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Showing 1 to 12 of 110 entries
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Acetylstrophanthidin-induced reflex inhibition of canine renal sympathetic nerve activity mediated by cardiac receptors with vagal afferents.

Circulation research

Thames MD.
PMID: 758235
Circ Res. 1979 Jan;44(1):8-15. doi: 10.1161/01.res.44.1.8.

The present experiments were performed to determine whether digitalis-induced augmentation of cardiac receptor discharge could induce reflex reductions in renal sympathetic nerve activity. Intracoronary injection or epicardial application of acetylstrophanthidin (AS) in chloraloseanesthetized dogs caused large decreases in renal...

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Thames MD. Acetylstrophanthidin-induced reflex inhibition of canine renal sympathetic nerve activity mediated by cardiac receptors with vagal afferents. Circ Res. 1979;44(1):8-15doi: 10.1161/01.res.44.1.8.
Thames, M. D. (1979). Acetylstrophanthidin-induced reflex inhibition of canine renal sympathetic nerve activity mediated by cardiac receptors with vagal afferents. Circulation research, 44(1), 8-15. https://doi.org/10.1161/01.res.44.1.8
Thames, M D. "Acetylstrophanthidin-induced reflex inhibition of canine renal sympathetic nerve activity mediated by cardiac receptors with vagal afferents." Circulation research vol. 44,1 (1979): 8-15. doi: https://doi.org/10.1161/01.res.44.1.8
Thames MD. Acetylstrophanthidin-induced reflex inhibition of canine renal sympathetic nerve activity mediated by cardiac receptors with vagal afferents. Circ Res. 1979 Jan;44(1):8-15. doi: 10.1161/01.res.44.1.8. PMID: 758235.
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The circulatory influences of vagal afferents at rest and during coronary occlusion in conscious dogs.

Circulation research

Bishop VS, Peterson DF.
PMID: 709745
Circ Res. 1978 Dec;43(6):840-7. doi: 10.1161/01.res.43.6.840.

We studied the role of cardiopulmonary vagal afferents in the cardiovascular responses to coronary artery occlusion in conscious dogs with intact carotid sinuses and following functional denervation of the arterial baroreceptors. The contributions of vagal afferents were determined by...

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Bishop VS, Peterson DF. The circulatory influences of vagal afferents at rest and during coronary occlusion in conscious dogs. Circ Res. 1978;43(6):840-7doi: 10.1161/01.res.43.6.840.
Bishop, V. S., & Peterson, D. F. (1978). The circulatory influences of vagal afferents at rest and during coronary occlusion in conscious dogs. Circulation research, 43(6), 840-7. https://doi.org/10.1161/01.res.43.6.840
Bishop, V S, and Peterson, D F. "The circulatory influences of vagal afferents at rest and during coronary occlusion in conscious dogs." Circulation research vol. 43,6 (1978): 840-7. doi: https://doi.org/10.1161/01.res.43.6.840
Bishop VS, Peterson DF. The circulatory influences of vagal afferents at rest and during coronary occlusion in conscious dogs. Circ Res. 1978 Dec;43(6):840-7. doi: 10.1161/01.res.43.6.840. PMID: 709745.
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6-Keto-prostaglandin E1 is a potent coronary vasodilator and stimulates a vagal reflex in dogs.

The Journal of pharmacology and experimental therapeutics

Panzenbeck MJ, Hintze TH, Kaley G.
PMID: 3075239
J Pharmacol Exp Ther. 1988 Mar;244(3):814-9.

These studies were performed to examine the effects of 6-keto-prostaglandin E1 (6-keto-PGE1) on coronary artery blood flow and reflex cardiovascular regulation. In seven chloralose-anesthetized dogs 6-keto-PGE1 and prostacyclin (PGI2) were injected as a bolus into the left circumflex coronary...

