apr 15. PMID: 28443179; PMCID: PMC5402866.
Copy Download .nbib Format: NLM
Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma.

Oncogenesis

Chauhan R, Churchill ND, Mulrooney-Cousins PM, Michalak TI.
PMID: 28414318
Oncogenesis. 2017 Apr 17;6(4):e317. doi: 10.1038/oncsis.2017.22.

Hepatitis B virus (HBV) and the closely related woodchuck hepatitis virus (WHV) are potent carcinogens that trigger development of primary hepatocellular carcinoma (HCC). The initial sites of hepadnavirus-host genome integration, their diversity and kinetics of formation can be central...

Cite
Chauhan R, Churchill ND, Mulrooney-Cousins PM, et al. Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma. Oncogenesis. 2017;6(4):e317doi: 10.1038/oncsis.2017.22.
Chauhan, R., Churchill, N. D., Mulrooney-Cousins, P. M., & Michalak, T. I. (2017). Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma. Oncogenesis, 6(4), e317. https://doi.org/10.1038/oncsis.2017.22
Chauhan, R, et al. "Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma." Oncogenesis vol. 6,4 (2017): e317. doi: https://doi.org/10.1038/oncsis.2017.22
Chauhan R, Churchill ND, Mulrooney-Cousins PM, Michalak TI. Initial sites of hepadnavirus integration into host genome in human hepatocytes and in the woodchuck model of hepatitis B-associated hepatocellular carcinoma. Oncogenesis. 2017 Apr 17;6(4):e317. doi: 10.1038/oncsis.2017.22. PMID: 28414318; PMCID: PMC5520499.
Copy Download .nbib Format: NLM
Cytochrome P450 2E1 RsaI/PstI polymorphism is associated with urologic cancer risk: evidence from a meta-analysis.

International journal of clinical and experimental medicine

Lin YC, Wu X, Zhou XQ, Ren R, Su ZX, Liu CX.
PMID: 26309545
Int J Clin Exp Med. 2015 Jun 15;8(6):8927-37. eCollection 2015.

Cytochrome P450 2E1 (CYP2E1) is involved in the metabolic activation of various carcinogens. CYP2E1 RsaI/PstI polymorphism has been identified in urologic cancer patients, while studies of the polymorphism have shown inconclusive trends in the risk of urologic cancers. Therefore,...

Cite
Lin YC, Wu X, Zhou XQ, et al. Cytochrome P450 2E1 RsaI/PstI polymorphism is associated with urologic cancer risk: evidence from a meta-analysis. Int J Clin Exp Med. 2015;8(6):8927-37
Lin, Y. C., Wu, X., Zhou, X. Q., Ren, R., Su, Z. X., & Liu, C. X. (2015). Cytochrome P450 2E1 RsaI/PstI polymorphism is associated with urologic cancer risk: evidence from a meta-analysis. International journal of clinical and experimental medicine, 8(6), 8927-37.
Lin, You-Cheng, et al. "Cytochrome P450 2E1 RsaI/PstI polymorphism is associated with urologic cancer risk: evidence from a meta-analysis." International journal of clinical and experimental medicine vol. 8,6 (2015): 8927-37.
Lin YC, Wu X, Zhou XQ, Ren R, Su ZX, Liu CX. Cytochrome P450 2E1 RsaI/PstI polymorphism is associated with urologic cancer risk: evidence from a meta-analysis. Int J Clin Exp Med. 2015 Jun 15;8(6):8927-37. eCollection 2015. PMID: 26309545; PMCID: PMC4538138.
Copy Download .nbib Format: NLM
Minor Salivary Gland Changes in Oral Epithelial Dysplasia and Oral Squamous Cell Carcinoma - A Histopathological Study.

Journal of clinical and diagnostic research : JCDR

Mohan SP, Chitturi RT, Ragunathan YT, Lakshmi SJ, Nallusamy J, Joseph I.
PMID: 27630945
J Clin Diagn Res. 2016 Jul;10(7):ZC12-5. doi: 10.7860/JCDR/2016/20218.8116. Epub 2016 Jul 01.

INTRODUCTION: The most common etiology for Oral Squamous Cell Carcinoma (OSCC) is tobacco and tobacco related products which cause nuclear damage to the keratinocytes. The chemical carcinogens not only affect the lining of oral epithelium but also affect the...

