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Showing 1 to 12 of 132 entries
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Extracellular potassium ion dynamics and ventricular arrhythmias in the canine heart.

Journal of the American College of Cardiology

Pelleg A, Mitamura H, Price R, Kaplinsky E, Menduke H, Dreifus LS, Michelson EL.
PMID: 2926046
J Am Coll Cardiol. 1989 Mar 15;13(4):941-50. doi: 10.1016/0735-1097(89)90240-4.
Free Article

The relation between extracellular potassium ion activity [( K+]o) and ventricular tachyarrhythmias was studied in an open chest canine model with the use of two protocols. In Protocol I, potassium chloride was administered into the proximal left anterior descending...

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Pelleg A, Mitamura H, Price R, et al. Extracellular potassium ion dynamics and ventricular arrhythmias in the canine heart. J Am Coll Cardiol. 1989;13(4):941-50doi: 10.1016/0735-1097(89)90240-4.
Pelleg, A., Mitamura, H., Price, R., Kaplinsky, E., Menduke, H., Dreifus, L. S., & Michelson, E. L. (1989). Extracellular potassium ion dynamics and ventricular arrhythmias in the canine heart. Journal of the American College of Cardiology, 13(4), 941-50. https://doi.org/10.1016/0735-1097(89)90240-4
Pelleg, A, et al. "Extracellular potassium ion dynamics and ventricular arrhythmias in the canine heart." Journal of the American College of Cardiology vol. 13,4 (1989): 941-50. doi: https://doi.org/10.1016/0735-1097(89)90240-4
Pelleg A, Mitamura H, Price R, Kaplinsky E, Menduke H, Dreifus LS, Michelson EL. Extracellular potassium ion dynamics and ventricular arrhythmias in the canine heart. J Am Coll Cardiol. 1989 Mar 15;13(4):941-50. doi: 10.1016/0735-1097(89)90240-4. PMID: 2926046.
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Myocardial K+ repletion and rise in contractility after brief ischemic periods in the pig.

Journal of molecular and cellular cardiology

Aksnes G, Ellingsen O, Rutlen DL, Ilebekk A.
PMID: 2795661
J Mol Cell Cardiol. 1989 Jul;21(7):681-90. doi: 10.1016/0022-2828(89)90609-3.

Potassium loss from the myocardium during brief ischemic periods is well documented, but whether intrinsic myocardial mechanisms restore this loss during reperfusion is unclear. To address this question, we established a shunt from the coronary sinus to the right...

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Aksnes G, Ellingsen O, Rutlen DL, et al. Myocardial K+ repletion and rise in contractility after brief ischemic periods in the pig. J Mol Cell Cardiol. 1989;21(7):681-90doi: 10.1016/0022-2828(89)90609-3.
Aksnes, G., Ellingsen, O., Rutlen, D. L., & Ilebekk, A. (1989). Myocardial K+ repletion and rise in contractility after brief ischemic periods in the pig. Journal of molecular and cellular cardiology, 21(7), 681-90. https://doi.org/10.1016/0022-2828(89)90609-3
Aksnes, G, et al. "Myocardial K+ repletion and rise in contractility after brief ischemic periods in the pig." Journal of molecular and cellular cardiology vol. 21,7 (1989): 681-90. doi: https://doi.org/10.1016/0022-2828(89)90609-3
Aksnes G, Ellingsen O, Rutlen DL, Ilebekk A. Myocardial K+ repletion and rise in contractility after brief ischemic periods in the pig. J Mol Cell Cardiol. 1989 Jul;21(7):681-90. doi: 10.1016/0022-2828(89)90609-3. PMID: 2795661.
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Passive electrical properties, mechanical activity, and extracellular potassium in arterially perfused and ischemic rabbit ventricular muscle. Effects of calcium entry blockade or hypocalcemia.

Circulation research

Cascio WE, Yan GX, Kléber AG.
PMID: 2344662
Circ Res. 1990 Jun;66(6):1461-73. doi: 10.1161/01.res.66.6.1461.

The relation among passive electrical resistive properties, longitudinal conduction velocity, extracellular potassium concentration, [K+]o, and mechanical activity was investigated in the isolated rabbit papillary muscle during normal arterial perfusion and no-flow ischemia in the presence and absence of verapamil,...