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Panzenbeck MJ, Hintze TH, Kaley G. 6-Keto-prostaglandin E1 is a potent coronary vasodilator and stimulates a vagal reflex in dogs. J Pharmacol Exp Ther. 1988;244(3):814-9
Panzenbeck, M. J., Hintze, T. H., & Kaley, G. (1988). 6-Keto-prostaglandin E1 is a potent coronary vasodilator and stimulates a vagal reflex in dogs. The Journal of pharmacology and experimental therapeutics, 244(3), 814-9.
Panzenbeck, M J, et al. "6-Keto-prostaglandin E1 is a potent coronary vasodilator and stimulates a vagal reflex in dogs." The Journal of pharmacology and experimental therapeutics vol. 244,3 (1988): 814-9.
Panzenbeck MJ, Hintze TH, Kaley G. 6-Keto-prostaglandin E1 is a potent coronary vasodilator and stimulates a vagal reflex in dogs. J Pharmacol Exp Ther. 1988 Mar;244(3):814-9. PMID: 3075239.
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Analysis of the action of angiotensin II on the baroreflex control of heart rate in conscious rabbits.

Endocrinology

Reid IA, Chou L.
PMID: 2328705
Endocrinology. 1990 May;126(5):2749-56. doi: 10.1210/endo-126-5-2749.

There is considerable evidence that angiotensin II (Ang II) attenuates the baroreflex control of heart rate (HR), but the mechanism and site of this action have not been precisely defined. In the present study the effects of systemically and...

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Reid IA, Chou L. Analysis of the action of angiotensin II on the baroreflex control of heart rate in conscious rabbits. Endocrinology. 1990;126(5):2749-56doi: 10.1210/endo-126-5-2749.
Reid, I. A., & Chou, L. (1990). Analysis of the action of angiotensin II on the baroreflex control of heart rate in conscious rabbits. Endocrinology, 126(5), 2749-56. https://doi.org/10.1210/endo-126-5-2749
Reid, I A, and Chou, L. "Analysis of the action of angiotensin II on the baroreflex control of heart rate in conscious rabbits." Endocrinology vol. 126,5 (1990): 2749-56. doi: https://doi.org/10.1210/endo-126-5-2749
Reid IA, Chou L. Analysis of the action of angiotensin II on the baroreflex control of heart rate in conscious rabbits. Endocrinology. 1990 May;126(5):2749-56. doi: 10.1210/endo-126-5-2749. PMID: 2328705.
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Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs.

Archives internationales de pharmacodynamie et de therapie

Abiko Y, Gotoh H, Yokoyama T, Abiko T, Hashizume H, Akiyama K.
PMID: 7944827
Arch Int Pharmacodyn Ther. 1994 Jan-Feb;327(1):40-55.

The effects of bopindolol (a new, nonselective beta-adrenoceptor antagonist) and its active metabolite, 18-502, on the regional myocardial pH during coronary occlusion were studied. Mongrel dogs were anesthetized with pentobarbital and the thorax was opened to expose the heart....

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Abiko Y, Gotoh H, Yokoyama T, et al. Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Arch Int Pharmacodyn Ther. 1994;327(1):40-55
Abiko, Y., Gotoh, H., Yokoyama, T., Abiko, T., Hashizume, H., & Akiyama, K. (1994). Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Archives internationales de pharmacodynamie et de therapie, 327(1), 40-55.
Abiko, Y, et al. "Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs." Archives internationales de pharmacodynamie et de therapie vol. 327,1 (1994): 40-55.
Abiko Y, Gotoh H, Yokoyama T, Abiko T, Hashizume H, Akiyama K. Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Arch Int Pharmacodyn Ther. 1994 Jan-Feb;327(1):40-55. PMID: 7944827.
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Characterization of the baroreceptor heart rate reflex during development in spontaneously hypertensive rats.

Clinical and experimental pharmacology & physiology

Head GA, Adams MA.
PMID: 1526065
Clin Exp Pharmacol Physiol. 1992 Aug;19(8):587-97. doi: 10.1111/j.1440-1681.1992.tb00509.x.

