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Showing 1 to 12 of 96 entries
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Evidence against the adenosine-catecholamine antagonism in the canine heart in situ.

Arzneimittel-Forschung

Schipke J, Heusch G, Thämer V.
PMID: 3449061
Arzneimittelforschung. 1987 Dec;37(12):1345-7.

Adenosine has been reported to attenuate the positive inotropic effects of catecholamines in isolated heart preparations of rodents. These results were not confirmed in anesthetized dogs during intracoronary infusion of isoprenaline. However, these experiments did not establish the effects...

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Schipke J, Heusch G, Thämer V. Evidence against the adenosine-catecholamine antagonism in the canine heart in situ. Arzneimittelforschung. 1987;37(12):1345-7
Schipke, J., Heusch, G., & Thämer, V. (1987). Evidence against the adenosine-catecholamine antagonism in the canine heart in situ. Arzneimittel-Forschung, 37(12), 1345-7.
Schipke, J, et al. "Evidence against the adenosine-catecholamine antagonism in the canine heart in situ." Arzneimittel-Forschung vol. 37,12 (1987): 1345-7.
Schipke J, Heusch G, Thämer V. Evidence against the adenosine-catecholamine antagonism in the canine heart in situ. Arzneimittelforschung. 1987 Dec;37(12):1345-7. PMID: 3449061.
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Decreased flow reserve in "stunned" myocardium after a 10-min coronary occlusion.

The American journal of physiology

Triana JF, Bolli R.
PMID: 1887925
Am J Physiol. 1991 Sep;261(3):H793-804. doi: 10.1152/ajpheart.1991.261.3.H793.

The effect of brief reversible ischemia on coronary vascular function remains controversial. Studies using a 15-min coronary occlusion have reported an impairment of vasodilator reserve, whereas studies using shorter occlusions have not. To elucidate the relation between postischemic vascular...

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Triana JF, Bolli R. Decreased flow reserve in "stunned" myocardium after a 10-min coronary occlusion. Am J Physiol. 1991;261(3):H793-804doi: 10.1152/ajpheart.1991.261.3.H793.
Triana, J. F., & Bolli, R. (1991). Decreased flow reserve in "stunned" myocardium after a 10-min coronary occlusion. The American journal of physiology, 261(3), H793-804. https://doi.org/10.1152/ajpheart.1991.261.3.H793
Triana, J F, and Bolli, R. "Decreased flow reserve in "stunned" myocardium after a 10-min coronary occlusion." The American journal of physiology vol. 261,3 (1991): H793-804. doi: https://doi.org/10.1152/ajpheart.1991.261.3.H793
Triana JF, Bolli R. Decreased flow reserve in "stunned" myocardium after a 10-min coronary occlusion. Am J Physiol. 1991 Sep;261(3):H793-804. doi: 10.1152/ajpheart.1991.261.3.H793. PMID: 1887925.
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Coronary pressure-flow, pressure-function, and function-myocardial oxygen consumption relations in postischaemic myocardium.

Cardiovascular research

Kawashima S, Satani A, Tsumoto S, Kondo T, Ikeoka K, Morita M, Iwasaki T.
PMID: 1747877
Cardiovasc Res. 1991 Oct;25(10):837-43. doi: 10.1093/cvr/25.10.837.

STUDY OBJECTIVE: The aim was to study coronary pressure-flow, pressure-myocardial wall motion and myocardial wall motion-myocardial oxygen consumption relations in postischaemic myocardium with prolonged myocardial dysfunction (stunned myocardium) in comparison with normal myocardium.DESIGN: Regional myocardial wall thickening was measured...