Cite
Mohan SP, Chitturi RT, Ragunathan YT, et al. Minor Salivary Gland Changes in Oral Epithelial Dysplasia and Oral Squamous Cell Carcinoma - A Histopathological Study. J Clin Diagn Res. 2016;10(7):ZC12-5doi: 10.7860/JCDR/2016/20218.8116.
Mohan, S. P., Chitturi, R. T., Ragunathan, Y. T., Lakshmi, S. J., Nallusamy, J., & Joseph, I. (2016). Minor Salivary Gland Changes in Oral Epithelial Dysplasia and Oral Squamous Cell Carcinoma - A Histopathological Study. Journal of clinical and diagnostic research : JCDR, 10(7), ZC12-5. https://doi.org/10.7860/JCDR/2016/20218.8116
Mohan, Sunil Paramel, et al. "Minor Salivary Gland Changes in Oral Epithelial Dysplasia and Oral Squamous Cell Carcinoma - A Histopathological Study." Journal of clinical and diagnostic research : JCDR vol. 10,7 (2016): ZC12-5. doi: https://doi.org/10.7860/JCDR/2016/20218.8116
Mohan SP, Chitturi RT, Ragunathan YT, Lakshmi SJ, Nallusamy J, Joseph I. Minor Salivary Gland Changes in Oral Epithelial Dysplasia and Oral Squamous Cell Carcinoma - A Histopathological Study. J Clin Diagn Res. 2016 Jul;10(7):ZC12-5. doi: 10.7860/JCDR/2016/20218.8116. Epub 2016 Jul 01. PMID: 27630945; PMCID: PMC5020236.
Copy Download .nbib Format: NLM
Cancer Risks among Welders and Occasional Welders in a National Population-Based Cohort Study: Canadian Census Health and Environmental Cohort.

Safety and health at work

MacLeod JS, Harris MA, Tjepkema M, Peters PA, Demers PA.
PMID: 28951802
Saf Health Work. 2017 Sep;8(3):258-266. doi: 10.1016/j.shaw.2016.12.001. Epub 2017 Jan 12.

BACKGROUND: Welders are exposed to many known and suspected carcinogens. An excess lung cancer risk among welders is well established, but whether this is attributable to welding fumes is unclear. Excess risks of other cancers have been suggested, but...

Cite
MacLeod JS, Harris MA, Tjepkema M, et al. Cancer Risks among Welders and Occasional Welders in a National Population-Based Cohort Study: Canadian Census Health and Environmental Cohort. Saf Health Work. 2017;8(3):258-266doi: 10.1016/j.shaw.2016.12.001.
MacLeod, J. S., Harris, M. A., Tjepkema, M., Peters, P. A., & Demers, P. A. (2017). Cancer Risks among Welders and Occasional Welders in a National Population-Based Cohort Study: Canadian Census Health and Environmental Cohort. Safety and health at work, 8(3), 258-266. https://doi.org/10.1016/j.shaw.2016.12.001
MacLeod, Jill S, et al. "Cancer Risks among Welders and Occasional Welders in a National Population-Based Cohort Study: Canadian Census Health and Environmental Cohort." Safety and health at work vol. 8,3 (2017): 258-266. doi: https://doi.org/10.1016/j.shaw.2016.12.001
MacLeod JS, Harris MA, Tjepkema M, Peters PA, Demers PA. Cancer Risks among Welders and Occasional Welders in a National Population-Based Cohort Study: Canadian Census Health and Environmental Cohort. Saf Health Work. 2017 Sep;8(3):258-266. doi: 10.1016/j.shaw.2016.12.001. Epub 2017 Jan 12. PMID: 28951802; PMCID: PMC5605892.
Copy Download .nbib Format: NLM
Association of mitochondrial deoxyribonucleic acid mutation with polymorphism in CYP2E1 gene in oral carcinogenesis.

Journal of oral biology and craniofacial research

Pandey R, Mehrotra D, Catapano C, Choubey V, Sarin R, Mahdi AA, Singh S.
PMID: 25756024
J Oral Biol Craniofac Res. 2012 Jan-Apr;2(1):4-9. doi: 10.1016/S2212-4268(12)60003-7.

BACKGROUND: Oral carcinogenesis is a complex process affected by genetic as well as environmental factors. CYP2E1 gene is involved in metabolism of number of compounds and carcinogens. Its normal functioning is required for homeostasis of free radical. Mitochondrial deoxyribonucleic...

Cite
Pandey R, Mehrotra D, Catapano C, et al. Association of mitochondrial deoxyribonucleic acid mutation with polymorphism in CYP2E1 gene in oral carcinogenesis. J Oral Biol Craniofac Res. 2012;2(1):4-9doi: 10.1016/S2212-4268(12)60003-7.
Pandey, R., Mehrotra, D., Catapano, C., Choubey, V., Sarin, R., Mahdi, A. A., & Singh, S. (2012). Association of mitochondrial deoxyribonucleic acid mutation with polymorphism in CYP2E1 gene in oral carcinogenesis. Journal of oral biology and craniofacial research, 2(1), 4-9. https://doi.org/10.1016/S2212-4268(12)60003-7
Pandey, Rahul, et al. "Association of mitochondrial deoxyribonucleic acid mutation with polymorphism in CYP2E1 gene in oral carcinogenesis." Journal of oral biology and craniofacial research vol. 2,1 (2012): 4-9. doi: https://doi.org/10.1016/S2212-4268(12)60003-7
Pandey R, Mehrotra D, Catapano C, Choubey V, Sarin R, Mahdi AA, Singh S. Association of mitochondrial deoxyribonucleic acid mutation with polymorphism in CYP2E1 gene in oral carcinogenesis. J Oral Biol Craniofac Res. 2012 Jan-Apr;2(1):4-9. doi: 10.1016/S2212-4268(12)60003-7. PMID: 25756024; PMCID: PMC3941659.
Copy Download .nbib Format: NLM
Assessing the Effect of Waterpipe Smoking on Cancer Outcome - a Systematic Review of Current Evidence.