1. We have examined the baroreceptor-heart rate (HR) reflex in weight-matched conscious spontaneously hypertensive rats (SHR) and normotensive Wistar-Kyoto (WKY) rats during development. 2. Graded steady-state changes in mean arterial pressure (MAP) and the corresponding HR responses before and...

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Head GA, Adams MA. Characterization of the baroreceptor heart rate reflex during development in spontaneously hypertensive rats. Clin Exp Pharmacol Physiol. 1992;19(8):587-97doi: 10.1111/j.1440-1681.1992.tb00509.x.
Head, G. A., & Adams, M. A. (1992). Characterization of the baroreceptor heart rate reflex during development in spontaneously hypertensive rats. Clinical and experimental pharmacology & physiology, 19(8), 587-97. https://doi.org/10.1111/j.1440-1681.1992.tb00509.x
Head, G A, and Adams, M A. "Characterization of the baroreceptor heart rate reflex during development in spontaneously hypertensive rats." Clinical and experimental pharmacology & physiology vol. 19,8 (1992): 587-97. doi: https://doi.org/10.1111/j.1440-1681.1992.tb00509.x
Head GA, Adams MA. Characterization of the baroreceptor heart rate reflex during development in spontaneously hypertensive rats. Clin Exp Pharmacol Physiol. 1992 Aug;19(8):587-97. doi: 10.1111/j.1440-1681.1992.tb00509.x. PMID: 1526065.
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Hemodynamic responses during acute normovolemic hemodilution in anesthetized dogs.

The Japanese journal of physiology

Fahim M, Singh M.
PMID: 1337127
Jpn J Physiol. 1992;42(5):753-63. doi: 10.2170/jjphysiol.42.753.

Acute normovolemic hemodilution was induced by progressive replacement of blood by dextran (molecular weight 150,000) in anesthetized artificially ventilated dogs. Experiments were performed on dogs with intact autonomic innervation, dogs with beta-adrenergic blockade, dogs with cholinergic blockade, dogs with...

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Fahim M, Singh M. Hemodynamic responses during acute normovolemic hemodilution in anesthetized dogs. Jpn J Physiol. 1992;42(5):753-63doi: 10.2170/jjphysiol.42.753.
Fahim, M., & Singh, M. (1992). Hemodynamic responses during acute normovolemic hemodilution in anesthetized dogs. The Japanese journal of physiology, 42(5), 753-63. https://doi.org/10.2170/jjphysiol.42.753
Fahim, M, and Singh, M. "Hemodynamic responses during acute normovolemic hemodilution in anesthetized dogs." The Japanese journal of physiology vol. 42,5 (1992): 753-63. doi: https://doi.org/10.2170/jjphysiol.42.753
Fahim M, Singh M. Hemodynamic responses during acute normovolemic hemodilution in anesthetized dogs. Jpn J Physiol. 1992;42(5):753-63. doi: 10.2170/jjphysiol.42.753. PMID: 1337127.
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Attenuation of the acute cardiovascular responses to haemorrhage by tissue injury in the conscious rat.

Quarterly journal of experimental physiology (Cambridge, England)

Little RA, Marshall HW, Kirkman E.
PMID: 2594935
Q J Exp Physiol. 1989 Nov;74(6):825-33. doi: 10.1113/expphysiol.1989.sp003352.
Free Article

A moderate haemorrhage of 0.75 +/- 0.09 ml (100 g body weight)-1 (11.1 +/- 1.3% of estimated blood volume) in the conscious rat produces a tachycardia, possibly mediated by the baroreflex, which serves to maintain mean arterial blood pressure....