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Kawashima S, Satani A, Tsumoto S, et al. Coronary pressure-flow, pressure-function, and function-myocardial oxygen consumption relations in postischaemic myocardium. Cardiovasc Res. 1991;25(10):837-43doi: 10.1093/cvr/25.10.837.
Kawashima, S., Satani, A., Tsumoto, S., Kondo, T., Ikeoka, K., Morita, M., & Iwasaki, T. (1991). Coronary pressure-flow, pressure-function, and function-myocardial oxygen consumption relations in postischaemic myocardium. Cardiovascular research, 25(10), 837-43. https://doi.org/10.1093/cvr/25.10.837
Kawashima, S, et al. "Coronary pressure-flow, pressure-function, and function-myocardial oxygen consumption relations in postischaemic myocardium." Cardiovascular research vol. 25,10 (1991): 837-43. doi: https://doi.org/10.1093/cvr/25.10.837
Kawashima S, Satani A, Tsumoto S, Kondo T, Ikeoka K, Morita M, Iwasaki T. Coronary pressure-flow, pressure-function, and function-myocardial oxygen consumption relations in postischaemic myocardium. Cardiovasc Res. 1991 Oct;25(10):837-43. doi: 10.1093/cvr/25.10.837. PMID: 1747877.
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Sympathetic stimulation and norepinephrine infusion modulate extracellular potassium concentration during acute myocardial ischemia.

Circulation research

Warner MR, Kroeker TS, Zipes DP.
PMID: 1394871
Circ Res. 1992 Nov;71(5):1078-87. doi: 10.1161/01.res.71.5.1078.

The purpose of this study was to investigate whether sympathetic stimulation modulated the rise in extracellular K+ concentration ([K+]o) evoked by acute myocardial ischemia. In 35 alpha-chloralose-anesthetized dogs, we measured changes in [K+]o during acute myocardial ischemia in the...

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Warner MR, Kroeker TS, Zipes DP. Sympathetic stimulation and norepinephrine infusion modulate extracellular potassium concentration during acute myocardial ischemia. Circ Res. 1992;71(5):1078-87doi: 10.1161/01.res.71.5.1078.
Warner, M. R., Kroeker, T. S., & Zipes, D. P. (1992). Sympathetic stimulation and norepinephrine infusion modulate extracellular potassium concentration during acute myocardial ischemia. Circulation research, 71(5), 1078-87. https://doi.org/10.1161/01.res.71.5.1078
Warner, M R, et al. "Sympathetic stimulation and norepinephrine infusion modulate extracellular potassium concentration during acute myocardial ischemia." Circulation research vol. 71,5 (1992): 1078-87. doi: https://doi.org/10.1161/01.res.71.5.1078
Warner MR, Kroeker TS, Zipes DP. Sympathetic stimulation and norepinephrine infusion modulate extracellular potassium concentration during acute myocardial ischemia. Circ Res. 1992 Nov;71(5):1078-87. doi: 10.1161/01.res.71.5.1078. PMID: 1394871.
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Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content.

Cardiovascular research

Dae MW, O'Connell JW, Botvinick EH, Chin MC.
PMID: 7585815
Cardiovasc Res. 1995 Aug;30(2):270-80.

OBJECTIVES: The sympathetic nervous system has profound influences on myocardial function, particularly during ischemia. There is controversy, however, as to whether myocardial ischemia results in damage to myocardial sympathetic nerves coursing through the ischemic territory. To further evaluate these...

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Dae MW, O'Connell JW, Botvinick EH, et al. Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovasc Res. 1995;30(2):270-80
Dae, M. W., O'Connell, J. W., Botvinick, E. H., & Chin, M. C. (1995). Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovascular research, 30(2), 270-80.
Dae, M W, et al. "Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content." Cardiovascular research vol. 30,2 (1995): 270-80.
Dae MW, O'Connell JW, Botvinick EH, Chin MC. Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovasc Res. 1995 Aug;30(2):270-80. PMID: 7585815.
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Parenteral magnesium sulphate restores regional contractile function in the post-ischaemic canine myocardium.

Magnesium research

Mass H, Santoni F, Feliciano L, Santiago O, Albino D, De Leon N, Sifonte M.
PMID: 7786688
Magnes Res. 1994 Dec;7(3):255-66.

The effects of parenteral magnesium sulphate (MS) on the regional contractile response of stunned myocardium was examined in 45 pentobarbital anaesthetized dogs. The hearts were instrumented to measure left ventricular pressure (LVP), coronary flow velocity (CFV), mean arterial blood...