Asian Pacific journal of cancer prevention : APJCP

Awan KH, Siddiqi K, Patil Sh, Hussain QA.
PMID: 28345836
Asian Pac J Cancer Prev. 2017 Feb 01;18(2):495-502. doi: 10.22034/APJCP.2017.18.2.495.

Background: Waterpipe smoking (WPS) is widely believed to be a safe and hazard-free tobacco habit. However, a number of studies have indicated that exposure to several toxicants and carcinogens through WPS is strongly related to serious health hazards. The...

Cite
Awan KH, Siddiqi K, Patil Sh, et al. Assessing the Effect of Waterpipe Smoking on Cancer Outcome - a Systematic Review of Current Evidence. Asian Pac J Cancer Prev. 2017;18(2):495-502doi: 10.22034/APJCP.2017.18.2.495.
Awan, K. H., Siddiqi, K., Patil, S. h., & Hussain, Q. A. (2017). Assessing the Effect of Waterpipe Smoking on Cancer Outcome - a Systematic Review of Current Evidence. Asian Pacific journal of cancer prevention : APJCP, 18(2), 495-502. https://doi.org/10.22034/APJCP.2017.18.2.495
Awan, Kamran Habib, et al. "Assessing the Effect of Waterpipe Smoking on Cancer Outcome - a Systematic Review of Current Evidence." Asian Pacific journal of cancer prevention : APJCP vol. 18,2 (2017): 495-502. doi: https://doi.org/10.22034/APJCP.2017.18.2.495
Awan KH, Siddiqi K, Patil Sh, Hussain QA. Assessing the Effect of Waterpipe Smoking on Cancer Outcome - a Systematic Review of Current Evidence. Asian Pac J Cancer Prev. 2017 Feb 01;18(2):495-502. doi: 10.22034/APJCP.2017.18.2.495. PMID: 28345836; PMCID: PMC5454749.
Copy Download .nbib Format: NLM
Mesenchymal stem cells inhibited development of lung cancer induced by chemical carcinogens in a rat model.

American journal of translational research

Liu T, Zhu K, Ke C, Yang S, Yang F, Li Z, Zhang Z.
PMID: 28670377
Am J Transl Res. 2017 Jun 15;9(6):2891-2900. eCollection 2017.

Mesenchymal stem cells (MSCs) may play a significant role in carcinogenesis; however, data have shown that MSCs can both promote and inhibit tumor growth. We investigated the effect of MSCs on the development of lung cancer in a rat...

Cite
Liu T, Zhu K, Ke C, et al. Mesenchymal stem cells inhibited development of lung cancer induced by chemical carcinogens in a rat model. Am J Transl Res. 2017;9(6):2891-2900
Liu, T., Zhu, K., Ke, C., Yang, S., Yang, F., Li, Z., & Zhang, Z. (2017). Mesenchymal stem cells inhibited development of lung cancer induced by chemical carcinogens in a rat model. American journal of translational research, 9(6), 2891-2900.
Liu, Tonggang, et al. "Mesenchymal stem cells inhibited development of lung cancer induced by chemical carcinogens in a rat model." American journal of translational research vol. 9,6 (2017): 2891-2900.
Liu T, Zhu K, Ke C, Yang S, Yang F, Li Z, Zhang Z. Mesenchymal stem cells inhibited development of lung cancer induced by chemical carcinogens in a rat model. Am J Transl Res. 2017 Jun 15;9(6):2891-2900. eCollection 2017. PMID: 28670377; PMCID: PMC5489889.
Copy Download .nbib Format: NLM
Theoretical and Experimental Study of Light-assisted Polymerization by Multimechanism Action.

Scientific reports

Zhou H, Song D, Zhong C, Ye G.
PMID: 27958300
Sci Rep. 2016 Dec 13;6:38473. doi: 10.1038/srep38473.

A novel bicomponent alkyd system was designed to decrease the usage of Cobalt-based drier due to its latent carcinogensis. The system was polymerized using a Cobalt-salt complex as an air-sensitive drier and an isopropylthioxanthone photoinitiator as a light-sensitive accelerator,...

Cite
Zhou H, Song D, Zhong C, et al. Theoretical and Experimental Study of Light-assisted Polymerization by Multimechanism Action. Sci Rep. 2016;6:38473doi: 10.1038/srep38473.
Zhou, H., Song, D., Zhong, C., & Ye, G. (2016). Theoretical and Experimental Study of Light-assisted Polymerization by Multimechanism Action. Scientific reports, 638473. https://doi.org/10.1038/srep38473
Zhou, Hua, et al. "Theoretical and Experimental Study of Light-assisted Polymerization by Multimechanism Action." Scientific reports vol. 6 (2016): 38473. doi: https://doi.org/10.1038/srep38473
Zhou H, Song D, Zhong C, Ye G. Theoretical and Experimental Study of Light-assisted Polymerization by Multimechanism Action. Sci Rep. 2016 Dec 13;6:38473. doi: 10.1038/srep38473. PMID: 27958300; PMCID: PMC5153847.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 1334 entries