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Little RA, Marshall HW, Kirkman E. Attenuation of the acute cardiovascular responses to haemorrhage by tissue injury in the conscious rat. Q J Exp Physiol. 1989;74(6):825-33doi: 10.1113/expphysiol.1989.sp003352.
Little, R. A., Marshall, H. W., & Kirkman, E. (1989). Attenuation of the acute cardiovascular responses to haemorrhage by tissue injury in the conscious rat. Quarterly journal of experimental physiology (Cambridge, England), 74(6), 825-33. https://doi.org/10.1113/expphysiol.1989.sp003352
Little, R A, et al. "Attenuation of the acute cardiovascular responses to haemorrhage by tissue injury in the conscious rat." Quarterly journal of experimental physiology (Cambridge, England) vol. 74,6 (1989): 825-33. doi: https://doi.org/10.1113/expphysiol.1989.sp003352
Little RA, Marshall HW, Kirkman E. Attenuation of the acute cardiovascular responses to haemorrhage by tissue injury in the conscious rat. Q J Exp Physiol. 1989 Nov;74(6):825-33. doi: 10.1113/expphysiol.1989.sp003352. PMID: 2594935.
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Endogenous angiotensin II and bradykinin delay and attenuate the hypotension after N-type calcium channel blockade in conscious rabbits.

Journal of cardiovascular pharmacology

Whorlow SL, Angus JA, Wright CE.
PMID: 9869501
J Cardiovasc Pharmacol. 1998 Dec;32(6):951-61. doi: 10.1097/00005344-199812000-00012.

The effects of N-type calcium channel inhibition with omega-conotoxin GVIA (omega-CTX) on cardiovascular parameters and vagally mediated autonomic reflexes and the role of the renin-angiotensin system were assessed in conscious rabbits. Omega-CTX (10 microg/kg, i.v.) resulted in hypotension, tachycardia,...

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Whorlow SL, Angus JA, Wright CE. Endogenous angiotensin II and bradykinin delay and attenuate the hypotension after N-type calcium channel blockade in conscious rabbits. J Cardiovasc Pharmacol. 1998;32(6):951-61doi: 10.1097/00005344-199812000-00012.
Whorlow, S. L., Angus, J. A., & Wright, C. E. (1998). Endogenous angiotensin II and bradykinin delay and attenuate the hypotension after N-type calcium channel blockade in conscious rabbits. Journal of cardiovascular pharmacology, 32(6), 951-61. https://doi.org/10.1097/00005344-199812000-00012
Whorlow, S L, et al. "Endogenous angiotensin II and bradykinin delay and attenuate the hypotension after N-type calcium channel blockade in conscious rabbits." Journal of cardiovascular pharmacology vol. 32,6 (1998): 951-61. doi: https://doi.org/10.1097/00005344-199812000-00012
Whorlow SL, Angus JA, Wright CE. Endogenous angiotensin II and bradykinin delay and attenuate the hypotension after N-type calcium channel blockade in conscious rabbits. J Cardiovasc Pharmacol. 1998 Dec;32(6):951-61. doi: 10.1097/00005344-199812000-00012. PMID: 9869501.
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Relationship between cardiovascular hypertrophy and cardiac baroreflex function in spontaneously hypertensive and stroke-prone rats.

Journal of hypertension

Minami N, Head GA.
PMID: 8390524
J Hypertens. 1993 May;11(5):523-33. doi: 10.1097/00004872-199305000-00008.

OBJECTIVE: To determine whether the reduced baroreceptor-heart rate reflex sensitivity in genetically hypertensive rats is related to the level of cardiac or vascular hypertrophy.DESIGN: Young spontaneously hypertensive rats (SHR), stroke-prone hypertensive rats (SHRSP) and Wistar-Kyoto (WKY) rats were treated...

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Minami N, Head GA. Relationship between cardiovascular hypertrophy and cardiac baroreflex function in spontaneously hypertensive and stroke-prone rats. J Hypertens. 1993;11(5):523-33doi: 10.1097/00004872-199305000-00008.
Minami, N., & Head, G. A. (1993). Relationship between cardiovascular hypertrophy and cardiac baroreflex function in spontaneously hypertensive and stroke-prone rats. Journal of hypertension, 11(5), 523-33. https://doi.org/10.1097/00004872-199305000-00008
Minami, N, and Head, G A. "Relationship between cardiovascular hypertrophy and cardiac baroreflex function in spontaneously hypertensive and stroke-prone rats." Journal of hypertension vol. 11,5 (1993): 523-33. doi: https://doi.org/10.1097/00004872-199305000-00008
Minami N, Head GA. Relationship between cardiovascular hypertrophy and cardiac baroreflex function in spontaneously hypertensive and stroke-prone rats. J Hypertens. 1993 May;11(5):523-33. doi: 10.1097/00004872-199305000-00008. PMID: 8390524.
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Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control.