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Mass H, Santoni F, Feliciano L, et al. Parenteral magnesium sulphate restores regional contractile function in the post-ischaemic canine myocardium. Magnes Res. 1994;7(3):255-66
Mass, H., Santoni, F., Feliciano, L., Santiago, O., Albino, D., De Leon, N., & Sifonte, M. (1994). Parenteral magnesium sulphate restores regional contractile function in the post-ischaemic canine myocardium. Magnesium research, 7(3), 255-66.
Mass, H, et al. "Parenteral magnesium sulphate restores regional contractile function in the post-ischaemic canine myocardium." Magnesium research vol. 7,3 (1994): 255-66.
Mass H, Santoni F, Feliciano L, Santiago O, Albino D, De Leon N, Sifonte M. Parenteral magnesium sulphate restores regional contractile function in the post-ischaemic canine myocardium. Magnes Res. 1994 Dec;7(3):255-66. PMID: 7786688.
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Controlled intermittent asystole cardiac therapy induced by pharmacologically potentiated vagus nerve stimulation in normal and hibernating myocardium.

The Annals of thoracic surgery

Ronson RS, Puskas JD, Thourani VH, Velez DA, Bufkin BL, Glass J, Guyton RA, Vinten-Johansen J.
PMID: 12822638
Ann Thorac Surg. 2003 Jun;75(6):1929-36. doi: 10.1016/s0003-4975(03)00115-2.

BACKGROUND: Pharmacologically potentiated electrical stimulation of the right vagus nerve achieves controlled intermittent asystole cardiac therapy. The present study examined pathophysiologic consequences of repetitive intermittent asystoles on contractile function, myocardial blood flow, and vagus nerve function and morphology.METHODS: Open-chest...

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Ronson RS, Puskas JD, Thourani VH, et al. Controlled intermittent asystole cardiac therapy induced by pharmacologically potentiated vagus nerve stimulation in normal and hibernating myocardium. Ann Thorac Surg. 2003;75(6):1929-36doi: 10.1016/s0003-4975(03)00115-2.
Ronson, R. S., Puskas, J. D., Thourani, V. H., Velez, D. A., Bufkin, B. L., Glass, J., Guyton, R. A., & Vinten-Johansen, J. (2003). Controlled intermittent asystole cardiac therapy induced by pharmacologically potentiated vagus nerve stimulation in normal and hibernating myocardium. The Annals of thoracic surgery, 75(6), 1929-36. https://doi.org/10.1016/s0003-4975(03)00115-2
Ronson, Russell S, et al. "Controlled intermittent asystole cardiac therapy induced by pharmacologically potentiated vagus nerve stimulation in normal and hibernating myocardium." The Annals of thoracic surgery vol. 75,6 (2003): 1929-36. doi: https://doi.org/10.1016/s0003-4975(03)00115-2
Ronson RS, Puskas JD, Thourani VH, Velez DA, Bufkin BL, Glass J, Guyton RA, Vinten-Johansen J. Controlled intermittent asystole cardiac therapy induced by pharmacologically potentiated vagus nerve stimulation in normal and hibernating myocardium. Ann Thorac Surg. 2003 Jun;75(6):1929-36. doi: 10.1016/s0003-4975(03)00115-2. PMID: 12822638.
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Blunted functional responses to pre- and postjunctional sympathetic stimulation in hibernating myocardium.

American journal of physiology. Heart and circulatory physiology

Ovchinnikov V, Suzuki G, Canty JM, Fallavollita JA.
PMID: 15923318
Am J Physiol Heart Circ Physiol. 2005 Oct;289(4):H1719-28. doi: 10.1152/ajpheart.00273.2005. Epub 2005 May 27.

Regional reductions in norepinephrine-tracer uptake are found in pigs with hibernating myocardium. Clinical studies would suggest that this is evidence for denervation; however, the functional responses to sympathetic stimulation have not been evaluated, and our previous studies with beta-adrenergic...

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Ovchinnikov V, Suzuki G, Canty JM, et al. Blunted functional responses to pre- and postjunctional sympathetic stimulation in hibernating myocardium. Am J Physiol Heart Circ Physiol. 2005;289(4):H1719-28doi: 10.1152/ajpheart.00273.2005.
Ovchinnikov, V., Suzuki, G., Canty, J. M., & Fallavollita, J. A. (2005). Blunted functional responses to pre- and postjunctional sympathetic stimulation in hibernating myocardium. American journal of physiology. Heart and circulatory physiology, 289(4), H1719-28. https://doi.org/10.1152/ajpheart.00273.2005
Ovchinnikov, Vladislav, et al. "Blunted functional responses to pre- and postjunctional sympathetic stimulation in hibernating myocardium." American journal of physiology. Heart and circulatory physiology vol. 289,4 (2005): H1719-28. doi: https://doi.org/10.1152/ajpheart.00273.2005
Ovchinnikov V, Suzuki G, Canty JM, Fallavollita JA. Blunted functional responses to pre- and postjunctional sympathetic stimulation in hibernating myocardium. Am J Physiol Heart Circ Physiol. 2005 Oct;289(4):H1719-28. doi: 10.1152/ajpheart.00273.2005. Epub 2005 May 27. PMID: 15923318.
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Mechanism of myocardial protection by isoflurane. Role of adenosine triphosphate-regulated potassium (KATP) channels.