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Cascio WE, Yan GX, Kléber AG. Passive electrical properties, mechanical activity, and extracellular potassium in arterially perfused and ischemic rabbit ventricular muscle. Effects of calcium entry blockade or hypocalcemia. Circ Res. 1990;66(6):1461-73doi: 10.1161/01.res.66.6.1461.
Cascio, W. E., Yan, G. X., & Kléber, A. G. (1990). Passive electrical properties, mechanical activity, and extracellular potassium in arterially perfused and ischemic rabbit ventricular muscle. Effects of calcium entry blockade or hypocalcemia. Circulation research, 66(6), 1461-73. https://doi.org/10.1161/01.res.66.6.1461
Cascio, W E, et al. "Passive electrical properties, mechanical activity, and extracellular potassium in arterially perfused and ischemic rabbit ventricular muscle. Effects of calcium entry blockade or hypocalcemia." Circulation research vol. 66,6 (1990): 1461-73. doi: https://doi.org/10.1161/01.res.66.6.1461
Cascio WE, Yan GX, Kléber AG. Passive electrical properties, mechanical activity, and extracellular potassium in arterially perfused and ischemic rabbit ventricular muscle. Effects of calcium entry blockade or hypocalcemia. Circ Res. 1990 Jun;66(6):1461-73. doi: 10.1161/01.res.66.6.1461. PMID: 2344662.
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Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs.

Archives internationales de pharmacodynamie et de therapie

Abiko Y, Gotoh H, Yokoyama T, Abiko T, Hashizume H, Akiyama K.
PMID: 7944827
Arch Int Pharmacodyn Ther. 1994 Jan-Feb;327(1):40-55.

The effects of bopindolol (a new, nonselective beta-adrenoceptor antagonist) and its active metabolite, 18-502, on the regional myocardial pH during coronary occlusion were studied. Mongrel dogs were anesthetized with pentobarbital and the thorax was opened to expose the heart....

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Abiko Y, Gotoh H, Yokoyama T, et al. Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Arch Int Pharmacodyn Ther. 1994;327(1):40-55
Abiko, Y., Gotoh, H., Yokoyama, T., Abiko, T., Hashizume, H., & Akiyama, K. (1994). Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Archives internationales de pharmacodynamie et de therapie, 327(1), 40-55.
Abiko, Y, et al. "Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs." Archives internationales de pharmacodynamie et de therapie vol. 327,1 (1994): 40-55.
Abiko Y, Gotoh H, Yokoyama T, Abiko T, Hashizume H, Akiyama K. Bopindolol and its metabolite 18-502 attenuate regional myocardial acidosis during partial occlusion of the coronary artery in dogs. Arch Int Pharmacodyn Ther. 1994 Jan-Feb;327(1):40-55. PMID: 7944827.
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Effect of isoflurane on the extent of myocardial necrosis and on systemic hemodynamics, regional myocardial blood flow, and regional myocardial metabolism in dogs after coronary artery occlusion.

Anesthesia and analgesia

Davis RF, Sidi A.
PMID: 2802193
Anesth Analg. 1989 Nov;69(5):575-86.

Anesthetized dogs were studied in two protocols to determine the effect of isoflurane on the extent of myocardial injury resulting from left anterior descending coronary artery (LAD) occlusion. In 22 dogs (11 treated with isoflurane 1% inspired, beginning 1...

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Davis RF, Sidi A. Effect of isoflurane on the extent of myocardial necrosis and on systemic hemodynamics, regional myocardial blood flow, and regional myocardial metabolism in dogs after coronary artery occlusion. Anesth Analg. 1989;69(5):575-86
Davis, R. F., & Sidi, A. (1989). Effect of isoflurane on the extent of myocardial necrosis and on systemic hemodynamics, regional myocardial blood flow, and regional myocardial metabolism in dogs after coronary artery occlusion. Anesthesia and analgesia, 69(5), 575-86.
Davis, R F, and Sidi, A. "Effect of isoflurane on the extent of myocardial necrosis and on systemic hemodynamics, regional myocardial blood flow, and regional myocardial metabolism in dogs after coronary artery occlusion." Anesthesia and analgesia vol. 69,5 (1989): 575-86.
Davis RF, Sidi A. Effect of isoflurane on the extent of myocardial necrosis and on systemic hemodynamics, regional myocardial blood flow, and regional myocardial metabolism in dogs after coronary artery occlusion. Anesth Analg. 1989 Nov;69(5):575-86. PMID: 2802193.
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Protection against autonomic denervation following acute myocardial infarction by preconditioning ischemia.