The Journal of pharmacology and experimental therapeutics

El-Mas MM, Afify EA, Omar AG, Sharabi FM.
PMID: 11907192
J Pharmacol Exp Ther. 2002 Apr;301(1):346-54. doi: 10.1124/jpet.301.1.346.

Previous studies have shown that the immunosuppressant drug cyclosporine A attenuates arterial baroreceptor function. This study investigated whether the modulatory effect of cyclosporine on baroreceptor function involves inhibition of the baroreflex-facilitatory effect of testosterone. The role of cardiac autonomic...

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El-Mas MM, Afify EA, Omar AG, et al. Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control. J Pharmacol Exp Ther. 2002;301(1):346-54doi: 10.1124/jpet.301.1.346.
El-Mas, M. M., Afify, E. A., Omar, A. G., & Sharabi, F. M. (2002). Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control. The Journal of pharmacology and experimental therapeutics, 301(1), 346-54. https://doi.org/10.1124/jpet.301.1.346
El-Mas, Mahmoud M, et al. "Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control." The Journal of pharmacology and experimental therapeutics vol. 301,1 (2002): 346-54. doi: https://doi.org/10.1124/jpet.301.1.346
El-Mas MM, Afify EA, Omar AG, Sharabi FM. Cyclosporine adversely affects baroreflexes via inhibition of testosterone modulation of cardiac vagal control. J Pharmacol Exp Ther. 2002 Apr;301(1):346-54. doi: 10.1124/jpet.301.1.346. PMID: 11907192.
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Baroreceptor control of heart rate in the awake toad: peripheral autonomic effectors and arterial baroreceptor areas.

Journal of the autonomic nervous system

Bianchi-da-Silva LM, Menescal-de-Oliveira L, Hoffmann A.
PMID: 10742537
J Auton Nerv Syst. 2000 Apr 12;80(1):31-9. doi: 10.1016/s0165-1838(99)00083-1.

Systemic injection of sodium nitroprusside (30 microg/kg, i.v.) in the awake Bufo paracnemis toad induced a fall in arterial blood pressure and tachycardia. This tachycardia, but not the hypotension, was significantly reduced in toads with bilateral electrolytic lesion of...

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Bianchi-da-Silva LM, Menescal-de-Oliveira L, Hoffmann A. Baroreceptor control of heart rate in the awake toad: peripheral autonomic effectors and arterial baroreceptor areas. J Auton Nerv Syst. 2000;80(1):31-9doi: 10.1016/s0165-1838(99)00083-1.
Bianchi-da-Silva, L. M., Menescal-de-Oliveira, L., & Hoffmann, A. (2000). Baroreceptor control of heart rate in the awake toad: peripheral autonomic effectors and arterial baroreceptor areas. Journal of the autonomic nervous system, 80(1), 31-9. https://doi.org/10.1016/s0165-1838(99)00083-1
Bianchi-da-Silva, L M, et al. "Baroreceptor control of heart rate in the awake toad: peripheral autonomic effectors and arterial baroreceptor areas." Journal of the autonomic nervous system vol. 80,1 (2000): 31-9. doi: https://doi.org/10.1016/s0165-1838(99)00083-1
Bianchi-da-Silva LM, Menescal-de-Oliveira L, Hoffmann A. Baroreceptor control of heart rate in the awake toad: peripheral autonomic effectors and arterial baroreceptor areas. J Auton Nerv Syst. 2000 Apr 12;80(1):31-9. doi: 10.1016/s0165-1838(99)00083-1. PMID: 10742537.
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Showing 1 to 12 of 110 entries