Anesthesiology

Kersten JR, Schmeling TJ, Hettrick DA, Pagel PS, Gross GJ, Warltier DC.
PMID: 8873550
Anesthesiology. 1996 Oct;85(4):794-807; discussion 27A. doi: 10.1097/00000542-199610000-00015.
Free Article

BACKGROUND: The mechanism of the protective actions of volatile anesthetics in ischemic myocardium has not been clearly elucidated. The role of myocardial adenosine triphosphate-regulated potassium (KATP) channels in isoflurane-induced enhancement of recovery of regional contractile function after multiple brief...

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Kersten JR, Schmeling TJ, Hettrick DA, et al. Mechanism of myocardial protection by isoflurane. Role of adenosine triphosphate-regulated potassium (KATP) channels. Anesthesiology. 1996;85(4):794-807; discussion 27Adoi: 10.1097/00000542-199610000-00015.
Kersten, J. R., Schmeling, T. J., Hettrick, D. A., Pagel, P. S., Gross, G. J., & Warltier, D. C. (1996). Mechanism of myocardial protection by isoflurane. Role of adenosine triphosphate-regulated potassium (KATP) channels. Anesthesiology, 85(4), 794-807; discussion 27A. https://doi.org/10.1097/00000542-199610000-00015
Kersten, J R, et al. "Mechanism of myocardial protection by isoflurane. Role of adenosine triphosphate-regulated potassium (KATP) channels." Anesthesiology vol. 85,4 (1996): 794-807; discussion 27A. doi: https://doi.org/10.1097/00000542-199610000-00015
Kersten JR, Schmeling TJ, Hettrick DA, Pagel PS, Gross GJ, Warltier DC. Mechanism of myocardial protection by isoflurane. Role of adenosine triphosphate-regulated potassium (KATP) channels. Anesthesiology. 1996 Oct;85(4):794-807; discussion 27A. doi: 10.1097/00000542-199610000-00015. PMID: 8873550.
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Intracoronary amiloride prevents contractile dysfunction of postischemic "stunned" myocardium: role of hemodynamic alterations and inhibition of Na+/H+ exchange and L-type Ca2+ channels.

Journal of the American College of Cardiology

Smart SC, LoCurto A, el Schultz J, Sagar KB, Warltier DC.
PMID: 7594055
J Am Coll Cardiol. 1995 Nov 01;26(5):1365-73. doi: 10.1016/0735-1097(95)00326-6.
Free Article

OBJECTIVES: This study sought to establish the effect of amiloride on stunned myocardium and to determine the role of hemodynamic alterations and inhibition of sodium/proton (Na+/H+) exchange and L-type cytosolic calcium (Ca2+) channels.BACKGROUND: Amiloride is a nonspecific agent that...

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Smart SC, LoCurto A, el Schultz J, et al. Intracoronary amiloride prevents contractile dysfunction of postischemic "stunned" myocardium: role of hemodynamic alterations and inhibition of Na+/H+ exchange and L-type Ca2+ channels. J Am Coll Cardiol. 1995;26(5):1365-73doi: 10.1016/0735-1097(95)00326-6.
Smart, S. C., LoCurto, A., el Schultz, J., Sagar, K. B., & Warltier, D. C. (1995). Intracoronary amiloride prevents contractile dysfunction of postischemic "stunned" myocardium: role of hemodynamic alterations and inhibition of Na+/H+ exchange and L-type Ca2+ channels. Journal of the American College of Cardiology, 26(5), 1365-73. https://doi.org/10.1016/0735-1097(95)00326-6
Smart, S C, et al. "Intracoronary amiloride prevents contractile dysfunction of postischemic "stunned" myocardium: role of hemodynamic alterations and inhibition of Na+/H+ exchange and L-type Ca2+ channels." Journal of the American College of Cardiology vol. 26,5 (1995): 1365-73. doi: https://doi.org/10.1016/0735-1097(95)00326-6
Smart SC, LoCurto A, el Schultz J, Sagar KB, Warltier DC. Intracoronary amiloride prevents contractile dysfunction of postischemic "stunned" myocardium: role of hemodynamic alterations and inhibition of Na+/H+ exchange and L-type Ca2+ channels. J Am Coll Cardiol. 1995 Nov 01;26(5):1365-73. doi: 10.1016/0735-1097(95)00326-6. PMID: 7594055.
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Lack of modulation by dietary unsaturated fats on sympathetic neurotransmission in rat hearts.