Circulation research

Miyazaki T, Zipes DP.
PMID: 2917377
Circ Res. 1989 Mar;64(3):437-48. doi: 10.1161/01.res.64.3.437.

To examine the effects of ischemic preconditioning on efferent autonomic responses following acute transmural myocardial ischemia/infarction (MI), the time course and extent of efferent sympathetic and vagal denervation were compared between control dogs that received a one-stage sustained coronary...

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Miyazaki T, Zipes DP. Protection against autonomic denervation following acute myocardial infarction by preconditioning ischemia. Circ Res. 1989;64(3):437-48doi: 10.1161/01.res.64.3.437.
Miyazaki, T., & Zipes, D. P. (1989). Protection against autonomic denervation following acute myocardial infarction by preconditioning ischemia. Circulation research, 64(3), 437-48. https://doi.org/10.1161/01.res.64.3.437
Miyazaki, T, and Zipes, D P. "Protection against autonomic denervation following acute myocardial infarction by preconditioning ischemia." Circulation research vol. 64,3 (1989): 437-48. doi: https://doi.org/10.1161/01.res.64.3.437
Miyazaki T, Zipes DP. Protection against autonomic denervation following acute myocardial infarction by preconditioning ischemia. Circ Res. 1989 Mar;64(3):437-48. doi: 10.1161/01.res.64.3.437. PMID: 2917377.
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Distribution of extracellular potassium and electrophysiologic changes during two-stage coronary ligation in the isolated, perfused canine heart.

Circulation

Coronel R, Fiolet JW, Wilms-Schopman JG, Opthof T, Schaapherder AF, Janse MJ.
PMID: 2736748
Circulation. 1989 Jul;80(1):165-77. doi: 10.1161/01.cir.80.1.165.

We studied the relation between [K+]o and the electrophysiologic changes during a "Harris two-stage ligation," which is an occlusion of a coronary artery, preceded by a 30-minute period of 50% reduction of flow through the artery. This two-stage ligation...

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Coronel R, Fiolet JW, Wilms-Schopman JG, et al. Distribution of extracellular potassium and electrophysiologic changes during two-stage coronary ligation in the isolated, perfused canine heart. Circulation. 1989;80(1):165-77doi: 10.1161/01.cir.80.1.165.
Coronel, R., Fiolet, J. W., Wilms-Schopman, J. G., Opthof, T., Schaapherder, A. F., & Janse, M. J. (1989). Distribution of extracellular potassium and electrophysiologic changes during two-stage coronary ligation in the isolated, perfused canine heart. Circulation, 80(1), 165-77. https://doi.org/10.1161/01.cir.80.1.165
Coronel, R, et al. "Distribution of extracellular potassium and electrophysiologic changes during two-stage coronary ligation in the isolated, perfused canine heart." Circulation vol. 80,1 (1989): 165-77. doi: https://doi.org/10.1161/01.cir.80.1.165
Coronel R, Fiolet JW, Wilms-Schopman JG, Opthof T, Schaapherder AF, Janse MJ. Distribution of extracellular potassium and electrophysiologic changes during two-stage coronary ligation in the isolated, perfused canine heart. Circulation. 1989 Jul;80(1):165-77. doi: 10.1161/01.cir.80.1.165. PMID: 2736748.
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The effect of cardiac sympathetic nerve stimulation on the right ventricle in canine heart.

Japanese circulation journal

Abe Y, Saito D, Tani H, Nakatsu T, Kusachi S, Haraoka S, Nagashima H.
PMID: 3626013
Jpn Circ J. 1987 May;51(5):535-42. doi: 10.1253/jcj.51.535.

The change in coronary hemodynamics during right or left cardiac sympathetic nerve stimulation was studied in anesthetized open chest dogs. No difference in the increasing rate of mean coronary blood flow between right coronary artery (RCA) and left anterior...

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Abe Y, Saito D, Tani H, et al. The effect of cardiac sympathetic nerve stimulation on the right ventricle in canine heart. Jpn Circ J. 1987;51(5):535-42doi: 10.1253/jcj.51.535.
Abe, Y., Saito, D., Tani, H., Nakatsu, T., Kusachi, S., Haraoka, S., & Nagashima, H. (1987). The effect of cardiac sympathetic nerve stimulation on the right ventricle in canine heart. Japanese circulation journal, 51(5), 535-42. https://doi.org/10.1253/jcj.51.535
Abe, Y, et al. "The effect of cardiac sympathetic nerve stimulation on the right ventricle in canine heart." Japanese circulation journal vol. 51,5 (1987): 535-42. doi: https://doi.org/10.1253/jcj.51.535
Abe Y, Saito D, Tani H, Nakatsu T, Kusachi S, Haraoka S, Nagashima H. The effect of cardiac sympathetic nerve stimulation on the right ventricle in canine heart. Jpn Circ J. 1987 May;51(5):535-42. doi: 10.1253/jcj.51.535. PMID: 3626013.
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Fundamental electrophysiological differences between low-dose intracoronary endothelin-1 infusion and myocardial ischemia revealed by multiple monophasic action potential recording.

Journal of cardiovascular pharmacology

Gellér L, Szabó T, Kiss O, Solti F, Juhász-Nagy A, Merkely B.
PMID: 11078368
J Cardiovasc Pharmacol. 2000 Nov;36(5):S167-71. doi: 10.1097/00005344-200036051-00052.

The supposed direct arrhythmogenic property of endothelin-1 (ET-1) has not yet been clearly proven. Our study aimed to characterize the electrophysiological changes during left anterior descending artery (LAD) occlusion and intracoronary (i.c.) ET-1 infusion, and to differentiate between the...

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Gellér L, Szabó T, Kiss O, et al. Fundamental electrophysiological differences between low-dose intracoronary endothelin-1 infusion and myocardial ischemia revealed by multiple monophasic action potential recording. J Cardiovasc Pharmacol. 2000;36(5):S167-71doi: 10.1097/00005344-200036051-00052.
Gellér, L., Szabó, T., Kiss, O., Solti, F., Juhász-Nagy, A., & Merkely, B. (2000). Fundamental electrophysiological differences between low-dose intracoronary endothelin-1 infusion and myocardial ischemia revealed by multiple monophasic action potential recording. Journal of cardiovascular pharmacology, 36(5), S167-71. https://doi.org/10.1097/00005344-200036051-00052
Gellér, L, et al. "Fundamental electrophysiological differences between low-dose intracoronary endothelin-1 infusion and myocardial ischemia revealed by multiple monophasic action potential recording." Journal of cardiovascular pharmacology vol. 36,5 (2000): S167-71. doi: https://doi.org/10.1097/00005344-200036051-00052
Gellér L, Szabó T, Kiss O, Solti F, Juhász-Nagy A, Merkely B. Fundamental electrophysiological differences between low-dose intracoronary endothelin-1 infusion and myocardial ischemia revealed by multiple monophasic action potential recording. J Cardiovasc Pharmacol. 2000 Nov;36(5):S167-71. doi: 10.1097/00005344-200036051-00052. PMID: 11078368.
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Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content.

Cardiovascular research

Dae MW, O'Connell JW, Botvinick EH, Chin MC.
PMID: 7585815
Cardiovasc Res. 1995 Aug;30(2):270-80.

OBJECTIVES: The sympathetic nervous system has profound influences on myocardial function, particularly during ischemia. There is controversy, however, as to whether myocardial ischemia results in damage to myocardial sympathetic nerves coursing through the ischemic territory. To further evaluate these...

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Dae MW, O'Connell JW, Botvinick EH, et al. Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovasc Res. 1995;30(2):270-80
Dae, M. W., O'Connell, J. W., Botvinick, E. H., & Chin, M. C. (1995). Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovascular research, 30(2), 270-80.
Dae, M W, et al. "Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content." Cardiovascular research vol. 30,2 (1995): 270-80.
Dae MW, O'Connell JW, Botvinick EH, Chin MC. Acute and chronic effects of transient myocardial ischemia on sympathetic nerve activity, density, and norepinephrine content. Cardiovasc Res. 1995 Aug;30(2):270-80. PMID: 7585815.
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An intramyocardial catheter for repeated in vivo sampling of interstitial fluid.