The American journal of physiology

Du XJ, Dart AM, Riemersma RA.
PMID: 8214123
Am J Physiol. 1993 Sep;265(3):H886-92. doi: 10.1152/ajpheart.1993.265.3.H886.

We examined the effect of dietary polyunsaturated fatty acids (PUFA) on cardiac sympathetic neurotransmission. Rats were fed semisynthetic diets (18.5% fat wt/wt) high in saturated fatty acids (control diet), high in n-6 PUFA (corn oil), or enriched with n-3...

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Du XJ, Dart AM, Riemersma RA. Lack of modulation by dietary unsaturated fats on sympathetic neurotransmission in rat hearts. Am J Physiol. 1993;265(3):H886-92doi: 10.1152/ajpheart.1993.265.3.H886.
Du, X. J., Dart, A. M., & Riemersma, R. A. (1993). Lack of modulation by dietary unsaturated fats on sympathetic neurotransmission in rat hearts. The American journal of physiology, 265(3), H886-92. https://doi.org/10.1152/ajpheart.1993.265.3.H886
Du, X J, et al. "Lack of modulation by dietary unsaturated fats on sympathetic neurotransmission in rat hearts." The American journal of physiology vol. 265,3 (1993): H886-92. doi: https://doi.org/10.1152/ajpheart.1993.265.3.H886
Du XJ, Dart AM, Riemersma RA. Lack of modulation by dietary unsaturated fats on sympathetic neurotransmission in rat hearts. Am J Physiol. 1993 Sep;265(3):H886-92. doi: 10.1152/ajpheart.1993.265.3.H886. PMID: 8214123.
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The effects of neuronal uptake blockade on the cardiac responses to sympathetic nerve stimulation and norepinephrine infusion in anesthetized dogs.

Journal of the autonomic nervous system

Masuda Y, Levy MN.
PMID: 6327795
J Auton Nerv Syst. 1984 Mar;10(1):1-17. doi: 10.1016/0165-1838(84)90063-8.

In anesthetized dogs, cocaine ( COC ) was administered intravenously in order to block the neuronal uptake of norepinephrine (NE) by the heart. COC had relatively little effect on the magnitudes of the inotropic and chronotropic responses to sympathetic...

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Masuda Y, Levy MN. The effects of neuronal uptake blockade on the cardiac responses to sympathetic nerve stimulation and norepinephrine infusion in anesthetized dogs. J Auton Nerv Syst. 1984;10(1):1-17doi: 10.1016/0165-1838(84)90063-8.
Masuda, Y., & Levy, M. N. (1984). The effects of neuronal uptake blockade on the cardiac responses to sympathetic nerve stimulation and norepinephrine infusion in anesthetized dogs. Journal of the autonomic nervous system, 10(1), 1-17. https://doi.org/10.1016/0165-1838(84)90063-8
Masuda, Y, and Levy, M N. "The effects of neuronal uptake blockade on the cardiac responses to sympathetic nerve stimulation and norepinephrine infusion in anesthetized dogs." Journal of the autonomic nervous system vol. 10,1 (1984): 1-17. doi: https://doi.org/10.1016/0165-1838(84)90063-8
Masuda Y, Levy MN. The effects of neuronal uptake blockade on the cardiac responses to sympathetic nerve stimulation and norepinephrine infusion in anesthetized dogs. J Auton Nerv Syst. 1984 Mar;10(1):1-17. doi: 10.1016/0165-1838(84)90063-8. PMID: 6327795.
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Showing 1 to 12 of 96 entries