Journal of pharmacological and toxicological methods

Tessmer JP, Pagel PS, Weihrauch D, Ludwig LM, Chilian WM, Kersten JR, Warltier DC.
PMID: 12459145
J Pharmacol Toxicol Methods. 2002 Mar-Apr;47(2):73-8. doi: 10.1016/s1056-8719(02)00220-4.

INTRODUCTION: Coronary collateral development is an important adaptive response to chronic myocardial ischemia. Characterization of mitogenic factors responsible for collateral formation has been an elusive goal because these substances are difficult to sample from the myocardial interstitium at multiple...

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Tessmer JP, Pagel PS, Weihrauch D, et al. An intramyocardial catheter for repeated in vivo sampling of interstitial fluid. J Pharmacol Toxicol Methods. 2002;47(2):73-8doi: 10.1016/s1056-8719(02)00220-4.
Tessmer, J. P., Pagel, P. S., Weihrauch, D., Ludwig, L. M., Chilian, W. M., Kersten, J. R., & Warltier, D. C. (2002). An intramyocardial catheter for repeated in vivo sampling of interstitial fluid. Journal of pharmacological and toxicological methods, 47(2), 73-8. https://doi.org/10.1016/s1056-8719(02)00220-4
Tessmer, John P, et al. "An intramyocardial catheter for repeated in vivo sampling of interstitial fluid." Journal of pharmacological and toxicological methods vol. 47,2 (2002): 73-8. doi: https://doi.org/10.1016/s1056-8719(02)00220-4
Tessmer JP, Pagel PS, Weihrauch D, Ludwig LM, Chilian WM, Kersten JR, Warltier DC. An intramyocardial catheter for repeated in vivo sampling of interstitial fluid. J Pharmacol Toxicol Methods. 2002 Mar-Apr;47(2):73-8. doi: 10.1016/s1056-8719(02)00220-4. PMID: 12459145.
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Ischemic preconditioning attenuates cardiac sympathetic nerve injury via ATP-sensitive potassium channels during myocardial ischemia.

Circulation

Miura T, Kawamura S, Tatsuno H, Ikeda Y, Mikami S, Iwamoto H, Okamura T, Iwatate M, Kimura M, Dairaku Y, Maekawa T, Matsuzaki M.
PMID: 11524401
Circulation. 2001 Aug 28;104(9):1053-8. doi: 10.1161/hc3501.093800.

BACKGROUND: During myocardial ischemia, massive norepinephrine (NE) is released from the cardiac sympathetic nerve terminals, reflecting the sympathetic nerve injury. A brief preceding ischemia can reduce infarct size; this is known as ischemic preconditioning (PC). The effect of PC...

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Miura T, Kawamura S, Tatsuno H, et al. Ischemic preconditioning attenuates cardiac sympathetic nerve injury via ATP-sensitive potassium channels during myocardial ischemia. Circulation. 2001;104(9):1053-8doi: 10.1161/hc3501.093800.
Miura, T., Kawamura, S., Tatsuno, H., Ikeda, Y., Mikami, S., Iwamoto, H., Okamura, T., Iwatate, M., Kimura, M., Dairaku, Y., Maekawa, T., & Matsuzaki, M. (2001). Ischemic preconditioning attenuates cardiac sympathetic nerve injury via ATP-sensitive potassium channels during myocardial ischemia. Circulation, 104(9), 1053-8. https://doi.org/10.1161/hc3501.093800
Miura, T, et al. "Ischemic preconditioning attenuates cardiac sympathetic nerve injury via ATP-sensitive potassium channels during myocardial ischemia." Circulation vol. 104,9 (2001): 1053-8. doi: https://doi.org/10.1161/hc3501.093800
Miura T, Kawamura S, Tatsuno H, Ikeda Y, Mikami S, Iwamoto H, Okamura T, Iwatate M, Kimura M, Dairaku Y, Maekawa T, Matsuzaki M. Ischemic preconditioning attenuates cardiac sympathetic nerve injury via ATP-sensitive potassium channels during myocardial ischemia. Circulation. 2001 Aug 28;104(9):1053-8. doi: 10.1161/hc3501.093800. PMID: 11524401.
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Showing 1 to 12 of 132 entries